Dépouillements

Effectiveness of interventions in preventing disorganized attachment: A meta-analysis / Christopher R. FACOMPRÉ in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.1-11 Titre : Effectiveness of interventions in preventing disorganized attachment: A meta-analysis Type de document : Texte imprimé et/ou numérique Auteurs : Christopher R. FACOMPRÉ, Auteur ; Kristin BERNARD, Auteur ; Theodore E. A. WATERS, Auteur Article en page(s) : p.1-11 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Disorganized attachment is associated with a host of negative developmental outcomes, leading to a growing interest in preventative interventions targeting the attachment relationship in infancy. The objective of this meta-analysis was to assess the effectiveness of interventions that aimed to prevent or reduce rates of disorganization among children at risk. We performed a literature search using PsycINFO, MEDLINE, and ProQuest databases for studies published between January 1989 and August 2016. All 16 studies (N = 1,360) included a control condition and reported postintervention rates of organized and disorganized attachments assessed by the Strange Situation Procedure. Results showed that, overall, interventions were effective in increasing rates of organized attachment compared to control conditions (d = 0.35, 95% CI [0.10–0.61]). Moderator analyses demonstrated that interventions were more effective (a) in more recently published studies than in older studies, (b) for maltreated samples than nonmaltreated samples, and (c) as children increased in age. These results have important implications for future development, tailoring, and implementation of attachment-based intervention programs. En ligne : https://doi.org/10.1017/S0954579417000426 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Effectiveness of interventions in preventing disorganized attachment: A meta-analysis [Texte imprimé et/ou numérique] / Christopher R. FACOMPRÉ, Auteur ; Kristin BERNARD, Auteur ; Theodore E. A. WATERS, Auteur . - p.1-11. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.1-11 Index. décimale : PER Périodiques Résumé : Disorganized attachment is associated with a host of negative developmental outcomes, leading to a growing interest in preventative interventions targeting the attachment relationship in infancy. The objective of this meta-analysis was to assess the effectiveness of interventions that aimed to prevent or reduce rates of disorganization among children at risk. We performed a literature search using PsycINFO, MEDLINE, and ProQuest databases for studies published between January 1989 and August 2016. All 16 studies (N = 1,360) included a control condition and reported postintervention rates of organized and disorganized attachments assessed by the Strange Situation Procedure. Results showed that, overall, interventions were effective in increasing rates of organized attachment compared to control conditions (d = 0.35, 95% CI [0.10–0.61]). Moderator analyses demonstrated that interventions were more effective (a) in more recently published studies than in older studies, (b) for maltreated samples than nonmaltreated samples, and (c) as children increased in age. These results have important implications for future development, tailoring, and implementation of attachment-based intervention programs. En ligne : https://doi.org/10.1017/S0954579417000426 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Chronic peer victimization heightens neural sensitivity to risk taking / Eva H. TELZER in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.13-26 Titre : Chronic peer victimization heightens neural sensitivity to risk taking Type de document : Texte imprimé et/ou numérique Auteurs : Eva H. TELZER, Auteur ; Michelle E. MIERNICKI, Auteur ; Karen D. RUDOLPH, Auteur Article en page(s) : p.13-26 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Although behavioral and experimental studies have shown links between victimization and antisocial behavior, the neural correlates explaining this link are relatively unknown. In the current study, we recruited adolescent girls from a longitudinal study that tracked youths’ reports of peer victimization experiences annually from the second through eighth grades. Based on these reports, 46 adolescents were recruited: 25 chronically victimized and 21 nonvictimized. During a functional magnetic resonance imaging scan, participants completed a risk-taking task. Chronic peer victimization was associated with greater risk-taking behavior during the task and higher levels of self-reported antisocial behavior in everyday life. At the neural level, chronically victimized girls showed greater activation in regions involved in affective sensitivity, social cognition, and cognitive control, which significantly mediated victimization group differences in self-reported antisocial behavior. En ligne : https://doi.org/10.1017/S0954579417000438 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Chronic peer victimization heightens neural sensitivity to risk taking [Texte imprimé et/ou numérique] / Eva H. TELZER, Auteur ; Michelle E. MIERNICKI, Auteur ; Karen D. RUDOLPH, Auteur . - p.13-26. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.13-26 Index. décimale : PER Périodiques Résumé : Although behavioral and experimental studies have shown links between victimization and antisocial behavior, the neural correlates explaining this link are relatively unknown. In the current study, we recruited adolescent girls from a longitudinal study that tracked youths’ reports of peer victimization experiences annually from the second through eighth grades. Based on these reports, 46 adolescents were recruited: 25 chronically victimized and 21 nonvictimized. During a functional magnetic resonance imaging scan, participants completed a risk-taking task. Chronic peer victimization was associated with greater risk-taking behavior during the task and higher levels of self-reported antisocial behavior in everyday life. At the neural level, chronically victimized girls showed greater activation in regions involved in affective sensitivity, social cognition, and cognitive control, which significantly mediated victimization group differences in self-reported antisocial behavior. En ligne : https://doi.org/10.1017/S0954579417000438 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Fathering moderates the effects of maternal depression on the family process / Adam VAKRAT in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.27-38 Titre : Fathering moderates the effects of maternal depression on the family process Type de document : Texte imprimé et/ou numérique Auteurs : Adam VAKRAT, Auteur ; Yael APTER-LEVY, Auteur ; Ruth FELDMAN, Auteur Article en page(s) : p.27-38 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Maternal depression negatively impacts children's development, yet few studies have focused on fathering and the family process in cases of maternal depression. A community cohort of married/cohabitating women was recruited on the second postbirth day (N = 1,983) and maternal depression repeatedly assessed across the first year and again at 6 years to form two cohorts: mothers chronically depressed from birth to 6 (N = 46) and nondepressed controls (N = 103). At 6 years, mother–child, father–child, and family interactions were observed. In families of depressed mothers, both mother and father exhibited lower sensitivity and higher intrusiveness, and children displayed lower social engagement during interactions with mother and father. Fathering moderated the effects of maternal depression on the family process. When fathers showed low sensitivity, high intrusiveness, and provided little opportunities for child social engagement, the family process was less cohesive, implying a decrease in the family's harmonious, warm, and collaborative style. However, in cases of high father sensitivity, low intrusiveness, and increased child engagement, the family process was unaffected by maternal depression. Findings describe both comparability and compensatory mechanisms in the effects of fathering on family life when maternal care is deficient, highlight the buffering role of fathers, and underscore the importance of father-focused interventions when mothers are depressed. En ligne : https://doi.org/10.1017/S095457941700044X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Fathering moderates the effects of maternal depression on the family process [Texte imprimé et/ou numérique] / Adam VAKRAT, Auteur ; Yael APTER-LEVY, Auteur ; Ruth FELDMAN, Auteur . - p.27-38. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.27-38 Index. décimale : PER Périodiques Résumé : Maternal depression negatively impacts children's development, yet few studies have focused on fathering and the family process in cases of maternal depression. A community cohort of married/cohabitating women was recruited on the second postbirth day (N = 1,983) and maternal depression repeatedly assessed across the first year and again at 6 years to form two cohorts: mothers chronically depressed from birth to 6 (N = 46) and nondepressed controls (N = 103). At 6 years, mother–child, father–child, and family interactions were observed. In families of depressed mothers, both mother and father exhibited lower sensitivity and higher intrusiveness, and children displayed lower social engagement during interactions with mother and father. Fathering moderated the effects of maternal depression on the family process. When fathers showed low sensitivity, high intrusiveness, and provided little opportunities for child social engagement, the family process was less cohesive, implying a decrease in the family's harmonious, warm, and collaborative style. However, in cases of high father sensitivity, low intrusiveness, and increased child engagement, the family process was unaffected by maternal depression. Findings describe both comparability and compensatory mechanisms in the effects of fathering on family life when maternal care is deficient, highlight the buffering role of fathers, and underscore the importance of father-focused interventions when mothers are depressed. En ligne : https://doi.org/10.1017/S095457941700044X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Potentially important periods of change in the development of social and role functioning in youth at clinical high risk for psychosis / Eva VELTHORST in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.39-47 Titre : Potentially important periods of change in the development of social and role functioning in youth at clinical high risk for psychosis Type de document : Texte imprimé et/ou numérique Auteurs : Eva VELTHORST, Auteur ; Jamie ZINBERG, Auteur ; Jean ADDINGTON, Auteur ; Kristin S. CADENHEAD, Auteur ; Tyrone D. CANNON, Auteur ; Ricardo E. CARRIÓN, Auteur ; Andrea M. AUTHER, Auteur ; Barbara A. CORNBLATT, Auteur ; Thomas H. MCGLASHAN, Auteur ; Daniel H. MATHALON, Auteur ; Diana O. PERKINS, Auteur ; Larry J. SEIDMAN, Auteur ; Ming T. TSUANG, Auteur ; Elaine F. WALKER, Auteur ; Scott W. WOODS, Auteur ; Abraham REICHENBERG, Auteur ; Carrie E. BEARDEN, Auteur Article en page(s) : p.39-47 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The developmental course of daily functioning prior to first psychosis-onset remains poorly understood. This study explored age-related periods of change in social and role functioning. The longitudinal study included youth (aged 12–23, mean follow-up years = 1.19) at clinical high risk (CHR) for psychosis (converters [CHR-C], n = 83; nonconverters [CHR-NC], n = 275) and a healthy control group (n = 164). Mixed-model analyses were performed to determine age-related differences in social and role functioning. We limited our analyses to functioning before psychosis conversion; thus, data of CHR-C participants gathered after psychosis onset were excluded. In controls, social and role functioning improved over time. From at least age 12, functioning in CHR was poorer than in controls, and this lag persisted over time. Between ages 15 and 18, social functioning in CHR-C stagnated and diverged from that of CHR-NC, who continued to improve (p = .001). Subsequently, CHR-C lagged behind in improvement between ages 21 and 23, further distinguishing them from CHR-NC (p < .001). A similar period of stagnation was apparent for role functioning, but to a lesser extent (p = .007). The results remained consistent when we accounted for the time to conversion. Our findings suggest that CHR-C start lagging behind CHR-NC in social and role functioning in adolescence, followed by a period of further stagnation in adulthood. En ligne : https://doi.org/10.1017/S0954579417000451 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Potentially important periods of change in the development of social and role functioning in youth at clinical high risk for psychosis [Texte imprimé et/ou numérique] / Eva VELTHORST, Auteur ; Jamie ZINBERG, Auteur ; Jean ADDINGTON, Auteur ; Kristin S. CADENHEAD, Auteur ; Tyrone D. CANNON, Auteur ; Ricardo E. CARRIÓN, Auteur ; Andrea M. AUTHER, Auteur ; Barbara A. CORNBLATT, Auteur ; Thomas H. MCGLASHAN, Auteur ; Daniel H. MATHALON, Auteur ; Diana O. PERKINS, Auteur ; Larry J. SEIDMAN, Auteur ; Ming T. TSUANG, Auteur ; Elaine F. WALKER, Auteur ; Scott W. WOODS, Auteur ; Abraham REICHENBERG, Auteur ; Carrie E. BEARDEN, Auteur . - p.39-47. