Distinct trajectories of separation anxiety in the preschool years: persistence at school entry and early-life associated factors / Marco BATTAGLIA in Journal of Child Psychology and Psychiatry, 57-1 (January 2016)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 57-1 (January 2016) . - p.39-46 Titre : Distinct trajectories of separation anxiety in the preschool years: persistence at school entry and early-life associated factors Type de document : Texte imprimé et/ou numérique Auteurs : Marco BATTAGLIA, Auteur ; Évelyne TOUCHETTE, Auteur ; Gabrielle GARON-CARRIER, Auteur ; Ginette DIONNE, Auteur ; Sylvana M. CÔTÉ, Auteur ; Frank VITARO, Auteur ; Richard E. TREMBLAY, Auteur ; Michel BOIVIN, Auteur Article en page(s) : p.39-46 Langues : Anglais (eng) Mots-clés : Separation anxiety  trajectories  risk factors  internalising disorders  development Index. décimale : PER Périodiques Résumé : Background Little is known about how children differ in the onset and evolution of separation anxiety (SA) symptoms during the preschool years, and how SA develops into separation anxiety disorder. In a large, representative population-based sample, we investigated the developmental trajectories of SA symptoms from infancy to school entry, their early associated risk factors, and their associations with teachers' ratings of SA in kindergarten. Methods Longitudinal assessment of SA trajectories and risk factors in a cohort of 1,933 families between the ages of 1.5 and 6 years. Results Analyses revealed a best-fitting, 4-trajectory solution, including a prevailing, unaffected Low-Persistent group (60.2%), and three smaller groups of distinct developmental course: a High-Increasing (6.9%), a High-Decreasing (10.8%), and a Low-Increasing group (22.1%). The High-Increasing group remained high throughout the preschool years and was the only trajectory to predict teacher-assessed SA at age 6 years. Except for the High-Increasing, all trajectories showed substantial reduction in symptoms by age 6 years. The High-Increasing and High-Decreasing groups shared several early risk factors, but the former was uniquely associated with higher maternal depression, maternal smoking during pregnancy, and parental unemployment. Conclusions Most children with high SA profile at age 1.5 years are expected to progressively recover by age 4–5. High SA at age 1.5 that persists over time deserves special attention, and may predict separation anxiety disorder. A host of child perinatal, parental and family-contextual risk factors were associated with the onset and developmental course of SA across the preschool years. En ligne : http://dx.doi.org/10.1111/jcpp.12424 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=273 [article] Distinct trajectories of separation anxiety in the preschool years: persistence at school entry and early-life associated factors [Texte imprimé et/ou numérique] / Marco BATTAGLIA, Auteur ; Évelyne TOUCHETTE, Auteur ; Gabrielle GARON-CARRIER, Auteur ; Ginette DIONNE, Auteur ; Sylvana M. CÔTÉ, Auteur ; Frank VITARO, Auteur ; Richard E. TREMBLAY, Auteur ; Michel BOIVIN, Auteur . - p.39-46. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 57-1 (January 2016) . - p.39-46 Mots-clés : Separation anxiety  trajectories  risk factors  internalising disorders  development Index. décimale : PER Périodiques Résumé : Background Little is known about how children differ in the onset and evolution of separation anxiety (SA) symptoms during the preschool years, and how SA develops into separation anxiety disorder. In a large, representative population-based sample, we investigated the developmental trajectories of SA symptoms from infancy to school entry, their early associated risk factors, and their associations with teachers' ratings of SA in kindergarten. Methods Longitudinal assessment of SA trajectories and risk factors in a cohort of 1,933 families between the ages of 1.5 and 6 years. Results Analyses revealed a best-fitting, 4-trajectory solution, including a prevailing, unaffected Low-Persistent group (60.2%), and three smaller groups of distinct developmental course: a High-Increasing (6.9%), a High-Decreasing (10.8%), and a Low-Increasing group (22.1%). The High-Increasing group remained high throughout the preschool years and was the only trajectory to predict teacher-assessed SA at age 6 years. Except for the High-Increasing, all trajectories showed substantial reduction in symptoms by age 6 years. The High-Increasing and High-Decreasing groups shared several early risk factors, but the former was uniquely associated with higher maternal depression, maternal smoking during pregnancy, and parental unemployment. Conclusions Most children with high SA profile at age 1.5 years are expected to progressively recover by age 4–5. High SA at age 1.5 that persists over time deserves special attention, and may predict separation anxiety disorder. A host of child perinatal, parental and family-contextual risk factors were associated with the onset and developmental course of SA across the preschool years. En ligne : http://dx.doi.org/10.1111/jcpp.12424 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=273

