Cognitive and emotional associations to positive schizotypy during adolescence / Martin DEBBANE in Journal of Child Psychology and Psychiatry, 50-3 (March 2009)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 50-3 (March 2009) . - p.326-334 Titre : Cognitive and emotional associations to positive schizotypy during adolescence Type de document : texte imprimé Auteurs : Martin DEBBANE, Auteur ; Martial VAN DER LINDEN, Auteur ; Marianne GEX-FABRY, Auteur ; Stephan ELIEZ, Auteur Année de publication : 2009 Article en page(s) : p.326-334 Langues : Anglais (eng) Mots-clés : Adolescence anxiety depression psychosis metacognitive beliefs hallucinations Index. décimale : PER Périodiques Résumé : Background: Sub-clinical symptoms of psychosis such as hallucinations and delusions, known as positive schizotypy, constitute one of the strongest predictive factors for adult psychotic disorders. Recent cognitive models suggest that the expression of positive schizotypy is associated with depression, anxiety, metacognitive beliefs and self-monitoring deficits. In this study, we present empirical data on the relationships positive schizotypy hold with both emotional and cognitive factors. Methods: A sample of 163 adolescents (77 females) participated in this study (mean age 15.3, 12–18 years). Subjects filled out self-report questionnaires assessing the emotional dimensions of depression and anxiety, as well as metacognitive beliefs. Self-monitoring skills were assessed using an action monitoring paradigm sensitive to dysfunctions associated with psychosis-proneness. Multivariate regression models were employed to examine emotional and cognitive contributions to positive schizotypy during adolescence. Results: Analyses revealed that dimensions of depression, anxiety, and metacognitive beliefs significantly correlated with the expression of positive schizotypy. When accounting for the effects of depression and metacognitive beliefs in adolescents reporting hallucinations, self-monitoring dysfunctions seemed to represent a significant factor in the expression of positive schizotypy. Conclusion: The present results suggest that the expression of positive schizotypy during adolescence is modulated by emotional factors of depression and anxiety, as well as metacognitive beliefs and self-monitoring dysfunctions. The current data lend some evidence that supports the cognitive-developmental account of positive symptom formation before the onset of a psychotic disorder. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2008.01961.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=719 [article] Cognitive and emotional associations to positive schizotypy during adolescence [texte imprimé] / Martin DEBBANE, Auteur ; Martial VAN DER LINDEN, Auteur ; Marianne GEX-FABRY, Auteur ; Stephan ELIEZ, Auteur . - 2009 . - p.326-334. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 50-3 (March 2009) . - p.326-334 Mots-clés : Adolescence anxiety depression psychosis metacognitive beliefs hallucinations Index. décimale : PER Périodiques Résumé : Background: Sub-clinical symptoms of psychosis such as hallucinations and delusions, known as positive schizotypy, constitute one of the strongest predictive factors for adult psychotic disorders. Recent cognitive models suggest that the expression of positive schizotypy is associated with depression, anxiety, metacognitive beliefs and self-monitoring deficits. In this study, we present empirical data on the relationships positive schizotypy hold with both emotional and cognitive factors. Methods: A sample of 163 adolescents (77 females) participated in this study (mean age 15.3, 12–18 years). Subjects filled out self-report questionnaires assessing the emotional dimensions of depression and anxiety, as well as metacognitive beliefs. Self-monitoring skills were assessed using an action monitoring paradigm sensitive to dysfunctions associated with psychosis-proneness. Multivariate regression models were employed to examine emotional and cognitive contributions to positive schizotypy during adolescence. Results: Analyses revealed that dimensions of depression, anxiety, and metacognitive beliefs significantly correlated with the expression of positive schizotypy. When accounting for the effects of depression and metacognitive beliefs in adolescents reporting hallucinations, self-monitoring dysfunctions seemed to represent a significant factor in the expression of positive schizotypy. Conclusion: The present results suggest that the expression of positive schizotypy during adolescence is modulated by emotional factors of depression and anxiety, as well as metacognitive beliefs and self-monitoring dysfunctions. The current data lend some evidence that supports the cognitive-developmental account of positive symptom formation before the onset of a psychotic disorder. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2008.01961.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=719

