Are there negative cycles of peer victimization and rejection sensitivity? Testing ri-CLPMs in two longitudinal samples of young adolescents / Gerine M.A. LODDER ; Matteo GILETTA ; Melanie J. ZIMMER-GEMBECK ; Berna GÜROĞLU ; René VEENSTRA in Development and Psychopathology, 36-2 (May 2024)  

Public ISBD [article]  inDevelopment and Psychopathology > 36-2 (May 2024) . - p.844-856 Titre : Are there negative cycles of peer victimization and rejection sensitivity? Testing ri-CLPMs in two longitudinal samples of young adolescents Type de document : texte imprimé Auteurs : Gerine M.A. LODDER, Auteur ; Matteo GILETTA, Auteur ; Melanie J. ZIMMER-GEMBECK, Auteur ; Berna GÜROĞLU, Auteur ; René VEENSTRA, Auteur Article en page(s) : p.844-856 Langues : Anglais (eng) Mots-clés : Social Information Processing Theory  between- and within-person effects  bullying  peer victimization  rejection sensitivity Index. décimale : PER Périodiques Résumé : This study?s aim was to examine whether there are negative increasing cycles of peer victimization and rejection sensitivity over time. Drawing from Social Information Processing Theory, we hypothesized that victimization leads to higher levels of rejection sensitivity, which would put adolescents at risk for higher future victimization. Data were collected in a four-wave study with 233 Dutch adolescents starting secondary education (Mage = 12.7 years), and a three-wave study with 711 Australian adolescents in the last years of primary school (Mage = 10.8 years). Random-intercept cross-lagged panel models were used to disentangle between-person from within-person effects. In each sample, a significant between-person association was found: adolescents with higher levels of victimization as compared to their peers also reported higher levels of rejection sensitivity. At the within-person level, all concurrent associations between individual fluctuations of victimization and rejection sensitivity were significant, but there were no significant cross-lagged effects (except in some sensitivity analyses). These findings demonstrate that victimization and rejection sensitivity are interrelated, but there may not be negative victimization-rejection sensitivity cycles during the early-middle adolescent years. Possibly, cycles establish earlier in life or results are due to shared underlying factors. Further research is needed examining different time lags between assessments, age groups, and contexts. En ligne : https://dx.doi.org/10.1017/S0954579423000123 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=528 [article] Are there negative cycles of peer victimization and rejection sensitivity? Testing ri-CLPMs in two longitudinal samples of young adolescents [texte imprimé] / Gerine M.A. LODDER, Auteur ; Matteo GILETTA, Auteur ; Melanie J. ZIMMER-GEMBECK, Auteur ; Berna GÜROĞLU, Auteur ; René VEENSTRA, Auteur . - p.844-856. Langues : Anglais (eng) in Development and Psychopathology > 36-2 (May 2024) . - p.844-856 Mots-clés : Social Information Processing Theory  between- and within-person effects  bullying  peer victimization  rejection sensitivity Index. décimale : PER Périodiques Résumé : This study?s aim was to examine whether there are negative increasing cycles of peer victimization and rejection sensitivity over time. Drawing from Social Information Processing Theory, we hypothesized that victimization leads to higher levels of rejection sensitivity, which would put adolescents at risk for higher future victimization. Data were collected in a four-wave study with 233 Dutch adolescents starting secondary education (Mage = 12.7 years), and a three-wave study with 711 Australian adolescents in the last years of primary school (Mage = 10.8 years). Random-intercept cross-lagged panel models were used to disentangle between-person from within-person effects. In each sample, a significant between-person association was found: adolescents with higher levels of victimization as compared to their peers also reported higher levels of rejection sensitivity. At the within-person level, all concurrent associations between individual fluctuations of victimization and rejection sensitivity were significant, but there were no significant cross-lagged effects (except in some sensitivity analyses). These findings demonstrate that victimization and rejection sensitivity are interrelated, but there may not be negative victimization-rejection sensitivity cycles during the early-middle adolescent years. Possibly, cycles establish earlier in life or results are due to shared underlying factors. Further research is needed examining different time lags between assessments, age groups, and contexts. En ligne : https://dx.doi.org/10.1017/S0954579423000123 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=528