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.39-47 Index. décimale : PER Périodiques Résumé : The developmental course of daily functioning prior to first psychosis-onset remains poorly understood. This study explored age-related periods of change in social and role functioning. The longitudinal study included youth (aged 12–23, mean follow-up years = 1.19) at clinical high risk (CHR) for psychosis (converters [CHR-C], n = 83; nonconverters [CHR-NC], n = 275) and a healthy control group (n = 164). Mixed-model analyses were performed to determine age-related differences in social and role functioning. We limited our analyses to functioning before psychosis conversion; thus, data of CHR-C participants gathered after psychosis onset were excluded. In controls, social and role functioning improved over time. From at least age 12, functioning in CHR was poorer than in controls, and this lag persisted over time. Between ages 15 and 18, social functioning in CHR-C stagnated and diverged from that of CHR-NC, who continued to improve (p = .001). Subsequently, CHR-C lagged behind in improvement between ages 21 and 23, further distinguishing them from CHR-NC (p < .001). A similar period of stagnation was apparent for role functioning, but to a lesser extent (p = .007). The results remained consistent when we accounted for the time to conversion. Our findings suggest that CHR-C start lagging behind CHR-NC in social and role functioning in adolescence, followed by a period of further stagnation in adulthood. En ligne : https://doi.org/10.1017/S0954579417000451 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Genetic and environmental influences on the codevelopment among borderline personality disorder traits, major depression symptoms, and substance use disorder symptoms from adolescence to young adulthood / Marina A. BORNOVALOVA in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.49-65 Titre : Genetic and environmental influences on the codevelopment among borderline personality disorder traits, major depression symptoms, and substance use disorder symptoms from adolescence to young adulthood Type de document : Texte imprimé et/ou numérique Auteurs : Marina A. BORNOVALOVA, Auteur ; Brad VERHULST, Auteur ; Troy WEBBER, Auteur ; Matt MCGUE, Auteur ; William G. IACONO, Auteur ; Brian M. HICKS, Auteur Article en page(s) : p.49-65 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Although borderline personality disorder (BPD) traits decline from adolescence to adulthood, comorbid psychopathology such as symptoms of major depressive disorder (MDD), alcohol use disorder (AUD), and drug use disorders (DUDs) likely disrupt this normative decline. Using a longitudinal sample of female twins (N = 1,763), we examined if levels of BPD traits were correlated with changes in MDD, AUD, and DUD symptoms from ages 14 to 24. A parallel process biometric latent growth model examined the contributions of genetic and environmental factors to the relationships between developmental components of these phenotypes. Higher BPD trait levels predicted a greater rate of increase in AUD and DUD symptoms, and higher AUD and DUD symptoms predicted a slower rate of decline of BPD traits from ages 14 to 24. Common genetic influences accounted for the associations between BPD traits and each disorder, as well as the interrelationships of AUD and DUD symptoms. Both genetic and nonshared environmental influences accounted for the correlated levels between BPD traits and MDD symptoms, but solely environmental influences accounted for the correlated changes between the two over time. Results indicate that higher levels of BPD traits may contribute to an earlier onset and faster escalation of AUD and DUD symptoms, and substance use problems slow the normative decline in BPD traits. Overall, our data suggests that primarily genetic influences contribute to the comorbidity between BPD features and substance use disorder symptoms. We discuss our data in the context of two major theories of developmental psychopathology and comorbidity. En ligne : https://doi.org/10.1017/S0954579417000463 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Genetic and environmental influences on the codevelopment among borderline personality disorder traits, major depression symptoms, and substance use disorder symptoms from adolescence to young adulthood [Texte imprimé et/ou numérique] / Marina A. BORNOVALOVA, Auteur ; Brad VERHULST, Auteur ; Troy WEBBER, Auteur ; Matt MCGUE, Auteur ; William G. IACONO, Auteur ; Brian M. HICKS, Auteur . - p.49-65. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.49-65 Index. décimale : PER Périodiques Résumé : Although borderline personality disorder (BPD) traits decline from adolescence to adulthood, comorbid psychopathology such as symptoms of major depressive disorder (MDD), alcohol use disorder (AUD), and drug use disorders (DUDs) likely disrupt this normative decline. Using a longitudinal sample of female twins (N = 1,763), we examined if levels of BPD traits were correlated with changes in MDD, AUD, and DUD symptoms from ages 14 to 24. A parallel process biometric latent growth model examined the contributions of genetic and environmental factors to the relationships between developmental components of these phenotypes. Higher BPD trait levels predicted a greater rate of increase in AUD and DUD symptoms, and higher AUD and DUD symptoms predicted a slower rate of decline of BPD traits from ages 14 to 24. Common genetic influences accounted for the associations between BPD traits and each disorder, as well as the interrelationships of AUD and DUD symptoms. Both genetic and nonshared environmental influences accounted for the correlated levels between BPD traits and MDD symptoms, but solely environmental influences accounted for the correlated changes between the two over time. Results indicate that higher levels of BPD traits may contribute to an earlier onset and faster escalation of AUD and DUD symptoms, and substance use problems slow the normative decline in BPD traits. Overall, our data suggests that primarily genetic influences contribute to the comorbidity between BPD features and substance use disorder symptoms. We discuss our data in the context of two major theories of developmental psychopathology and comorbidity. En ligne : https://doi.org/10.1017/S0954579417000463 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Are impairments in emotion recognition a core feature of callous–unemotional traits? Testing the primary versus secondary variants model in children / Mark R. DADDS in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.67-77 Titre : Are impairments in emotion recognition a core feature of callous–unemotional traits? Testing the primary versus secondary variants model in children Type de document : Texte imprimé et/ou numérique Auteurs : Mark R. DADDS, Auteur ; Eva R. KIMONIS, Auteur ; Olivia SCHOLLAR-ROOT, Auteur ; Caroline MOUL, Auteur ; David J. HAWES, Auteur Article en page(s) : p.67-77 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The role of environmental adversity in the development of high callous–unemotional (CU) traits in children is controversial. Evidence speaks to the traits being largely independent of adversity; however, recent data shows that those with high CU traits and high adversity and/or high anxiety might differ in important ways from those with no such history. We tested this using emotion recognition (ER) skills. We tested whether maltreatment history and anxiety levels moderated the relationship between level of CU traits and ER skills in N = 364 children with behavioral problems who were 3 to 16 years old. As hypothesised, in the full sample, the relationship between CU traits and ER differed according to maltreatment history, such that CU traits were associated with poorer recognition for those with zero or negligible history of maltreatment. This moderation of the CU-ER relationship by maltreatment was inconsistent across subgroups, however, and for the cohort utilizing youth self-report of maltreatment, high CU traits were associated with poor ER in those with lower anxiety levels. Maltreatment history and/or anxiety levels can identify different emotional impairments associated with high CU traits, and the impairments might be characteristic of “primary” high CU traits defined as occurring independently of maltreatment and/or high anxiety. En ligne : https://doi.org/10.1017/S0954579417000475 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Are impairments in emotion recognition a core feature of callous–unemotional traits? Testing the primary versus secondary variants model in children [Texte imprimé et/ou numérique] / Mark R. DADDS, Auteur ; Eva R. KIMONIS, Auteur ; Olivia SCHOLLAR-ROOT, Auteur ; Caroline MOUL, Auteur ; David J. HAWES, Auteur . - p.67-77. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.67-77 Index. décimale : PER Périodiques Résumé : The role of environmental adversity in the development of high callous–unemotional (CU) traits in children is controversial. Evidence speaks to the traits being largely independent of adversity; however, recent data shows that those with high CU traits and high adversity and/or high anxiety might differ in important ways from those with no such history. We tested this using emotion recognition (ER) skills. We tested whether maltreatment history and anxiety levels moderated the relationship between level of CU traits and ER skills in N = 364 children with behavioral problems who were 3 to 16 years old. As hypothesised, in the full sample, the relationship between CU traits and ER differed according to maltreatment history, such that CU traits were associated with poorer recognition for those with zero or negligible history of maltreatment. This moderation of the CU-ER relationship by maltreatment was inconsistent across subgroups, however, and for the cohort utilizing youth self-report of maltreatment, high CU traits were associated with poor ER in those with lower anxiety levels. Maltreatment history and/or anxiety levels can identify different emotional impairments associated with high CU traits, and the impairments might be characteristic of “primary” high CU traits defined as occurring independently of maltreatment and/or high anxiety. En ligne : https://doi.org/10.1017/S0954579417000475 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Early maturation and substance use across adolescence and young adulthood: A longitudinal study of Finnish twins / Jeanne E. SAVAGE in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.79-92 Titre : Early maturation and substance use across adolescence and young adulthood: A longitudinal study of Finnish twins Type de document : Texte imprimé et/ou numérique Auteurs : Jeanne E. SAVAGE, Auteur ; Richard J. ROSE, Auteur ; Lea PULKKINEN, Auteur ; Karri SILVENTOINEN, Auteur ; Tellervo KORHONEN, Auteur ; Jaakko KAPRIO, Auteur ; Nathan GILLESPIE, Auteur ; Danielle M. DICK, Auteur Article en page(s) : p.79-92 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Early maturation, indexed by pubertal development (PD), has been associated with earlier initiation and greater frequency of adolescent substance use, but this relationship may be biased by confounding factors and effects that change across development. Using a population-based Finnish twin sample (N = 3,632 individuals), we conducted twin modeling and multilevel structural equation modeling of the relationship between PD and substance use at ages 12–22. Shared environmental factors contributed to early PD and heavier substance use for females. Biological father absence was associated with early PD for boys but not girls, and did not account for the relationship between PD and substance use. The association between early PD and heavier substance use was partially due to between-family confounds, although early PD appeared to qualitatively alter long-term trajectories for some substances (nicotine), but not others (alcohol). Mediation by peer and parental factors did not explain this relationship within families. However, higher peer substance use and lower parental monitoring were themselves associated with heavier substance use, strengthening the existing evidence for these factors as targets for prevention/intervention efforts. Early maturation was not supported as a robust determinant of alcohol use trajectories in adolescence and young adulthood, but may require longer term follow-up. Subtle effects of early PD on nicotine and illicit drug use trajectories throughout adolescence and adulthood merit further investigation. En ligne : https://doi.org/10.1017/S0954579417000487 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Early maturation and substance use across adolescence and young adulthood: A longitudinal study of Finnish twins [Texte imprimé et/ou numérique] / Jeanne E. SAVAGE, Auteur ; Richard J. ROSE, Auteur ; Lea PULKKINEN, Auteur ; Karri SILVENTOINEN, Auteur ; Tellervo KORHONEN, Auteur ; Jaakko KAPRIO, Auteur ; Nathan GILLESPIE, Auteur ; Danielle M. DICK, Auteur . - p.79-92. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.79-92 Index. décimale : PER Périodiques Résumé : Early maturation, indexed by pubertal development (PD), has been associated with earlier initiation and greater frequency of adolescent substance use, but this relationship may be biased by confounding factors and effects that change across development. Using a population-based Finnish twin sample (N = 3,632 individuals), we conducted twin modeling and multilevel structural equation modeling of the relationship between PD and substance use at ages 12–22. Shared environmental factors contributed to early PD and heavier substance use for females. Biological father absence was associated with early PD for boys but not girls, and did not account for the relationship between PD and substance use. The association between early PD and heavier substance use was partially due to between-family confounds, although early PD appeared to qualitatively alter long-term trajectories for some substances (nicotine), but not others (alcohol). Mediation by peer and parental factors did not explain this relationship within families. However, higher peer substance use and lower parental monitoring were themselves associated with heavier substance use, strengthening the existing evidence for these factors as targets for prevention/intervention efforts. Early maturation was not supported as a robust determinant of alcohol use trajectories in adolescence and young adulthood, but may require longer term follow-up. Subtle effects of early PD on nicotine and illicit drug use trajectories throughout adolescence and adulthood merit further investigation. En ligne : https://doi.org/10.1017/S0954579417000487 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Does the Incredible Years reduce child externalizing problems through improved parenting? The role of child negative affectivity and serotonin transporter linked polymorphic region (5-HTTLPR) genotype / Joyce WEELAND in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.93-112 Titre : Does the Incredible Years reduce child externalizing problems through improved parenting? The role of child negative affectivity and serotonin transporter linked polymorphic region (5-HTTLPR) genotype Type de document : Texte imprimé et/ou numérique Auteurs : Joyce WEELAND, Auteur ; Rabia R. CHHANGUR, Auteur ; Sara R. JAFFEE, Auteur ; Danielle VAN DER GIESSEN, Auteur ; Walter MATTHYS, Auteur ; Bram OROBIO DE CASTRO, Auteur ; Geertjan OVERBEEK, Auteur Article en page(s) : p.93-112 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : In a randomized controlled trial, the Observational Randomized Controlled Trial of Childhood Differential Susceptibility (ORCHIDS study), we tested whether observed parental affect and observed and reported parenting behavior are mechanisms of change underlying the effects of the behavioral parent training program the Incredible Years (IY). Furthermore, we tested whether some children are more susceptible to these change mechanisms because of their temperamental negative affectivity and/or serotonin transporter linked polymorphic region (5-HTTLPR) genotype. Participants were 387 Dutch children between 4 and 8 years of age (M age = 6.31, SD = 1.33; 55.3% boys) and their parents. Results showed that although IY was successful in improving parenting behavior and increasing parental positive affect, these effects did not explain the significant decreases in child externalizing problems. We therefore found no evidence for changes in parenting behavior or parental affect being the putative mechanisms of IY effectiveness. Furthermore, intervention effects on child externalizing behavior were not moderated by child negative affectivity or 5-HTTLPR genotype. However, child 5-HTTLPR genotype did moderate intervention effects on negative parenting behavior. This suggests that in research on behavioral parent training programs, “what works for which parents” might also be an important question. En ligne : https://doi.org/10.1017/S0954579417000499 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Does the Incredible Years reduce child externalizing problems through improved parenting? The role of child negative affectivity and serotonin transporter linked polymorphic region (5-HTTLPR) genotype [Texte imprimé et/ou numérique] / Joyce WEELAND, Auteur ; Rabia R. CHHANGUR, Auteur ; Sara R. JAFFEE, Auteur ; Danielle VAN DER GIESSEN, Auteur ; Walter MATTHYS, Auteur ; Bram OROBIO DE CASTRO, Auteur ; Geertjan OVERBEEK, Auteur . - p.93-112. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.93-112 Index. décimale : PER Périodiques Résumé : In a randomized controlled trial, the Observational Randomized Controlled Trial of Childhood Differential Susceptibility (ORCHIDS study), we tested whether observed parental affect and observed and reported parenting behavior are mechanisms of change underlying the effects of the behavioral parent training program the Incredible Years (IY). Furthermore, we tested whether some children are more susceptible to these change mechanisms because of their temperamental negative affectivity and/or serotonin transporter linked polymorphic region (5-HTTLPR) genotype. Participants were 387 Dutch children between 4 and 8 years of age (M age = 6.31, SD = 1.33; 55.3% boys) and their parents. Results showed that although IY was successful in improving parenting behavior and increasing parental positive affect, these effects did not explain the significant decreases in child externalizing problems. We therefore found no evidence for changes in parenting behavior or parental affect being the putative mechanisms of IY effectiveness. Furthermore, intervention effects on child externalizing behavior were not moderated by child negative affectivity or 5-HTTLPR genotype. However, child 5-HTTLPR genotype did moderate intervention effects on negative parenting behavior. This suggests that in research on behavioral parent training programs, “what works for which parents” might also be an important question. En ligne : https://doi.org/10.1017/S0954579417000499 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Early developmental influences on self-esteem trajectories from adolescence through adulthood: Impact of birth weight and motor skills / Kristie L. POOLE in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.113-123 Titre : Early developmental influences on self-esteem trajectories from adolescence through adulthood: Impact of birth weight and motor skills Type de document : Texte imprimé et/ou numérique Auteurs : Kristie L. POOLE, Auteur ; Louis A. SCHMIDT, Auteur ; Mark A. FERRO, Auteur ; Cheryl MISSIUNA, Auteur ; Saroj SAIGAL, Auteur ; Michael H. BOYLE, Auteur ; Ryan J. VAN LIESHOUT, Auteur Article en page(s) : p.113-123 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : While the trajectory of self-esteem from adolescence to adulthood varies from person to person, little research has examined how differences in early developmental processes might affect these pathways. This study examined how early motor skill development interacted with preterm birth status to predict self-esteem from adolescence through the early 30s. We addressed this using the oldest known, prospectively followed cohort of extremely low birth weight (<1000 g) survivors (N = 179) and normal birth weight controls (N = 145) in the world, born between 1977 and 1982. Motor skills were measured using a performance-based assessment at age 8 and a retrospective self-report, and self-esteem was reported during three follow-up periods (age 12–16, age 22–26, and age 29–36). We found that birth weight status moderated the association between early motor skills and self-esteem. Stable over three decades, the self-esteem of normal birth weight participants was sensitive to early motor skills such that those with poorer motor functioning manifested lower self-esteem, while those with better motor skills manifested higher self-esteem. Conversely, differences in motor skill development did not affect the self-esteem from adolescence to adulthood in individuals born at extremely low birth weight. Early motor skill development may exert differential effects on self-esteem, depending on whether one is born at term or prematurely. En ligne : https://doi.org/10.1017/S0954579417000505 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Early developmental influences on self-esteem trajectories from adolescence through adulthood: Impact of birth weight and motor skills [Texte imprimé et/ou numérique] / Kristie L. POOLE, Auteur ; Louis A. SCHMIDT, Auteur ; Mark A. FERRO, Auteur ; Cheryl MISSIUNA, Auteur ; Saroj SAIGAL, Auteur ; Michael H. BOYLE, Auteur ; Ryan J. VAN LIESHOUT, Auteur . - p.113-123. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.113-123 Index. décimale : PER Périodiques Résumé : While the trajectory of self-esteem from adolescence to adulthood varies from person to person, little research has examined how differences in early developmental processes might affect these pathways. This study examined how early motor skill development interacted with preterm birth status to predict self-esteem from adolescence through the early 30s. We addressed this using the oldest known, prospectively followed cohort of extremely low birth weight (<1000 g) survivors (N = 179) and normal birth weight controls (N = 145) in the world, born between 1977 and 1982. Motor skills were measured using a performance-based assessment at age 8 and a retrospective self-report, and self-esteem was reported during three follow-up periods (age 12–16, age 22–26, and age 29–36). We found that birth weight status moderated the association between early motor skills and self-esteem. Stable over three decades, the self-esteem of normal birth weight participants was sensitive to early motor skills such that those with poorer motor functioning manifested lower self-esteem, while those with better motor skills manifested higher self-esteem. Conversely, differences in motor skill development did not affect the self-esteem from adolescence to adulthood in individuals born at extremely low birth weight. Early motor skill development may exert differential effects on self-esteem, depending on whether one is born at term or prematurely. En ligne : https://doi.org/10.1017/S0954579417000505 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

A genetic variant brain-derived neurotrophic factor (BDNF) polymorphism interacts with hostile parenting to predict error-related brain activity and thereby risk for internalizing disorders in children / Alexandria MEYER in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.125-141 Titre : A genetic variant brain-derived neurotrophic factor (BDNF) polymorphism interacts with hostile parenting to predict error-related brain activity and thereby risk for internalizing disorders in children Type de document : Texte imprimé et/ou numérique Auteurs : Alexandria MEYER, Auteur ; Greg HAJCAK, Auteur ; Elizabeth HAYDEN, Auteur ; Haroon I. SHEIKH, Auteur ; Shiva M. SINGH, Auteur ; Daniel N. KLEIN, Auteur Article en page(s) : p.125-141 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The error-related negativity (ERN) is a negative deflection in the event-related potential occurring when individuals make mistakes, and is increased in children with internalizing psychopathology. We recently found that harsh parenting predicts a larger ERN in children, and recent work has suggested that variation in the brain-derived neurotrophic factor (BDNF) gene may moderate the impact of early life adversity. Parents and children completed measures of parenting when children were 3 years old (N = 201); 3 years later, the ERN was measured and diagnostic interviews as well as dimensional symptom measures were completed. We found that harsh parenting predicted an increased ERN only among children with a methionine allele of the BDNF genotype, and evidence of moderated mediation: the ERN mediated the relationship between parenting and internalizing diagnoses and dimensional symptoms only if children had a methionine allele. We tested this model with externalizing disorders, and found that harsh parenting predicted externalizing outcomes, but the ERN did not mediate this association. These findings suggest that harsh parenting predicts both externalizing and internalizing outcomes in children; however, this occurs through different pathways that uniquely implicate error-related brain activity in the development of internalizing disorders. En ligne : https://doi.org/10.1017/S0954579417000517 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] A genetic variant brain-derived neurotrophic factor (BDNF) polymorphism interacts with hostile parenting to predict error-related brain activity and thereby risk for internalizing disorders in children [Texte imprimé et/ou numérique] / Alexandria MEYER, Auteur ; Greg HAJCAK, Auteur ; Elizabeth HAYDEN, Auteur ; Haroon I. SHEIKH, Auteur ; Shiva M. SINGH, Auteur ; Daniel N. KLEIN, Auteur . - p.125-141. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.125-141 Index. décimale : PER Périodiques Résumé : The error-related negativity (ERN) is a negative deflection in the event-related potential occurring when individuals make mistakes, and is increased in children with internalizing psychopathology. We recently found that harsh parenting predicts a larger ERN in children, and recent work has suggested that variation in the brain-derived neurotrophic factor (BDNF) gene may moderate the impact of early life adversity. Parents and children completed measures of parenting when children were 3 years old (N = 201); 3 years later, the ERN was measured and diagnostic interviews as well as dimensional symptom measures were completed. We found that harsh parenting predicted an increased ERN only among children with a methionine allele of the BDNF genotype, and evidence of moderated mediation: the ERN mediated the relationship between parenting and internalizing diagnoses and dimensional symptoms only if children had a methionine allele. We tested this model with externalizing disorders, and found that harsh parenting predicted externalizing outcomes, but the ERN did not mediate this association. These findings suggest that harsh parenting predicts both externalizing and internalizing outcomes in children; however, this occurs through different pathways that uniquely implicate error-related brain activity in the development of internalizing disorders. En ligne : https://doi.org/10.1017/S0954579417000517 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Association of cognitive function and liability to addiction with childhood herpesvirus infections: A prospective cohort study / Michael M. VANYUKOV in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.143-152 Titre : Association of cognitive function and liability to addiction with childhood herpesvirus infections: A prospective cohort study Type de document : Texte imprimé et/ou numérique Auteurs : Michael M. VANYUKOV, Auteur ; Vishwajit L. NIMGAONKAR, Auteur ; Levent KIRISCI, Auteur ; Galina P. KIRILLOVA, Auteur ; Maureen D. REYNOLDS, Auteur ; Konasale PRASAD, Auteur ; Ralph E. TARTER, Auteur ; Robert H. YOLKEN, Auteur Article en page(s) : p.143-152 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Liability to substance use disorder (SUD) is largely nonspecific to particular drugs and is related to behavior dysregulation, including reduced cognitive control. Recent data suggest that cognitive mechanisms may be influenced by exposure to neurotropic infections, such as human herpesviruses. In this study, serological evidence of exposure to human herpesvirus Herpes simplex virus Type 1 (HSV-1), cytomegalovirus (CMV), and Epstein–Barr virus (EBV) as well as Toxoplasma gondii was determined in childhood (age ~11 years) in 395 sons and 174 daughters of fathers with or without SUD. Its relationships with a cognitive characteristic (IQ) in childhood and with risk for SUD in adulthood were examined using correlation, regression, survival, and path analyses. Exposure to HSV-1, EBV, and T. gondii in males and females, and CMV in males, was associated with lower IQ. Independent of that relationship, EBV in females and possibly in males, and CMV and possibly HSV-1 in females were associated with elevated risk for SUD. Therefore, childhood neurotropic infections may influence cognitive development and risk for behavior disorders such as SUD. The results may point to new avenues for alleviating cognitive impairment and SUD risk. En ligne : https://doi.org/10.1017/S0954579417000529 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Association of cognitive function and liability to addiction with childhood herpesvirus infections: A prospective cohort study [Texte imprimé et/ou numérique] / Michael M. VANYUKOV, Auteur ; Vishwajit L. NIMGAONKAR, Auteur ; Levent KIRISCI, Auteur ; Galina P. KIRILLOVA, Auteur ; Maureen D. REYNOLDS, Auteur ; Konasale PRASAD, Auteur ; Ralph E. TARTER, Auteur ; Robert H. YOLKEN, Auteur . - p.143-152. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.143-152 Index. décimale : PER Périodiques Résumé : Liability to substance use disorder (SUD) is largely nonspecific to particular drugs and is related to behavior dysregulation, including reduced cognitive control. Recent data suggest that cognitive mechanisms may be influenced by exposure to neurotropic infections, such as human herpesviruses. In this study, serological evidence of exposure to human herpesvirus Herpes simplex virus Type 1 (HSV-1), cytomegalovirus (CMV), and Epstein–Barr virus (EBV) as well as Toxoplasma gondii was determined in childhood (age ~11 years) in 395 sons and 174 daughters of fathers with or without SUD. Its relationships with a cognitive characteristic (IQ) in childhood and with risk for SUD in adulthood were examined using correlation, regression, survival, and path analyses. Exposure to HSV-1, EBV, and T. gondii in males and females, and CMV in males, was associated with lower IQ. Independent of that relationship, EBV in females and possibly in males, and CMV and possibly HSV-1 in females were associated with elevated risk for SUD. Therefore, childhood neurotropic infections may influence cognitive development and risk for behavior disorders such as SUD. The results may point to new avenues for alleviating cognitive impairment and SUD risk. En ligne : https://doi.org/10.1017/S0954579417000529 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Interpersonal harm aversion as a necessary foundation for morality: A developmental neuroscience perspective / Jean DECETY in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.153-164 Titre : Interpersonal harm aversion as a necessary foundation for morality: A developmental neuroscience perspective Type de document : Texte imprimé et/ou numérique Auteurs : Jean DECETY, Auteur ; Jason M. COWELL, Auteur Article en page(s) : p.153-164 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Growing evidence from developmental psychology and social neuroscience emphasizes the importance of third-party harm aversion for constructing morality. A sensitivity to interpersonal harm emerges very early in ontogeny, as reflected in both the capacity for implicit social evaluation and an aversion for antisocial agents. Yet it does not necessarily entail avoidance toward inflicting pain to others. Later, an understanding that harmful actions cause suffering emerges, followed by an integration of rules that can depend on social contexts and cultures. These developmental findings build on a burgeoning literature, which suggests that the fundamental nature of moral and social cognition, including their motivational and hedonic value, lies in general computational processes such as attention, approach–avoidance, social valuation, and decision making rather than in fully distinct, dedicated neural regions for morality. Bridging the gap between cognition and behaviors and the requisite affective, motivational, and cognitive mechanisms, a developmental neuroscience approach enriches our understanding of the emergence of morality. En ligne : https://doi.org/10.1017/S0954579417000530 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Interpersonal harm aversion as a necessary foundation for morality: A developmental neuroscience perspective [Texte imprimé et/ou numérique] / Jean DECETY, Auteur ; Jason M. COWELL, Auteur . - p.153-164. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.153-164 Index. décimale : PER Périodiques Résumé : Growing evidence from developmental psychology and social neuroscience emphasizes the importance of third-party harm aversion for constructing morality. A sensitivity to interpersonal harm emerges very early in ontogeny, as reflected in both the capacity for implicit social evaluation and an aversion for antisocial agents. Yet it does not necessarily entail avoidance toward inflicting pain to others. Later, an understanding that harmful actions cause suffering emerges, followed by an integration of rules that can depend on social contexts and cultures. These developmental findings build on a burgeoning literature, which suggests that the fundamental nature of moral and social cognition, including their motivational and hedonic value, lies in general computational processes such as attention, approach–avoidance, social valuation, and decision making rather than in fully distinct, dedicated neural regions for morality. Bridging the gap between cognition and behaviors and the requisite affective, motivational, and cognitive mechanisms, a developmental neuroscience approach enriches our understanding of the emergence of morality. En ligne : https://doi.org/10.1017/S0954579417000530 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Genetic moderation of multiple pathways linking early cumulative socioeconomic adversity and young adults' cardiometabolic disease risk / Kandauda A. S. WICKRAMA in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.165-177 Titre : Genetic moderation of multiple pathways linking early cumulative socioeconomic adversity and young adults' cardiometabolic disease risk Type de document : Texte imprimé et/ou numérique Auteurs : Kandauda A. S. WICKRAMA, Auteur ; Tae Kyoung LEE, Auteur ; Catherine Walker O'NEAL, Auteur Article en page(s) : p.165-177 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Recent research suggests that psychosocial resources and life stressors are mediating pathways explaining socioeconomic variation in young adults' health risks. However, less research has examined both these pathways simultaneously and their genetic moderation. A nationally representative sample of 11,030 respondents with prospective data collected over 13 years from the National Study of Adolescent to Adult Health was examined. First, the association between early cumulative socioeconomic adversity and young adults' (ages 25–34) cardiometabolic disease risk, as measured by 10 biomarkers, through psychosocial resources (educational attainment) and life stressors (accelerated transition to adulthood) was examined. Second, moderation of these pathways by the serotonin transporter linked polymorphic region gene (5-HTTLPR) was examined. There was evidence for the association between early socioeconomic adversity and young adults' cardiometabolic disease risk directly and indirectly through educational attainment and accelerated transitions. These direct and mediating pathways were amplified by the 5-HTTLPR polymorphism. These findings elucidate how early adversity can have an enduring influence on young adults' cardiometabolic disease risk directly and indirectly through psychosocial resources and life stressors and their genetic moderation. This information suggests that effective intervention and prevention programs should focus on early adversity, youth educational attainment, and their transition to young adulthood. En ligne : https://doi.org/10.1017/S0954579417000542 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335 [article] Genetic moderation of multiple pathways linking early cumulative socioeconomic adversity and young adults' cardiometabolic disease risk [Texte imprimé et/ou numérique] / Kandauda A. S. WICKRAMA, Auteur ; Tae Kyoung LEE, Auteur ; Catherine Walker O'NEAL, Auteur . - p.165-177. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.165-177 Index. décimale : PER Périodiques Résumé : Recent research suggests that psychosocial resources and life stressors are mediating pathways explaining socioeconomic variation in young adults' health risks. However, less research has examined both these pathways simultaneously and their genetic moderation. A nationally representative sample of 11,030 respondents with prospective data collected over 13 years from the National Study of Adolescent to Adult Health was examined. First, the association between early cumulative socioeconomic adversity and young adults' (ages 25–34) cardiometabolic disease risk, as measured by 10 biomarkers, through psychosocial resources (educational attainment) and life stressors (accelerated transition to adulthood) was examined. Second, moderation of these pathways by the serotonin transporter linked polymorphic region gene (5-HTTLPR) was examined. There was evidence for the association between early socioeconomic adversity and young adults' cardiometabolic disease risk directly and indirectly through educational attainment and accelerated transitions. These direct and mediating pathways were amplified by the 5-HTTLPR polymorphism. These findings elucidate how early adversity can have an enduring influence on young adults' cardiometabolic disease risk directly and indirectly through psychosocial resources and life stressors and their genetic moderation. This information suggests that effective intervention and prevention programs should focus on early adversity, youth educational attainment, and their transition to young adulthood. En ligne : https://doi.org/10.1017/S0954579417000542 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=335

