74 recherche sur le mot-clé 'Conduct disorder'

Conduct disorder symptomatology is associated with an altered functional connectome in a large national youth sample / Scott TILLEM in Development and Psychopathology, 34-4 (October 2022)  

Public ISBD [article]  inDevelopment and Psychopathology > 34-4 (October 2022) . - p.1573-1584 Titre : Conduct disorder symptomatology is associated with an altered functional connectome in a large national youth sample Type de document : texte imprimé Auteurs : Scott TILLEM, Auteur ; May I. CONLEY, Auteur ; Arielle BASKIN-SOMMERS, Auteur Article en page(s) : p.1573-1584 Langues : Anglais (eng) Mots-clés : Adolescent  Brain  Child  Cluster Analysis  Conduct Disorder/diagnostic imaging  Connectome  Humans  Magnetic Resonance Imaging/methods  conduct disorder  graph analysis  neural topology  neurocognitive functioning  subcortical structures Index. décimale : PER Périodiques Résumé : Conduct disorder (CD), characterized by youth antisocial behavior, is associated with a variety of neurocognitive impairments. However, questions remain regarding the neural underpinnings of these impairments. To investigate novel neural mechanisms that may support these neurocognitive abnormalities, the present study applied a graph analysis to resting-state functional magnetic resonance imaging (fMRI) data collected from a national sample of 4,781 youth, ages 9-10, who participated in the baseline session of the Adolescent Brain Cognitive Development(SM) Study (ABCD Study®). Analyses were then conducted to examine the relationships among levels of CD symptomatology, metrics of global topology, node-level metrics for subcortical structures, and performance on neurocognitive assessments. Youth higher on CD displayed higher global clustering (ÃŽ2= .039, 95% CI(corrected) [.0027 .0771]), but lower Degree(subcortical) (ÃŽ2= -.052, 95% CI(corrected) [-.0916 -.0152]). Youth higher on CD had worse performance on a general neurocognitive assessment (ÃŽ2= -.104, 95% CI [-.1328 -.0763]) and an emotion recognition memory assessment (ÃŽ2= -.061, 95% CI [-.0919 -.0290]). Finally, global clustering mediated the relationship between CD and general neurocognitive functioning (indirect ÃŽ2= -.002, 95% CI [-.0044 -.0002]), and Degree(subcortical) mediated the relationship between CD and emotion recognition memory performance (indirect ÃŽ2= -.002, 95% CI [-.0046 -.0005]). CD appears associated with neuro-topological abnormalities and these abnormalities may represent neural mechanisms supporting CD-related neurocognitive disruptions. En ligne : http://dx.doi.org/10.1017/s0954579421000237 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=489 [article] Conduct disorder symptomatology is associated with an altered functional connectome in a large national youth sample [texte imprimé] / Scott TILLEM, Auteur ; May I. CONLEY, Auteur ; Arielle BASKIN-SOMMERS, Auteur . - p.1573-1584. Langues : Anglais (eng) in Development and Psychopathology > 34-4 (October 2022) . - p.1573-1584 Mots-clés : Adolescent  Brain  Child  Cluster Analysis  Conduct Disorder/diagnostic imaging  Connectome  Humans  Magnetic Resonance Imaging/methods  conduct disorder  graph analysis  neural topology  neurocognitive functioning  subcortical structures Index. décimale : PER Périodiques Résumé : Conduct disorder (CD), characterized by youth antisocial behavior, is associated with a variety of neurocognitive impairments. However, questions remain regarding the neural underpinnings of these impairments. To investigate novel neural mechanisms that may support these neurocognitive abnormalities, the present study applied a graph analysis to resting-state functional magnetic resonance imaging (fMRI) data collected from a national sample of 4,781 youth, ages 9-10, who participated in the baseline session of the Adolescent Brain Cognitive Development(SM) Study (ABCD Study®). Analyses were then conducted to examine the relationships among levels of CD symptomatology, metrics of global topology, node-level metrics for subcortical structures, and performance on neurocognitive assessments. Youth higher on CD displayed higher global clustering (ÃŽ2= .039, 95% CI(corrected) [.0027 .0771]), but lower Degree(subcortical) (ÃŽ2= -.052, 95% CI(corrected) [-.0916 -.0152]). Youth higher on CD had worse performance on a general neurocognitive assessment (ÃŽ2= -.104, 95% CI [-.1328 -.0763]) and an emotion recognition memory assessment (ÃŽ2= -.061, 95% CI [-.0919 -.0290]). Finally, global clustering mediated the relationship between CD and general neurocognitive functioning (indirect ÃŽ2= -.002, 95% CI [-.0044 -.0002]), and Degree(subcortical) mediated the relationship between CD and emotion recognition memory performance (indirect ÃŽ2= -.002, 95% CI [-.0046 -.0005]). CD appears associated with neuro-topological abnormalities and these abnormalities may represent neural mechanisms supporting CD-related neurocognitive disruptions. En ligne : http://dx.doi.org/10.1017/s0954579421000237 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=489