Examining diurnal cortisol changes as a pathway linking childhood adversity to depressive symptoms during adolescence / Tamara LORENZ in Development and Psychopathology, 38-2 (May 2026)  

Public ISBD [article]  inDevelopment and Psychopathology > 38-2 (May 2026) . - p.974-985 Titre : Examining diurnal cortisol changes as a pathway linking childhood adversity to depressive symptoms during adolescence Type de document : texte imprimé Auteurs : Tamara LORENZ, Auteur ; Nathalie MICHELS, Auteur ; Matteo GILETTA, Auteur Article en page(s) : p.974-985 Langues : Anglais (eng) Mots-clés : Adolescents  HPA axis  childhood adversity  depression  salivary diurnal cortisol Index. décimale : PER Périodiques Résumé : This study examined whether childhood adversity, specifically threat-related adversity, was associated with within-person changes in the cortisol awakening response (CAR) and diurnal cortisol slope (DCS), and whether these changes predicted increased depressive symptoms during adolescence. We also explored sex differences. In total, 283 first-year secondary school students in Belgium (M = 12.48 years; SD = 0.39; 42.8% female) participated in six assessments over 2.5 years. Childhood adversity (psychological, physical, and sexual victimization) reported at the first three waves was averaged. CAR and DCS latent residual change scores were derived from salivary cortisol samples collected during waves 1 and 3. Depressive symptom changes were assessed in linear growth curve models using self-reports from waves 3 to 6. The childhood adversity × sex interaction significantly predicted CAR and DCS changes, indicating a blunted CAR across waves for victimized boys, and a blunted DCS for victimized girls. Childhood adversity predicted the depressive symptoms intercept. No other predictors were associated with the depressive symptoms intercept, and none were linked to the depressive symptoms slope. Thus, childhood adversity may be linked to changes in diurnal cortisol patterns that differ by sex. Evidence for diurnal cortisol changes as a pathway to increased depressive symptoms remains inconclusive. En ligne : https://dx.doi.org/10.1017/S0954579425100916 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=586 [article] Examining diurnal cortisol changes as a pathway linking childhood adversity to depressive symptoms during adolescence [texte imprimé] / Tamara LORENZ, Auteur ; Nathalie MICHELS, Auteur ; Matteo GILETTA, Auteur . - p.974-985. Langues : Anglais (eng) in Development and Psychopathology > 38-2 (May 2026) . - p.974-985 Mots-clés : Adolescents  HPA axis  childhood adversity  depression  salivary diurnal cortisol Index. décimale : PER Périodiques Résumé : This study examined whether childhood adversity, specifically threat-related adversity, was associated with within-person changes in the cortisol awakening response (CAR) and diurnal cortisol slope (DCS), and whether these changes predicted increased depressive symptoms during adolescence. We also explored sex differences. In total, 283 first-year secondary school students in Belgium (M = 12.48 years; SD = 0.39; 42.8% female) participated in six assessments over 2.5 years. Childhood adversity (psychological, physical, and sexual victimization) reported at the first three waves was averaged. CAR and DCS latent residual change scores were derived from salivary cortisol samples collected during waves 1 and 3. Depressive symptom changes were assessed in linear growth curve models using self-reports from waves 3 to 6. The childhood adversity × sex interaction significantly predicted CAR and DCS changes, indicating a blunted CAR across waves for victimized boys, and a blunted DCS for victimized girls. Childhood adversity predicted the depressive symptoms intercept. No other predictors were associated with the depressive symptoms intercept, and none were linked to the depressive symptoms slope. Thus, childhood adversity may be linked to changes in diurnal cortisol patterns that differ by sex. Evidence for diurnal cortisol changes as a pathway to increased depressive symptoms remains inconclusive. En ligne : https://dx.doi.org/10.1017/S0954579425100916 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=586

