Association of subcortical gray-matter volumes with life-course-persistent antisocial behavior in a population-representative longitudinal birth cohort / Christina O. CARLISI in Development and Psychopathology, 34-5 (December 2022)  

Public ISBD [article]  inDevelopment and Psychopathology > 34-5 (December 2022) . - p.2012-2022 Titre : Association of subcortical gray-matter volumes with life-course-persistent antisocial behavior in a population-representative longitudinal birth cohort Type de document : texte imprimé Auteurs : Christina O. CARLISI, Auteur ; Terrie E. MOFFITT, Auteur ; Annchen R. KNODT, Auteur ; Honalee HARRINGTON, Auteur ; Stéphanie LANGEVIN, Auteur ; David IRELAND, Auteur ; Tracy R. MELZER, Auteur ; Richie POULTON, Auteur ; Sandhya RAMRAKHA, Auteur ; Avshalom CASPI, Auteur ; Ahmad R. HARIRI, Auteur ; Essi VIDING, Auteur Article en page(s) : p.2012-2022 Langues : Anglais (eng) Mots-clés : antisocial behavior  conduct disorder  development  longitudinal  structural MRI Index. décimale : PER Périodiques Résumé : Neuropsychological evidence supports the developmental taxonomy theory of antisocial behavior, suggesting that abnormal brain development distinguishes life-course-persistent from adolescence-limited antisocial behavior. Recent neuroimaging work confirmed that prospectively-measured life-course-persistent antisocial behavior is associated with differences in cortical brain structure. Whether this extends to subcortical brain structures remains uninvestigated. This study compared subcortical gray-matter volumes between 672 members of the Dunedin Study previously defined as exhibiting life-course-persistent, adolescence-limited or low-level antisocial behavior based on repeated assessments at ages 7 “26 years. Gray-matter volumes of 10 subcortical structures were compared across groups. The life-course-persistent group had lower volumes of amygdala, brain stem, cerebellum, hippocampus, pallidum, thalamus, and ventral diencephalon compared to the low-antisocial group. Differences between life-course-persistent and adolescence-limited individuals were comparable in effect size to differences between life-course-persistent and low-antisocial individuals, but were not statistically significant due to less statistical power. Gray-matter volumes in adolescence-limited individuals were near the norm in this population-representative cohort and similar to volumes in low-antisocial individuals. Although this study could not establish causal links between brain volume and antisocial behavior, it constitutes new biological evidence that all people with antisocial behavior are not the same, supporting a need for greater developmental and diagnostic precision in clinical, forensic, and policy-based interventions. En ligne : http://dx.doi.org/10.1017/S0954579421000377 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=492 [article] Association of subcortical gray-matter volumes with life-course-persistent antisocial behavior in a population-representative longitudinal birth cohort [texte imprimé] / Christina O. CARLISI, Auteur ; Terrie E. MOFFITT, Auteur ; Annchen R. KNODT, Auteur ; Honalee HARRINGTON, Auteur ; Stéphanie LANGEVIN, Auteur ; David IRELAND, Auteur ; Tracy R. MELZER, Auteur ; Richie POULTON, Auteur ; Sandhya RAMRAKHA, Auteur ; Avshalom CASPI, Auteur ; Ahmad R. HARIRI, Auteur ; Essi VIDING, Auteur . - p.2012-2022. Langues : Anglais (eng) in Development and Psychopathology > 34-5 (December 2022) . - p.2012-2022 Mots-clés : antisocial behavior  conduct disorder  development  longitudinal  structural MRI Index. décimale : PER Périodiques Résumé : Neuropsychological evidence supports the developmental taxonomy theory of antisocial behavior, suggesting that abnormal brain development distinguishes life-course-persistent from adolescence-limited antisocial behavior. Recent neuroimaging work confirmed that prospectively-measured life-course-persistent antisocial behavior is associated with differences in cortical brain structure. Whether this extends to subcortical brain structures remains uninvestigated. This study compared subcortical gray-matter volumes between 672 members of the Dunedin Study previously defined as exhibiting life-course-persistent, adolescence-limited or low-level antisocial behavior based on repeated assessments at ages 7 “26 years. Gray-matter volumes of 10 subcortical structures were compared across groups. The life-course-persistent group had lower volumes of amygdala, brain stem, cerebellum, hippocampus, pallidum, thalamus, and ventral diencephalon compared to the low-antisocial group. Differences between life-course-persistent and adolescence-limited individuals were comparable in effect size to differences between life-course-persistent and low-antisocial individuals, but were not statistically significant due to less statistical power. Gray-matter volumes in adolescence-limited individuals were near the norm in this population-representative cohort and similar to volumes in low-antisocial individuals. Although this study could not establish causal links between brain volume and antisocial behavior, it constitutes new biological evidence that all people with antisocial behavior are not the same, supporting a need for greater developmental and diagnostic precision in clinical, forensic, and policy-based interventions. En ligne : http://dx.doi.org/10.1017/S0954579421000377 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=492

