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Auteur Edmund J. S. SONUGA-BARKE |
Documents disponibles écrits par cet auteur (76)
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Delay aversion in attention deficit/hyperactivity disorder is mediated by amygdala and prefrontal cortex hyper-activation / J. VAN DESSEL in Journal of Child Psychology and Psychiatry, 59-8 (August 2018)
[article]
Titre : Delay aversion in attention deficit/hyperactivity disorder is mediated by amygdala and prefrontal cortex hyper-activation Type de document : Texte imprimé et/ou numérique Auteurs : J. VAN DESSEL, Auteur ; Edmund J. S. SONUGA-BARKE, Auteur ; G. MIES, Auteur ; J. LEMIERE, Auteur ; S. VAN DER OORD, Auteur ; S. MORSINK, Auteur ; M. DANCKAERTS, Auteur Article en page(s) : p.888-899 Langues : Anglais (eng) Mots-clés : fMRI Attention deficit/hyperactivity disorder affective network amygdala delay aversion dorsolateral prefrontal cortex Index. décimale : PER Périodiques Résumé : BACKGROUND: Experimental research supports delay aversion as a motivational feature of attention deficit/hyperactivity disorder (ADHD). To investigate the neurobiology of delay aversion in ADHD, this study examined whether adolescents with ADHD display an unusually strong activation in affective brain regions in response to cues predicting forthcoming delay and whether these effects are (a) delay-dose dependent and (b) statistically mediate the association between ADHD and self-reported delay aversion. METHODS: Twenty-nine right-handed male adolescents with combined type ADHD and 32 typically developing controls (ages 10-18 years) performed a reaction time task in an MRI scanner. Pretarget cues indicated delay-related response consequences. One indicated that delay would follow the response irrespective of response speed (CERTAIN DELAY), a second that delay would only follow if the response was too slow (CONDITIONAL DELAY), and a third that no delay would follow the response whatever its speed (NO DELAY). Delay levels were 2, 6, or 14 s. Participants also rated their own delay aversion in everyday life. RESULTS: Individuals with ADHD rated themselves as more delay averse than controls. Significantly greater activation to CERTAIN DELAY cues relative to NO DELAY cues was found in participants with ADHD compared to controls (bilaterally) in amygdala, anterior insula, temporal pole, dorsolateral prefrontal cortex (DLPFC), and ventromedial prefrontal cortex. Amygdala and DLPFC activation strength were strongly and delay-dose dependently correlated with delay aversion ratings, and statistically mediated the relationship between ADHD status and delay aversion. CONCLUSIONS: When presented with cues predicting impending delay, adolescents with ADHD, relative to controls, displayed a delay-related increase in activation in amygdala and DLPFC, regions known to be implicated in the processing of aversive events. Future studies should examine the specificity of these effects to delay aversion compared to aversive events in general. En ligne : http://dx.doi.org/10.1111/jcpp.12868 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=368
in Journal of Child Psychology and Psychiatry > 59-8 (August 2018) . - p.888-899[article] Delay aversion in attention deficit/hyperactivity disorder is mediated by amygdala and prefrontal cortex hyper-activation [Texte imprimé et/ou numérique] / J. VAN DESSEL, Auteur ; Edmund J. S. SONUGA-BARKE, Auteur ; G. MIES, Auteur ; J. LEMIERE, Auteur ; S. VAN DER OORD, Auteur ; S. MORSINK, Auteur ; M. DANCKAERTS, Auteur . - p.888-899.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 59-8 (August 2018) . - p.888-899
Mots-clés : fMRI Attention deficit/hyperactivity disorder affective network amygdala delay aversion dorsolateral prefrontal cortex Index. décimale : PER Périodiques Résumé : BACKGROUND: Experimental research supports delay aversion as a motivational feature of attention deficit/hyperactivity disorder (ADHD). To investigate the neurobiology of delay aversion in ADHD, this study examined whether adolescents with ADHD display an unusually strong activation in affective brain regions in response to cues predicting forthcoming delay and whether these effects are (a) delay-dose dependent and (b) statistically mediate the association between ADHD and self-reported delay aversion. METHODS: Twenty-nine right-handed male adolescents with combined type ADHD and 32 typically developing controls (ages 10-18 years) performed a reaction time task in an MRI scanner. Pretarget cues indicated delay-related response consequences. One indicated that delay would follow the response irrespective of response speed (CERTAIN DELAY), a second that delay would only follow if the response was too slow (CONDITIONAL DELAY), and a third that no delay would follow the response whatever its speed (NO DELAY). Delay levels were 2, 6, or 14 s. Participants also rated their own delay aversion in