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Résultat de la recherche
10 recherche sur le mot-clé 'family history'




Association between family history of suicide attempt and neurocognitive functioning in community youth / Jason D. JONES in Journal of Child Psychology and Psychiatry, 62-1 (January 2021)
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Titre : Association between family history of suicide attempt and neurocognitive functioning in community youth Type de document : Texte imprimé et/ou numérique Auteurs : Jason D. JONES, Auteur ; Rhonda C. BOYD, Auteur ; Monica E. CALKINS, Auteur ; Tyler M. MOORE, Auteur ; Annisa AHMED, Auteur ; Ran BARZILAY, Auteur ; Tami D. BENTON, Auteur ; Raquel E. GUR, Auteur ; Ruben C. GUR, Auteur Article en page(s) : p.58-65 Langues : Anglais (eng) Mots-clés : Family history cognition endophenotype suicide Index. décimale : PER Périodiques Résumé : BACKGROUND: Suicidal behavior is highly familial. Neurocognitive deficits have been proposed as an endophenotype for suicide risk that may contribute to the familial transmission of suicide. Yet, there is a lack of research on the neurocognitive functioning of first-degree biological relatives of suicide attempters. The aim of the present study is to conduct the largest investigation to date of neurocognitive functioning in community youth with a family history of a fatal or nonfatal suicide attempt (FH). METHODS: Participants aged 8-21 years from the Philadelphia Neurodevelopmental Cohort completed detailed clinical and neurocognitive evaluations. A subsample of 501 participants with a FH was matched to a comparison group of 3,006 participants without a family history of suicide attempt (no-FH) on age, sex, race, and lifetime depression. RESULTS: After adjusting for multiple comparisons and including relevant clinical and demographic covariates, youth with a FH had significantly lower executive function factor scores (F[1,3432] = 6.63, p = .010) and performed worse on individual tests of attention (F[1,3382] = 7.08, p = .008) and language reasoning (F[1,3387] = 5.12, p = .024) than no-FH youth. CONCLUSIONS: Youth with a FH show small differences in executive function, attention, and language reasoning compared to youth without a FH. Further research is warranted to investigate neurocognitive functioning as an endophenotype for suicide risk. Implications for the prevention and treatment of suicidal behaviors are discussed. En ligne : http://dx.doi.org/10.1111/jcpp.13239 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=435
in Journal of Child Psychology and Psychiatry > 62-1 (January 2021) . - p.58-65[article] Association between family history of suicide attempt and neurocognitive functioning in community youth [Texte imprimé et/ou numérique] / Jason D. JONES, Auteur ; Rhonda C. BOYD, Auteur ; Monica E. CALKINS, Auteur ; Tyler M. MOORE, Auteur ; Annisa AHMED, Auteur ; Ran BARZILAY, Auteur ; Tami D. BENTON, Auteur ; Raquel E. GUR, Auteur ; Ruben C. GUR, Auteur . - p.58-65.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 62-1 (January 2021) . - p.58-65
Mots-clés : Family history cognition endophenotype suicide Index. décimale : PER Périodiques Résumé : BACKGROUND: Suicidal behavior is highly familial. Neurocognitive deficits have been proposed as an endophenotype for suicide risk that may contribute to the familial transmission of suicide. Yet, there is a lack of research on the neurocognitive functioning of first-degree biological relatives of suicide attempters. The aim of the present study is to conduct the largest investigation to date of neurocognitive functioning in community youth with a family history of a fatal or nonfatal suicide attempt (FH). METHODS: Participants aged 8-21 years from the Philadelphia Neurodevelopmental Cohort completed detailed clinical and neurocognitive evaluations. A subsample of 501 participants with a FH was matched to a comparison group of 3,006 participants without a family history of suicide attempt (no-FH) on age, sex, race, and lifetime depression. RESULTS: After adjusting for multiple comparisons and including relevant clinical and demographic covariates, youth with a FH had significantly lower executive function factor scores (F[1,3432] = 6.63, p = .010) and performed worse on individual tests of attention (F[1,3382] = 7.08, p = .008) and language reasoning (F[1,3387] = 5.12, p = .024) than no-FH youth. CONCLUSIONS: Youth with a FH show small differences in executive function, attention, and language reasoning compared to youth without a FH. Further research is warranted to investigate neurocognitive functioning as an endophenotype for suicide risk. Implications for the prevention and treatment of suicidal behaviors are discussed. En ligne : http://dx.doi.org/10.1111/jcpp.13239 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=435 Clinical utility of family history of depression for prognosis of adolescent depression severity and duration assessed with predictive modeling / Lisa S. GORHAM in Journal of Child Psychology and Psychiatry, 63-8 (August 2022)
