50 recherche sur le mot-clé 'Developmental psychopathology'

Annual Research Review: A developmental psychopathology approach to understanding callous-unemotional traits in children and adolescents with serious conduct problems / Paul J. FRICK in Journal of Child Psychology and Psychiatry, 55-6 (June 2014)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 55-6 (June 2014) . - p.532-548 Titre : Annual Research Review: A developmental psychopathology approach to understanding callous-unemotional traits in children and adolescents with serious conduct problems Type de document : Texte imprimé et/ou numérique Auteurs : Paul J. FRICK, Auteur ; James V. RAY, Auteur ; Laura C. THORNTON, Auteur ; Rachel E. KAHN, Auteur Année de publication : 2014 Article en page(s) : p.532-548 Langues : Anglais (eng) Mots-clés : Callous-unemotional traits  developmental psychopathology  conscience  empathy  guilt  stability Index. décimale : PER Périodiques Résumé : Recent research has suggested that the presence of significant levels of callous-unemotional (CU) traits designates a clinically important and etiologically distinct subgroup of children and adolescents with serious conduct problems. Based on this research, CU traits have been included in the most recent revision of the Diagnostic and Statistical Manual of Mental Disorders – 5th Edition (DSM-5; American Psychiatric Association, 2013) – as a specifier for the diagnosis of conduct disorder. In this review, we attempt to understand CU traits within a developmental psychopathological framework. Specifically, we summarize research on the normal development of the prosocial emotions of empathy and guilt (i.e., conscience) and we illustrate how the development of CU traits can be viewed as the normal development of conscience gone awry. Furthermore, we review research on the stability of CU traits across different developmental periods and highlight factors that can influence this stability. Finally, we highlight the implications of this developmental psychopathological framework for future etiological research, for assessment and diagnostic classification, and for treatment of children with serious conduct problems. En ligne : http://dx.doi.org/10.1111/jcpp.12152 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=233 [article] Annual Research Review: A developmental psychopathology approach to understanding callous-unemotional traits in children and adolescents with serious conduct problems [Texte imprimé et/ou numérique] / Paul J. FRICK, Auteur ; James V. RAY, Auteur ; Laura C. THORNTON, Auteur ; Rachel E. KAHN, Auteur . - 2014 . - p.532-548. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 55-6 (June 2014) . - p.532-548 Mots-clés : Callous-unemotional traits  developmental psychopathology  conscience  empathy  guilt  stability Index. décimale : PER Périodiques Résumé : Recent research has suggested that the presence of significant levels of callous-unemotional (CU) traits designates a clinically important and etiologically distinct subgroup of children and adolescents with serious conduct problems. Based on this research, CU traits have been included in the most recent revision of the Diagnostic and Statistical Manual of Mental Disorders – 5th Edition (DSM-5; American Psychiatric Association, 2013) – as a specifier for the diagnosis of conduct disorder. In this review, we attempt to understand CU traits within a developmental psychopathological framework. Specifically, we summarize research on the normal development of the prosocial emotions of empathy and guilt (i.e., conscience) and we illustrate how the development of CU traits can be viewed as the normal development of conscience gone awry. Furthermore, we review research on the stability of CU traits across different developmental periods and highlight factors that can influence this stability. Finally, we highlight the implications of this developmental psychopathological framework for future etiological research, for assessment and diagnostic classification, and for treatment of children with serious conduct problems. En ligne : http://dx.doi.org/10.1111/jcpp.12152 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=233