Childhood sexual abuse and pervasive problems across multiple life domains: Findings from a five-decade study / Hayley GUINEY in Development and Psychopathology, 36-1 (February 2024)  

Public ISBD [article]  inDevelopment and Psychopathology > 36-1 (February 2024) . - p.219-235 Titre : Childhood sexual abuse and pervasive problems across multiple life domains: Findings from a five-decade study Type de document : texte imprimé Auteurs : Hayley GUINEY, Auteur ; Avshalom CASPI, Auteur ; Antony AMBLER, Auteur ; Jay BELSKY, Auteur ; Jesse KOKAUA, Auteur ; Jonathan M. BROADBENT, Auteur ; Kirsten CHEYNE, Auteur ; Nigel DICKSON, Auteur ; Robert J. HANCOX, Auteur ; Honalee HARRINGTON, Auteur ; Sean HOGAN, Auteur ; Sandhya RAMRAKHA, Auteur ; Antoinette RIGHARTS, Auteur ; W. MURRAY THOMSON, Auteur ; Terrie E. MOFFITT, Auteur ; Richie POULTON, Auteur Article en page(s) : p.219-235 Langues : Anglais (eng) Mots-clés : child sexual abuseconsequences  long-term outcomes  longitudinal  multi-morbidity Index. décimale : PER Périodiques Résumé : The aim of this study was to use longitudinal population-based data to examine the associations between childhood sexual abuse (CSA) and risk for adverse outcomes in multiple life domains across adulthood. In 937 individuals followed from birth to age 45y, we assessed associations between CSA (retrospectively reported at age 26y) and the experience of 22 adverse outcomes in seven domains (physical, mental, sexual, interpersonal, economic, antisocial, multi-domain) from young adulthood to midlife (26 to 45y). Analyses controlled for sex, socioeconomic status, prospectively reported child harm and household dysfunction adverse childhood experiences, and adult sexual assault, and considered different definitions of CSA. After adjusting for confounders, CSA survivors were more likely than their peers to experience internalizing, externalizing, and thought disorders, suicide attempts, health risk behaviors, systemic inflammation, poor oral health, sexually transmitted diseases, high-conflict relationships, benefit use, financial difficulties, antisocial behavior, and cumulative problems across multiple domains in adulthood. In sum, CSA was associated with multiple persistent problems across adulthood, even after adjusting for confounding life stressors, and the risk for particular problems incremented with CSA severity. The higher risk for most specific problems was small to moderate, but the cumulative long-term effects across multiple domains reflect considerable individual and societal burden. En ligne : https://dx.doi.org/10.1017/S0954579422001146 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=523 [article] Childhood sexual abuse and pervasive problems across multiple life domains: Findings from a five-decade study [texte imprimé] / Hayley GUINEY, Auteur ; Avshalom CASPI, Auteur ; Antony AMBLER, Auteur ; Jay BELSKY, Auteur ; Jesse KOKAUA, Auteur ; Jonathan M. BROADBENT, Auteur ; Kirsten CHEYNE, Auteur ; Nigel DICKSON, Auteur ; Robert J. HANCOX, Auteur ; Honalee HARRINGTON, Auteur ; Sean HOGAN, Auteur ; Sandhya RAMRAKHA, Auteur ; Antoinette RIGHARTS, Auteur ; W. MURRAY THOMSON, Auteur ; Terrie E. MOFFITT, Auteur ; Richie POULTON, Auteur . - p.219-235. Langues : Anglais (eng) in Development and Psychopathology > 36-1 (February 2024) . - p.219-235 Mots-clés : child sexual abuseconsequences  long-term outcomes  longitudinal  multi-morbidity Index. décimale : PER Périodiques Résumé : The aim of this study was to use longitudinal population-based data to examine the associations between childhood sexual abuse (CSA) and risk for adverse outcomes in multiple life domains across adulthood. In 937 individuals followed from birth to age 45y, we assessed associations between CSA (retrospectively reported at age 26y) and the experience of 22 adverse outcomes in seven domains (physical, mental, sexual, interpersonal, economic, antisocial, multi-domain) from young adulthood to midlife (26 to 45y). Analyses controlled for sex, socioeconomic status, prospectively reported child harm and household dysfunction adverse childhood experiences, and adult sexual assault, and considered different definitions of CSA. After adjusting for confounders, CSA survivors were more likely than their peers to experience internalizing, externalizing, and thought disorders, suicide attempts, health risk behaviors, systemic inflammation, poor oral health, sexually transmitted diseases, high-conflict relationships, benefit use, financial difficulties, antisocial behavior, and cumulative problems across multiple domains in adulthood. In sum, CSA was associated with multiple persistent problems across adulthood, even after adjusting for confounding life stressors, and the risk for particular problems incremented with CSA severity. The higher risk for most specific problems was small to moderate, but the cumulative long-term effects across multiple domains reflect considerable individual and societal burden. En ligne : https://dx.doi.org/10.1017/S0954579422001146 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=523