Individual- and family-level associations between child psychopathology and parenting / Florence PERQUIER ; John D. HALTIGAN ; Li WANG ; Brendan F. ANDRADE ; Marco BATTAGLIA ; Peter SZATMARI ; Katholiki GEORGIADES in Development and Psychopathology, 36-2 (May 2024)  

Public ISBD [article]  inDevelopment and Psychopathology > 36-2 (May 2024) . - p.944-952 Titre : Individual- and family-level associations between child psychopathology and parenting Type de document : Texte imprimé et/ou numérique Auteurs : Florence PERQUIER, Auteur ; John D. HALTIGAN, Auteur ; Li WANG, Auteur ; Brendan F. ANDRADE, Auteur ; Marco BATTAGLIA, Auteur ; Peter SZATMARI, Auteur ; Katholiki GEORGIADES, Auteur Article en page(s) : p.944-952 Langues : Anglais (eng) Mots-clés : bifactor modeling  externalizing  family  internalizing  parenting Index. décimale : PER Périodiques Résumé : Parenting can protect against the development of, or increase risk for, child psychopathology; however, it is unclear if parenting is related to psychopathology symptoms in a specific domain, or to broad liability for psychopathology. Parenting differs between and within families, and both overall family-level parenting and the child-specific parenting a child receives may be important in estimating transdiagnostic associations with psychopathology. Data come from a cross-sectional epidemiological sample (N = 10,605 children ages 4-17, 6434 households). Parents rated child internalizing and externalizing symptoms and their parenting toward each child. General and specific (internalizing, externalizing) psychopathology factors, derived with bifactor modeling, were regressed on parenting using multilevel modeling. Less warmth and more aversive/inconsistent parenting in the family, and toward an individual child relative to family average, were associated with higher general psychopathology and specific externalizing problems. Unexpectedly, more warmth in the family, and toward an individual child relative to family average, was associated with higher specific internalizing problems in 4-11 (not 12-17) year-olds. Less warmth and more aversive/inconsistent parenting are broad correlates of child psychopathology. Aversive/inconsistent parenting, is also related to specific externalizing problems. Parents may behave more warmly when their younger children have specific internalizing problems, net of overall psychopathology. En ligne : https://dx.doi.org/10.1017/S0954579423000202 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=528 [article] Individual- and family-level associations between child psychopathology and parenting [Texte imprimé et/ou numérique] / Florence PERQUIER, Auteur ; John D. HALTIGAN, Auteur ; Li WANG, Auteur ; Brendan F. ANDRADE, Auteur ; Marco BATTAGLIA, Auteur ; Peter SZATMARI, Auteur ; Katholiki GEORGIADES, Auteur . - p.944-952. Langues : Anglais (eng) in Development and Psychopathology > 36-2 (May 2024) . - p.944-952 Mots-clés : bifactor modeling  externalizing  family  internalizing  parenting Index. décimale : PER Périodiques Résumé : Parenting can protect against the development of, or increase risk for, child psychopathology; however, it is unclear if parenting is related to psychopathology symptoms in a specific domain, or to broad liability for psychopathology. Parenting differs between and within families, and both overall family-level parenting and the child-specific parenting a child receives may be important in estimating transdiagnostic associations with psychopathology. Data come from a cross-sectional epidemiological sample (N = 10,605 children ages 4-17, 6434 households). Parents rated child internalizing and externalizing symptoms and their parenting toward each child. General and specific (internalizing, externalizing) psychopathology factors, derived with bifactor modeling, were regressed on parenting using multilevel modeling. Less warmth and more aversive/inconsistent parenting in the family, and toward an individual child relative to family average, were associated with higher general psychopathology and specific externalizing problems. Unexpectedly, more warmth in the family, and toward an individual child relative to family average, was associated with higher specific internalizing problems in 4-11 (not 12-17) year-olds. Less warmth and more aversive/inconsistent parenting are broad correlates of child psychopathology. Aversive/inconsistent parenting, is also related to specific externalizing problems. Parents may behave more warmly when their younger children have specific internalizing problems, net of overall psychopathology. En ligne : https://dx.doi.org/10.1017/S0954579423000202 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=528