Examining systemic inflammation as a pathway linking peer victimization to depressive symptoms in adolescence / Nathalie MICHELS ; George M. SLAVICH ; Matteo GILETTA in Journal of Child Psychology and Psychiatry, 66-3 (March 2025)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 66-3 (March 2025) . - p.311-321 Titre : Examining systemic inflammation as a pathway linking peer victimization to depressive symptoms in adolescence Type de document : texte imprimé Auteurs : Nathalie MICHELS, Auteur ; George M. SLAVICH, Auteur ; Matteo GILETTA, Auteur Article en page(s) : p.311-321 Langues : Anglais (eng) Mots-clés : Adolescence  depression  inflammation  interleukin-6  peer victimization  social stress Index. décimale : PER Périodiques Résumé : Background Adolescents exposed to victimization are at an increased risk for a variety of adverse mental health outcomes, including depressive symptoms. Yet, the biological pathways underlying these associations remain poorly understood. Focusing on within-person processes, we examined whether low-grade systemic inflammation mediated the longitudinal associations between peer victimization and depressive symptoms in adolescence. Methods 207 adolescents (at baseline Mage 12.69 years; SD 0.49; 43.5% female) participated in a multi-wave longitudinal study, with assessments repeated every 6 months over 1.5 years. At each assessment wave, participants self-reported their peer victimization experiences and depressive symptoms. Dried blood spots were collected at each wave using a finger prick procedure to assay a key marker of low-grade systemic inflammation, interkeukin-6 (IL-6). Data were analyzed using random-intercept cross-lagged panel models. Results The cross-lagged paths from IL-6 to depressive symptoms were significant across all models and waves ( 12 .13; 23 .12; 34 .08), indicating that when adolescents' levels of low-grade systemic inflammation were above their person-specific average, they reported increased levels of depressive symptoms in the subsequent months. However, no significant cross-lagged within-person associations emerged between peer victimization and either IL-6 or depressive symptoms. Conclusions The findings provide no evidence for the hypothesized mediating role of inflammation in the within-person associations between peer victimization and depressive symptoms. Nevertheless, they extend prior research by indicating that elevated levels of low-grade systemic inflammation predict the development of depressive symptoms in adolescence. En ligne : https://doi.org/10.1111/jcpp.14060 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=548 [article] Examining systemic inflammation as a pathway linking peer victimization to depressive symptoms in adolescence [texte imprimé] / Nathalie MICHELS, Auteur ; George M. SLAVICH, Auteur ; Matteo GILETTA, Auteur . - p.311-321. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 66-3 (March 2025) . - p.311-321 Mots-clés : Adolescence  depression  inflammation  interleukin-6  peer victimization  social stress Index. décimale : PER Périodiques Résumé : Background Adolescents exposed to victimization are at an increased risk for a variety of adverse mental health outcomes, including depressive symptoms. Yet, the biological pathways underlying these associations remain poorly understood. Focusing on within-person processes, we examined whether low-grade systemic inflammation mediated the longitudinal associations between peer victimization and depressive symptoms in adolescence. Methods 207 adolescents (at baseline Mage 12.69 years; SD 0.49; 43.5% female) participated in a multi-wave longitudinal study, with assessments repeated every 6 months over 1.5 years. At each assessment wave, participants self-reported their peer victimization experiences and depressive symptoms. Dried blood spots were collected at each wave using a finger prick procedure to assay a key marker of low-grade systemic inflammation, interkeukin-6 (IL-6). Data were analyzed using random-intercept cross-lagged panel models. Results The cross-lagged paths from IL-6 to depressive symptoms were significant across all models and waves ( 12 .13; 23 .12; 34 .08), indicating that when adolescents' levels of low-grade systemic inflammation were above their person-specific average, they reported increased levels of depressive symptoms in the subsequent months. However, no significant cross-lagged within-person associations emerged between peer victimization and either IL-6 or depressive symptoms. Conclusions The findings provide no evidence for the hypothesized mediating role of inflammation in the within-person associations between peer victimization and depressive symptoms. Nevertheless, they extend prior research by indicating that elevated levels of low-grade systemic inflammation predict the development of depressive symptoms in adolescence. En ligne : https://doi.org/10.1111/jcpp.14060 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=548