Cumulative childhood risk is associated with a new measure of chronic inflammation in adulthood / Line Jee Hartmann RASMUSSEN in Journal of Child Psychology and Psychiatry, 60-2 (February 2019)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 60-2 (February 2019) . - p.199-208 Titre : Cumulative childhood risk is associated with a new measure of chronic inflammation in adulthood Type de document : texte imprimé Auteurs : Line Jee Hartmann RASMUSSEN, Auteur ; Terrie E. MOFFITT, Auteur ; Jesper EUGEN-OLSEN, Auteur ; Daniel W. BELSKY, Auteur ; Andrea DANESE, Auteur ; Honalee HARRINGTON, Auteur ; Renate HOUTS, Auteur ; Richie POULTON, Auteur ; Karen SUGDEN, Auteur ; Benjamin S. WILLIAMS, Auteur ; Avshalom CASPI, Auteur Article en page(s) : p.199-208 Langues : Anglais (eng) Mots-clés : Adverse childhood experiences  inflammation  physical health  risk factors  self-control Index. décimale : PER Périodiques Résumé : BACKGROUND: Childhood risk factors are associated with elevated inflammatory biomarkers in adulthood, but it is unknown whether these risk factors are associated with increased adult levels of the chronic inflammation marker soluble urokinase plasminogen activator receptor (suPAR). We aimed to test the hypothesis that childhood exposure to risk factors for adult disease is associated with elevated suPAR in adulthood and to compare suPAR with the oft-reported inflammatory biomarker C-reactive protein (CRP). METHODS: Prospective study of a population-representative 1972-1973 birth cohort; the Dunedin Multidisciplinary Health and Development Study observed participants to age 38 years. Main childhood predictors were poor health, socioeconomic disadvantage, adverse childhood experiences (ACEs), low IQ, and poor self-control. Main adult outcomes were adulthood inflammation measured as suPAR and high-sensitivity CRP (hsCRP). RESULTS: Participants with available plasma samples at age 38 were included (N = 837, 50.5% male). suPAR (mean 2.40 ng/ml; SD 0.91) was positively correlated with hsCRP (r 0.15, p < .001). After controlling for sex, body mass index (BMI), and smoking, children who experienced more ACEs, lower IQ, or had poorer self-control showed elevated adult suPAR. When the five childhood risks were aggregated into a Cumulative Childhood Risk index, and controlling for sex, BMI, and smoking, Cumulative Childhood Risk was associated with higher suPAR (b 0.10; SE 0.03; p = .002). Cumulative Childhood Risk predicted elevated suPAR, after controlling for hsCRP (b 0.18; SE 0.03; p < .001). CONCLUSIONS: Exposure to more childhood risk factors was associated with higher suPAR levels, independent of CRP. suPAR is a useful addition to studies connecting childhood risk to adult