Socioeconomic status mediates the genetic contribution of the dopamine receptor D4 and serotonin transporter linked promoter region repeat polymorphisms to externalization in preadolescence / Maria NOBILE in Development and Psychopathology, 19-4 (Fall 2007)  

Public ISBD [article]  inDevelopment and Psychopathology > 19-4 (Fall 2007) . - p.1147-1160 Titre : Socioeconomic status mediates the genetic contribution of the dopamine receptor D4 and serotonin transporter linked promoter region repeat polymorphisms to externalization in preadolescence Type de document : Texte imprimé et/ou numérique Auteurs : Maria NOBILE, Auteur ; Roberto GIORDA, Auteur ; Cecilia MARINO, Auteur ; Ombretta CARLET, Auteur ; Valentina PASTORE, Auteur ; Laura VANZIN, Auteur ; Monica BELLINA, Auteur ; Massimo MOLTENI, Auteur ; Marco BATTAGLIA, Auteur Année de publication : 2007 Article en page(s) : p.1147-1160 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The impact of socioeconomic status (SES) and genetic polymorphisms on individual differences for externalized behaviors have often been investigated separately in studies of children and adults. In a general population sample of 607 Italian preadolescents, we examined the independent and joint effects of SES and the dopamine receptor D4 (DRD4) and serotonin transporter linked promoter region (5-HTTLPR) polymorphisms upon rule-breaking and aggressive behaviors measured with the Child Behavior CheckList/6–18. We found evidence, which was based on both one locus and two-loci genotype analyses, that low SES and DRD4 long and 5-HTTLPR long alleles, both alone and in interaction, are associated with higher aggressive behavior scores. The effects were similar but more modest and limited to one locus genotype analyses for rule-breaking behavior. Consistent with studies that showed the effects of societal moderators on the heritability of externalized behaviors across different segments of the population, we suggest that diminished social constraints associated with low parental SES may act as enhancers of the genetic influence of specific DRD4 and 5-HTTLPR alleles over aggressive behaviors in preadolescence. En ligne : http://dx.doi.org/10.1017/s0954579407000594 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=182 [article] Socioeconomic status mediates the genetic contribution of the dopamine receptor D4 and serotonin transporter linked promoter region repeat polymorphisms to externalization in preadolescence [Texte imprimé et/ou numérique] / Maria NOBILE, Auteur ; Roberto GIORDA, Auteur ; Cecilia MARINO, Auteur ; Ombretta CARLET, Auteur ; Valentina PASTORE, Auteur ; Laura VANZIN, Auteur ; Monica BELLINA, Auteur ; Massimo MOLTENI, Auteur ; Marco BATTAGLIA, Auteur . - 2007 . - p.1147-1160. Langues : Anglais (eng) in Development and Psychopathology > 19-4 (Fall 2007) . - p.1147-1160 Index. décimale : PER Périodiques Résumé : The impact of socioeconomic status (SES) and genetic polymorphisms on individual differences for externalized behaviors have often been investigated separately in studies of children and adults. In a general population sample of 607 Italian preadolescents, we examined the independent and joint effects of SES and the dopamine receptor D4 (DRD4) and serotonin transporter linked promoter region (5-HTTLPR) polymorphisms upon rule-breaking and aggressive behaviors measured with the Child Behavior CheckList/6–18. We found evidence, which was based on both one locus and two-loci genotype analyses, that low SES and DRD4 long and 5-HTTLPR long alleles, both alone and in interaction, are associated with higher aggressive behavior scores. The effects were similar but more modest and limited to one locus genotype analyses for rule-breaking behavior. Consistent with studies that showed the effects of societal moderators on the heritability of externalized behaviors across different segments of the population, we suggest that diminished social constraints associated with low parental SES may act as enhancers of the genetic influence of specific DRD4 and 5-HTTLPR alleles over aggressive behaviors in preadolescence. En ligne : http://dx.doi.org/10.1017/s0954579407000594 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=182