Conduct disorder with limited prosocial emotions: a multimethod study in criminal justice-involved boys / Olivier F. COLINS in Journal of Child Psychology and Psychiatry, 67-7 (July 2026)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 67-7 (July 2026) . - p.1062-1072 Titre : Conduct disorder with limited prosocial emotions: a multimethod study in criminal justice-involved boys Type de document : texte imprimé Auteurs : Olivier F. COLINS, Auteur ; Thomas CASSART, Auteur ; Emmely DELAMILLIEURE, Auteur ; Kostas A. FANTI, Auteur ; Melina Nicole KYRANIDES, Auteur ; Maria SIKKI, Auteur ; Maria PETRIDOU, Auteur ; Paul H.F. MEENS, Auteur Article en page(s) : p.1062-1072 Langues : Anglais (eng) Mots-clés : Callous-unemotional  Clinical Assessment of Prosocial Emotions  conduct disorder  subtyping Index. décimale : PER Périodiques Résumé : Background More than 10?years after its introduction in DSM-5, support for the ?limited prosocial emotions? specifier (LPE) for the diagnosis of conduct disorder (CD) remains scarce, possibly because studies failed to comprehensively assess the specifier and to include important characteristics. This study was designed to tackle both major limitations. Methods In a sample of 323 criminal justice-involved male adolescents (ages 16?17?years), diagnostic interviews were used to differentiate between males who meet criteria for both CD and the DSM-5 specifier (CD?+?LPE) and males who only meet criteria for CD (CD Only). Individual and environmental characteristics were measured by means of questionnaires and computerized tasks. Results The CD?+?LPE (vs. CD Only) group exhibited higher levels of aggression, delay discounting, and boldness, was more likely to be expelled from school, and had significantly more delinquent friends. Levels of treatment engagement and empathy also were lower in the CD?+?LPE group. The DSM-5 LPE specifier significantly added to the prediction of various of these characteristics beyond indices of CD severity (i.e., total number of CD symptoms and childhood-onset CD) and features of attention-deficit/hyperactivity disorder. However, no significant group differences emerged for other characteristics (e.g., emotion recognition accuracy, anxiety, sensation seeking, response inhibition, and warm parenting). Conclusions Findings provide important and novel evidence to support the utility of the DSM-5 LPE specifier for CD. Some findings were unexpected, though, underscoring the need to replicate and extend the current results when testing the viability of this specifier. En ligne : https://doi.org/10.1111/jcpp.70118 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=588 [article] Conduct disorder with limited prosocial emotions: a multimethod study in criminal justice-involved boys [texte imprimé] / Olivier F. COLINS, Auteur ; Thomas CASSART, Auteur ; Emmely DELAMILLIEURE, Auteur ; Kostas A. FANTI, Auteur ; Melina Nicole KYRANIDES, Auteur ; Maria SIKKI, Auteur ; Maria PETRIDOU, Auteur ; Paul H.F. MEENS, Auteur . - p.1062-1072. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 67-7 (July 2026) . - p.1062-1072 Mots-clés : Callous-unemotional  Clinical Assessment of Prosocial Emotions  conduct disorder  subtyping Index. décimale : PER Périodiques Résumé : Background More than 10?years after its introduction in DSM-5, support for the ?limited prosocial emotions? specifier (LPE) for the diagnosis of conduct disorder (CD) remains scarce, possibly because studies failed to comprehensively assess the specifier and to include important characteristics. This study was designed to tackle both major limitations. Methods In a sample of 323 criminal justice-involved male adolescents (ages 16?17?years), diagnostic interviews were used to differentiate between males who meet criteria for both CD and the DSM-5 specifier (CD?