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Auteur A. FINLAY |
Documents disponibles écrits par cet auteur (2)
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An Investigation of Functional Communication Training and Schedule Thinning Using a Multiple Schedule on Elopement to Access Stereotypy / J. QUIGLEY in Journal of Autism and Developmental Disorders, 51-9 (September 2021)
[article]
Titre : An Investigation of Functional Communication Training and Schedule Thinning Using a Multiple Schedule on Elopement to Access Stereotypy Type de document : Texte imprimé et/ou numérique Auteurs : J. QUIGLEY, Auteur ; A. DOWDY, Auteur ; K. TRUCKSESS, Auteur ; A. FINLAY, Auteur Article en page(s) : p.3224-3234 Langues : Anglais (eng) Mots-clés : Autism Spectrum Disorder Behavior Therapy Communication Humans Reinforcement Schedule Stereotyped Behavior Chained problem behavior Elopement Functional communication training Generalization Multiple schedules of reinforcement Signaled availability Stereotypy Index. décimale : PER Périodiques Résumé : Individuals diagnosed with autism spectrum disorder (ASD) who engage in stereotypy may also emit a prior, temporally contiguous, high-risk response to access stereotypic behaviors. For example, the participant in this study who was diagnosed with ASD engaged in a chained response that included elopement, often in unsafe locations, to access light switch flipping. Previous research indicates that functional communication training (FCT) with delay fading is a viable approach to reduce chained problem behavior. In this study, we extended previous research by (a) evaluating the generalized effect of FCT and schedule thinning using multiple schedule technology for an automatically maintained chained response, and (b) evaluating whether intervention effects maintained in the participant's optimal context. Results for the participant suggested that FCT with schedule thinning mitigated high-risk chained responding across settings and discrimination training using a multiple schedule assessment effectively signaled available and unavailable times for the participant to emit the chained response which matched the participant's natural schedule parameters. En ligne : http://dx.doi.org/10.1007/s10803-020-04788-7 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=453
in Journal of Autism and Developmental Disorders > 51-9 (September 2021) . - p.3224-3234[article] An Investigation of Functional Communication Training and Schedule Thinning Using a Multiple Schedule on Elopement to Access Stereotypy [Texte imprimé et/ou numérique] / J. QUIGLEY, Auteur ; A. DOWDY, Auteur ; K. TRUCKSESS, Auteur ; A. FINLAY, Auteur . - p.3224-3234.
Langues : Anglais (eng)
in Journal of Autism and Developmental Disorders > 51-9 (September 2021) . - p.3224-3234
Mots-clés : Autism Spectrum Disorder Behavior Therapy Communication Humans Reinforcement Schedule Stereotyped Behavior Chained problem behavior Elopement Functional communication training Generalization Multiple schedules of reinforcement Signaled availability Stereotypy Index. décimale : PER Périodiques Résumé : Individuals diagnosed with autism spectrum disorder (ASD) who engage in stereotypy may also emit a prior, temporally contiguous, high-risk response to access stereotypic behaviors. For example, the participant in this study who was diagnosed with ASD engaged in a chained response that included elopement, often in unsafe locations, to access light switch flipping. Previous research indicates that functional communication training (FCT) with delay fading is a viable approach to reduce chained problem behavior. In this study, we extended previous research by (a) evaluating the generalized effect of FCT and schedule thinning using multiple schedule technology for an automatically maintained chained response, and (b) evaluating whether intervention effects maintained in the participant's optimal context. Results for the participant suggested that FCT with schedule thinning mitigated high-risk chained responding across settings and discrimination training using a multiple schedule assessment effectively signaled available and unavailable times for the participant to emit the chained response which matched the participant's natural schedule parameters. En ligne : http://dx.doi.org/10.1007/s10803-020-04788-7 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=453 The Monash Autism-ADHD genetics and neurodevelopment (MAGNET) project design and methodologies: a dimensional approach to understanding neurobiological and genetic aetiology / R. KNOTT in Molecular Autism, 12 (2021)
[article]
