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Auteur Gabriel L. SCHLOMER |
Documents disponibles écrits par cet auteur (7)



Associations between alcohol dehydrogenase genes and alcohol use across early and middle adolescence: Moderation × Preventive intervention / H. Harrington CLEVELAND in Development and Psychopathology, 30-1 (February 2018)
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Titre : Associations between alcohol dehydrogenase genes and alcohol use across early and middle adolescence: Moderation × Preventive intervention Type de document : Texte imprimé et/ou numérique Auteurs : H. Harrington CLEVELAND, Auteur ; Gabriel L. SCHLOMER, Auteur ; David J. VANDENBERGH, Auteur ; Pedro S. A. WOLF, Auteur ; Mark E. FEINBERG, Auteur ; Mark T. GREENBERG, Auteur ; Richard L. SPOTH, Auteur ; Cleve REDMOND, Auteur Article en page(s) : p.297-313 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Data from the in-school sample of the PROSPER preventive intervention dissemination trial were used to investigate associations between alcohol dehydrogenase genes and alcohol use across adolescence, and whether substance misuse interventions in the 6th and 7th grades (targeting parenting, family functioning, social norms, youth decision making, and peer group affiliations) modified associations between these genes and adolescent use. Primary analyses were run on a sample of 1,885 individuals and included three steps. First, we estimated unconditional growth curve models with separate slopes for alcohol use from 6th to 9th grade and from 9th to 12th grade, as well as the intercept at Grade 9. Second, we used intervention condition and three alcohol dehydrogenase genes, 1B (ADH1B), 1C (ADH1C), and 4 (ADH4) to predict variance in slopes and intercept. Third, we examined whether genetic influences on model slopes and intercepts were moderated by intervention condition. The results indicated that the increase in alcohol use was greater in early adolescence than in middle adolescence; two of the genes, ADH1B and ADH1C, significantly predicted early adolescent slope and Grade 9 intercept, and associations between ADH1C and both early adolescent slope and intercept were significantly different across control and intervention conditions. En ligne : https://doi.org/10.1017/S0954579417000633 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336
in Development and Psychopathology > 30-1 (February 2018) . - p.297-313[article] Associations between alcohol dehydrogenase genes and alcohol use across early and middle adolescence: Moderation × Preventive intervention [Texte imprimé et/ou numérique] / H. Harrington CLEVELAND, Auteur ; Gabriel L. SCHLOMER, Auteur ; David J. VANDENBERGH, Auteur ; Pedro S. A. WOLF, Auteur ; Mark E. FEINBERG, Auteur ; Mark T. GREENBERG, Auteur ; Richard L. SPOTH, Auteur ; Cleve REDMOND, Auteur . - p.297-313.
Langues : Anglais (eng)
in Development and Psychopathology > 30-1 (February 2018) . - p.297-313
Index. décimale : PER Périodiques Résumé : Data from the in-school sample of the PROSPER preventive intervention dissemination trial were used to investigate associations between alcohol dehydrogenase genes and alcohol use across adolescence, and whether substance misuse interventions in the 6th and 7th grades (targeting parenting, family functioning, social norms, youth decision making, and peer group affiliations) modified associations between these genes and adolescent use. Primary analyses were run on a sample of 1,885 individuals and included three steps. First, we estimated unconditional growth curve models with separate slopes for alcohol use from 6th to 9th grade and from 9th to 12th grade, as well as the intercept at Grade 9. Second, we used intervention condition and three alcohol dehydrogenase genes, 1B (ADH1B), 1C (ADH1C), and 4 (ADH4) to predict variance in slopes and intercept. Third, we examined whether genetic influences on model slopes and intercepts were moderated by intervention condition. The results indicated that the increase in alcohol use was greater in early adolescence than in middle adolescence; two of the genes, ADH1B and ADH1C, significantly predicted early adolescent slope and Grade 9 intercept, and associations between ADH1C and both early adolescent slope and intercept were significantly different across control and intervention conditions. En ligne : https://doi.org/10.1017/S0954579417000633 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 Decomposing environmental unpredictability in forecasting adolescent and young adult development: A two-sample study / Sarah HARTMAN in Development and Psychopathology, 30-4 (October 2018)