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Titre : Clinical utility of family history of depression for prognosis of adolescent depression severity and duration assessed with predictive modeling Type de document : Texte imprimé et/ou numérique Auteurs : Lisa S. GORHAM, Auteur ; Neda SADEGHI, Auteur ; Lillian EISNER, Auteur ; Jeremy TAIGMAN, Auteur ; Katherine HAYNES, Auteur ; Karen QI, Auteur ; Christopher C. CAMP, Auteur ; Payton FORS, Auteur ; Diana RODRIGUEZ, Auteur ; Jerry MCGUIRE, Auteur ; Erin GARTH, Auteur ; Chana ENGEL, Auteur ; Mollie DAVIS, Auteur ; Kenneth TOWBIN, Auteur ; Argyris STRINGARIS, Auteur ; Dylan M. NIELSON, Auteur Article en page(s) : p.939-947 Langues : Anglais (eng) Mots-clés : Depression/psychology Humans Longitudinal Studies Prognosis Risk Factors Depression adolescence family history Index. décimale : PER Périodiques Résumé : BACKGROUND: Family history of depression (FHD) is a known risk factor for the new onset of depression. However, it is unclear if FHD is clinically useful for prognosis in adolescents with current, ongoing, or past depression. This preregistered study uses a longitudinal, multi-informant design to examine whether a child's FHD adds information about future depressive episodes and depression severity applying state-of-the-art predictive out-of-sample methodology. METHODS: We examined data in adolescents with current or past depression (age 11-17years) from the National Institute of Mental Health Characterization and Treatment of Adolescent Depression (CAT-D) study. We asked whether a history of depression in a first-degree relative was predictive of depressive episode duration (72 participants) and future depressive symptom severity in probands (129 participants, 1,439 total assessments). RESULTS: Family history of depression, while statistically associated with time spent depressed, did not improve predictions of time spent depressed, nor did it improve models of change in depression severity measured by self- or parent-report. CONCLUSIONS: Family history of depression does not improve the prediction of the course of depression in adolescents already diagnosed with depression. The difference between statistical association and predictive models highlights the importance of assessing predictive performance when evaluating questions of clinical utility. En ligne : http://dx.doi.org/10.1111/jcpp.13547 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=486
in Journal of Child Psychology and Psychiatry > 63-8 (August 2022) . - p.939-947[article] Clinical utility of family history of depression for prognosis of adolescent depression severity and duration assessed with predictive modeling [Texte imprimé et/ou numérique] / Lisa S. GORHAM, Auteur ; Neda SADEGHI, Auteur ; Lillian EISNER, Auteur ; Jeremy TAIGMAN, Auteur ; Katherine HAYNES, Auteur ; Karen QI, Auteur ; Christopher C. CAMP, Auteur ; Payton FORS, Auteur ; Diana RODRIGUEZ, Auteur ; Jerry MCGUIRE, Auteur ; Erin GARTH, Auteur ; Chana ENGEL, Auteur ; Mollie DAVIS, Auteur ; Kenneth TOWBIN, Auteur ; Argyris STRINGARIS, Auteur ; Dylan M. NIELSON, Auteur . - p.939-947.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 63-8 (August 2022) . - p.939-947
Mots-clés : Depression/psychology Humans Longitudinal Studies Prognosis Risk Factors Depression adolescence family history Index. décimale : PER Périodiques Résumé : BACKGROUND: Family history of depression (FHD) is a known risk factor for the new onset of depression. However, it is unclear if FHD is clinically useful for prognosis in adolescents with current, ongoing, or past depression. This preregistered study uses a longitudinal, multi-informant design to examine whether a child's FHD adds information about future depressive episodes and depression severity applying state-of-the-art predictive out-of-sample methodology. METHODS: We examined data in adolescents with current or past depression (age 11-17years) from the National Institute of Mental Health Characterization and Treatment of Adolescent Depression (CAT-D) study. We asked whether a history of depression in a first-degree relative was predictive of depressive episode duration (72 participants) and future depressive symptom severity in probands (129 participants, 1,439 total assessments). RESULTS: Family history of depression, while statistically associated with time spent depressed, did not improve predictions of time spent depressed, nor did it improve models of change in depression severity measured by self- or parent-report. CONCLUSIONS: Family history of depression does not improve the prediction of the course of depression in adolescents already diagnosed with depression. The difference between statistical association and predictive models highlights the importance of assessing predictive performance when evaluating questions of clinical utility. En ligne : http://dx.doi.org/10.1111/jcpp.13547 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=486 Evaluating the interrelations between the autism polygenic score and psychiatric family history in risk for autism / Diana SCHENDEL in Autism Research, 15-1 (January 2022)
