1. Bohm HV, Stewart MG. {{Brief report: on the concordance percentages for Autistic Spectrum Disorder of twins}}. {J Autism Dev Disord};2009 (May);39(5):806-808.
2. Casanova MF, El-Baz A, Mott M, Mannheim G, Hassan H, Fahmi R, Giedd J, Rumsey JM, Switala AE, Farag A. {{Reduced gyral window and corpus callosum size in autism: possible macroscopic correlates of a minicolumnopathy}}. {J Autism Dev Disord};2009 (May);39(5):751-764.
3. Groen WB, van Orsouw L, Huurne N, Swinkels S, van der Gaag RJ, Buitelaar JK, Zwiers MP. {{Intact spectral but abnormal temporal processing of auditory stimuli in autism}}. {J Autism Dev Disord};2009 (May);39(5):742-750.
4. Hadjikhani N, Joseph RM, Manoach DS, Naik P, Snyder J, Dominick K, Hoge R, Van den Stock J, Tager Flusberg H, de Gelder B. {{Body expressions of emotion do not trigger fear contagion in autism spectrum disorder}}. {Soc Cogn Affect Neurosci};2009 (Mar);4(1):70-78.
Although there is evidence of emotion perception deficits in autism spectrum disorder (ASD), research on this topic has been mostly confined to perception of emotions in faces. Using behavioral measures and 3T functional magnetic resonance imaging (fMRI), we examined whether such deficits extend to the perception of bodily expressed emotions. We found that individuals with ASD, in contrast to neurotypical (NT) individuals, did not exhibit a differential pattern of brain activation to bodies expressing fear as compared with emotionally neutral bodies. ASD and NT individuals showed similar patterns of activation in response to bodies engaged in emotionally neutral actions, with the exception of decreased activation in the inferior frontal cortex and the anterior insula in ASD. We discuss these findings in relation to possible abnormalities in a network of cortical and subcortical mechanisms involved in social orienting and emotion contagion. Our data suggest that emotion perception deficits in ASD may be due to compromised processing of the emotional component of observed actions.
5. Handen BL, Melmed RD, Hansen RL, Aman MG, Burnham DL, Bruss JB, McDougle CJ. {{A double-blind, placebo-controlled trial of oral human immunoglobulin for gastrointestinal dysfunction in children with autistic disorder}}. {J Autism Dev Disord};2009 (May);39(5):796-805.
6. Mooney EL, Gray KM, Tonge BJ, Sweeney DJ, Taffe JR. {{Factor analytic study of repetitive behaviours in young children with Pervasive Developmental Disorders}}. {J Autism Dev Disord};2009 (May);39(5):765-774.
7. Schwartzman F, Vitolo MR, Schwartzman JS, Morais MB. {{Eating practices, nutritional status and constipation in patients with Rett syndrome}}. {Arq Gastroenterol};2008 (Oct-Dec);45(4):284-289.
BACKGROUND: Disturbance in chewing, swallowing and digestive motility may predispose to feeding and nutritional abnormalities in patients with Rett syndrome. OBJECTIVE: To evaluate the dietary habits, nutritional status and the prevalence of constipation in patients with classical Rett syndrome. METHODS: Twenty seven female patients between the ages of 2.6 and 21.8 years were studied. The following parameters were evaluated: food register, weight, height and intestinal movement characteristics. Weight and height were compared with the National Center for Health Statistics standards. RESULTS: The inability to ingest solid foods was observed in 80.8% of the patients. A height-to-age deficit was observed in 13 (48.1%) of the girls, being more intense in patients at stage IV. Weight-for-height deficit was found in 10 (37.0%) patients, 15 (55.6%) showed normal weight and 2 (7.4%) were overweight for their height. The median ingestion of energy, according to weight-for-height, was equal to 106.6%. Insufficient iron ingestion was observed in 63.0% and insufficient calcium in 55.6% of the patients. Constipation was verified in 74.1% of the patients and did not show a relationship with the quantity of fiber in the diet. CONCLUSION: Various nutritional problems, as well as, intestinal constipation were observed in these patients with Rett syndrome, and they must be considered in the multidisciplinary therapeutic planning of these individuals.
8. Singh J, Illes J, Lazzeroni L, Hallmayer J. {{Trends in US autism research funding}}. {J Autism Dev Disord};2009 (May);39(5):788-795.
9. Wallace GL, Anderson M, Happe F. {{Brief report: information processing speed is intact in autism but not correlated with measured intelligence}}. {J Autism Dev Disord};2009 (May);39(5):809-814.