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Titre : Decomposing environmental unpredictability in forecasting adolescent and young adult development: A two-sample study Type de document : Texte imprimé et/ou numérique Auteurs : Sarah HARTMAN, Auteur ; Sooyeon SUNG, Auteur ; Jeffry A. SIMPSON, Auteur ; Gabriel L. SCHLOMER, Auteur ; Jay BELSKY, Auteur Article en page(s) : p.1321-1332 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : To illuminate which features of an unpredictable environment early in life best forecast adolescent and adult functioning, data from two longitudinal studies were examined. After decomposing a composite unpredictability construct found to predict later development, results of both studies revealed that paternal transitions predicted outcomes more consistently and strongly than did residential or occupational changes across the first 5 years of a child's life. These results derive from analyses of the NICHD Study of Early Child Care and Youth Development, which included diverse families from 10 different sites in the United States, and from the Minnesota Longitudinal Study of Risk and Adaptation, whose participants came from one site, were disproportionately economically disadvantaged, and were enrolled 15 years earlier than the NICHD Study sample. The finding that results from both studies are consistent with evolutionary, life history thinking regarding the importance of males in children's lives makes this general, cross-study replication noteworthy. En ligne : http://dx.doi.org/10.1017/S0954579417001729 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=368
in Development and Psychopathology > 30-4 (October 2018) . - p.1321-1332[article] Decomposing environmental unpredictability in forecasting adolescent and young adult development: A two-sample study [Texte imprimé et/ou numérique] / Sarah HARTMAN, Auteur ; Sooyeon SUNG, Auteur ; Jeffry A. SIMPSON, Auteur ; Gabriel L. SCHLOMER, Auteur ; Jay BELSKY, Auteur . - p.1321-1332.
Langues : Anglais (eng)
in Development and Psychopathology > 30-4 (October 2018) . - p.1321-1332
Index. décimale : PER Périodiques Résumé : To illuminate which features of an unpredictable environment early in life best forecast adolescent and adult functioning, data from two longitudinal studies were examined. After decomposing a composite unpredictability construct found to predict later development, results of both studies revealed that paternal transitions predicted outcomes more consistently and strongly than did residential or occupational changes across the first 5 years of a child's life. These results derive from analyses of the NICHD Study of Early Child Care and Youth Development, which included diverse families from 10 different sites in the United States, and from the Minnesota Longitudinal Study of Risk and Adaptation, whose participants came from one site, were disproportionately economically disadvantaged, and were enrolled 15 years earlier than the NICHD Study sample. The finding that results from both studies are consistent with evolutionary, life history thinking regarding the importance of males in children's lives makes this general, cross-study replication noteworthy. En ligne : http://dx.doi.org/10.1017/S0954579417001729 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=368 Environmental harshness and unpredictability: Do they affect the same parents and children? / Xiaoya ZHANG in Development and Psychopathology, 34-2 (May 2022)
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Titre : Environmental harshness and unpredictability: Do they affect the same parents and children? Type de document : Texte imprimé et/ou numérique Auteurs : Xiaoya ZHANG, Auteur ; Gabriel L. SCHLOMER, Auteur ; Bruce J. ELLIS, Auteur ; Jay BELSKY, Auteur Article en page(s) : 667-673 Langues : Anglais (eng) Mots-clés : differential susceptibility harshness life-history theory unpredictability Index. décimale : PER Périodiques Résumé : Differential susceptibility theory stipulates that individuals vary in their susceptibility to environmental effects, often implying that the same individuals differ in the same way in their susceptibility to different environmental exposures. The latter point is addressed herein by evaluating the extent to which early-life harshness and unpredictability affect mother's psychological well-being and parenting, as well as their adolescent's life-history strategy, as reflected in number of sexual partners by age 15 years, drawing on data from the National Institute of Child Health and Human Development (NICHD) Study of Early Child Care and Youth Development. Results indicated that mothers whose well-being and parenting proved more susceptible to harshness also proved somewhat more susceptible to environmental unpredictability, with the same being true of adolescent sexual behavior. Nevertheless, findings caution against overgeneralizing sample-level findings to all individuals. En ligne : http://dx.doi.org/10.1017/s095457942100095x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=474
in Development and Psychopathology > 34-2 (May 2022) . - 667-673[article] Environmental harshness and unpredictability: Do they affect the same parents and children? [Texte imprimé et/ou numérique] / Xiaoya ZHANG, Auteur ; Gabriel L. SCHLOMER, Auteur ; Bruce J. ELLIS, Auteur ; Jay BELSKY, Auteur . - 667-673.