The influence of family structure, the TPH2 G-703T and the 5-HTTLPR serotonergic genes upon affective problems in children aged 10–14 years / Maria NOBILE in Journal of Child Psychology and Psychiatry, 50-3 (March 2009)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 50-3 (March 2009) . - p.317-325 Titre : The influence of family structure, the TPH2 G-703T and the 5-HTTLPR serotonergic genes upon affective problems in children aged 10–14 years Type de document : Texte imprimé et/ou numérique Auteurs : Maria NOBILE, Auteur ; Roberto GIORDA, Auteur ; Cecilia MARINO, Auteur ; Ombretta CARLET, Auteur ; Laura VANZIN, Auteur ; Monica BELLINA, Auteur ; Massimo MOLTENI, Auteur ; Marco BATTAGLIA, Auteur ; Marianna RUSCONI, Auteur Année de publication : 2009 Article en page(s) : p.317-325 Langues : Anglais (eng) Mots-clés : Children depression gene–environment-interaction serotonin divorce family affective-disorders Index. décimale : PER Périodiques Résumé : Background: Both genetic and psychosocial risk factors influence the risk for depression in development. While the impacts of family structure and of serotonergic polymorphisms upon individual differences for affective problems have been investigated separately, they have never been considered together in a gene–environment interplay perspective. Methods: We examined the effects of family structure and two serotonergic polymorphisms (the TPH2 G-703T and the 5-HTTLPR) upon depressive symptoms assessed by the new CBCL/6-18 DSM-oriented Affective Problems scale in a general population sample of 607 Italian children aged 10–14 years. Results: Belonging to 'one-parent' families, the TPH2 G-703T 'G variant', and the 5-HTTLPR 'short' alleles were associated – both alone and in apparent gene-by-environment interaction – with higher Affective Problems scores. As predicted by quantitative genetics theory, both polymorphisms contributed with a small effect size, while 'family structure' had a moderate effect size. Conclusions: A putative hazard factor impinging on individual risk at the family-wide level, namely family structure, appears to act interactively with two pivotal serotonergic genes in heightening risk for Affective Problems. Although it remains to be demonstrated that belonging to a one- rather than a two-parent family has true environmental causal effects on Affective Problems, these data may contribute to identify/prevent risk for depression in childhood. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2008.01958.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=719 [article] The influence of family structure, the TPH2 G-703T and the 5-HTTLPR serotonergic genes upon affective problems in children aged 10–14 years [Texte imprimé et/ou numérique] / Maria NOBILE, Auteur ; Roberto GIORDA, Auteur ; Cecilia MARINO, Auteur ; Ombretta CARLET, Auteur ; Laura VANZIN, Auteur ; Monica BELLINA, Auteur ; Massimo MOLTENI, Auteur ; Marco BATTAGLIA, Auteur ; Marianna RUSCONI, Auteur . - 2009 . - p.317-325. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 50-3 (March 2009) . - p.317-325 Mots-clés : Children depression gene–environment-interaction serotonin divorce family affective-disorders Index. décimale : PER Périodiques Résumé : Background: Both genetic and psychosocial risk factors influence the risk for depression in development. While the impacts of family structure and of serotonergic polymorphisms upon individual differences for affective problems have been investigated separately, they have never been considered together in a gene–environment interplay perspective. Methods: We examined the effects of family structure and two serotonergic polymorphisms (the TPH2 G-703T and the 5-HTTLPR) upon depressive symptoms assessed by the new CBCL/6-18 DSM-oriented Affective Problems scale in a general population sample of 607 Italian children aged 10–14 years. Results: Belonging to 'one-parent' families, the TPH2 G-703T 'G variant', and the 5-HTTLPR 'short' alleles were associated – both alone and in apparent gene-by-environment interaction – with higher Affective Problems scores. As predicted by quantitative genetics theory, both polymorphisms contributed with a small effect size, while 'family structure' had a moderate effect size. Conclusions: A putative hazard factor impinging on individual risk at the family-wide level, namely family structure, appears to act interactively with two pivotal serotonergic genes in heightening risk for Affective Problems. Although it remains to be demonstrated that belonging to a one- rather than a two-parent family has true environmental causal effects on Affective Problems, these data may contribute to identify/prevent risk for depression in childhood. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2008.01958.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=719

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