+?LPE) and males who only meet criteria for CD (CD Only). Individual and environmental characteristics were measured by means of questionnaires and computerized tasks. Results The CD?+?LPE (vs. CD Only) group exhibited higher levels of aggression, delay discounting, and boldness, was more likely to be expelled from school, and had significantly more delinquent friends. Levels of treatment engagement and empathy also were lower in the CD?+?LPE group. The DSM-5 LPE specifier significantly added to the prediction of various of these characteristics beyond indices of CD severity (i.e., total number of CD symptoms and childhood-onset CD) and features of attention-deficit/hyperactivity disorder. However, no significant group differences emerged for other characteristics (e.g., emotion recognition accuracy, anxiety, sensation seeking, response inhibition, and warm parenting). Conclusions Findings provide important and novel evidence to support the utility of the DSM-5 LPE specifier for CD. Some findings were unexpected, though, underscoring the need to replicate and extend the current results when testing the viability of this specifier. En ligne : https://doi.org/10.1111/jcpp.70118 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=588

Age of onset and the subclassification of conduct/dissocial disorder / Judy SILBERG in Journal of Child Psychology and Psychiatry, 56-7 (July 2015)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 56-7 (July 2015) . - p.826-833 Titre : Age of onset and the subclassification of conduct/dissocial disorder Type de document : texte imprimé Auteurs : Judy SILBERG, Auteur ; Ashlee A. MOORE, Auteur ; Michael RUTTER, Auteur Article en page(s) : p.826-833 Langues : Anglais (eng) Mots-clés : Age of onset  conduct disorder  persistence into adult life Index. décimale : PER Périodiques Résumé : Background Conduct Disorder (CD) is a markedly heterogeneous psychiatric condition. Moffitt (1993) proposed that subclassification of CD should be according to age of onset. Our goals were to compare childhood-onset and adolescent-onset CD in terms of differences in phenotypic risk factors, genetic analyses, and factors associated with the persistence of antisocial behavior into young adulthood. Methods The data are from the Virginia Twin Study of Adolescent Behavioral Development (VTSABD) and Young Adult Follow-Up (YAFU). Childhood-onset CD was defined as CD beginning at or before age 11. Adolescent-onset CD was defined as having CD onset between ages 14 and 17. These subgroups were compared on ADHD, young adult antisocial behavior (ASB), family dysfunction, and parental depression. Genetic analyses compare childhood-onset and adolescent-onset CD, as well as their cooccurrence with ADHD and ASB. Finally, predictors of persistence were examined. Results Childhood-onset CD was significantly associated with ADHD, ASB, family dysfunction, and parental depression. Adolescent-onset CD was marginally associated with parental depression (p = .05) but not with any of the other risk factors. Univariate genetic models showed that both childhood-onset and adolescent-onset CD involve a large genetic liability accounting for 62% and 65% of the variance, respectively. A common genetic factor (as well as an ADHD-specific factor) accounted for the cooccurrence of childhood-onset CD and ADHD. The cooccurrence of childhood-onset CD and ASB are reflected by a common genetic factor with genetic specific effects on ASB. There was no etiological link between adolescent-onset CD and either ADHD or ASB. Both ADHD and family dysfunction were significantly associated with the persistence of antisocial behavior into young adulthood. Conclusions Phenotypic findings differentiated between childhood-onset and adolescent-onset CD. ADHD and family dysfunction predicted persistence of antisocial behavior into young adulthood. En ligne : http://dx.doi.org/10.1111/jcpp.12353 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=260 [article] Age of onset and the subclassification of conduct/dissocial disorder [texte imprimé] / Judy SILBERG, Auteur ; Ashlee A. MOORE, Auteur ; Michael RUTTER, Auteur . - p.826-833. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 56-7 (July 2015) . - p.826-833 Mots-clés : Age of onset  conduct disorder  persistence into adult life Index. décimale : PER Périodiques Résumé : Background Conduct Disorder (CD) is a markedly heterogeneous psychiatric condition. Moffitt (1993) proposed that subclassification of CD should be according to age of onset. Our goals were to compare childhood-onset and adolescent-onset CD in terms of differences in phenotypic risk factors, genetic analyses, and factors associated with the persistence of antisocial behavior into young adulthood. Methods The data are from the Virginia Twin Study of Adolescent Behavioral Development (VTSABD) and Young Adult Follow-Up (YAFU). Childhood-onset CD was defined as CD beginning at or before age 11. Adolescent-onset CD was defined as having CD onset between ages 14 and 17. These subgroups were compared on ADHD, young adult antisocial behavior (ASB), family dysfunction, and parental depression. Genetic analyses compare childhood-onset and adolescent-onset CD, as well as their cooccurrence with ADHD and ASB. Finally, predictors of persistence were examined. Results Childhood-onset CD was significantly associated with ADHD, ASB, family dysfunction, and parental depression. Adolescent-onset CD was marginally associated with parental depression (p = .05) but not with any of the other risk factors. Univariate genetic models showed that both childhood-onset and adolescent-onset CD involve a large genetic liability accounting for 62% and 65% of the variance, respectively. A common genetic factor (as well as an ADHD-specific factor) accounted for the cooccurrence of childhood-onset CD and ADHD. The cooccurrence of childhood-onset CD and ASB are reflected by a common genetic factor with genetic specific effects on ASB. There was no etiological link between adolescent-onset CD and either ADHD or ASB. Both ADHD and family dysfunction were significantly associated with the persistence of antisocial behavior into young adulthood. Conclusions Phenotypic findings differentiated between childhood-onset and adolescent-onset CD. ADHD and family dysfunction predicted persistence of antisocial behavior into young adulthood. En ligne : http://dx.doi.org/10.1111/jcpp.12353 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=260

Annual Research Review: Phenotypic and causal structure of conduct disorder in the broader context of prevalent forms of psychopathology / Benjamin B. LAHEY in Journal of Child Psychology and Psychiatry, 53-5 (May 2012)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 53-5 (May 2012) . - p.536-557 Titre : Annual Research Review: Phenotypic and causal structure of conduct disorder in the broader context of prevalent forms of psychopathology Type de document : texte imprimé Auteurs : Benjamin B. LAHEY, Auteur ; Irwin D. WALDMAN, Auteur Année de publication : 2012 Article en page(s) : p.536-557 Langues : Anglais (eng) Mots-clés : Conduct disorder  oppositional defiant disorder  taxonomy  etiology  homotypic and heterotypic continuity Index. décimale : PER Périodiques Résumé : Background:  A better understanding of the nature and etiology of conduct disorder (CD) can inform nosology and vice versa. We posit that any prevalent form of psychopathology, including CD, can be best understood if it is studied in the context of other correlated forms of child and adolescent psychopathology using formal models to guide inquiry. Methods:  Review of both cross-sectional and longitudinal studies of the place of CD in the phenotypic and causal structure of prevalent psychopathology, with an emphasis on similarities and differences between CD and oppositional defiant disorder (ODD). Papers were located using Web of Science by topic searches with no restriction on year of publication. Results:  Although some important nosologic questions remain unanswered, the dimensional phenotype of CD is well defined. CD differs from other disorders in its correlates, associated impairment, and course. Nonetheless, it is robustly correlated with many other prevalent dimensions of psychopathology both concurrently and predictively, including both other ‘externalizing’ disorders and some ‘internalizing’ disorders. Based on emerging evidence, we hypothesize that these concurrent and predictive correlations result primarily from widespread genetic pleiotropy, with some genetic factors nonspecifically influencing risk for multiple correlated dimensions of psychopathology. In contrast, environmental influences mostly act to differentiate dimensions of psychopathology from one another both concurrently and over time. CD and ODD share half of their genetic influences, but their genetic etiologies are distinct in other ways. Unlike most other dimensions