Langues : Anglais (eng)
in Development and Psychopathology > 34-2 (May 2022) . - 667-673
Mots-clés : differential susceptibility harshness life-history theory unpredictability Index. décimale : PER Périodiques Résumé : Differential susceptibility theory stipulates that individuals vary in their susceptibility to environmental effects, often implying that the same individuals differ in the same way in their susceptibility to different environmental exposures. The latter point is addressed herein by evaluating the extent to which early-life harshness and unpredictability affect mother's psychological well-being and parenting, as well as their adolescent's life-history strategy, as reflected in number of sexual partners by age 15 years, drawing on data from the National Institute of Child Health and Human Development (NICHD) Study of Early Child Care and Youth Development. Results indicated that mothers whose well-being and parenting proved more susceptible to harshness also proved somewhat more susceptible to environmental unpredictability, with the same being true of adolescent sexual behavior. Nevertheless, findings caution against overgeneralizing sample-level findings to all individuals. En ligne : http://dx.doi.org/10.1017/s095457942100095x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=474 Father absence, age at menarche, and genetic confounding: A replication and extension using a polygenic score / Gabriel L. SCHLOMER in Development and Psychopathology, 34-1 (February 2022)
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Titre : Father absence, age at menarche, and genetic confounding: A replication and extension using a polygenic score Type de document : Texte imprimé et/ou numérique Auteurs : Gabriel L. SCHLOMER, Auteur ; Kristine MARCEAU, Auteur Article en page(s) : p.355-366 Langues : Anglais (eng) Mots-clés : ALSPAC father absence LIN28B menarche PGS Index. décimale : PER Périodiques Résumé : Father absence has a small but robust association with earlier age at menarche (AAM), likely reflecting both genetic confounding and an environmental influence on life history strategy development. Studies that have attempted to disambiguate genetic versus environmental contributions to this association have shown conflicting findings, though genomic-based studies have begun to establish the role of gene?environment interplay in the father absence/AAM literature. The purpose of this study was to replicate and extend prior genomic work using the Avon Longitudinal Study of Parents and Children (ALSPAC), a prospective longitudinal cohort study (N = 2,685), by (a) testing if an AAM polygenic score (PGS) could account for the father absence/AAM association, (b) replicating G*E research on lin-28 homolog B (LIN28B) variation and father absence, and (c) testing the G*E hypothesis using the PGS. Results showed that the PGS could not explain the father absence/AAM association and there was no interaction between father absence and the PGS. Findings using LIN28B largely replicated prior work that showed LIN28B variants predicted later AAM in father-present girls, but this AAM-delaying effect was absent or reversed in father-absent girls. Findings are discussed in terms genetic confounding, the unique biological role of LIN28B, and using PGSs for G*E tests. En ligne : http://dx.doi.org/10.1017/s0954579420000929 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=474
in Development and Psychopathology > 34-1 (February 2022) . - p.355-366[article] Father absence, age at menarche, and genetic confounding: A replication and extension using a polygenic score [Texte imprimé et/ou numérique] / Gabriel L. SCHLOMER, Auteur ; Kristine MARCEAU, Auteur . - p.355-366.