inflammatory burden. En ligne : http://dx.doi.org/10.1111/jcpp.12928 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=381 [article] Cumulative childhood risk is associated with a new measure of chronic inflammation in adulthood [texte imprimé] / Line Jee Hartmann RASMUSSEN, Auteur ; Terrie E. MOFFITT, Auteur ; Jesper EUGEN-OLSEN, Auteur ; Daniel W. BELSKY, Auteur ; Andrea DANESE, Auteur ; Honalee HARRINGTON, Auteur ; Renate HOUTS, Auteur ; Richie POULTON, Auteur ; Karen SUGDEN, Auteur ; Benjamin S. WILLIAMS, Auteur ; Avshalom CASPI, Auteur . - p.199-208. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 60-2 (February 2019) . - p.199-208 Mots-clés : Adverse childhood experiences  inflammation  physical health  risk factors  self-control Index. décimale : PER Périodiques Résumé : BACKGROUND: Childhood risk factors are associated with elevated inflammatory biomarkers in adulthood, but it is unknown whether these risk factors are associated with increased adult levels of the chronic inflammation marker soluble urokinase plasminogen activator receptor (suPAR). We aimed to test the hypothesis that childhood exposure to risk factors for adult disease is associated with elevated suPAR in adulthood and to compare suPAR with the oft-reported inflammatory biomarker C-reactive protein (CRP). METHODS: Prospective study of a population-representative 1972-1973 birth cohort; the Dunedin Multidisciplinary Health and Development Study observed participants to age 38 years. Main childhood predictors were poor health, socioeconomic disadvantage, adverse childhood experiences (ACEs), low IQ, and poor self-control. Main adult outcomes were adulthood inflammation measured as suPAR and high-sensitivity CRP (hsCRP). RESULTS: Participants with available plasma samples at age 38 were included (N = 837, 50.5% male). suPAR (mean 2.40 ng/ml; SD 0.91) was positively correlated with hsCRP (r 0.15, p < .001). After controlling for sex, body mass index (BMI), and smoking, children who experienced more ACEs, lower IQ, or had poorer self-control showed elevated adult suPAR. When the five childhood risks were aggregated into a Cumulative Childhood Risk index, and controlling for sex, BMI, and smoking, Cumulative Childhood Risk was associated with higher suPAR (b 0.10; SE 0.03; p = .002). Cumulative Childhood Risk predicted elevated suPAR, after controlling for hsCRP (b 0.18; SE 0.03; p < .001). CONCLUSIONS: Exposure to more childhood risk factors was associated with higher suPAR levels, independent of CRP. suPAR is a useful addition to studies connecting childhood risk to adult inflammatory burden. En ligne : http://dx.doi.org/10.1111/jcpp.12928 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=381