Annual Research Review: Impact of advances in genetics in understanding developmental psychopathology / Anjene M. ADDINGTON in Journal of Child Psychology and Psychiatry, 53-5 (May 2012)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 53-5 (May 2012) . - p.510-518 Titre : Annual Research Review: Impact of advances in genetics in understanding developmental psychopathology Type de document : Texte imprimé et/ou numérique Auteurs : Anjene M. ADDINGTON, Auteur ; Judith RAPOPORT, Auteur Année de publication : 2012 Article en page(s) : p.510-518 Langues : Anglais (eng) Mots-clés : Developmental psychopathology  genetics  copy number variantspre-natal diagnosis  nosology  prevention Index. décimale : PER Périodiques Résumé : It was hoped that diagnostic guidelines for, and treatment of, child psychiatric disorders in DSM-5 would be informed by the wealth of clinical genetic research related to neurodevelopmental disorders. In spite of remarkable advances in genetic technology, this has not been the case. Candidate gene, genome-wide association, and rare copy number variant (CNV) studies have been carried out for attention-deficit/hyperactivity disorder (ADHD), Autism, Tourette’s Syndrome, and schizophrenia, with intriguing results, but environmental factors, incomplete penetrance, pleiotropy, and genetic heterogeneity, underlying any given phenotype have limited clinical translation. One promising approach may be the use of developmental brain imaging measures as more relevant phenotypes. This is particularly important, as subtle abnormalities in timing and expression of gene pathways underlying brain development may well link these disorders and be the ultimate target of treatments. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2011.02478.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=154 [article] Annual Research Review: Impact of advances in genetics in understanding developmental psychopathology [Texte imprimé et/ou numérique] / Anjene M. ADDINGTON, Auteur ; Judith RAPOPORT, Auteur . - 2012 . - p.510-518. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 53-5 (May 2012) . - p.510-518 Mots-clés : Developmental psychopathology  genetics  copy number variantspre-natal diagnosis  nosology  prevention Index. décimale : PER Périodiques Résumé : It was hoped that diagnostic guidelines for, and treatment of, child psychiatric disorders in DSM-5 would be informed by the wealth of clinical genetic research related to neurodevelopmental disorders. In spite of remarkable advances in genetic technology, this has not been the case. Candidate gene, genome-wide association, and rare copy number variant (CNV) studies have been carried out for attention-deficit/hyperactivity disorder (ADHD), Autism, Tourette’s Syndrome, and schizophrenia, with intriguing results, but environmental factors, incomplete penetrance, pleiotropy, and genetic heterogeneity, underlying any given phenotype have limited clinical translation. One promising approach may be the use of developmental brain imaging measures as more relevant phenotypes. This is particularly important, as subtle abnormalities in timing and expression of gene pathways underlying brain development may well link these disorders and be the ultimate target of treatments. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2011.02478.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=154

Annual Research Review: The neuroinflammation hypothesis for stress and psychopathology in children – developmental psychoneuroimmunology / Thomas G. O'CONNOR in Journal of Child Psychology and Psychiatry, 55-6 (June 2014)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 55-6 (June 2014) . - p.615-631 Titre : Annual Research Review: The neuroinflammation hypothesis for stress and psychopathology in children – developmental psychoneuroimmunology Type de document : Texte imprimé et/ou numérique Auteurs : Thomas G. O'CONNOR, Auteur ; Jan A. MOYNIHAN, Auteur ; Mary T. CASERTA, Auteur Année de publication : 2014 Article en page(s) : p.615-631 Langues : Anglais (eng) Mots-clés : Immunology  psychoneuroimmunology  neuroinflammation  stress  developmental psychopathology Index. décimale : PER Périodiques Résumé : Abstract Experimental animal and adult human data suggest that stress exposure is associated with alterations in immune system function that may underlie increased susceptibility to disease and behavioral disorders. The implications of these data for child psychology and psychiatry are not yet clear. The current review seeks to distil and translate the relevant animal and adult human work to children to advance a developmental model of psychoneuroimmunology. In addition to reviewing key specific findings, we consider biological/conceptual models and technical aspects of psychoneuroimmunology work in pediatric populations, and outline the rationales and advantages of integrating hypotheses concerning neuroinflammation in developmental studies of psychopathology. En ligne : http://dx.doi.org/10.1111/jcpp.12187 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=234 [article] Annual Research Review: The neuroinflammation hypothesis for stress and psychopathology in children – developmental psychoneuroimmunology [Texte imprimé et/ou numérique] / Thomas G. O'CONNOR, Auteur ; Jan A. MOYNIHAN, Auteur ; Mary T. CASERTA, Auteur . - 2014 . - p.615-631. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 55-6 (June 2014) . - p.615-631 Mots-clés : Immunology  psychoneuroimmunology  neuroinflammation  stress  developmental psychopathology Index. décimale : PER Périodiques Résumé : Abstract Experimental animal and adult human data suggest that stress exposure is associated with alterations in immune system function that may underlie increased susceptibility to disease and behavioral disorders. The implications of these data for child psychology and psychiatry are not yet clear. The current review seeks to distil and translate the relevant animal and adult human work to children to advance a developmental model of psychoneuroimmunology. In addition to reviewing key specific findings, we consider biological/conceptual models and technical aspects of psychoneuroimmunology work in pediatric populations, and outline the rationales and advantages of integrating hypotheses concerning neuroinflammation in developmental studies of psychopathology. En ligne : http://dx.doi.org/10.1111/jcpp.12187 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=234