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Titre : Evaluating the interrelations between the autism polygenic score and psychiatric family history in risk for autism Type de document : Texte imprimé et/ou numérique Auteurs : Diana SCHENDEL, Auteur ; T. MUNK LAURSEN, Auteur ; C. ALBIÑANA, Auteur ; B. VILHJALMSSON, Auteur ; Christine LADD-ACOSTA, Auteur ; M. D. FALLIN, Auteur ; Kelly S. BENKE, Auteur ; B. LEE, Auteur ; J. GROVE, Auteur ; Amy E. KALKBRENNER, Auteur ; L. EJLSKOV, Auteur ; D. HOUGAARD, Auteur ; Jonas BYBJERG-GRAUHOLM, Auteur ; M. BAEKVAD-HANSEN, Auteur ; A. D. BØRGLUM, Auteur ; T. WERGE, Auteur ; M. NORDENTOFT, Auteur ; P. B. MORTENSEN, Auteur ; E. AGERBO, Auteur Article en page(s) : p.171-182 Langues : Anglais (eng) Mots-clés : Autism Spectrum Disorder/genetics Autistic Disorder/genetics Case-Control Studies Humans Multifactorial Inheritance/genetics Risk Factors Siblings autism spectrum disorder case-control studies family history genetic risk factors polygenic risk score Index. décimale : PER Périodiques Résumé : Psychiatric family history or a high autism polygenic risk score (PRS) have been separately linked to autism spectrum disorder (ASD) risk. The study aimed to simultaneously consider psychiatric family history and individual autism genetic liability (PRS) in autism risk. We performed a case-control study of all Denmark singleton births, May 1981-December 2005, in Denmark at their first birthday and a known mother. Cases were diagnosed with ASD before 2013 and controls comprised a random sample of 30,000 births without ASD, excluding persons with non-Denmark-born parents, missing ASD PRS, non-European ancestry. Adjusted odds ratios (aOR) were estimated for ASD by PRS decile and by psychiatric history in parents or full siblings (8 mutually-exclusive categories) using logistic regression. Adjusted ASD PRS z-score least-squares means were estimated by psychiatric family history category. ASD risk (11,339 ASD cases; 20,175 controls) from ASD PRS was not substantially altered after accounting for psychiatric family history (e.g., ASD PRS 10th decile aOR: 2.35 (95% CI 2.11-2.63) before vs 2.11 (95% CI 1.91-2.40) after adjustment) nor from psychiatric family history after accounting for ASD PRS (e.g., ASD family history aOR: 6.73 (95% CI 5.89-7.68) before vs 6.32 (95% CI 5.53-7.22) after adjustment). ASD risk from ASD PRS varied slightly by psychiatric family history. While ASD risk from psychiatric family history was not accounted for by ASD PRS and vice versa, risk overlap between the two factors will likely increase as measures of genetic risk improve. The two factors are best viewed as complementary measures of family-based autism risk. LAY SUMMARY: Autism risk from a history of mental disorders in the immediate family was not explained by a measure of individual genetic risk (autism polygenic risk score) and vice versa. That is, genetic risk did not appear to overlap family history risk. As genetic measures for autism improve then the overlap in autism risk from family history versus genetic factors will likely increase, but further study may be needed to fully determine the components of risk and how they are inter-related between these key family factors. Meanwhile, the two factors may be best viewed as complementary measures of autism family-based risk. En ligne : http://dx.doi.org/10.1002/aur.2629 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=450
in Autism Research > 15-1 (January 2022) . - p.171-182[article] Evaluating the interrelations between the autism polygenic score and psychiatric family history in risk for autism [Texte imprimé et/ou numérique] / Diana SCHENDEL, Auteur ; T. MUNK LAURSEN, Auteur ; C. ALBIÑANA, Auteur ; B. VILHJALMSSON, Auteur ; Christine LADD-ACOSTA, Auteur ; M. D. FALLIN, Auteur ; Kelly S. BENKE, Auteur ; B. LEE, Auteur ; J. GROVE, Auteur ; Amy E. KALKBRENNER, Auteur ; L. EJLSKOV, Auteur ; D. HOUGAARD, Auteur ; Jonas BYBJERG-GRAUHOLM, Auteur ; M. BAEKVAD-HANSEN, Auteur ; A. D. BØRGLUM, Auteur ; T. WERGE, Auteur ; M. NORDENTOFT, Auteur ; P. B. MORTENSEN, Auteur ; E. AGERBO, Auteur . - p.171-182.