Cortical thickness of the insula and prefrontal cortex relates to externalizing behavior: Cross-sectional and prospective findings / Michal TANZER in Development and Psychopathology, 33-4 (October 2021)  

Public ISBD [article]  inDevelopment and Psychopathology > 33-4 (October 2021) . - p.1437-1447 Titre : Cortical thickness of the insula and prefrontal cortex relates to externalizing behavior: Cross-sectional and prospective findings Type de document : texte imprimé Auteurs : Michal TANZER, Auteur ; Mélodie DEROME, Auteur ; Larisa MOROSAN, Auteur ; George SALAMINIOS, Auteur ; Martin DEBBANE, Auteur Article en page(s) : p.1437-1447 Langues : Anglais (eng) Mots-clés : adolescence  development  externalizing dimension  interoception Index. décimale : PER Périodiques Résumé : Externalizing behaviors (EBs) pertain to a diverse set of aggressive, antisocial, and potentially destructive behaviors directed toward the external environment. They range from nonclinical to clinical in severity, associated with opposition, aggression, hyperactivity, or impulsivity, and are considered a risk factor for the emergence of psychopathology later in adulthood. Focusing on community adolescents (N = 102; 49 female and 53 male adolescents; age range 12–19 years), this study aimed to explore the relations between EBs and the cortical thickness of regions of interest as well as to identify possible risk markers that could improve understanding of the EB construct. Using a mixed cross-sectional and prospective design (1-year follow-up), we report specific associations with cortical thickness of the left insular, right orbitofrontal, and left anterior cingulate cortex. Specifically, thinner left insular and right orbitofrontal cortex was associated with higher EBs, and thinner left anterior cingulate cortex predicted less reduction in EBs 1 year later. In addition, further examination of the aggression and rule-breaking subscales of the Youth/Adult Self-Report, used to assess EBs, revealed specific associations with insular subregions. Findings suggest that cortical structure morphology may significantly relate to the expression and maintenance of EBs within the general population of adolescents. En ligne : http://dx.doi.org/10.1017/S0954579420000619 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457 [article] Cortical thickness of the insula and prefrontal cortex relates to externalizing behavior: Cross-sectional and prospective findings [texte imprimé] / Michal TANZER, Auteur ; Mélodie DEROME, Auteur ; Larisa MOROSAN, Auteur ; George SALAMINIOS, Auteur ; Martin DEBBANE, Auteur . - p.1437-1447. Langues : Anglais (eng) in Development and Psychopathology > 33-4 (October 2021) . - p.1437-1447 Mots-clés : adolescence  development  externalizing dimension  interoception Index. décimale : PER Périodiques Résumé : Externalizing behaviors (EBs) pertain to a diverse set of aggressive, antisocial, and potentially destructive behaviors directed toward the external environment. They range from nonclinical to clinical in severity, associated with opposition, aggression, hyperactivity, or impulsivity, and are considered a risk factor for the emergence of psychopathology later in adulthood. Focusing on community adolescents (N = 102; 49 female and 53 male adolescents; age range 12–19 years), this study aimed to explore the relations between EBs and the cortical thickness of regions of interest as well as to identify possible risk markers that could improve understanding of the EB construct. Using a mixed cross-sectional and prospective design (1-year follow-up), we report specific associations with cortical thickness of the left insular, right orbitofrontal, and left anterior cingulate cortex. Specifically, thinner left insular and right orbitofrontal cortex was associated with higher EBs, and thinner left anterior cingulate cortex predicted less reduction in EBs 1 year later. In addition, further examination of the aggression and rule-breaking subscales of the Youth/Adult Self-Report, used to assess EBs, revealed specific associations with insular subregions. Findings suggest that cortical structure morphology may significantly relate to the expression and maintenance of EBs within the general population of adolescents. En ligne : http://dx.doi.org/10.1017/S0954579420000619 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457

Developmental trajectories of executive functions in 22q11.2 deletion syndrome / J. MAEDER in Journal of Neurodevelopmental Disorders, 8-1 (December 2016)  