Annual Research Review: The role of the environment in the developmental psychopathology of autism spectrum condition / William MANDY in Journal of Child Psychology and Psychiatry, 57-3 (March 2016)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 57-3 (March 2016) . - p.271-292 Titre : Annual Research Review: The role of the environment in the developmental psychopathology of autism spectrum condition Type de document : Texte imprimé et/ou numérique Auteurs : William MANDY, Auteur ; Meng-Chuan LAI, Auteur Article en page(s) : p.271-292 Langues : Anglais (eng) Mots-clés : Autism spectrum condition  autism spectrum disorder  autism  Asperger's syndrome  genetics  environment  developmental psychopathology Index. décimale : PER Périodiques Résumé : Background Although autism spectrum condition (ASC) is strongly genetic in origin, accumulating evidence points to the critical roles of various environmental influences on its emergence and subsequent developmental course. Methods A developmental psychopathology framework was used to synthesise literature on environmental factors associated with the onset and course of ASC (based on a systematic search of the literature using PubMed, PsychInfo and Google Scholar databases). Particular emphasis was placed on gene–environment interplay, including gene–environment interaction (G × E) and gene–environment correlation (rGE). Results Before conception, advanced paternal and maternal ages may independently enhance offspring risk for ASC. Exogenous prenatal risks are evident (e.g. valproate and toxic chemicals) or possible (e.g. selective serotonin reuptake inhibitors), and processes endogenous to the materno-foeto-placental unit (e.g. maternal diabetes, enhanced steroidogenic activities and maternal immune activation) likely heighten offspring vulnerability to ASC. Folate intake is a prenatal protective factor, with a particular window of action around 4 weeks preconception and during the first trimester. These prenatal risks and protective mechanisms appear to involve G × E and potentially rGE. A variety of perinatal risks are related to offspring ASC risk, possibly reflecting rGE. Postnatal social factors (e.g. caregiver–infant interaction, severe early deprivation) during the first years of life may operate through rGE to influence the likelihood of manifesting a full ASC phenotype from a ‘prodromal’ phase (a proposal distinct to the discredited and harmful ‘refrigerator mother hypothesis’); and later postnatal risks, after the full manifestation of ASC, shape life span development through transactions mediated by rGE. There is no evidence that vaccination is a postnatal risk for ASC. Conclusions Future investigations should consider the specificity of risks for ASC versus other atypical neurodevelopmental trajectories, timing of risk and protective mechanisms, animal model systems to study mechanisms underlying gene–environment interplay, large-sample genome–envirome designs to address G × E and longitudinal studies to elucidate how rGE plays out over time. Clinical and public health implications are discussed. En ligne : http://dx.doi.org/10.1111/jcpp.12501 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=282 [article] Annual Research Review: The role of the environment in the developmental psychopathology of autism spectrum condition [Texte imprimé et/ou numérique] / William MANDY, Auteur ; Meng-Chuan LAI, Auteur . - p.271-292. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 57-3 (March 2016) . - p.271-292 Mots-clés : Autism spectrum condition  autism spectrum disorder  autism  Asperger's syndrome  genetics  environment  developmental psychopathology Index. décimale : PER Périodiques Résumé : Background Although autism spectrum condition (ASC) is strongly genetic in origin, accumulating evidence points to the critical roles of various environmental influences on its emergence and subsequent developmental course. Methods A developmental psychopathology framework was used to synthesise literature on environmental factors associated with the onset and course of ASC (based on a systematic search of the literature using PubMed, PsychInfo and Google Scholar databases). Particular emphasis was placed on gene–environment interplay, including gene–environment interaction (G × E) and gene–environment correlation (rGE). Results Before conception, advanced paternal and maternal ages may independently enhance offspring risk for ASC. Exogenous prenatal risks are evident (e.g. valproate and toxic chemicals) or possible (e.g. selective serotonin reuptake inhibitors), and processes endogenous to the materno-foeto-placental unit (e.g. maternal diabetes, enhanced steroidogenic activities and maternal immune activation) likely heighten offspring vulnerability to ASC. Folate intake is a prenatal protective factor, with a particular window of action around 4 weeks preconception and during the first trimester. These prenatal risks and protective mechanisms appear to involve G × E and potentially rGE. A variety of perinatal risks are related to offspring ASC risk, possibly reflecting rGE. Postnatal social factors (e.g. caregiver–infant interaction, severe early deprivation) during the first years of life may operate through rGE to influence the likelihood of manifesting a full ASC phenotype from a ‘prodromal’ phase (a proposal distinct to the discredited and harmful ‘refrigerator mother hypothesis’); and later postnatal risks, after the full manifestation of ASC, shape life span development through transactions mediated by rGE. There is no evidence that vaccination is a postnatal risk for ASC. Conclusions Future investigations should consider the specificity of risks for ASC versus other atypical neurodevelopmental trajectories, timing of risk and protective mechanisms, animal model systems to study mechanisms underlying gene–environment interplay, large-sample genome–envirome designs to address G × E and longitudinal studies to elucidate how rGE plays out over time. Clinical and public health implications are discussed. En ligne : http://dx.doi.org/10.1111/jcpp.12501 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=282