Langues : Anglais (eng)
in Development and Psychopathology > 34-1 (February 2022) . - p.355-366
Mots-clés : ALSPAC father absence LIN28B menarche PGS Index. décimale : PER Périodiques Résumé : Father absence has a small but robust association with earlier age at menarche (AAM), likely reflecting both genetic confounding and an environmental influence on life history strategy development. Studies that have attempted to disambiguate genetic versus environmental contributions to this association have shown conflicting findings, though genomic-based studies have begun to establish the role of gene?environment interplay in the father absence/AAM literature. The purpose of this study was to replicate and extend prior genomic work using the Avon Longitudinal Study of Parents and Children (ALSPAC), a prospective longitudinal cohort study (N = 2,685), by (a) testing if an AAM polygenic score (PGS) could account for the father absence/AAM association, (b) replicating G*E research on lin-28 homolog B (LIN28B) variation and father absence, and (c) testing the G*E hypothesis using the PGS. Results showed that the PGS could not explain the father absence/AAM association and there was no interaction between father absence and the PGS. Findings using LIN28B largely replicated prior work that showed LIN28B variants predicted later AAM in father-present girls, but this AAM-delaying effect was absent or reversed in father-absent girls. Findings are discussed in terms genetic confounding, the unique biological role of LIN28B, and using PGSs for G*E tests. En ligne : http://dx.doi.org/10.1017/s0954579420000929 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=474 Impact of fathers on risky sexual behavior in daughters: A genetically and environmentally controlled sibling study / Bruce J. ELLIS in Development and Psychopathology, 24-1 (January 2012)
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Titre : Impact of fathers on risky sexual behavior in daughters: A genetically and environmentally controlled sibling study Type de document : Texte imprimé et/ou numérique Auteurs : Bruce J. ELLIS, Auteur ; Gabriel L. SCHLOMER, Auteur ; Elizabeth H. TILLEY, Auteur ; Emily A. BUTLER, Auteur Année de publication : 2012 Article en page(s) : p.317-332 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Girls receiving lower quality paternal investment tend to engage in more risky sexual behavior (RSB) than peers. Whereas paternal investment theory posits that this effect is causal, it could arise from environmental or genetic confounds. To distinguish between these competing explanations, the current authors employed a genetically and environmentally controlled sibling design (N = 101 sister pairs; ages 18–36), which retrospectively examined the effects of differential sibling exposure to family disruption/father absence and quality of fathering. Consistent with a causal explanation, differences between older and younger sisters in the effects of quality of fathering on RSB were greatest in biologically disrupted families when there was a large age gap between the sisters (thus maximizing differential exposure to fathers), with greater exposure within families to higher quality fathering serving as a protective factor against RSB. Further, variation around the lower end of fathering quality appeared to have the most influence on RSB. In contrast, differential sibling exposure to family disruption/father absence (irrespective of quality of fathering) was not associated with RSB. The differential sibling-exposure design affords a new quasi-experimental method for evaluating the causal effects of fathers within families. En ligne : http://dx.doi.org/10.1017/S095457941100085X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=152
in Development and Psychopathology > 24-1 (January 2012) . - p.317-332[article] Impact of fathers on risky sexual behavior in daughters: A genetically and environmentally controlled sibling study [Texte imprimé et/ou numérique] / Bruce J. ELLIS, Auteur ; Gabriel L. SCHLOMER, Auteur ; Elizabeth H. TILLEY, Auteur ; Emily A. BUTLER, Auteur . - 2012 . - p.317-332.
Langues : Anglais (eng)
in Development and Psychopathology > 24-1 (January 2012) . - p.317-332
Index. décimale : PER Périodiques Résumé : Girls receiving lower quality paternal investment tend to engage in more risky sexual behavior (RSB) than peers. Whereas paternal investment theory posits that this effect is causal, it could arise from environmental or genetic confounds. To distinguish between these competing explanations, the current authors employed a genetically and environmentally controlled sibling design (N = 101 sister pairs; ages 18–36), which retrospectively examined the effects of differential sibling exposure to family disruption/father absence and quality of fathering. Consistent with a causal explanation, differences between older and younger sisters in the effects of quality of fathering on RSB were greatest in biologically disrupted families when there was a large age gap between the sisters (thus maximizing differential exposure to fathers), with greater exposure within families to higher quality fathering serving as a protective factor against RSB. Further, variation around the lower end of fathering quality appeared to have the most influence on RSB. In contrast, differential sibling exposure to family disruption/father absence (irrespective of quality of fathering) was not associated with RSB. The differential sibling-exposure design affords a new quasi-experimental method for evaluating the causal effects of fathers within families. En ligne : http://dx.doi.org/10.1017/S095457941100085X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=152 The conditioning of intervention effects on early adolescent alcohol use by maternal involvement and dopamine receptor D4 (DRD4) and serotonin transporter linked polymorphic region (5-HTTLPR) genetic variants / H. Harrington CLEVELAND in Development and Psychopathology, 27-1 (February 2015)
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PermalinkThe influence of harshness and unpredictability on female sexual development: Addressing gene?environment interplay using a polygenic score / Gabriel L. SCHLOMER in Development and Psychopathology, 34-2 (May 2022)
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