Peer victimization predicts heightened inflammatory reactivity to social stress in cognitively vulnerable adolescents / Matteo GILETTA in Journal of Child Psychology and Psychiatry, 59-2 (February 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-2 (February 2018) . - p.129-139 Titre : Peer victimization predicts heightened inflammatory reactivity to social stress in cognitively vulnerable adolescents Type de document : texte imprimé Auteurs : Matteo GILETTA, Auteur ; George M. SLAVICH, Auteur ; Karen D. RUDOLPH, Auteur ; Paul D. HASTINGS, Auteur ; Matthew K. NOCK, Auteur ; Mitchell J. PRINSTEIN, Auteur Article en page(s) : p.129-139 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Background During adolescence, peer victimization is a potent type of social stressor that can confer enduring risk for poor mental and physical health. Given recent research implicating inflammation in promoting a variety of serious mental and physical health problems, this study examined the role that peer victimization and cognitive vulnerability (i.e. negative cognitive styles and hopelessness) play in shaping adolescents’ pro‐inflammatory cytokine responses to an acute social stressor. Methods Adolescent girls at risk for psychopathology (n = 157; Mage = 14.73 years; SD = 1.38) were exposed to a laboratory‐based social stressor before and after which we assessed salivary levels of three key pro‐inflammatory cytokines – interleukin‐6 (IL‐6), interleukin‐1β (IL‐1β), and tumor necrosis factor‐α (TNF‐α). Results As hypothesized, adolescents with greater peer victimization exposure exhibited greater increases in IL‐6 and IL1‐β in response to the laboratory‐based social stressor. Moreover, for all three cytokines individually, as well as for a combined latent factor of inflammation, peer victimization predicted enhanced inflammatory responding most strongly for adolescents with high levels of hopelessness. Conclusions The findings reveal a biological pathway by which peer victimization may interact with cognitive vulnerability to influence health in adolescence. En ligne : https://doi.org/10.1111/jcpp.12804 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=339 [article] Peer victimization predicts heightened inflammatory reactivity to social stress in cognitively vulnerable adolescents [texte imprimé] / Matteo GILETTA, Auteur ; George M. SLAVICH, Auteur ; Karen D. RUDOLPH, Auteur ; Paul D. HASTINGS, Auteur ; Matthew K. NOCK, Auteur ; Mitchell J. PRINSTEIN, Auteur . - p.129-139. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-2 (February 2018) . - p.129-139 Index. décimale : PER Périodiques Résumé : Background During adolescence, peer victimization is a potent type of social stressor that can confer enduring risk for poor mental and physical health. Given recent research implicating inflammation in promoting a variety of serious mental and physical health problems, this study examined the role that peer victimization and cognitive vulnerability (i.e. negative cognitive styles and hopelessness) play in shaping adolescents’ pro‐inflammatory cytokine responses to an acute social stressor. Methods Adolescent girls at risk for psychopathology (n = 157; Mage = 14.73 years; SD = 1.38) were exposed to a laboratory‐based social stressor before and after which we assessed salivary levels of three key pro‐inflammatory cytokines – interleukin‐6 (IL‐6), interleukin‐1β (IL‐1β), and tumor necrosis factor‐α (TNF‐α). Results As hypothesized, adolescents with greater peer victimization exposure exhibited greater increases in IL‐6 and IL1‐β in response to the laboratory‐based social stressor. Moreover, for all three cytokines individually, as well as for a combined latent factor of inflammation, peer victimization predicted enhanced inflammatory responding most strongly for adolescents with high levels of hopelessness. Conclusions The findings reveal a biological pathway by which peer victimization may interact with cognitive vulnerability to influence health in adolescence. En ligne : https://doi.org/10.1111/jcpp.12804 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=339

Proinflammatory gene expression is associated with prospective risk for adolescent suicidal thoughts and behaviors over twelve months / Matthew G. CLAYTON in Development and Psychopathology, 37-3 (August 2025)  