everyday life. RESULTS: Individuals with ADHD rated themselves as more delay averse than controls. Significantly greater activation to CERTAIN DELAY cues relative to NO DELAY cues was found in participants with ADHD compared to controls (bilaterally) in amygdala, anterior insula, temporal pole, dorsolateral prefrontal cortex (DLPFC), and ventromedial prefrontal cortex. Amygdala and DLPFC activation strength were strongly and delay-dose dependently correlated with delay aversion ratings, and statistically mediated the relationship between ADHD status and delay aversion. CONCLUSIONS: When presented with cues predicting impending delay, adolescents with ADHD, relative to controls, displayed a delay-related increase in activation in amygdala and DLPFC, regions known to be implicated in the processing of aversive events. Future studies should examine the specificity of these effects to delay aversion compared to aversive events in general. En ligne : http://dx.doi.org/10.1111/jcpp.12868 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=368 Developmental phenotypes and causal pathways in attention deficit/hyperactivity disorder: potential targets for early intervention? / Edmund J. S. SONUGA-BARKE in Journal of Child Psychology and Psychiatry, 51-4 (April 2010)
[article]
Titre : Developmental phenotypes and causal pathways in attention deficit/hyperactivity disorder: potential targets for early intervention? Type de document : Texte imprimé et/ou numérique Auteurs : Edmund J. S. SONUGA-BARKE, Auteur ; Jeffrey M. HALPERIN, Auteur Année de publication : 2010 Article en page(s) : p.368-389 Langues : Anglais (eng) Mots-clés : Attention-deficit/hyperactivity-disorder preschool early-intervention translational development treatment longitudinal-studies Index. décimale : PER Périodiques Résumé : Early intervention approaches have rarely been implemented for the prevention of attention deficit/hyperactivity disorder (ADHD). In this paper we explore whether such an approach may represent an important new direction for therapeutic innovation. We propose that such an approach is most likely to be of value when grounded in and informed by developmental models of the dynamic, complex and heterogeneous nature of the condition. First, we set out a rationale for early intervention grounded in the science of ADHD viewed through developmental models. Second, we re-examine the concept of disorder-onset from the perspective of developmental trajectories and phenotypes. Third, we examine potential causal pathways to ADHD with regard to originating risk, pathophysiological mediators, environmental moderators and developmental continuities. Finally, we explore the potential value of strategies for identifying young children at risk for ADHD, and implementing interventions in ways that can target these underlying pathogenic processes. The utility of such an approach represents an important area for future research but still requires 'proof of concept'. Therefore prior to widespread clinical implementation, far greater knowledge is required of (i) developmental pathways into ADHD, (ii) the value of identifying neuropsychological mediators of these pathways, and (iii) the extent to which targeting mediating mechanisms will improve treatment outcomes for children with ADHD. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02195.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=989
in Journal of Child Psychology and Psychiatry > 51-4 (April 2010) . - p.368-389[article] Developmental phenotypes and causal pathways in attention deficit/hyperactivity disorder: potential targets for early intervention? [Texte imprimé et/ou numérique] / Edmund J. S. SONUGA-BARKE, Auteur ; Jeffrey M. HALPERIN, Auteur . - 2010 . - p.368-389.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 51-4 (April 2010) . - p.368-389
Mots-clés : Attention-deficit/hyperactivity-disorder preschool early-intervention translational development treatment longitudinal-studies Index. décimale : PER Périodiques Résumé : Early intervention approaches have rarely been implemented for the prevention of attention deficit/hyperactivity disorder (ADHD). In this paper we explore whether such an approach may represent an important new direction for therapeutic innovation. We propose that such an approach is most likely to be of value when grounded in and informed by developmental models of the dynamic, complex and heterogeneous nature of the condition. First, we set out a rationale for early intervention grounded in the science of ADHD viewed through developmental models. Second, we re-examine the concept of disorder-onset from the perspective of developmental trajectories and phenotypes. Third, we examine potential causal pathways to ADHD with regard to originating risk, pathophysiological mediators, environmental moderators and developmental continuities. Finally, we explore the potential value of strategies for identifying young children at risk for ADHD, and implementing interventions in ways that can target these underlying pathogenic processes. The utility of such an approach represents an important area for future research but still requires 'proof of concept'. Therefore prior to widespread clinical implementation, far greater knowledge is required of (i) developmental pathways into ADHD, (ii) the value of identifying neuropsychological mediators of these pathways, and (iii) the extent to which targeting mediating mechanisms will improve treatment outcomes for children with ADHD. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02195.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=989 Do maternal attention-deficit/hyperactivity disorder symptoms exacerbate or ameliorate the negative effect of child attention-deficit/hyperactivity disorder symptoms on parenting? / Lamprini PSYCHOGIOU in Development and Psychopathology, 20-1 (Winter 2008)
[article]
Titre : Do maternal attention-deficit/hyperactivity disorder symptoms exacerbate or ameliorate the negative effect of child attention-deficit/hyperactivity disorder symptoms on parenting? Type de document : Texte imprimé et/ou numérique Auteurs : Lamprini PSYCHOGIOU, Auteur ; Edmund J. S. SONUGA-BARKE, Auteur ; Dave M. DALEY, Auteur ; Margaret J. THOMPSON, Auteur Année de publication : 2008 Article en page(s) : p.121-137 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The impact of similarity in parent and child characteristics on the quality of parenting is underresearched. The current study examined the interaction between mother and child attention-deficit/hyperactivity disorder (ADHD) symptoms on parenting. Two hypotheses were tested: the similarity-fit hypothesis, which predicted that parent and child similarity will improve parenting, and the similarity-misfit hypothesis, which predicted the opposite. Study 1 examined the associations between maternal and child ADHD symptoms and child-specific rearing attitudes of 95 mothers with school-aged children. In Study 2 this analysis was extended to more objective observer-rated mother–child interaction and maternal expressed emotion in 192 mothers of preschool children. Child ADHD symptoms were associated with negative maternal comments and maternal ADHD symptoms with negative expressed emotion. In both studies maternal ADHD symptoms appeared to ameliorate the effects of child ADHD symptoms on negative parenting. Parental response to children with high ADHD symptoms was more positive and affectionate when the mother also had high ADHD symptoms. The results support the similarity-fit hypothesis and highlight the importance of considering both child and maternal ADHD symptoms in studies of parenting. En ligne : http://dx.doi.org/10.1017/s0954579408000060 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=332
in Development and Psychopathology > 20-1 (Winter 2008) . - p.121-137[article] Do maternal attention-deficit/hyperactivity disorder symptoms exacerbate or ameliorate the negative effect of child attention-deficit/hyperactivity disorder symptoms on parenting? [Texte imprimé et/ou numérique] / Lamprini PSYCHOGIOU, Auteur ; Edmund J. S. SONUGA-BARKE, Auteur ; Dave M. DALEY, Auteur ; Margaret J. THOMPSON, Auteur . - 2008 . - p.121-137.
Langues : Anglais (eng)
in Development and Psychopathology > 20-1 (Winter 2008) . - p.121-137
Index. décimale : PER Périodiques Résumé : The impact of similarity in parent and child characteristics on the quality of parenting is underresearched. The current study examined the interaction between mother and child attention-deficit/hyperactivity disorder (ADHD) symptoms on parenting. Two hypotheses were tested: the similarity-fit hypothesis, which predicted that parent and child similarity will improve parenting, and the similarity-misfit hypothesis, which predicted the opposite. Study 1 examined the associations between maternal and child ADHD symptoms and child-specific rearing attitudes of 95 mothers with school-aged children. In Study 2 this analysis was extended to more objective observer-rated mother–child interaction and maternal expressed emotion in 192 mothers of preschool children. Child ADHD symptoms were associated with negative maternal comments and maternal ADHD symptoms with negative expressed emotion. In both studies maternal ADHD symptoms appeared to ameliorate the effects of child ADHD symptoms on negative parenting. Parental response to children with high ADHD symptoms was more positive and affectionate when the mother also had high ADHD symptoms. The results support the similarity-fit hypothesis and highlight the importance of considering both child and maternal ADHD symptoms in studies of parenting. En ligne : http://dx.doi.org/10.1017/s0954579408000060 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=332 Does comorbid anxiety counteract emotion recognition deficits in conduct disorder? / Roxanna M. L. SHORT in Journal of Child Psychology and Psychiatry, 57-8 (August 2016)