Public ISBD [article]  inJournal of Neurodevelopmental Disorders > 8-1 (December 2016) . - p.10 Titre : Developmental trajectories of executive functions in 22q11.2 deletion syndrome Type de document : texte imprimé Auteurs : J. MAEDER, Auteur ; Maude SCHNEIDER, Auteur ; Mathilde BOSTELMANN, Auteur ; Martin DEBBANE, Auteur ; Bronwyn GLASER, Auteur ; S. MENGHETTI, Auteur ; M. SCHAER, Auteur ; Stephan ELIEZ, Auteur Année de publication : 2016 Article en page(s) : p.10 Langues : Anglais (eng) Mots-clés : 22q11.2 deletion syndrome  Adaptive functioning  Development  Executive functions Index. décimale : PER Périodiques Résumé : BACKGROUND: 22q11.2 deletion syndrome (22q11.2DS) is a genetic disorder associated with a specific cognitive profile. Higher-order cognitive skills like executive functions (EF) are reported as a relative weakness in this population. The present study aimed to delineate the developmental trajectories of multiple EF domains in a longitudinal sample using a broader age range than previous studies. Given the high incidence of psychotic symptoms in 22q11.2DS, we also compared the development of EF in participants with/without comorbid psychotic symptoms. Given the importance of EF in daily life, the third aim of the study was to characterize the link between EF and adaptive functioning. METHODS: The sample consisted of 95 individuals with 22q11.2DS and 100 typically developing controls aged 6-26 years. A large proportion of the sample (55.38 %) had multiple time points available. Between-group differences in the developmental trajectories of three subdomains of EF (verbal fluency, working memory, and inhibition) were examined using mixed models regression analyses. Analyses were repeated comparing only the 22q11.2DS group based on the presence/absence of psychotic symptoms to investigate the influence of executive dysfunction on the emergence of psychotic symptoms. Hierarchical stepwise regression analyses were also conducted to investigate the predictive value of EF on adaptive functioning. RESULTS: We observed lower performance on EF domains, as well as atypical development of working memory and verbal fluency. Participants who presented with negative symptoms exhibited different developmental trajectories of inhibition and working memory. Adaptive functioning level was not significantly predicted by EF scores. CONCLUSIONS: The present study highlighted domain-specific atypical trajectories of EF in individuals with 22q11.DS and explored the link with psychotic symptoms. However, no relation between EF and adaptive functioning was observed. En ligne : http://dx.doi.org/10.1186/s11689-016-9141-1 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=348 [article] Developmental trajectories of executive functions in 22q11.2 deletion syndrome [texte imprimé] / J. MAEDER, Auteur ; Maude SCHNEIDER, Auteur ; Mathilde BOSTELMANN, Auteur ; Martin DEBBANE, Auteur ; Bronwyn GLASER, Auteur ; S. MENGHETTI, Auteur ; M. SCHAER, Auteur ; Stephan ELIEZ, Auteur . - 2016 . - p.10. Langues : Anglais (eng) in Journal of Neurodevelopmental Disorders > 8-1 (December 2016) . - p.10 Mots-clés : 22q11.2 deletion syndrome  Adaptive functioning  Development  Executive functions Index. décimale : PER Périodiques Résumé : BACKGROUND: 22q11.2 deletion syndrome (22q11.2DS) is a genetic disorder associated with a specific cognitive profile. Higher-order cognitive skills like executive functions (EF) are reported as a relative weakness in this population. The present study aimed to delineate the developmental trajectories of multiple EF domains in a longitudinal sample using a broader age range than previous studies. Given the high incidence of psychotic symptoms in 22q11.2DS, we also compared the development of EF in participants with/without comorbid psychotic symptoms. Given the importance of EF in daily life, the third aim of the study was to characterize the link between EF and adaptive functioning. METHODS: The sample consisted of 95 individuals with 22q11.2DS and 100 typically developing controls aged 6-26 years. A large proportion of the sample (55.38 %) had multiple time points available. Between-group differences in the developmental trajectories of three subdomains of EF (verbal fluency, working memory, and inhibition) were examined using mixed models regression analyses. Analyses were repeated comparing only the 22q11.2DS group based on the presence/absence of psychotic symptoms to investigate the influence of executive dysfunction on the emergence of psychotic symptoms. Hierarchical stepwise regression analyses were also conducted to investigate the predictive value of EF on adaptive functioning. RESULTS: We observed lower performance on EF domains, as well as atypical development of working memory and verbal fluency. Participants who presented with negative symptoms exhibited different developmental trajectories of inhibition and working memory. Adaptive functioning level was not significantly predicted by EF scores. CONCLUSIONS: The present study highlighted domain-specific atypical trajectories of EF in individuals with 22q11.DS and explored the link with psychotic symptoms. However, no relation between EF and adaptive functioning was observed. En ligne : http://dx.doi.org/10.1186/s11689-016-9141-1 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=348