Childhood vaccination as a protective factor for developmental psychopathology / Bora KIM in Research in Autism Spectrum Disorders, 79 (November 2020)  

Public ISBD [article]  inResearch in Autism Spectrum Disorders > 79 (November 2020) . - 101653 Titre : Childhood vaccination as a protective factor for developmental psychopathology Type de document : Texte imprimé et/ou numérique Auteurs : Bora KIM, Auteur ; Mina HA, Auteur ; Young Shin KIM, Auteur ; Yun-Joo KOH, Auteur ; Ho-Jang KWON, Auteur ; Myung-Ho LIM, Auteur ; Ki-Chung PAIK, Auteur ; Hosanna KIM, Auteur ; Patricia HONG, Auteur ; Bennett L. LEVENTHAL, Auteur Article en page(s) : 101653 Langues : Anglais (eng) Mots-clés : ASD  Developmental psychopathology  Vaccination  Protective effects Index. décimale : PER Périodiques Résumé : Backgrounds Despite multiple studies demonstrating no relationship between childhood vaccination and increasing Autism Spectrum Disorder (ASD) prevalence, parental fear for vaccination and subsequent refusal to vaccinate their children continue, resulting in recent outbreaks of childhood infections such as measles in the US. We examine the relationship between the completion of 6 recommended vaccinations in childhood and the likelihood for having developmental psychopathology. Methods Two large-scale South Korean epidemiologic samples were used to examine whether completion of childhood vaccinations decrease likelihood of having ASD as assessed by Autism Spectrum Screening Questionnaire (ASSQ) and behavioral problems scores. Parental reports on vaccination completion were categorized in groups: <3, 4?5, & 6. The primary outcome is the likelihood of having ASD and/or, internalizing, and externalizing behavioral symptoms. Likelihood of having ASD was categorized as: low (ASSQ < 10), intermediate (ASSQ = 10–14), and high (ASSQ ? 15). The risk for externalizing/internalizing symptoms was assessed with the Behavior Assessment System for Children-Parent Rating Scale. We examined the hypothesis in a Discovery Sample (DS) (N = 10,006) and verified findings in a Replication Sample (RS) (N = 29,381). Results 84.3 % of DS and 80.1 % of RS participants were fully vaccinated. In the DS, after adjusting for demographics and confounders, children with incomplete-vaccinations were at greater risk for ASD when compared to those fully vaccinated (adjusted odds ratio [aOR] = 1.42, 95 % Confidence Interval [CI] 1.17?1.73 with 4?5 vaccinations; aOR = 2.33, CI 1.53?3.56 with vaccination <3). The DS finding was confirmed in the RS (aOR = 1.44, CI 1.32?1.58 with 4?5 vaccinations and aOR = 2.19, CI 1.80?2.67 with < 3 vaccinations). In the DS, those with incomplete-vaccinations were at a greater risk for internalizing and externalizing symptoms. Conclusions We replicate our own and prior findings that vaccination does not increase ASD risk. Further, completing recommended vaccinations may offer protection against the risk of having ASD and other developmental psychopathology. En ligne : https://doi.org/10.1016/j.rasd.2020.101653 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Childhood vaccination as a protective factor for developmental psychopathology [Texte imprimé et/ou numérique] / Bora KIM, Auteur ; Mina HA, Auteur ; Young Shin KIM, Auteur ; Yun-Joo KOH, Auteur ; Ho-Jang KWON, Auteur ; Myung-Ho LIM, Auteur ; Ki-Chung PAIK, Auteur ; Hosanna KIM, Auteur ; Patricia HONG, Auteur ; Bennett L. LEVENTHAL, Auteur . - 101653. Langues : Anglais (eng) in Research in Autism Spectrum Disorders > 79 (November 2020) . - 101653 Mots-clés : ASD  Developmental psychopathology  Vaccination  Protective effects Index. décimale : PER Périodiques Résumé : Backgrounds Despite multiple studies demonstrating no relationship between childhood vaccination and increasing Autism Spectrum Disorder (ASD) prevalence, parental fear for vaccination and subsequent refusal to vaccinate their children continue, resulting in recent outbreaks of childhood infections such as measles in the US. We examine the relationship between the completion of 6 recommended vaccinations in childhood and the likelihood for having developmental psychopathology. Methods Two large-scale South Korean epidemiologic samples were used to examine whether completion of childhood vaccinations decrease likelihood of having ASD as assessed by Autism Spectrum Screening Questionnaire (ASSQ) and behavioral problems scores. Parental reports on vaccination completion were categorized in groups: <3, 4?5, & 6. The primary outcome is the likelihood of having ASD and/or, internalizing, and externalizing behavioral symptoms. Likelihood of having ASD was categorized as: low (ASSQ < 10), intermediate (ASSQ = 10–14), and high (ASSQ ? 15). The risk for externalizing/internalizing symptoms was assessed with the Behavior Assessment System for Children-Parent Rating Scale. We examined the hypothesis in a Discovery Sample (DS) (N = 10,006) and verified findings in a Replication Sample (RS) (N = 29,381). Results 84.3 % of DS and 80.1 % of RS participants were fully vaccinated. In the DS, after adjusting for demographics and confounders, children with incomplete-vaccinations were at greater risk for ASD when compared to those fully vaccinated (adjusted odds ratio [aOR] = 1.42, 95 % Confidence Interval [CI] 1.17?1.73 with 4?5 vaccinations; aOR = 2.33, CI 1.53?3.56 with vaccination <3). The DS finding was confirmed in the RS (aOR = 1.44, CI 1.32?1.58 with 4?5 vaccinations and aOR = 2.19, CI 1.80?2.67 with < 3 vaccinations). In the DS, those with incomplete-vaccinations were at a greater risk for internalizing and externalizing symptoms. Conclusions We replicate our own and prior findings that vaccination does not increase ASD risk. Further, completing recommended vaccinations may offer protection against the risk of having ASD and other developmental psychopathology. En ligne : https://doi.org/10.1016/j.rasd.2020.101653 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Conduct problems among children in low-income, urban neighborhoods: A developmental psychopathology- and RDoC-informed approach / Deborah A.G. DRABICK in Development and Psychopathology, 33-5 (December 2021)  

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Defining the developmental parameters of temper loss in early childhood: implications for developmental psychopathology / Lauren S. WAKSCHLAG in Journal of Child Psychology and Psychiatry, 53-11 (November 2012)  

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Developmental stability of general and specific factors of psychopathology from early childhood to adolescence: dynamic mutualism or p-differentiation? / E. MCELROY in Journal of Child Psychology and Psychiatry, 59-6 (June 2018)  

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Editorial: The shape of the nosology to come in developmental psychopathology / Joel T. NIGG in Journal of Child Psychology and Psychiatry, 56-4 (April 2015)  

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A multilevel developmental psychopathology model of childbirth and the perinatal transition / Elisabeth CONRADT ; Patricia K. KERIG ; Paula G. WILLIAMS ; Sheila E. CROWELL in Development and Psychopathology, 36-2 (May 2024)  

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