Langues : Anglais (eng)
in Autism Research > 15-1 (January 2022) . - p.171-182
Mots-clés : Autism Spectrum Disorder/genetics Autistic Disorder/genetics Case-Control Studies Humans Multifactorial Inheritance/genetics Risk Factors Siblings autism spectrum disorder case-control studies family history genetic risk factors polygenic risk score Index. décimale : PER Périodiques Résumé : Psychiatric family history or a high autism polygenic risk score (PRS) have been separately linked to autism spectrum disorder (ASD) risk. The study aimed to simultaneously consider psychiatric family history and individual autism genetic liability (PRS) in autism risk. We performed a case-control study of all Denmark singleton births, May 1981-December 2005, in Denmark at their first birthday and a known mother. Cases were diagnosed with ASD before 2013 and controls comprised a random sample of 30,000 births without ASD, excluding persons with non-Denmark-born parents, missing ASD PRS, non-European ancestry. Adjusted odds ratios (aOR) were estimated for ASD by PRS decile and by psychiatric history in parents or full siblings (8 mutually-exclusive categories) using logistic regression. Adjusted ASD PRS z-score least-squares means were estimated by psychiatric family history category. ASD risk (11,339 ASD cases; 20,175 controls) from ASD PRS was not substantially altered after accounting for psychiatric family history (e.g., ASD PRS 10th decile aOR: 2.35 (95% CI 2.11-2.63) before vs 2.11 (95% CI 1.91-2.40) after adjustment) nor from psychiatric family history after accounting for ASD PRS (e.g., ASD family history aOR: 6.73 (95% CI 5.89-7.68) before vs 6.32 (95% CI 5.53-7.22) after adjustment). ASD risk from ASD PRS varied slightly by psychiatric family history. While ASD risk from psychiatric family history was not accounted for by ASD PRS and vice versa, risk overlap between the two factors will likely increase as measures of genetic risk improve. The two factors are best viewed as complementary measures of family-based autism risk. LAY SUMMARY: Autism risk from a history of mental disorders in the immediate family was not explained by a measure of individual genetic risk (autism polygenic risk score) and vice versa. That is, genetic risk did not appear to overlap family history risk. As genetic measures for autism improve then the overlap in autism risk from family history versus genetic factors will likely increase, but further study may be needed to fully determine the components of risk and how they are inter-related between these key family factors. Meanwhile, the two factors may be best viewed as complementary measures of autism family-based risk. En ligne : http://dx.doi.org/10.1002/aur.2629 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=450 Relationship of family history conditions and early signs of autism spectrum disorder in low and high-risk infants / Maurice A. FELDMAN in Research in Autism Spectrum Disorders, 65 (September 2019)
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Titre : Relationship of family history conditions and early signs of autism spectrum disorder in low and high-risk infants Type de document : Texte imprimé et/ou numérique Auteurs : Maurice A. FELDMAN, Auteur ; Alicia AZZANO, Auteur ; Rebecca A. WARD, Auteur ; Melissa HUDSON, Auteur ; Calvin P. SJAARDA, Auteur ; Xudong LIU, Auteur Article en page(s) : p.25-33 Langues : Anglais (eng) Mots-clés : Infants Siblings Family history Prediction Early screening Index. décimale : PER Périodiques Résumé : Background Early identification and understanding of Autism Spectrum Disorder (ASD) could be facilitated by knowledge of family history of medical, developmental and psychiatric conditions associated with showing early signs of ASD. Method The current study used nonparametric analysis of covariance to compare the number of family history conditions n 69 high-risk (biological sibling with ASD) and 108 low-risk (no family history of ASD) infants. Spearman correlation was used to assess the relationship between family history of various conditions and early ASD signs as measured by an early screener, the Parent Observation of Early Markers Scale (POEMS). Results There were significantly more family history conditions in the families of the high-risk infants, and significant positive relationships between the number of family history problems and early markers of ASD in both groups. Conclusions The results suggest that family history conditions may play an important role in screening infants not yet diagnosed with ASD and reveal etiological pathways. En ligne : https://doi.org/10.1016/j.rasd.2019.05.002 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=401
in Research in Autism Spectrum Disorders > 65 (September 2019) . - p.25-33[article] Relationship of family history conditions and early signs of autism spectrum disorder in low and high-risk infants [Texte imprimé et/ou numérique] / Maurice A. FELDMAN, Auteur ; Alicia AZZANO, Auteur ; Rebecca A. WARD, Auteur ; Melissa HUDSON, Auteur ; Calvin P. SJAARDA, Auteur ; Xudong LIU, Auteur . - p.25-33.