Face processing in 22q11.2 deletion syndrome: atypical development and visual scanning alterations / A. ZAHARIA in Journal of Neurodevelopmental Disorders, 10-1 (December 2018)  

Public ISBD [article]  inJournal of Neurodevelopmental Disorders > 10-1 (December 2018) . - 26 p. Titre : Face processing in 22q11.2 deletion syndrome: atypical development and visual scanning alterations Type de document : texte imprimé Auteurs : A. ZAHARIA, Auteur ; Maude SCHNEIDER, Auteur ; Bronwyn GLASER, Auteur ; Martina FRANCHINI, Auteur ; S. MENGHETTI, Auteur ; M. SCHAER, Auteur ; Martin DEBBANE, Auteur ; Stephan ELIEZ, Auteur Année de publication : 2018 Article en page(s) : 26 p. Langues : Anglais (eng) Mots-clés : Configural face processing  Eye-tracking  Featural face processing  Neurodevelopmental disorders  Social difficulties Index. décimale : PER Périodiques Résumé : BACKGROUND: Previous research links social difficulties to atypical face exploration in 22q11.2 deletion syndrome (22q11.2DS). Two types of face processing are distinguished: configural (CFP) and featural (FFP). CFP develops later in life and plays an important role in face and emotion recognition abilities. Recent studies reported atypical development of CFP in several neurodevelopmental disorders. Taking previous reports of atypical face exploration one step further, our study aims at characterizing face processing in children and adolescents with 22q11.2DS. First, we sought to identify biases in the first two fixation positions on faces and to detect differences between CFP and FFP in 22q11.2DS using eye-tracking technology. Second, we investigated the developmental trajectories of CFP and FFP using accuracy data from follow-up evaluation. METHODS: Seventy-five individuals with 22q11.2DS and 84 typically developed (TD) individuals (aged 6-21 years) completed a discrimination task ("Jane task") inducing CFP and FFP in an eye-tracking setting. Thirty-six individuals with 22q11DS and 30 TD from our sample completed a longitudinal follow-up evaluation. RESULTS: Findings revealed that individuals with 22q11.2DS demonstrate an early bias toward the mouth region during the initial fixations on the faces and reduced flexibility exploration of the faces, with a reduced number of transitions between faces and longer fixations compared to the TD group. Further, scanpaths did not differ between CFP and FFP in the 22q11.2DS group. Longitudinal analysis of accuracy data provided evidence for atypical development of CFP in 22q11.2DS. CONCLUSIONS: The current study brings new evidence of altered face exploration in 22q11.2DS and identifies developmental mechanisms that may contribute to difficulties impacting social interactions in the syndrome. En ligne : http://dx.doi.org/10.1186/s11689-018-9245-x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=386 [article] Face processing in 22q11.2 deletion syndrome: atypical development and visual scanning alterations [texte imprimé] / A. ZAHARIA, Auteur ; Maude SCHNEIDER, Auteur ; Bronwyn GLASER, Auteur ; Martina FRANCHINI, Auteur ; S. MENGHETTI, Auteur ; M. SCHAER, Auteur ; Martin DEBBANE, Auteur ; Stephan ELIEZ, Auteur . - 2018 . - 26 p. Langues : Anglais (eng) in Journal of Neurodevelopmental Disorders > 10-1 (December 2018) . - 26 p. Mots-clés : Configural face processing  Eye-tracking  Featural face processing  Neurodevelopmental disorders  Social difficulties Index. décimale : PER Périodiques Résumé : BACKGROUND: Previous research links social difficulties to atypical face exploration in 22q11.2 deletion syndrome (22q11.2DS). Two types of face processing are distinguished: configural (CFP) and featural (FFP). CFP develops later in life and plays an important role in face and emotion recognition abilities. Recent studies reported atypical development of CFP in several neurodevelopmental disorders. Taking previous reports of atypical face exploration one step further, our study aims at characterizing face processing in children and adolescents with 22q11.2DS. First, we sought to identify biases in the first two fixation positions on faces and to detect differences between CFP and FFP in 22q11.2DS using eye-tracking technology. Second, we investigated the developmental trajectories of CFP and FFP using accuracy data from follow-up evaluation. METHODS: Seventy-five individuals with 22q11.2DS and 84 typically developed (TD) individuals (aged 6-21 years) completed a discrimination task ("Jane task") inducing CFP and FFP in an eye-tracking setting. Thirty-six individuals with 22q11DS and 30 TD from our sample completed a longitudinal follow-up evaluation. RESULTS: Findings revealed that individuals with 22q11.2DS demonstrate an early bias toward the mouth region during the initial fixations on the faces and reduced flexibility exploration of the faces, with a reduced number of transitions between faces and longer fixations compared to the TD group. Further, scanpaths did not differ between CFP and FFP in the 22q11.2DS group. Longitudinal analysis of accuracy data provided evidence for atypical development of CFP in 22q11.2DS. CONCLUSIONS: The current study brings new evidence of altered face exploration in 22q11.2DS and identifies developmental mechanisms that may contribute to difficulties impacting social interactions in the syndrome. En ligne : http://dx.doi.org/10.1186/s11689-018-9245-x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=386