Female and male antisocial trajectories: From childhood origins to adult outcomes / Candice L. ODGERS in Development and Psychopathology, 20-2 (Spring 2008)  

Public ISBD [article]  inDevelopment and Psychopathology > 20-2 (Spring 2008) . - p.673-716 Titre : Female and male antisocial trajectories: From childhood origins to adult outcomes Type de document : texte imprimé Auteurs : Candice L. ODGERS, Auteur ; Terrie E. MOFFITT, Auteur ; Malcolm R. SEARS, Auteur ; Richie POULTON, Auteur ; Honalee HARRINGTON, Auteur ; Robert J. HANCOX, Auteur ; Nigel DICKSON, Auteur ; Jonathan M. BROADBENT, Auteur ; Avshalom CASPI, Auteur ; W. MURRAY THOMSON, Auteur Année de publication : 2008 Article en page(s) : p.673-716 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : This article reports on the childhood origins and adult outcomes of female versus male antisocial behavior trajectories in the Dunedin longitudinal study. Four antisocial behavior trajectory groups were identified among females and males using general growth mixture modeling and included life-course persistent (LCP), adolescent-onset, childhood-limited, and low trajectory groups. During childhood, both LCP females and males were characterized by social, familial and neurodevelopmental risk factors, whereas those on the adolescent-onset pathway were not. At age 32, women and men on the LCP pathway were engaging in serious violence and experiencing significant mental health, physical health, and economic problems. Females and males on the adolescent-onset pathway were also experiencing difficulties at age 32, although to a lesser extent. Although more males than females followed the LCP trajectory, findings support similarities across gender with respect to developmental trajectories of antisocial behavior and their associated childhood origins and adult consequences. Implications for theory, research, and practice are discussed. En ligne : http://dx.doi.org/10.1017/s0954579408000333 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=413 [article] Female and male antisocial trajectories: From childhood origins to adult outcomes [texte imprimé] / Candice L. ODGERS, Auteur ; Terrie E. MOFFITT, Auteur ; Malcolm R. SEARS, Auteur ; Richie POULTON, Auteur ; Honalee HARRINGTON, Auteur ; Robert J. HANCOX, Auteur ; Nigel DICKSON, Auteur ; Jonathan M. BROADBENT, Auteur ; Avshalom CASPI, Auteur ; W. MURRAY THOMSON, Auteur . - 2008 . - p.673-716. Langues : Anglais (eng) in Development and Psychopathology > 20-2 (Spring 2008) . - p.673-716 Index. décimale : PER Périodiques Résumé : This article reports on the childhood origins and adult outcomes of female versus male antisocial behavior trajectories in the Dunedin longitudinal study. Four antisocial behavior trajectory groups were identified among females and males using general growth mixture modeling and included life-course persistent (LCP), adolescent-onset, childhood-limited, and low trajectory groups. During childhood, both LCP females and males were characterized by social, familial and neurodevelopmental risk factors, whereas those on the adolescent-onset pathway were not. At age 32, women and men on the LCP pathway were engaging in serious violence and experiencing significant mental health, physical health, and economic problems. Females and males on the adolescent-onset pathway were also experiencing difficulties at age 32, although to a lesser extent. Although more males than females followed the LCP trajectory, findings support similarities across gender with respect to developmental trajectories of antisocial behavior and their associated childhood origins and adult consequences. Implications for theory, research, and practice are discussed. En ligne : http://dx.doi.org/10.1017/s0954579408000333 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=413

Is low cognitive functioning a predictor or consequence of major depressive disorder? A test in two longitudinal birth cohorts / Jonathan D. SCHAEFER in Development and Psychopathology, 37-5 (December 2025)  