[article]
Titre : Does comorbid anxiety counteract emotion recognition deficits in conduct disorder? Type de document : Texte imprimé et/ou numérique Auteurs : Roxanna M. L. SHORT, Auteur ; Edmund J. S. SONUGA-BARKE, Auteur ; Wendy J. ADAMS, Auteur ; Graeme FAIRCHILD, Auteur Article en page(s) : p.917-926 Langues : Anglais (eng) Mots-clés : Conduct disorder anxiety disorder callous-unemotional traits comorbidity emotion recognition response biases social information processing Index. décimale : PER Périodiques Résumé : Background Previous research has reported altered emotion recognition in both conduct disorder (CD) and anxiety disorders (ADs) – but these effects appear to be of different kinds. Adolescents with CD often show a generalised pattern of deficits, while those with ADs show hypersensitivity to specific negative emotions. Although these conditions often cooccur, little is known regarding emotion recognition performance in comorbid CD+ADs. Here, we test the hypothesis that in the comorbid case, anxiety-related emotion hypersensitivity counteracts the emotion recognition deficits typically observed in CD. Method We compared facial emotion recognition across four groups of adolescents aged 12–18 years: those with CD alone (n = 28), ADs alone (n = 23), cooccurring CD+ADs (n = 20) and typically developing controls (n = 28). The emotion recognition task we used systematically manipulated the emotional intensity of facial expressions as well as fixation location (eye, nose or mouth region). Results Conduct disorder was associated with a generalised impairment in emotion recognition; however, this may have been modulated by group differences in IQ. AD was associated with increased sensitivity to low-intensity happiness, disgust and sadness. In general, the comorbid CD+ADs group performed similarly to typically developing controls. Conclusions Although CD alone was associated with emotion recognition impairments, ADs and comorbid CD+ADs were associated with normal or enhanced emotion recognition performance. The presence of comorbid ADs appeared to counteract the effects of CD, suggesting a potentially protective role, although future research should examine the contribution of IQ and gender to these effects. En ligne : http://dx.doi.org/10.1111/jcpp.12544 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=292
in Journal of Child Psychology and Psychiatry > 57-8 (August 2016) . - p.917-926[article] Does comorbid anxiety counteract emotion recognition deficits in conduct disorder? [Texte imprimé et/ou numérique] / Roxanna M. L. SHORT, Auteur ; Edmund J. S. SONUGA-BARKE, Auteur ; Wendy J. ADAMS, Auteur ; Graeme FAIRCHILD, Auteur . - p.917-926.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 57-8 (August 2016) . - p.917-926
Mots-clés : Conduct disorder anxiety disorder callous-unemotional traits comorbidity emotion recognition response biases social information processing Index. décimale : PER Périodiques Résumé : Background Previous research has reported altered emotion recognition in both conduct disorder (CD) and anxiety disorders (ADs) – but these effects appear to be of different kinds. Adolescents with CD often show a generalised pattern of deficits, while those with ADs show hypersensitivity to specific negative emotions. Although these conditions often cooccur, little is known regarding emotion recognition performance in comorbid CD+ADs. Here, we test the hypothesis that in the comorbid case, anxiety-related emotion hypersensitivity counteracts the emotion recognition deficits typically observed in CD. Method We compared facial emotion recognition across four groups of adolescents aged 12–18 years: those with CD alone (n = 28), ADs alone (n = 23), cooccurring CD+ADs (n = 20) and typically developing controls (n = 28). The emotion recognition task we used systematically manipulated the emotional intensity of facial expressions as well as fixation location (eye, nose or mouth region). Results Conduct disorder was associated with a generalised impairment in emotion recognition; however, this may have been modulated by group differences in IQ. AD was associated with increased sensitivity to low-intensity happiness, disgust and sadness. In general, the comorbid CD+ADs group performed similarly to typically developing controls. Conclusions Although CD alone was associated with emotion recognition impairments, ADs and comorbid CD+ADs were associated with normal or enhanced emotion recognition performance. The presence of comorbid ADs appeared to counteract the effects of CD, suggesting a potentially protective role, although future research should examine the contribution of IQ and gender to these effects. En ligne : http://dx.doi.org/10.1111/jcpp.12544 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=292 Dopamine and serotonin transporter genotypes moderate sensitivity to maternal expressed emotion: the case of conduct and emotional problems in attention deficit/hyperactivity disorder / Edmund J. S. SONUGA-BARKE in Journal of Child Psychology and Psychiatry, 50-9 (September 2009)