Mentaliser. De la théorie à la pratique clinique / Martin DEBBANE

Public ISBD Titre : Mentaliser. De la théorie à la pratique clinique Type de document : texte imprimé Auteurs : Martin DEBBANE, Auteur ; Anthony W. BATEMAN, Préfacier, etc. ; Peter FONAGY, Préfacier, etc. ; Mario SPERANZA, Auteur de la postface, du colophon, etc. Editeur : Louvain [Belgique] : De Boeck Université Année de publication : 2018 Collection : Carrefour des psychothérapies, ISSN 1780-9517 Importance : 255 p. Format : 16,0 cm × 24,0 cm × 1,4 cm ISBN/ISSN/EAN : 978-2-8073-1515-0 Note générale : Bibliogr. Langues : Français (fre) Mots-clés : Mentalisation Index. décimale : ALT-E ALT-E - Autres Approches - Médiation - Divers Résumé : Les thérapies basées sur la capacité à mentaliser guident le clinicien dans les moments les plus corsés de sa pratique clinique afin d'optimiser son impact thérapeutique. Notre capacité à mentaliser - faire émerger les probables intentions, émotions, besoins qui se cachent derrière les actions d'autrui mais aussi les siennes propres - est un pilier de notre santé mentale. Sa défaillance ouvre la porte à des états psychopathologiques caractérisés par des tempêtes affectives, des ruptures relationnelles et des conduites auto-destructrices. Cet ouvrage, suivant les développements conceptuels des trente dernières années, en éclaire les mécanismes en montrant comment cette faculté se développe en lien aux relations d'attachement. Mais il pose aussi les bases cliniques des TBM, les thérapies basées sur la mentalisation - celle du patient comme celle du thérapeute. Construites sur des fondements théoriques issus notamment de la psychanalyse, de la psychologie du développement et des neurosciences, les TBM proposent un modèle intégré de la communication humaine, centré sur la mentalisation, et en tirent le postulat suivant : soutenir et solidifier le processus mentalisant permet d'optimiser l'effet de toute intervention visant des souffrances psychologiques chroniques. Ce livre guidera le professionnel pour optimiser son impact thérapeutique, y compris dans les moments critiques. [Résumé d'Auteur/Editeur] Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=391 Mentaliser. De la théorie à la pratique clinique [texte imprimé] / Martin DEBBANE, Auteur ; Anthony W. BATEMAN, Préfacier, etc. ; Peter FONAGY, Préfacier, etc. ; Mario SPERANZA, Auteur de la postface, du colophon, etc. . - Louvain [Belgique] : De Boeck Université, 2018 . - 255 p. ; 16,0 cm × 24,0 cm × 1,4 cm. - (Carrefour des psychothérapies, ISSN 1780-9517) . ISBN : 978-2-8073-1515-0 Bibliogr. Langues : Français (fre) Mots-clés : Mentalisation Index. décimale : ALT-E ALT-E - Autres Approches - Médiation - Divers Résumé : Les thérapies basées sur la capacité à mentaliser guident le clinicien dans les moments les plus corsés de sa pratique clinique afin d'optimiser son impact thérapeutique. Notre capacité à mentaliser - faire émerger les probables intentions, émotions, besoins qui se cachent derrière les actions d'autrui mais aussi les siennes propres - est un pilier de notre santé mentale. Sa défaillance ouvre la porte à des états psychopathologiques caractérisés par des tempêtes affectives, des ruptures relationnelles et des conduites auto-destructrices. Cet ouvrage, suivant les développements conceptuels des trente dernières années, en éclaire les mécanismes en montrant comment cette faculté se développe en lien aux relations d'attachement. Mais il pose aussi les bases cliniques des TBM, les thérapies basées sur la mentalisation - celle du patient comme celle du thérapeute. Construites sur des fondements théoriques issus notamment de la psychanalyse, de la psychologie du développement et des neurosciences, les TBM proposent un modèle intégré de la communication humaine, centré sur la mentalisation, et en tirent le postulat suivant : soutenir et solidifier le processus mentalisant permet d'optimiser l'effet de toute intervention visant des souffrances psychologiques chroniques. Ce livre guidera le professionnel pour optimiser son impact thérapeutique, y compris dans les moments critiques. [Résumé d'Auteur/Editeur] Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=391