Public ISBD [article]  inDevelopment and Psychopathology > 37-5 (December 2025) . - p.2251-2265 Titre : Is low cognitive functioning a predictor or consequence of major depressive disorder? A test in two longitudinal birth cohorts Type de document : texte imprimé Auteurs : Jonathan D. SCHAEFER, Auteur ; Matthew A. SCULT, Auteur ; Avshalom CASPI, Auteur ; Louise ARSENEAULT, Auteur ; Daniel W. BELSKY, Auteur ; Ahmad R. HARIRI, Auteur ; Honalee HARRINGTON, Auteur ; Renate HOUTS, Auteur ; Sandhya RAMRAKHA, Auteur ; Richie POULTON, Auteur ; Terrie E. MOFFITT, Auteur Article en page(s) : p.2251-2265 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Cognitive impairment has been identified as an important aspect of major depressive disorder (MDD). We tested two theories regarding the association between MDD and cognitive functioning using data from longitudinal cohort studies. One theory, the cognitive reserve hypothesis, suggests that higher cognitive ability in childhood decreases risk of later MDD. The second, the scarring hypothesis, instead suggests that MDD leads to persistent cognitive deficits following disorder onset. We tested both theories in the Dunedin Study, a population-representative cohort followed from birth to midlife and assessed repeatedly for both cognitive functioning and psychopathology. We also used data from the Environmental Risk Longitudinal Twin Study to test whether childhood cognitive functioning predicts future MDD risk independent of family-wide and genetic risk using a discordant twin design. Contrary to both hypotheses, we found that childhood cognitive functioning did not predict future risk of MDD, nor did study members with a past history of MDD show evidence of greater cognitive decline unless MDD was accompanied by other comorbid psychiatric conditions. Our results thus suggest that low cognitive functioning is related to comorbidity, but is neither an antecedent nor an enduring consequence of MDD. Future research may benefit from considering cognitive deficits that occur during depressive episodes from a transdiagnostic perspective. En ligne : https://dx.doi.org/10.1017/S095457941700164X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=572 [article] Is low cognitive functioning a predictor or consequence of major depressive disorder? A test in two longitudinal birth cohorts [texte imprimé] / Jonathan D. SCHAEFER, Auteur ; Matthew A. SCULT, Auteur ; Avshalom CASPI, Auteur ; Louise ARSENEAULT, Auteur ; Daniel W. BELSKY, Auteur ; Ahmad R. HARIRI, Auteur ; Honalee HARRINGTON, Auteur ; Renate HOUTS, Auteur ; Sandhya RAMRAKHA, Auteur ; Richie POULTON, Auteur ; Terrie E. MOFFITT, Auteur . - p.2251-2265. Langues : Anglais (eng) in Development and Psychopathology > 37-5 (December 2025) . - p.2251-2265 Index. décimale : PER Périodiques Résumé : Cognitive impairment has been identified as an important aspect of major depressive disorder (MDD). We tested two theories regarding the association between MDD and cognitive functioning using data from longitudinal cohort studies. One theory, the cognitive reserve hypothesis, suggests that higher cognitive ability in childhood decreases risk of later MDD. The second, the scarring hypothesis, instead suggests that MDD leads to persistent cognitive deficits following disorder onset. We tested both theories in the Dunedin Study, a population-representative cohort followed from birth to midlife and assessed repeatedly for both cognitive functioning and psychopathology. We also used data from the Environmental Risk Longitudinal Twin Study to test whether childhood cognitive functioning predicts future MDD risk independent of family-wide and genetic risk using a discordant twin design. Contrary to both hypotheses, we found that childhood cognitive functioning did not predict future risk of MDD, nor did study members with a past history of MDD show evidence of greater cognitive decline unless MDD was accompanied by other comorbid psychiatric conditions. Our results thus suggest that low cognitive functioning is related to comorbidity, but is neither an antecedent nor an enduring consequence of MDD. Future research may benefit from considering cognitive deficits that occur during depressive episodes from a transdiagnostic perspective. En ligne : https://dx.doi.org/10.1017/S095457941700164X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=572

Lest we forget: comparing retrospective and prospective assessments of adverse childhood experiences in the prediction of adult health / Aaron REUBEN in Journal of Child Psychology and Psychiatry, 57-10 (October 2016)  

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A polygenic score for age-at-first-birth predicts disinhibition / Leah S. RICHMOND-RAKERD in Journal of Child Psychology and Psychiatry, 61-12 (December 2020)  

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Prospective developmental subtypes of alcohol dependence from age 18 to 32 years: Implications for nosology, etiology, and intervention / Madeline H. MEIER in Development and Psychopathology, 25-3 (August 2013)  

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The high societal costs of childhood conduct problems: evidence from administrative records up to age 38 in a longitudinal birth cohort / Joshua G. RIVENBARK in Journal of Child Psychology and Psychiatry, 59-6 (June 2018)  

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