Public ISBD [article]  inDevelopment and Psychopathology > 37-3 (August 2025) . - p.1676-1684 Titre : Proinflammatory gene expression is associated with prospective risk for adolescent suicidal thoughts and behaviors over twelve months Type de document : texte imprimé Auteurs : Matthew G. CLAYTON, Auteur ; Steven W. COLE, Auteur ; Matteo GILETTA, Auteur ; Paul D. HASTINGS, Auteur ; Matthew K. NOCK, Auteur ; Karen D. RUDOLPH, Auteur ; George M. SLAVICH, Auteur ; Mitchell J. PRINSTEIN, Auteur Article en page(s) : p.1676-1684 Langues : Anglais (eng) Mots-clés : adolescence  biomarkers  childhood trauma  inflammation  suicide Index. décimale : PER Périodiques Résumé : Objective:Recent theories have implicated inflammatory biology in the development of psychopathology and maladaptive behaviors in adolescence, including suicidal thoughts and behaviors (STB). Examining specific biological markers related to inflammation is thus warranted to better understand risk for STB in adolescents, for whom suicide is a leading cause of death.Method:Participants were 211 adolescent females (ages 9-14 years; Mage = 11.8 years, SD = 1.8 years) at increased risk for STB. This study examined the prospective association between basal levels of inflammatory gene expression (average of 15 proinflammatory mRNA transcripts) and subsequent risk for suicidal ideation and suicidal behavior over a 12-month follow-up period.Results:Controlling for past levels of STB, greater proinflammatory gene expression was associated with prospective risk for STB in these youth. Similar effects were observed for CD14 mRNA level, a marker of monocyte abundance within the blood sample. Sensitivity analyses controlling for other relevant covariates, including history of trauma, depressive symptoms, and STB prior to data collection, yielded similar patterns of results.Conclusions:Upregulated inflammatory signaling in the immune system is prospectively associated with STB among at-risk adolescent females, even after controlling for history of trauma, depressive symptoms, and STB prior to data collection. Additional research is needed to identify the sources of inflammatory up-regulation in adolescents (e.g., stress psychobiology, physiological development, microbial exposures) and strategies for mitigating such effects to reduce STB. En ligne : https://www.cambridge.org/core/product/25AD4CF3A7BD16D263771246B7E45F56 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=564 [article] Proinflammatory gene expression is associated with prospective risk for adolescent suicidal thoughts and behaviors over twelve months [texte imprimé] / Matthew G. CLAYTON, Auteur ; Steven W. COLE, Auteur ; Matteo GILETTA, Auteur ; Paul D. HASTINGS, Auteur ; Matthew K. NOCK, Auteur ; Karen D. RUDOLPH, Auteur ; George M. SLAVICH, Auteur ; Mitchell J. PRINSTEIN, Auteur . - p.1676-1684. Langues : Anglais (eng) in Development and Psychopathology > 37-3 (August 2025) . - p.1676-1684 Mots-clés : adolescence  biomarkers  childhood trauma  inflammation  suicide Index. décimale : PER Périodiques Résumé : Objective:Recent theories have implicated inflammatory biology in the development of psychopathology and maladaptive behaviors in adolescence, including suicidal thoughts and behaviors (STB). Examining specific biological markers related to inflammation is thus warranted to better understand risk for STB in adolescents, for whom suicide is a leading cause of death.Method:Participants were 211 adolescent females (ages 9-14 years; Mage = 11.8 years, SD = 1.8 years) at increased risk for STB. This study examined the prospective association between basal levels of inflammatory gene expression (average of 15 proinflammatory mRNA transcripts) and subsequent risk for suicidal ideation and suicidal behavior over a 12-month follow-up period.Results:Controlling for past levels of STB, greater proinflammatory gene expression was associated with prospective risk for STB in these youth. Similar effects were observed for CD14 mRNA level, a marker of monocyte abundance within the blood sample. Sensitivity analyses controlling for other relevant covariates, including history of trauma, depressive symptoms, and STB prior to data collection, yielded similar patterns of results.Conclusions:Upregulated inflammatory signaling in the immune system is prospectively associated with STB among at-risk adolescent females, even after controlling for history of trauma, depressive symptoms, and STB prior to data collection. Additional research is needed to identify the sources of inflammatory up-regulation in adolescents (e.g., stress psychobiology, physiological development, microbial exposures) and strategies for mitigating such effects to reduce STB. En ligne : https://www.cambridge.org/core/product/25AD4CF3A7BD16D263771246B7E45F56 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=564

Reciprocal associations between peer problems and non-suicidal self-injury throughout adolescence / Lisa DE LUCA in Journal of Child Psychology and Psychiatry, 63-12 (December 2022)  

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Suicide ideation among high-risk adolescent females: Examining the interplay between parasympathetic regulation and friendship support / Matteo GILETTA in Development and Psychopathology, 29-4 (October 2017)  

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