Differences in neural response to extinction recall in young adults with or without history of behavioral inhibition / Tomer SHECHNER in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.179-189 Titre : Differences in neural response to extinction recall in young adults with or without history of behavioral inhibition Type de document : Texte imprimé et/ou numérique Auteurs : Tomer SHECHNER, Auteur ; Nathan A. FOX, Auteur ; Jamie A. MASH, Auteur ; Johanna M. JARCHO, Auteur ; Gang CHEN, Auteur ; Ellen LEIBENLUFT, Auteur ; Daniel S. PINE, Auteur ; Jennifer C. BRITTON, Auteur Article en page(s) : p.179-189 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Behavioral inhibition (BI) is a temperament identified in early childhood that is associated with risk for anxiety disorders, yet only about half of behaviorally inhibited children manifest anxiety later in life. We compared brain function and behavior during extinction recall in a sample of nonanxious young adults characterized in childhood with BI (n = 22) or with no BI (n = 28). Three weeks after undergoing fear conditioning and extinction, participants completed a functional magnetic resonance imaging extinction recall task assessing memory and threat differentiation for conditioned stimuli. While self-report and psychophysiological measures of differential conditioning and extinction were similar across groups, BI-related differences in brain function emerged during extinction recall. Childhood BI was associated with greater activation in subgenual anterior cingulate cortex in response to cues signaling safety. This pattern of results may reflect neural correlates that promote resilience against anxiety in a temperamentally at-risk population. En ligne : https://doi.org/10.1017/S0954579417000554 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Differences in neural response to extinction recall in young adults with or without history of behavioral inhibition [Texte imprimé et/ou numérique] / Tomer SHECHNER, Auteur ; Nathan A. FOX, Auteur ; Jamie A. MASH, Auteur ; Johanna M. JARCHO, Auteur ; Gang CHEN, Auteur ; Ellen LEIBENLUFT, Auteur ; Daniel S. PINE, Auteur ; Jennifer C. BRITTON, Auteur . - p.179-189. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.179-189 Index. décimale : PER Périodiques Résumé : Behavioral inhibition (BI) is a temperament identified in early childhood that is associated with risk for anxiety disorders, yet only about half of behaviorally inhibited children manifest anxiety later in life. We compared brain function and behavior during extinction recall in a sample of nonanxious young adults characterized in childhood with BI (n = 22) or with no BI (n = 28). Three weeks after undergoing fear conditioning and extinction, participants completed a functional magnetic resonance imaging extinction recall task assessing memory and threat differentiation for conditioned stimuli. While self-report and psychophysiological measures of differential conditioning and extinction were similar across groups, BI-related differences in brain function emerged during extinction recall. Childhood BI was associated with greater activation in subgenual anterior cingulate cortex in response to cues signaling safety. This pattern of results may reflect neural correlates that promote resilience against anxiety in a temperamentally at-risk population. En ligne : https://doi.org/10.1017/S0954579417000554 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Externalizing behavior severity in youths with callous–unemotional traits corresponds to patterns of amygdala activity and connectivity during judgments of causing fear / Elise M. CARDINALE in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.191-201 Titre : Externalizing behavior severity in youths with callous–unemotional traits corresponds to patterns of amygdala activity and connectivity during judgments of causing fear Type de document : Texte imprimé et/ou numérique Auteurs : Elise M. CARDINALE, Auteur ; Andrew L. BREEDEN, Auteur ; Emily L. ROBERTSON, Auteur ; Leah M. LOZIER, Auteur ; John W. VANMETER, Auteur ; Abigail A. MARSH, Auteur Article en page(s) : p.191-201 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Callous–unemotional (CU) traits characterize a subgroup of youths with conduct problems who exhibit low empathy, fearlessness, and elevated externalizing behaviors. The current study examines the role of aberrant amygdala activity and functional connectivity during a socioemotional judgment task in youths with CU traits, and links these deficits to externalizing behaviors. Functional magnetic resonance imaging was used to compare neural responses in 18 healthy youths and 30 youths with conduct problems and varying levels of CU traits as they evaluated the acceptability of causing another person to experience each of several emotions, including fear. Neuroimaging analyses examined blood oxygenation level dependent responses and task-dependent functional connectivity. High-CU youths exhibited left amygdala hypoactivation relative to healthy controls and low-CU youths primarily during evaluations of causing others fear. CU traits moderated the relationship between externalizing behavior and both amygdala activity and patterns of functional connectivity. The present data suggest that CU youths' aberrant amygdala activity and connectivity affect how they make judgments about the acceptability of causing others emotional distress, and that these aberrations represent risk factors for externalizing behaviors like rule breaking and aggression. These findings suggest that reducing externalizing behaviors in high-CU youths may require interventions that influence affective sensitivity. En ligne : https://doi.org/10.1017/S0954579417000566 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Externalizing behavior severity in youths with callous–unemotional traits corresponds to patterns of amygdala activity and connectivity during judgments of causing fear [Texte imprimé et/ou numérique] / Elise M. CARDINALE, Auteur ; Andrew L. BREEDEN, Auteur ; Emily L. ROBERTSON, Auteur ; Leah M. LOZIER, Auteur ; John W. VANMETER, Auteur ; Abigail A. MARSH, Auteur . - p.191-201. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.191-201 Index. décimale : PER Périodiques Résumé : Callous–unemotional (CU) traits characterize a subgroup of youths with conduct problems who exhibit low empathy, fearlessness, and elevated externalizing behaviors. The current study examines the role of aberrant amygdala activity and functional connectivity during a socioemotional judgment task in youths with CU traits, and links these deficits to externalizing behaviors. Functional magnetic resonance imaging was used to compare neural responses in 18 healthy youths and 30 youths with conduct problems and varying levels of CU traits as they evaluated the acceptability of causing another person to experience each of several emotions, including fear. Neuroimaging analyses examined blood oxygenation level dependent responses and task-dependent functional connectivity. High-CU youths exhibited left amygdala hypoactivation relative to healthy controls and low-CU youths primarily during evaluations of causing others fear. CU traits moderated the relationship between externalizing behavior and both amygdala activity and patterns of functional connectivity. The present data suggest that CU youths' aberrant amygdala activity and connectivity affect how they make judgments about the acceptability of causing others emotional distress, and that these aberrations represent risk factors for externalizing behaviors like rule breaking and aggression. These findings suggest that reducing externalizing behaviors in high-CU youths may require interventions that influence affective sensitivity. En ligne : https://doi.org/10.1017/S0954579417000566 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Birth weight interacts with a functional variant of the oxytocin receptor gene (OXTR) to predict executive functioning in children / Mark WADE in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.203-211 Titre : Birth weight interacts with a functional variant of the oxytocin receptor gene (OXTR) to predict executive functioning in children Type de document : Texte imprimé et/ou numérique Auteurs : Mark WADE, Auteur ; Heather PRIME, Auteur ; Thomas J. HOFFMANN, Auteur ; Louis A. SCHMIDT, Auteur ; Thomas G. O'CONNOR, Auteur ; Jennifer M. JENKINS, Auteur Article en page(s) : p.203-211 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Genetic variation in the oxytocin receptor gene (OXTR) is associated with several psychiatric conditions characterized by deficits in executive functioning (EF). A specific OXTR variant, rs2254298, has previously been associated with brain functioning in regions implicated in EF. Moreover, birth weight variation across the entire range is associated with individual differences in cortical structure and function that underlie EF. This is the first study to examine the main and interactive effect between rs2254298 and birth weight on EF in children. The sample consisted of 310 children from an ongoing longitudinal study. EF was measured at age 4.5 using observational tasks indexing working memory, cognitive flexibility, and inhibitory control. A family-based design that controlled for population admixture, stratification, and nongenomic confounds was employed. A significant genetic association between rs2254298 and EF was observed, with more copies of the major allele (G) associated with higher EF. There was also a significant interaction between rs2254298 and birth weight, such that more copies of the major allele in combination with higher birth weight predicted better EF. Findings suggest that OXTR may be associated with discrete neurocognitive abilities in childhood, and these effects may be modulated by intrauterine conditions related to fetal growth and development. En ligne : https://doi.org/10.1017/S0954579417000578 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Birth weight interacts with a functional variant of the oxytocin receptor gene (OXTR) to predict executive functioning in children [Texte imprimé et/ou numérique] / Mark WADE, Auteur ; Heather PRIME, Auteur ; Thomas J. HOFFMANN, Auteur ; Louis A. SCHMIDT, Auteur ; Thomas G. O'CONNOR, Auteur ; Jennifer M. JENKINS, Auteur . - p.203-211. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.203-211 Index. décimale : PER Périodiques Résumé : Genetic variation in the oxytocin receptor gene (OXTR) is associated with several psychiatric conditions characterized by deficits in executive functioning (EF). A specific OXTR variant, rs2254298, has previously been associated with brain functioning in regions implicated in EF. Moreover, birth weight variation across the entire range is associated with individual differences in cortical structure and function that underlie EF. This is the first study to examine the main and interactive effect between rs2254298 and birth weight on EF in children. The sample consisted of 310 children from an ongoing longitudinal study. EF was measured at age 4.5 using observational tasks indexing working memory, cognitive flexibility, and inhibitory control. A family-based design that controlled for population admixture, stratification, and nongenomic confounds was employed. A significant genetic association between rs2254298 and EF was observed, with more copies of the major allele (G) associated with higher EF. There was also a significant interaction between rs2254298 and birth weight, such that more copies of the major allele in combination with higher birth weight predicted better EF. Findings suggest that OXTR may be associated with discrete neurocognitive abilities in childhood, and these effects may be modulated by intrauterine conditions related to fetal growth and development. En ligne : https://doi.org/10.1017/S0954579417000578 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Serotonin functioning and adolescents' alcohol use: A genetically informed study examining mechanisms of risk / Frances L. WANG in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.213-233 Titre : Serotonin functioning and adolescents' alcohol use: A genetically informed study examining mechanisms of risk Type de document : Texte imprimé et/ou numérique Auteurs : Frances L. WANG, Auteur ; Laurie A. CHASSIN, Auteur ; John E. BATES, Auteur ; Danielle DICK, Auteur ; Jennifer E. LANSFORD, Auteur ; Gregory S. PETTIT, Auteur ; Kenneth A. DODGE, Auteur Article en page(s) : p.213-233 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The current study used data from two longitudinal samples to test whether self-regulation, depressive symptoms, and aggression/antisociality were mediators in the relation between a polygenic score indexing serotonin (5-HT) functioning and alcohol use in adolescence. The results from an independent genome-wide association study of 5-hydroxyindoleacetic acid in the cerebrospinal fluid were used to create 5-HT polygenic risk scores. Adolescents and/or parents reported on adolescents’ self-regulation (Time 1), depressive symptoms (Time 2), aggression/antisociality (Time 2), and alcohol use (Time 3). The results showed that 5-HT polygenic risk did not predict self-regulation. However, adolescents with higher levels of 5-HT polygenic risk showed greater depression and aggression/antisociality. Adolescents’ aggression/antisociality mediated the relation between 5-HT polygenic risk and later alcohol use. Deficits in self-regulation also predicted depression and aggression/antisociality, and indirectly predicted alcohol use through aggression/antisociality. Pathways to alcohol use were especially salient for males from families with low parental education in one of the two samples. The results provide insights into the longitudinal mechanisms underlying the relation between 5-HT functioning and alcohol use (i.e., earlier aggression/antisociality). There was no evidence that genetically based variation in 5-HT functioning predisposed individuals to deficits in self-regulation. Genetically based variation in 5-HT functioning and self-regulation might be separate, transdiagnostic risk factors for several types of psychopathology. En ligne : https://doi.org/10.1017/S095457941700058X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Serotonin functioning and adolescents' alcohol use: A genetically informed study examining mechanisms of risk [Texte imprimé et/ou numérique] / Frances L. WANG, Auteur ; Laurie A. CHASSIN, Auteur ; John E. BATES, Auteur ; Danielle DICK, Auteur ; Jennifer E. LANSFORD, Auteur ; Gregory S. PETTIT, Auteur ; Kenneth A. DODGE, Auteur . - p.213-233. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.213-233 Index. décimale : PER Périodiques Résumé : The current study used data from two longitudinal samples to test whether self-regulation, depressive symptoms, and aggression/antisociality were mediators in the relation between a polygenic score indexing serotonin (5-HT) functioning and alcohol use in adolescence. The results from an independent genome-wide association study of 5-hydroxyindoleacetic acid in the cerebrospinal fluid were used to create 5-HT polygenic risk scores. Adolescents and/or parents reported on adolescents’ self-regulation (Time 1), depressive symptoms (Time 2), aggression/antisociality (Time 2), and alcohol use (Time 3). The results showed that 5-HT polygenic risk did not predict self-regulation. However, adolescents with higher levels of 5-HT polygenic risk showed greater depression and aggression/antisociality. Adolescents’ aggression/antisociality mediated the relation between 5-HT polygenic risk and later alcohol use. Deficits in self-regulation also predicted depression and aggression/antisociality, and indirectly predicted alcohol use through aggression/antisociality. Pathways to alcohol use were especially salient for males from families with low parental education in one of the two samples. The results provide insights into the longitudinal mechanisms underlying the relation between 5-HT functioning and alcohol use (i.e., earlier aggression/antisociality). There was no evidence that genetically based variation in 5-HT functioning predisposed individuals to deficits in self-regulation. Genetically based variation in 5-HT functioning and self-regulation might be separate, transdiagnostic risk factors for several types of psychopathology. En ligne : https://doi.org/10.1017/S095457941700058X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Family environments and leukocyte transcriptome indicators of a proinflammatory phenotype in children and parents / Theodore F. ROBLES in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.235-253 Titre : Family environments and leukocyte transcriptome indicators of a proinflammatory phenotype in children and parents Type de document : Texte imprimé et/ou numérique Auteurs : Theodore F. ROBLES, Auteur ; Rena L. REPETTI, Auteur ; Bridget M. REYNOLDS, Auteur ; Paul J. CHUNG, Auteur ; Jesusa M. G. AREVALO, Auteur ; Steven W. COLE, Auteur Article en page(s) : p.235-253 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : High conflict and low warmth in families may contribute to immune cells developing a tendency to respond to threats with exaggerated inflammation that is insensitive to inhibitory signaling. We tested associations between family environments and expression of genes bearing response elements for transcription factors that regulate inflammation: nuclear factor kappa B (NF-?B) and glucocorticoid receptor. The overall sample (47 families) completed interviews, questionnaires, and 8-week daily diary assessments of conflict and warmth, which were used to create composite family conflict and warmth scores. The diaries assessed upper respiratory infection (URI) symptoms, and URI episodes were clinically verified. Leukocyte RNA was extracted from whole blood samples provided by a subsample of 42 children (8–13 years of age) and 73 parents. In children, higher conflict and lower warmth were related to greater expression of genes bearing response elements for the proinflammatory transcription factor NF-?B, and more severe URI symptoms. In parents, higher conflict and lower warmth were also related to greater NF-?B–associated gene expression. Monocytes and dendritic cells were implicated as primary cellular sources of differential gene expression in the sample. Consistent with existing conceptual frameworks, stressful family environments were related to a proinflammatory phenotype at the level of the circulating leukocyte transcriptome. En ligne : https://doi.org/10.1017/S0954579417000591 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Family environments and leukocyte transcriptome indicators of a proinflammatory phenotype in children and parents [Texte imprimé et/ou numérique] / Theodore F. ROBLES, Auteur ; Rena L. REPETTI, Auteur ; Bridget M. REYNOLDS, Auteur ; Paul J. CHUNG, Auteur ; Jesusa M. G. AREVALO, Auteur ; Steven W. COLE, Auteur . - p.235-253. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.235-253 Index. décimale : PER Périodiques Résumé : High conflict and low warmth in families may contribute to immune cells developing a tendency to respond to threats with exaggerated inflammation that is insensitive to inhibitory signaling. We tested associations between family environments and expression of genes bearing response elements for transcription factors that regulate inflammation: nuclear factor kappa B (NF-?B) and glucocorticoid receptor. The overall sample (47 families) completed interviews, questionnaires, and 8-week daily diary assessments of conflict and warmth, which were used to create composite family conflict and warmth scores. The diaries assessed upper respiratory infection (URI) symptoms, and URI episodes were clinically verified. Leukocyte RNA was extracted from whole blood samples provided by a subsample of 42 children (8–13 years of age) and 73 parents. In children, higher conflict and lower warmth were related to greater expression of genes bearing response elements for the proinflammatory transcription factor NF-?B, and more severe URI symptoms. In parents, higher conflict and lower warmth were also related to greater NF-?B–associated gene expression. Monocytes and dendritic cells were implicated as primary cellular sources of differential gene expression in the sample. Consistent with existing conceptual frameworks, stressful family environments were related to a proinflammatory phenotype at the level of the circulating leukocyte transcriptome. En ligne : https://doi.org/10.1017/S0954579417000591 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Social–ecological predictors of externalizing behavior trajectories in at-risk youth / Caleb J. FIGGE in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.255-266 Titre : Social–ecological predictors of externalizing behavior trajectories in at-risk youth Type de document : Texte imprimé et/ou numérique Auteurs : Caleb J. FIGGE, Auteur ; Cecilia MARTINEZ-TORTEYA, Auteur ; Jessica E. WEEKS, Auteur Article en page(s) : p.255-266 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Extant research consistently links youth externalizing problems and later maladaptive outcomes, and these behaviors are particularly detrimental given their relative stability across development. Although an array of risk and protective factors for externalizing problems have been identified, few studies have examined factors reflecting the multiple social–ecological levels that influence child development and used them to predict longitudinal trajectories of externalizing problems. The current study examined externalizing behavior trajectories in a sample of 1,094 at-risk youth (539 boys, 555 girls) from the Longitudinal Studies in Child Abuse and Neglect multisite longitudinal study of child maltreatment. Normed Child Behavior Checklist externalizing scores were used to estimate group trajectories via growth-based trajectory modeling at ages 10, 12, 14, and 16 using the SAS PROC TRAJ procedure. Model fit was assessed using the Bayes information criterion and the Akaike information criterion statistics. Analyses revealed optimal fit for five distinct behavioral trajectories: low stable, mid-increasing, mid-decreasing, medium high, and high stable. Multinomial logistic regressions revealed that a combination of risk and protective factors at individual, family, school, and neighborhood levels contribute to distinct trajectories of externalizing problems over time. Predictors of low and decreasing trajectories can inform interventions aimed at addressing externalizing problems among high-risk adolescents. En ligne : https://doi.org/10.1017/S0954579417000608 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Social–ecological predictors of externalizing behavior trajectories in at-risk youth [Texte imprimé et/ou numérique] / Caleb J. FIGGE, Auteur ; Cecilia MARTINEZ-TORTEYA, Auteur ; Jessica E. WEEKS, Auteur . - p.255-266. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.255-266 Index. décimale : PER Périodiques Résumé : Extant research consistently links youth externalizing problems and later maladaptive outcomes, and these behaviors are particularly detrimental given their relative stability across development. Although an array of risk and protective factors for externalizing problems have been identified, few studies have examined factors reflecting the multiple social–ecological levels that influence child development and used them to predict longitudinal trajectories of externalizing problems. The current study examined externalizing behavior trajectories in a sample of 1,094 at-risk youth (539 boys, 555 girls) from the Longitudinal Studies in Child Abuse and Neglect multisite longitudinal study of child maltreatment. Normed Child Behavior Checklist externalizing scores were used to estimate group trajectories via growth-based trajectory modeling at ages 10, 12, 14, and 16 using the SAS PROC TRAJ procedure. Model fit was assessed using the Bayes information criterion and the Akaike information criterion statistics. Analyses revealed optimal fit for five distinct behavioral trajectories: low stable, mid-increasing, mid-decreasing, medium high, and high stable. Multinomial logistic regressions revealed that a combination of risk and protective factors at individual, family, school, and neighborhood levels contribute to distinct trajectories of externalizing problems over time. Predictors of low and decreasing trajectories can inform interventions aimed at addressing externalizing problems among high-risk adolescents. En ligne : https://doi.org/10.1017/S0954579417000608 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Personality risk for antisocial behavior: Testing the intersections between callous–unemotional traits, sensation seeking, and impulse control in adolescence / Frank D. MANN in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.267-282 Titre : Personality risk for antisocial behavior: Testing the intersections between callous–unemotional traits, sensation seeking, and impulse control in adolescence Type de document : Texte imprimé et/ou numérique Auteurs : Frank D. MANN, Auteur ; Sara L. PAUL, Auteur ; Jennifer L. TACKETT, Auteur ; Elliot M. TUCKER-DROB, Auteur ; K. Paige HARDEN, Auteur Article en page(s) : p.267-282 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The current project seeks to integrate literatures on personality risk for antisocial behavior (ASB) by examining how callous–unemotional traits relate to (a) the development of disinhibited traits and (b) the association between disinhibited traits and ASB. In Study 1, using a nationally representative sample of youth (N > 7,000), we examined whether conduct problems and lack of guilt assessed during ages 4–10 years predicted levels of and changes in disinhibited traits over the course of adolescence, and moderated associations between these traits and ASB. High levels of childhood conduct problems were associated with higher levels of impulsivity, sensation seeking, and ASB in early adolescence, whereas lack of guilt was associated with lower levels of sensation seeking. Neither conduct problems nor lack of guilt significantly predicted changes in impulsivity or sensation seeking, and associations among changes in sensation seeking, impulsivity, and ASB were also consistent across levels of conduct problems and lack of guilt. In Study 2, using a cross-sectional sample of adolescents (N = 970), we tested whether callous–unemotional traits moderated associations between disinhibited traits and ASB. Consistent with the results of Study 1, associations between disinhibited personality and ASB were consistent across a continuous range of callous–unemotional traits. En ligne : https://doi.org/10.1017/S095457941700061X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Personality risk for antisocial behavior: Testing the intersections between callous–unemotional traits, sensation seeking, and impulse control in adolescence [Texte imprimé et/ou numérique] / Frank D. MANN, Auteur ; Sara L. PAUL, Auteur ; Jennifer L. TACKETT, Auteur ; Elliot M. TUCKER-DROB, Auteur ; K. Paige HARDEN, Auteur . - p.267-282. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.267-282 Index. décimale : PER Périodiques Résumé : The current project seeks to integrate literatures on personality risk for antisocial behavior (ASB) by examining how callous–unemotional traits relate to (a) the development of disinhibited traits and (b) the association between disinhibited traits and ASB. In Study 1, using a nationally representative sample of youth (N > 7,000), we examined whether conduct problems and lack of guilt assessed during ages 4–10 years predicted levels of and changes in disinhibited traits over the course of adolescence, and moderated associations between these traits and ASB. High levels of childhood conduct problems were associated with higher levels of impulsivity, sensation seeking, and ASB in early adolescence, whereas lack of guilt was associated with lower levels of sensation seeking. Neither conduct problems nor lack of guilt significantly predicted changes in impulsivity or sensation seeking, and associations among changes in sensation seeking, impulsivity, and ASB were also consistent across levels of conduct problems and lack of guilt. In Study 2, using a cross-sectional sample of adolescents (N = 970), we tested whether callous–unemotional traits moderated associations between disinhibited traits and ASB. Consistent with the results of Study 1, associations between disinhibited personality and ASB were consistent across a continuous range of callous–unemotional traits. En ligne : https://doi.org/10.1017/S095457941700061X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Out of harm's way: Secure versus insecure–disorganized attachment predicts less adolescent risk taking related to childhood poverty / Brianna C. DELKER in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.283-296 Titre : Out of harm's way: Secure versus insecure–disorganized attachment predicts less adolescent risk taking related to childhood poverty Type de document : Texte imprimé et/ou numérique Auteurs : Brianna C. DELKER, Auteur ; Rosemary E. BERNSTEIN, Auteur ; Heidemarie K. LAURENT, Auteur Article en page(s) : p.283-296 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Although some risk taking in adolescence is normative, evidence suggests that adolescents raised in conditions of socioeconomic disadvantage are disproportionately burdened with risk taking and its negative consequences. Using longitudinal data from the NICHD Study of Early Child Care and Youth Development, we investigated quality of the early caregiving environment as a potential prospective buffer against the long-term association between childhood poverty and adolescent risk taking. Multicategorical moderation model results indicated that if raised in poverty across age 1–54 months (average family income to needs ratio ? 1.02), relative to affluence (income to needs ratio ? 6.16), adolescents with histories of secure attachment to caregivers exhibited two times the number of risk behaviors at age 15, whereas adolescents with insecure–disorganized histories exhibited nearly five times the number of risk behaviors. Both early family economic hardship and history of insecure–disorganized attachment remained significant predictors of increased adolescent risk taking, alongside the interactive effect. Probing the interaction's region of significance revealed that history of secure (vs. insecure–disorganized) attachment is associated with protective reductions in risk taking below a family income to needs ratio of 2.24, or about 220% poverty level. Findings support a diathesis–stress model in which children with secure attachment histories are less deleteriously impacted by early socioeconomic adversity than their insecure–disorganized peers. En ligne : https://doi.org/10.1017/S0954579417000621 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Out of harm's way: Secure versus insecure–disorganized attachment predicts less adolescent risk taking related to childhood poverty [Texte imprimé et/ou numérique] / Brianna C. DELKER, Auteur ; Rosemary E. BERNSTEIN, Auteur ; Heidemarie K. LAURENT, Auteur . - p.283-296. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.283-296 Index. décimale : PER Périodiques Résumé : Although some risk taking in adolescence is normative, evidence suggests that adolescents raised in conditions of socioeconomic disadvantage are disproportionately burdened with risk taking and its negative consequences. Using longitudinal data from the NICHD Study of Early Child Care and Youth Development, we investigated quality of the early caregiving environment as a potential prospective buffer against the long-term association between childhood poverty and adolescent risk taking. Multicategorical moderation model results indicated that if raised in poverty across age 1–54 months (average family income to needs ratio ? 1.02), relative to affluence (income to needs ratio ? 6.16), adolescents with histories of secure attachment to caregivers exhibited two times the number of risk behaviors at age 15, whereas adolescents with insecure–disorganized histories exhibited nearly five times the number of risk behaviors. Both early family economic hardship and history of insecure–disorganized attachment remained significant predictors of increased adolescent risk taking, alongside the interactive effect. Probing the interaction's region of significance revealed that history of secure (vs. insecure–disorganized) attachment is associated with protective reductions in risk taking below a family income to needs ratio of 2.24, or about 220% poverty level. Findings support a diathesis–stress model in which children with secure attachment histories are less deleteriously impacted by early socioeconomic adversity than their insecure–disorganized peers. En ligne : https://doi.org/10.1017/S0954579417000621 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Associations between alcohol dehydrogenase genes and alcohol use across early and middle adolescence: Moderation × Preventive intervention / H. Harrington CLEVELAND in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.297-313 Titre : Associations between alcohol dehydrogenase genes and alcohol use across early and middle adolescence: Moderation × Preventive intervention Type de document : Texte imprimé et/ou numérique Auteurs : H. Harrington CLEVELAND, Auteur ; Gabriel L. SCHLOMER, Auteur ; David J. VANDENBERGH, Auteur ; Pedro S. A. WOLF, Auteur ; Mark E. FEINBERG, Auteur ; Mark T. GREENBERG, Auteur ; Richard L. SPOTH, Auteur ; Cleve REDMOND, Auteur Article en page(s) : p.297-313 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Data from the in-school sample of the PROSPER preventive intervention dissemination trial were used to investigate associations between alcohol dehydrogenase genes and alcohol use across adolescence, and whether substance misuse interventions in the 6th and 7th grades (targeting parenting, family functioning, social norms, youth decision making, and peer group affiliations) modified associations between these genes and adolescent use. Primary analyses were run on a sample of 1,885 individuals and included three steps. First, we estimated unconditional growth curve models with separate slopes for alcohol use from 6th to 9th grade and from 9th to 12th grade, as well as the intercept at Grade 9. Second, we used intervention condition and three alcohol dehydrogenase genes, 1B (ADH1B), 1C (ADH1C), and 4 (ADH4) to predict variance in slopes and intercept. Third, we examined whether genetic influences on model slopes and intercepts were moderated by intervention condition. The results indicated that the increase in alcohol use was greater in early adolescence than in middle adolescence; two of the genes, ADH1B and ADH1C, significantly predicted early adolescent slope and Grade 9 intercept, and associations between ADH1C and both early adolescent slope and intercept were significantly different across control and intervention conditions. En ligne : https://doi.org/10.1017/S0954579417000633 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Associations between alcohol dehydrogenase genes and alcohol use across early and middle adolescence: Moderation × Preventive intervention [Texte imprimé et/ou numérique] / H. Harrington CLEVELAND, Auteur ; Gabriel L. SCHLOMER, Auteur ; David J. VANDENBERGH, Auteur ; Pedro S. A. WOLF, Auteur ; Mark E. FEINBERG, Auteur ; Mark T. GREENBERG, Auteur ; Richard L. SPOTH, Auteur ; Cleve REDMOND, Auteur . - p.297-313. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.297-313 Index. décimale : PER Périodiques Résumé : Data from the in-school sample of the PROSPER preventive intervention dissemination trial were used to investigate associations between alcohol dehydrogenase genes and alcohol use across adolescence, and whether substance misuse interventions in the 6th and 7th grades (targeting parenting, family functioning, social norms, youth decision making, and peer group affiliations) modified associations between these genes and adolescent use. Primary analyses were run on a sample of 1,885 individuals and included three steps. First, we estimated unconditional growth curve models with separate slopes for alcohol use from 6th to 9th grade and from 9th to 12th grade, as well as the intercept at Grade 9. Second, we used intervention condition and three alcohol dehydrogenase genes, 1B (ADH1B), 1C (ADH1C), and 4 (ADH4) to predict variance in slopes and intercept. Third, we examined whether genetic influences on model slopes and intercepts were moderated by intervention condition. The results indicated that the increase in alcohol use was greater in early adolescence than in middle adolescence; two of the genes, ADH1B and ADH1C, significantly predicted early adolescent slope and Grade 9 intercept, and associations between ADH1C and both early adolescent slope and intercept were significantly different across control and intervention conditions. En ligne : https://doi.org/10.1017/S0954579417000633 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Adolescents from upper middle class communities: Substance misuse and addiction across early adulthood / Suniya S. LUTHAR in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.315-335 Titre : Adolescents from upper middle class communities: Substance misuse and addiction across early adulthood Type de document : Texte imprimé et/ou numérique Auteurs : Suniya S. LUTHAR, Auteur ; Phillip J. SMALL, Auteur ; Lucia CICIOLLA, Auteur Article en page(s) : p.315-335 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : In this prospective study of upper middle class youth, we document frequency of alcohol and drug use, as well as diagnoses of abuse and dependence, during early adulthood. Two cohorts were assessed as high school seniors and then annually across 4 college years (New England Study of Suburban Youth younger cohort [NESSY-Y]), and across ages 23–27 (NESSY older cohort [NESSY-O]; ns = 152 and 183 at final assessments, respectively). Across gender and annual assessments, results showed substantial elevations, relative to norms, for frequency of drunkenness and using marijuana, stimulants, and cocaine. Of more concern were psychiatric diagnoses of alcohol/drug dependence: among women and men, respectively, lifetime rates ranged between 19%–24% and 23%–40% among NESSY-Os at age 26; and 11%–16% and 19%–27% among NESSY-Ys at 22. Relative to norms, these rates among NESSY-O women and men were three and two times as high, respectively, and among NESSY-Y, close to one among women but twice as high among men. Findings also showed the protective power of parents’ containment (anticipated stringency of repercussions for substance use) at age 18; this was inversely associated with frequency of drunkenness and marijuana and stimulant use in adulthood. Results emphasize the need to take seriously the elevated rates of substance documented among adolescents in affluent American school communities. En ligne : https://doi.org/10.1017/S0954579417000645 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Adolescents from upper middle class communities: Substance misuse and addiction across early adulthood [Texte imprimé et/ou numérique] / Suniya S. LUTHAR, Auteur ; Phillip J. SMALL, Auteur ; Lucia CICIOLLA, Auteur . - p.315-335. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.315-335 Index. décimale : PER Périodiques Résumé : In this prospective study of upper middle class youth, we document frequency of alcohol and drug use, as well as diagnoses of abuse and dependence, during early adulthood. Two cohorts were assessed as high school seniors and then annually across 4 college years (New England Study of Suburban Youth younger cohort [NESSY-Y]), and across ages 23–27 (NESSY older cohort [NESSY-O]; ns = 152 and 183 at final assessments, respectively). Across gender and annual assessments, results showed substantial elevations, relative to norms, for frequency of drunkenness and using marijuana, stimulants, and cocaine. Of more concern were psychiatric diagnoses of alcohol/drug dependence: among women and men, respectively, lifetime rates ranged between 19%–24% and 23%–40% among NESSY-Os at age 26; and 11%–16% and 19%–27% among NESSY-Ys at 22. Relative to norms, these rates among NESSY-O women and men were three and two times as high, respectively, and among NESSY-Y, close to one among women but twice as high among men. Findings also showed the protective power of parents’ containment (anticipated stringency of repercussions for substance use) at age 18; this was inversely associated with frequency of drunkenness and marijuana and stimulant use in adulthood. Results emphasize the need to take seriously the elevated rates of substance documented among adolescents in affluent American school communities. En ligne : https://doi.org/10.1017/S0954579417000645 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Girls’ and boys’ trajectories of appearance anxiety from age 10 to 15 years are associated with earlier maturation and appearance-related teasing / Melanie J. ZIMMER-GEMBECK in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.337-350 Titre : Girls’ and boys’ trajectories of appearance anxiety from age 10 to 15 years are associated with earlier maturation and appearance-related teasing Type de document : Texte imprimé et/ou numérique Auteurs : Melanie J. ZIMMER-GEMBECK, Auteur ; Haley J. WEBB, Auteur ; Lara J. FARRELL, Auteur ; Allison M. WATERS, Auteur Article en page(s) : p.337-350 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Adolescents’ appearance-related concerns can provoke increasing emotional, social, and eating-related problems. The aims of this five-wave (2.5-year), multiple-informant longitudinal study were to (a) examine growth trajectories of appearance anxiety symptoms and appearance esteem, (b) identify whether trajectories differed by gender, and (c) examine several launching factors including parent-reported physical maturation, peer-rated physical appearance, body mass index, and appearance teasing by parents and peers. Participants were 387 adolescents (44% boys) aged 10 to 13 years at the first assessment. Steep growth in appearance anxiety symptoms was found for both girls and boys, but there was no average change in appearance esteem. Girls had more elevated appearance anxiety symptoms and lower appearance esteem than boys, girls’ body mass index was associated with symptoms, and earlier physical maturation and teasing about appearance, alone and in combination, were associated with growth in appearance anxiety symptoms for girls and boys. Earlier maturing boys who were highly teased by parents, but even more so when teased by peers, were at utmost risk for elevated appearance anxiety symptoms and increasing symptoms over time. In contrast, all girls exhibited elevated or increasing appearance anxiety symptoms across time, with the exception of girls with the latest maturation who also reported little teasing about their appearance. En ligne : https://doi.org/10.1017/S0954579417000657 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Girls’ and boys’ trajectories of appearance anxiety from age 10 to 15 years are associated with earlier maturation and appearance-related teasing [Texte imprimé et/ou numérique] / Melanie J. ZIMMER-GEMBECK, Auteur ; Haley J. WEBB, Auteur ; Lara J. FARRELL, Auteur ; Allison M. WATERS, Auteur . - p.337-350. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.337-350 Index. décimale : PER Périodiques Résumé : Adolescents’ appearance-related concerns can provoke increasing emotional, social, and eating-related problems. The aims of this five-wave (2.5-year), multiple-informant longitudinal study were to (a) examine growth trajectories of appearance anxiety symptoms and appearance esteem, (b) identify whether trajectories differed by gender, and (c) examine several launching factors including parent-reported physical maturation, peer-rated physical appearance, body mass index, and appearance teasing by parents and peers. Participants were 387 adolescents (44% boys) aged 10 to 13 years at the first assessment. Steep growth in appearance anxiety symptoms was found for both girls and boys, but there was no average change in appearance esteem. Girls had more elevated appearance anxiety symptoms and lower appearance esteem than boys, girls’ body mass index was associated with symptoms, and earlier physical maturation and teasing about appearance, alone and in combination, were associated with growth in appearance anxiety symptoms for girls and boys. Earlier maturing boys who were highly teased by parents, but even more so when teased by peers, were at utmost risk for elevated appearance anxiety symptoms and increasing symptoms over time. In contrast, all girls exhibited elevated or increasing appearance anxiety symptoms across time, with the exception of girls with the latest maturation who also reported little teasing about their appearance. En ligne : https://doi.org/10.1017/S0954579417000657 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Quantifying respiratory sinus arrhythmia: Effects of misspecifying breathing frequencies across development / Tiffany M. SHADER in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.351-366 Titre : Quantifying respiratory sinus arrhythmia: Effects of misspecifying breathing frequencies across development Type de document : Texte imprimé et/ou numérique Auteurs : Tiffany M. SHADER, Auteur ; Lisa M. GATZKE-KOPP, Auteur ; Sheila E. CROWELL, Auteur ; M. JAMILA REID, Auteur ; Julian F. THAYER, Auteur ; Michael W. VASEY, Auteur ; Carolyn WEBSTER-STRATTON, Auteur ; Ziv BELL, Auteur ; Theodore P. BEAUCHAINE, Auteur Article en page(s) : p.351-366 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Low resting respiratory sinus arrhythmia (RSA), and to a lesser extent excessive RSA reactivity to emotion evocation, are observed in many psychiatric disorders characterized by emotion dysregulation, including syndromes spanning the internalizing and externalizing spectra, and other conditions such as nonsuicidal self-injury. Nevertheless, some inconsistencies exist. For example, null outcomes in studies of RSA–emotion dysregulation relations are sometimes observed among younger participants. Such findings may derive from use of age inappropriate frequency bands in calculating RSA. We combine data from five published samples (N = 559) spanning ages 4 to 17 years, and reanalyze RSA data using age-appropriate respiratory frequencies. Misspecifying respiratory frequencies results in overestimates of resting RSA and underestimates of RSA reactivity, particularly among young children. Underestimates of developmental shifts in RSA and RSA reactivity from preschool to adolescence were also observed. Although correlational analyses revealed weak negative associations between resting RSA and aggression, those with clinical levels of externalizing exhibited lower resting RSA than their peers. No associations between RSA reactivity and externalizing were observed. Results confirm that age-corrected frequency bands should be used when estimating RSA, and that literature-wide overestimates of resting RSA, underestimates of RSA reactivity, and underestimates of developmental shifts in RSA and RSA reactivity may exist. En ligne : https://doi.org/10.1017/S0954579417000669 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Quantifying respiratory sinus arrhythmia: Effects of misspecifying breathing frequencies across development [Texte imprimé et/ou numérique] / Tiffany M. SHADER, Auteur ; Lisa M. GATZKE-KOPP, Auteur ; Sheila E. CROWELL, Auteur ; M. JAMILA REID, Auteur ; Julian F. THAYER, Auteur ; Michael W. VASEY, Auteur ; Carolyn WEBSTER-STRATTON, Auteur ; Ziv BELL, Auteur ; Theodore P. BEAUCHAINE, Auteur . - p.351-366. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.351-366 Index. décimale : PER Périodiques Résumé : Low resting respiratory sinus arrhythmia (RSA), and to a lesser extent excessive RSA reactivity to emotion evocation, are observed in many psychiatric disorders characterized by emotion dysregulation, including syndromes spanning the internalizing and externalizing spectra, and other conditions such as nonsuicidal self-injury. Nevertheless, some inconsistencies exist. For example, null outcomes in studies of RSA–emotion dysregulation relations are sometimes observed among younger participants. Such findings may derive from use of age inappropriate frequency bands in calculating RSA. We combine data from five published samples (N = 559) spanning ages 4 to 17 years, and reanalyze RSA data using age-appropriate respiratory frequencies. Misspecifying respiratory frequencies results in overestimates of resting RSA and underestimates of RSA reactivity, particularly among young children. Underestimates of developmental shifts in RSA and RSA reactivity from preschool to adolescence were also observed. Although correlational analyses revealed weak negative associations between resting RSA and aggression, those with clinical levels of externalizing exhibited lower resting RSA than their peers. No associations between RSA reactivity and externalizing were observed. Results confirm that age-corrected frequency bands should be used when estimating RSA, and that literature-wide overestimates of resting RSA, underestimates of RSA reactivity, and underestimates of developmental shifts in RSA and RSA reactivity may exist. En ligne : https://doi.org/10.1017/S0954579417000669 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Childhood abuse and neglect and insecure attachment states of mind in adulthood: Prospective, longitudinal evidence from a high-risk sample—CORRIGENDUM / K. Lee RABY in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.367-370 Titre : Childhood abuse and neglect and insecure attachment states of mind in adulthood: Prospective, longitudinal evidence from a high-risk sample—CORRIGENDUM Type de document : Texte imprimé et/ou numérique Auteurs : K. Lee RABY, Auteur ; Madelyn H. LABELLA, Auteur ; Jodi MARTIN, Auteur ; Elizabeth A. CARLSON, Auteur ; Glenn I. ROISMAN, Auteur Article en page(s) : p.367-370 Langues : Anglais (eng) Index. décimale : PER Périodiques En ligne : https://doi.org/10.1017/S095457941700089X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Childhood abuse and neglect and insecure attachment states of mind in adulthood: Prospective, longitudinal evidence from a high-risk sample—CORRIGENDUM [Texte imprimé et/ou numérique] / K. Lee RABY, Auteur ; Madelyn H. LABELLA, Auteur ; Jodi MARTIN, Auteur ; Elizabeth A. CARLSON, Auteur ; Glenn I. ROISMAN, Auteur . - p.367-370. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.367-370 Index. décimale : PER Périodiques En ligne : https://doi.org/10.1017/S095457941700089X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336

Association of cognitive function and liability to addiction with childhood herpesvirus infections: A prospective cohort study—ERRATUM / Michael M. VANYUKOV in Development and Psychopathology, 30-1 (February 2018)  

Public ISBD [article]  inDevelopment and Psychopathology > 30-1 (February 2018) . - p.371-372 Titre : Association of cognitive function and liability to addiction with childhood herpesvirus infections: A prospective cohort study—ERRATUM Type de document : Texte imprimé et/ou numérique Auteurs : Michael M. VANYUKOV, Auteur ; Vishwajit L. NIMGAONKAR, Auteur ; Levent KIRISCI, Auteur ; Galina P. KIRILLOVA, Auteur ; Maureen D. REYNOLDS, Auteur ; Konasale PRASAD, Auteur ; Ralph E. TARTER, Auteur ; Robert H. YOLKEN, Auteur Article en page(s) : p.371-372 Langues : Anglais (eng) Index. décimale : PER Périodiques En ligne : https://doi.org/10.1017/S0954579417000906 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336 [article] Association of cognitive function and liability to addiction with childhood herpesvirus infections: A prospective cohort study—ERRATUM [Texte imprimé et/ou numérique] / Michael M. VANYUKOV, Auteur ; Vishwajit L. NIMGAONKAR, Auteur ; Levent KIRISCI, Auteur ; Galina P. KIRILLOVA, Auteur ; Maureen D. REYNOLDS, Auteur ; Konasale PRASAD, Auteur ; Ralph E. TARTER, Auteur ; Robert H. YOLKEN, Auteur . - p.371-372. Langues : Anglais (eng) in Development and Psychopathology > 30-1 (February 2018) . - p.371-372 Index. décimale : PER Périodiques En ligne : https://doi.org/10.1017/S0954579417000906 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=336