Langues : Anglais (eng)
in Research in Autism Spectrum Disorders > 65 (September 2019) . - p.25-33
Mots-clés : Infants Siblings Family history Prediction Early screening Index. décimale : PER Périodiques Résumé : Background Early identification and understanding of Autism Spectrum Disorder (ASD) could be facilitated by knowledge of family history of medical, developmental and psychiatric conditions associated with showing early signs of ASD. Method The current study used nonparametric analysis of covariance to compare the number of family history conditions n 69 high-risk (biological sibling with ASD) and 108 low-risk (no family history of ASD) infants. Spearman correlation was used to assess the relationship between family history of various conditions and early ASD signs as measured by an early screener, the Parent Observation of Early Markers Scale (POEMS). Results There were significantly more family history conditions in the families of the high-risk infants, and significant positive relationships between the number of family history problems and early markers of ASD in both groups. Conclusions The results suggest that family history conditions may play an important role in screening infants not yet diagnosed with ASD and reveal etiological pathways. En ligne : https://doi.org/10.1016/j.rasd.2019.05.002 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=401 3-generation family histories of mental, neurologic, cardiometabolic, birth defect, asthma, allergy, and autoimmune conditions associated with autism: An open-source catalog of findings / Diana SCHENDEL in Autism Research, 17-10 (October 2024)
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Titre : 3-generation family histories of mental, neurologic, cardiometabolic, birth defect, asthma, allergy, and autoimmune conditions associated with autism: An open-source catalog of findings Type de document : Texte imprimé et/ou numérique Auteurs : Diana SCHENDEL, Auteur ; Linda EJLSKOV, Auteur ; Morten OVERGAARD, Auteur ; Zeal JINWALA, Auteur ; Viktor KIM, Auteur ; Erik PARNER, Auteur ; Amy E. KALKBRENNER, Auteur ; Christine LADD ACOSTA, Auteur ; M. Danielle FALLIN, Auteur ; Sherlly XIE, Auteur ; Preben Bo MORTENSEN, Auteur ; Brian K. LEE, Auteur Article en page(s) : p.2144-2155 Langues : Anglais (eng) Mots-clés : allergy asthma autism autoimmune birth defect cardiometabolic family history mental disorder neurologic Index. décimale : PER Périodiques Résumé : Abstract The relatively few conditions and family member types (e.g., sibling, parent) considered in investigations of family health history in autism spectrum disorder (ASD, or autism) limits understanding of the role of family history in autism etiology. For more comprehensive understanding and hypothesis-generation, we produced an open-source catalog of autism associations with family histories of mental, neurologic, cardiometabolic, birth defect, asthma, allergy, and autoimmune conditions. All live births in Denmark, 1980?2012, of Denmark-born parents (1,697,231 births), and their 3-generation family members were followed through April 10, 2017 for each of 90 diagnoses (including autism), emigration or death. Adjusted hazard ratios (aHR) were estimated via Cox regression for each diagnosis-family member type combination, adjusting for birth year, sex, birth weight, gestational age, parental ages at birth, and number of family member types of index person; aHRs also calculated for sex-specific co-occurrence of each disorder. We obtained 6462 individual family history aHRS across autism overall (26,840 autistic persons; 1.6% of births), by sex, and considering intellectual disability (ID); and 350 individual co-occurrence aHRS. Results are cataloged in interactive heat maps and down-loadable data files: https://ncrr-au.shinyapps.io/asd-riskatlas/ and interactive graphic summaries: https://public.tableau.com/app/profile/diana.schendel/viz/ASDPlots_16918786403110/e-Figure5. While primarily for reference material or use in other studies (e.g., meta-analyses), results revealed considerable breadth and variation in magnitude of familial health history associations with autism by type of condition, family member type, sex of the family member, side of the family, sex of the index person, and ID status, indicative of diverse genetic, familial, and nongenetic autism etiologic pathways. Careful attention to sources of autism likelihood in family health history, aided by our open data resource, may accelerate understanding of factors underlying neurodiversity. En ligne : https://doi.org/10.1002/aur.3232 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=536