[article]
Titre : Dopamine and serotonin transporter genotypes moderate sensitivity to maternal expressed emotion: the case of conduct and emotional problems in attention deficit/hyperactivity disorder Type de document : Texte imprimé et/ou numérique Auteurs : Edmund J. S. SONUGA-BARKE, Auteur ; Herbert ROEYERS, Auteur ; Michael GIL, Auteur ; Ana MIRANDA, Auteur ; Robert D. OADES, Auteur ; Wai CHEN, Auteur ; Richard ANNEY, Auteur ; Barbara FRANKE, Auteur ; Margaret J. THOMPSON, Auteur ; Lamprini PSYCHOGIOU, Auteur ; Jan K. BUITELAAR, Auteur ; Philip ASHERSON, Auteur ; Stephen V. FARAONE, Auteur ; Hans-Christoph STEINHAUSEN, Auteur ; Joseph A. SERGEANT, Auteur ; Tobias BANASCHEWSKI, Auteur ; Aribert ROTHENBERGER, Auteur ; Richard P. EBSTEIN, Auteur Année de publication : 2009 Article en page(s) : p.1052-1063 Langues : Anglais (eng) Mots-clés : ADHD gene-x-environment-interaction conduct-problems behaviour-problems emotional-problems emotional-symptoms expressed-emotion mothers teachers genetics serotonin-transports dopamine-transport Index. décimale : PER Périodiques Résumé : Background: Mothers' positive emotions expressed about their children with attention deficit/hyperactivity disorder (ADHD) are associated with a reduced likelihood of comorbid conduct problems (CP). We examined whether this association with CP, and one with emotional problems (EMO), is moderated by variants within three genes, previously reported to be associated with ADHD and to moderate the impact of environmental risks on conduct and/or emotional problems; the dopamine transporter gene (SLC6A3/DAT1), the dopamine D4 receptor gene (DRD4) and the serotonin transporter gene (SLC6A4/5HTT).
Methods: Seven hundred and twenty-eight males between the ages of 5 and 17 with a DSM-IV research diagnosis of combined type ADHD were included in these analyses. Parents and teachers rated children's conduct and emotional problems. Positive maternal expressed emotion (PMEE) was coded by independent observers on comments made during a clinical assessment with the mother based on current or recent medication-free periods.
Results: Sensitivity to the effects of PMEE on CP was moderated by variants of the DAT1 and 5HTT genes. Only children who did not carry the DAT1 10R/10R or the 5HTT l/l genotypes showed altered levels of CP when exposed to PMEE. The effect was most marked where the child with ADHD had both these genotypes. For EMO, sensitivity to PMEE was found only with those who carried the DAT1 9R/9R. There was no effect of DRD4 on CP or EMO.
Conclusion: The gene–environment interactions observed suggested that genetic make-up can alter the degree of sensitivity an ADHD patients has to their family environment. Further research should focus on distinguishing general sensitivity genotypes from those conferring risk or protective qualities.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02095.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=828
in Journal of Child Psychology and Psychiatry > 50-9 (September 2009) . - p.1052-1063[article] Dopamine and serotonin transporter genotypes moderate sensitivity to maternal expressed emotion: the case of conduct and emotional problems in attention deficit/hyperactivity disorder [Texte imprimé et/ou numérique] / Edmund J. S. SONUGA-BARKE, Auteur ; Herbert ROEYERS, Auteur ; Michael GIL, Auteur ; Ana MIRANDA, Auteur ; Robert D. OADES, Auteur ; Wai CHEN, Auteur ; Richard ANNEY, Auteur ; Barbara FRANKE, Auteur ; Margaret J. THOMPSON, Auteur ; Lamprini PSYCHOGIOU, Auteur ; Jan K. BUITELAAR, Auteur ; Philip ASHERSON, Auteur ; Stephen V. FARAONE, Auteur ; Hans-Christoph STEINHAUSEN, Auteur ; Joseph A. SERGEANT, Auteur ; Tobias BANASCHEWSKI, Auteur ; Aribert ROTHENBERGER, Auteur ; Richard P. EBSTEIN, Auteur . - 2009 . - p.1052-1063.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 50-9 (September 2009) . - p.1052-1063
Mots-clés : ADHD gene-x-environment-interaction conduct-problems behaviour-problems emotional-problems emotional-symptoms expressed-emotion mothers teachers genetics serotonin-transports dopamine-transport Index. décimale : PER Périodiques Résumé : Background: Mothers' positive emotions expressed about their children with attention deficit/hyperactivity disorder (ADHD) are associated with a reduced likelihood of comorbid conduct problems (CP). We examined whether this association with CP, and one with emotional problems (EMO), is moderated by variants within three genes, previously reported to be associated with ADHD and to moderate the impact of environmental risks on conduct and/or emotional problems; the dopamine transporter gene (SLC6A3/DAT1), the dopamine D4 receptor gene (DRD4) and the serotonin transporter gene (SLC6A4/5HTT).