Exemplaires(1)

Code-barres	Cote	Support	Localisation	Section	Disponibilité
DOC0004868	ALT-E DEB	Livre	Centre d'Information et de Documentation du CRA Rhône-Alpes	ALT - Thérapies à Médiation / Alternatives	Disponible

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Le superguide pour enfin oser être soi !	BOURNEL, Alda
L'aspinaute	BRESSON, Laura
TDA/H : Trouble Déficit de l'Attention / Hyperactivité	BANGE, François
Troubles de l'attention avec ou sans hyperactivité (TDAH)	GRAMOND, Anne
Neuropsychologie de l'enfant	LUSSIER, Francine
Oui, tu peux y arriver !	CAMBORDE, Mélanie

La microdélétion 22q11.2 / Maude SCHNEIDER

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Neural correlates of socio-emotional perception in 22q11.2 deletion syndrome / Lydia DUBOURG in Journal of Neurodevelopmental Disorders, 10-1 (December 2018)  

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Une perspective développementale et intégrative du profil neuropsychologique des jeunes porteurs de la microdélétion 22q11.2 / Johanna MAEDER

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Regional cortical volumes and congenital heart disease: a MRI study in 22q11.2 deletion syndrome / M. SCHAER in Journal of Neurodevelopmental Disorders, 2-4 (December 2010)  

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Structural and functional connectivity in the default mode network in 22q11.2 deletion syndrome / M.C. PADULA in Journal of Neurodevelopmental Disorders, 7-1 (December 2015)  

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Understanding others: a pilot investigation of cognitive and affective facets of social cognition in patients with 22q11.2 deletion syndrome (22q11DS) / D. BADOUD in Journal of Neurodevelopmental Disorders, 9-1 (December 2017)  

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Visual memory profile in 22q11.2 microdeletion syndrome: are there differences in performance and neurobiological substrates between tasks linked to ventral and dorsal visual brain structures? A cross-sectional and longitudinal study / Mathilde BOSTELMANN in Journal of Neurodevelopmental Disorders, 8-1 (December 2016)  

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