in Autism Research > 17-10 (October 2024) . - p.2144-2155[article] 3-generation family histories of mental, neurologic, cardiometabolic, birth defect, asthma, allergy, and autoimmune conditions associated with autism: An open-source catalog of findings [Texte imprimé et/ou numérique] / Diana SCHENDEL, Auteur ; Linda EJLSKOV, Auteur ; Morten OVERGAARD, Auteur ; Zeal JINWALA, Auteur ; Viktor KIM, Auteur ; Erik PARNER, Auteur ; Amy E. KALKBRENNER, Auteur ; Christine LADD ACOSTA, Auteur ; M. Danielle FALLIN, Auteur ; Sherlly XIE, Auteur ; Preben Bo MORTENSEN, Auteur ; Brian K. LEE, Auteur . - p.2144-2155.
Langues : Anglais (eng)
in Autism Research > 17-10 (October 2024) . - p.2144-2155
Mots-clés : allergy asthma autism autoimmune birth defect cardiometabolic family history mental disorder neurologic Index. décimale : PER Périodiques Résumé : Abstract The relatively few conditions and family member types (e.g., sibling, parent) considered in investigations of family health history in autism spectrum disorder (ASD, or autism) limits understanding of the role of family history in autism etiology. For more comprehensive understanding and hypothesis-generation, we produced an open-source catalog of autism associations with family histories of mental, neurologic, cardiometabolic, birth defect, asthma, allergy, and autoimmune conditions. All live births in Denmark, 1980?2012, of Denmark-born parents (1,697,231 births), and their 3-generation family members were followed through April 10, 2017 for each of 90 diagnoses (including autism), emigration or death. Adjusted hazard ratios (aHR) were estimated via Cox regression for each diagnosis-family member type combination, adjusting for birth year, sex, birth weight, gestational age, parental ages at birth, and number of family member types of index person; aHRs also calculated for sex-specific co-occurrence of each disorder. We obtained 6462 individual family history aHRS across autism overall (26,840 autistic persons; 1.6% of births), by sex, and considering intellectual disability (ID); and 350 individual co-occurrence aHRS. Results are cataloged in interactive heat maps and down-loadable data files: https://ncrr-au.shinyapps.io/asd-riskatlas/ and interactive graphic summaries: https://public.tableau.com/app/profile/diana.schendel/viz/ASDPlots_16918786403110/e-Figure5. While primarily for reference material or use in other studies (e.g., meta-analyses), results revealed considerable breadth and variation in magnitude of familial health history associations with autism by type of condition, family member type, sex of the family member, side of the family, sex of the index person, and ID status, indicative of diverse genetic, familial, and nongenetic autism etiologic pathways. Careful attention to sources of autism likelihood in family health history, aided by our open data resource, may accelerate understanding of factors underlying neurodiversity. En ligne : https://doi.org/10.1002/aur.3232 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=536 Child and parental literacy levels within families with a history of dyslexia / Elsje VAN BERGEN in Journal of Child Psychology and Psychiatry, 53-1 (January 2012)
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PermalinkDo infant vocabulary skills predict school-age language and literacy outcomes? / Fiona J. DUFF in Journal of Child Psychology and Psychiatry, 56-8 (August 2015)
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PermalinkUnique prediction of developmental psychopathology from genetic and familial risk / Robert J. LOUGHNAN in Journal of Child Psychology and Psychiatry, 63-12 (December 2022)
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PermalinkParental psychopathology in families of children with attention-deficit/hyperactivity disorder and exposed to maternal smoking during pregnancy / Sarojini M. SENGUPTA in Journal of Child Psychology and Psychiatry, 56-2 (February 2015)
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PermalinkEarly-onset and recurrent depression in parents increases risk of intergenerational transmission to adolescent offspring / Sara R. JAFFEE in Journal of Child Psychology and Psychiatry, 62-8 (August 2021)
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