Titre : The Monash Autism-ADHD genetics and neurodevelopment (MAGNET) project design and methodologies: a dimensional approach to understanding neurobiological and genetic aetiology Type de document : Texte imprimé et/ou numérique Auteurs : R. KNOTT, Auteur ; Beth P. JOHNSON, Auteur ; J. TIEGO, Auteur ; O. MELLAHN, Auteur ; A. FINLAY, Auteur ; K. KALLADY, Auteur ; M. KOUSPOS, Auteur ; V. P. MOHANAKUMAR SINDHU, Auteur ; Z. HAWI, Auteur ; A. ARNATKEVICIUTE, Auteur ; T. CHAU, Auteur ; D. MARON, Auteur ; E. C. MERCIECA, Auteur ; K. FURLEY, Auteur ; K. HARRIS, Auteur ; K. WILLIAMS, Auteur ; A. URE, Auteur ; A. FORNITO, Auteur ; K. GRAY, Auteur ; D. COGHILL, Auteur ; A. NICHOLSON, Auteur ; D. PHUNG, Auteur ; E. LOTH, Auteur ; L. MASON, Auteur ; D. MURPHY, Auteur ; Jan K. BUITELAAR, Auteur ; Mark A. BELLGROVE, Auteur Article en page(s) : 55 p. Langues : Anglais (eng) Mots-clés : Adhd Asd Cognition Eye-tracking Genetics HiTOP Neuroimaging RDoC Index. décimale : PER Périodiques Résumé : BACKGROUND: ASD and ADHD are prevalent neurodevelopmental disorders that frequently co-occur and have strong evidence for a degree of shared genetic aetiology. Behavioural and neurocognitive heterogeneity in ASD and ADHD has hampered attempts to map the underlying genetics and neurobiology, predict intervention response, and improve diagnostic accuracy. Moving away from categorical conceptualisations of psychopathology to a dimensional approach is anticipated to facilitate discovery of data-driven clusters and enhance our understanding of the neurobiological and genetic aetiology of these conditions. The Monash Autism-ADHD genetics and neurodevelopment (MAGNET) project is one of the first large-scale, family-based studies to take a truly transdiagnostic approach to ASD and ADHD. Using a comprehensive phenotyping protocol capturing dimensional traits central to ASD and ADHD, the MAGNET project aims to identify data-driven clusters across ADHD-ASD spectra using deep phenotyping of symptoms and behaviours; investigate the degree of familiality for different dimensional ASD-ADHD phenotypes and clusters; and map the neurocognitive, brain imaging, and genetic correlates of these data-driven symptom-based clusters. METHODS: The MAGNET project will recruit 1,200 families with children who are either typically developing, or who display elevated ASD, ADHD, or ASD-ADHD traits, in addition to affected and unaffected biological siblings of probands, and parents. All children will be comprehensively phenotyped for behavioural symptoms, comorbidities, neurocognitive and neuroimaging traits and genetics. CONCLUSION: The MAGNET project will be the first large-scale family study to take a transdiagnostic approach to ASD-ADHD, utilising deep phenotyping across behavioural, neurocognitive, brain imaging and genetic measures. En ligne : http://dx.doi.org/10.1186/s13229-021-00457-3 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=459
in Molecular Autism > 12 (2021) . - 55 p.[article] The Monash Autism-ADHD genetics and neurodevelopment (MAGNET) project design and methodologies: a dimensional approach to understanding neurobiological and genetic aetiology [Texte imprimé et/ou numérique] / R. KNOTT, Auteur ; Beth P. JOHNSON, Auteur ; J. TIEGO, Auteur ; O. MELLAHN, Auteur ; A. FINLAY, Auteur ; K. KALLADY, Auteur ; M. KOUSPOS, Auteur ; V. P. MOHANAKUMAR SINDHU, Auteur ; Z. HAWI, Auteur ; A. ARNATKEVICIUTE, Auteur ; T. CHAU, Auteur ; D. MARON, Auteur ; E. C. MERCIECA, Auteur ; K. FURLEY, Auteur ; K. HARRIS, Auteur ; K. WILLIAMS, Auteur ; A. URE, Auteur ; A. FORNITO, Auteur ; K. GRAY, Auteur ; D. COGHILL, Auteur ; A. NICHOLSON, Auteur ; D. PHUNG, Auteur ; E. LOTH, Auteur ; L. MASON, Auteur ; D. MURPHY, Auteur ; Jan K. BUITELAAR, Auteur ; Mark A. BELLGROVE, Auteur . - 55 p.
Langues : Anglais (eng)
in Molecular Autism > 12 (2021) . - 55 p.
Mots-clés : Adhd Asd Cognition Eye-tracking Genetics HiTOP Neuroimaging RDoC Index. décimale : PER Périodiques Résumé : BACKGROUND: ASD and ADHD are prevalent neurodevelopmental disorders that frequently co-occur and have strong evidence for a degree of shared genetic aetiology. Behavioural and neurocognitive heterogeneity in ASD and ADHD has hampered attempts to map the underlying genetics and neurobiology, predict intervention response, and improve diagnostic accuracy. Moving away from categorical conceptualisations of psychopathology to a dimensional approach is anticipated to facilitate discovery of data-driven clusters and enhance our understanding of the neurobiological and genetic aetiology of these conditions. The Monash Autism-ADHD genetics and neurodevelopment (MAGNET) project is one of the first large-scale, family-based studies to take a truly transdiagnostic approach to ASD and ADHD. Using a comprehensive phenotyping protocol capturing dimensional traits central to ASD and ADHD, the MAGNET project aims to identify data-driven clusters across ADHD-ASD spectra using deep phenotyping of symptoms and behaviours; investigate the degree of familiality for different dimensional ASD-ADHD phenotypes and clusters; and map the neurocognitive, brain imaging, and genetic correlates of these data-driven symptom-based clusters. METHODS: The MAGNET project will recruit 1,200 families with children who are either typically developing, or who display elevated ASD, ADHD, or ASD-ADHD traits, in addition to affected and unaffected biological siblings of probands, and parents. All children will be comprehensively phenotyped for behavioural symptoms, comorbidities, neurocognitive and neuroimaging traits and genetics. CONCLUSION: The MAGNET project will be the first large-scale family study to take a transdiagnostic approach to ASD-ADHD, utilising deep phenotyping across behavioural, neurocognitive, brain imaging and genetic measures. En ligne : http://dx.doi.org/10.1186/s13229-021-00457-3 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=459