Methods: Seven hundred and twenty-eight males between the ages of 5 and 17 with a DSM-IV research diagnosis of combined type ADHD were included in these analyses. Parents and teachers rated children's conduct and emotional problems. Positive maternal expressed emotion (PMEE) was coded by independent observers on comments made during a clinical assessment with the mother based on current or recent medication-free periods.
Results: Sensitivity to the effects of PMEE on CP was moderated by variants of the DAT1 and 5HTT genes. Only children who did not carry the DAT1 10R/10R or the 5HTT l/l genotypes showed altered levels of CP when exposed to PMEE. The effect was most marked where the child with ADHD had both these genotypes. For EMO, sensitivity to PMEE was found only with those who carried the DAT1 9R/9R. There was no effect of DRD4 on CP or EMO.
Conclusion: The gene–environment interactions observed suggested that genetic make-up can alter the degree of sensitivity an ADHD patients has to their family environment. Further research should focus on distinguishing general sensitivity genotypes from those conferring risk or protective qualities.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02095.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=828 Early adolescent outcomes for institutionally-deprived and non-deprived adoptees. I: Disinhibited attachment / Michael RUTTER in Journal of Child Psychology and Psychiatry, 48-1 (January 2007)
PermalinkEarly adolescent outcomes of institutionally-deprived and non-deprived adoptees. II: Language as a protective factor and a vulnerable outcome / Carla CROFT in Journal of Child Psychology and Psychiatry, 48-1 (January 2007)
PermalinkEarly adolescent outcomes of institutionally deprived and non-deprived adoptees. III. Quasi-autism / Michael RUTTER in Journal of Child Psychology and Psychiatry, 48-12 (December 2007)
PermalinkEarly severe institutional deprivation is associated with a persistent variant of adult attention-deficit/hyperactivity disorder: clinical presentation, developmental continuities and life circumstances in the English and Romanian Adoptees study / Mark KENNEDY in Journal of Child Psychology and Psychiatry, 57-10 (October 2016)
PermalinkEditorial: ADHD as a reinforcement disorder – moving from general effects to identifying (six) specific models to test / Edmund J. S. SONUGA-BARKE in Journal of Child Psychology and Psychiatry, 52-9 (September 2011)
PermalinkEditorial: Building global science capacity in child psychology and psychiatry – between the etic and emic of cross-cultural enquiry / Edmund J. S. SONUGA-BARKE in Journal of Child Psychology and Psychiatry, 55-4 (April 2014)
PermalinkEditorial: Building therapeutic innovation on scientific foundations in child psychology and psychiatry / Edmund J. S. SONUGA-BARKE in Journal of Child Psychology and Psychiatry, 48-7 (July 2007)
PermalinkEditorial: Developmental foundations of mental health and disorder – moving beyond ‘Towards…’ / Edmund J. S. SONUGA-BARKE in Journal of Child Psychology and Psychiatry, 55-6 (June 2014)
PermalinkEditorial: Diet and children's behaviour problems – disentangling urban myth from clinical reality / Edmund J. S. SONUGA-BARKE in Journal of Child Psychology and Psychiatry, 56-5 (May 2015)
PermalinkEditorial: Distinguishing between the challenges posed by surface and deep forms of heterogeneity to diagnostic systems: do we need a new approach to subtyping of child and adolescent psychiatric disorders / Edmund J. S. SONUGA-BARKE in Journal of Child Psychology and Psychiatry, 57-1 (January 2016)
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