Pubmed (TDAH) du 17/03/26
1. Hashizoe K, Nakai A, Nobusako S. Impaired motor imagery in children with developmental coordination disorder: task-specific deficits and links to ADHD and ASD traits. Exp Brain Res. 2026; 244(4).
Lien vers le texte intégral (Open Access ou abonnement)
2. Hueg TK, Lim YH, Holmboe SA, Micali N, Juul A. Precocious Puberty and Risk of Psychiatric Disorders: A Nationwide Cohort Study Using Prospective Registry Data. J Clin Endocrinol Metab. 2026; 111(4): e1088-e96.
CONTEXT: Early menarche within the general population has previously been linked to increased risk of mental health disorders, and such associations are likely to be even stronger in children diagnosed with extremely early puberty (precocious puberty); however, information is sparse. OBJECTIVE: Investigate the association between precocious puberty and psychiatric disorders. DESIGN: A large cohort study included individuals in the National Danish Patient Registry with a diagnosis of precocious puberty from January 1, 1995, to December 31, 2020 (n = 9315, 11% boys), comprising patients registered with an ICD-10 diagnosis of central precocious puberty (n = 2955), unspecific premature puberty (n = 3948), premature thelarche (n = 937), and premature adrenarche (n = 1475). All cases were matched with 5 randomly selected referents from the background population (n = 46 566) according to age, sex, and calendar time (at diagnosis). Incident psychiatric disorders were identified in national health registries (average follow-up time of 7.9 years). RESULTS: Precocious puberty (all cases) was associated with a statistically significant increased risk of attention-deficit/hyperactivity disorder (ADHD) (hazard ratio [HR]: 1.58; 95% CI: 1.36-1.84), anxiety (HR: 1.60; 95% CI: 1.48-1.74), autism (HR: 1.94; 95% CI: 1.67-2.25), depression (HR: 1.41; 95% CI: 1.29-1.53), eating disorders (HR: 1.23; 95% CI: 1.03-1.47), and schizophrenia (HR: 1.72; 95% CI: 1.40-2.10) compared to matched referents. Similar results were observed when stratified by sex, but with greatest effect size among boys. The associations were even stronger among individuals with central precocious puberty. CONCLUSION: Our large register-based cohort study highlights a markedly increased risk of psychiatric disorders among individuals with precocious puberty.
Lien vers le texte intégral (Open Access ou abonnement)
3. Irmak DE, Bumin G. The Turkish adaptation of the Child Occupational Self-Assessment Scale (COSA) and its psychometric properties in children with attention-deficit/hyperactivity disorder. BMC Psychol. 2026.
Lien vers le texte intégral (Open Access ou abonnement)
4. Osborne JB, Sellers J, Zhang H, Wang S, Liberatore S, Sripada C, Shah P, Jonides J. Delayed goal-directed processing underlies inhibitory control challenges in adult ADHD. Sci Rep. 2026.
Lien vers le texte intégral (Open Access ou abonnement)
5. Thomson P, Rakesh D. Editorial: ADHD persistence – the interplay of genes, socioeconomic context, and symptom domains over development. J Child Psychol Psychiatry. 2026.
Persistence of childhood ADHD symptoms into adolescence and adulthood is common. However, persistence is not simply a continuation of early high severity ADHD. Rather, it is the product of influences from individual-level genetic liability, one’s environmental context, and their interplay. The field has often focused on cross-sectional ADHD severity and genetic load. However, environments – such as one’s socioeconomic context – exert their own influence over development independently of genetics, as well as modulate genetic influences. Importantly, these genetic and environmental effects vary significantly between inattention and hyperactivity/impulsivity symptom domains, emphasizing the need to consider these domains separately when studying persistence risk. This article outlines a unifying persistence framework reflecting the changing contributions of genes, environmental context, and their interaction over time, offering a path to a more complete understanding of risk for symptom persistence.