of psychopathology, half of the genetic influences on CD appear to be unique to CD. In contrast, ODD broadly shares nearly all of its genetic influences with other disorders and has little unique genetic variance. Conclusions:  Conduct disorder is a relatively distinct syndrome at both phenotypic and etiologic levels, but much is revealed by studying CD in the context of its causal and phenotypic associations with other disorders over time. Advancing and refining formal causal models that specify the common and unique causes and biological mechanisms underlying each correlated dimension of psychopathology should facilitate research on the fundamental nature and nosology of CD. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2011.02509.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=154 [article] Annual Research Review: Phenotypic and causal structure of conduct disorder in the broader context of prevalent forms of psychopathology [texte imprimé] / Benjamin B. LAHEY, Auteur ; Irwin D. WALDMAN, Auteur . - 2012 . - p.536-557. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 53-5 (May 2012) . - p.536-557 Mots-clés : Conduct disorder  oppositional defiant disorder  taxonomy  etiology  homotypic and heterotypic continuity Index. décimale : PER Périodiques Résumé : Background:  A better understanding of the nature and etiology of conduct disorder (CD) can inform nosology and vice versa. We posit that any prevalent form of psychopathology, including CD, can be best understood if it is studied in the context of other correlated forms of child and adolescent psychopathology using formal models to guide inquiry. Methods:  Review of both cross-sectional and longitudinal studies of the place of CD in the phenotypic and causal structure of prevalent psychopathology, with an emphasis on similarities and differences between CD and oppositional defiant disorder (ODD). Papers were located using Web of Science by topic searches with no restriction on year of publication. Results:  Although some important nosologic questions remain unanswered, the dimensional phenotype of CD is well defined. CD differs from other disorders in its correlates, associated impairment, and course. Nonetheless, it is robustly correlated with many other prevalent dimensions of psychopathology both concurrently and predictively, including both other ‘externalizing’ disorders and some ‘internalizing’ disorders. Based on emerging evidence, we hypothesize that these concurrent and predictive correlations result primarily from widespread genetic pleiotropy, with some genetic factors nonspecifically influencing risk for multiple correlated dimensions of psychopathology. In contrast, environmental influences mostly act to differentiate dimensions of psychopathology from one another both concurrently and over time. CD and ODD share half of their genetic influences, but their genetic etiologies are distinct in other ways. Unlike most other dimensions of psychopathology, half of the genetic influences on CD appear to be unique to CD. In contrast, ODD broadly shares nearly all of its genetic influences with other disorders and has little unique genetic variance. Conclusions:  Conduct disorder is a relatively distinct syndrome at both phenotypic and etiologic levels, but much is revealed by studying CD in the context of its causal and phenotypic associations with other disorders over time. Advancing and refining formal causal models that specify the common and unique causes and biological mechanisms underlying each correlated dimension of psychopathology should facilitate research on the fundamental nature and nosology of CD. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2011.02509.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=154

Brain response to viewing others being harmed in children with conduct disorder symptoms / Kalina J. MICHALSKA in Journal of Child Psychology and Psychiatry, 57-4 (April 2016)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 57-4 (April 2016) . - p.510-519 Titre : Brain response to viewing others being harmed in children with conduct disorder symptoms Type de document : texte imprimé Auteurs : Kalina J. MICHALSKA, Auteur ; Thomas A. ZEFFIRO, Auteur ; Jean DECETY, Auteur Article en page(s) : p.510-519 Langues : Anglais (eng) Mots-clés : Conduct disorder  callousness  affective arousal  emotional empathy  insula  anterior cingulate cortex Index. décimale : PER Périodiques Résumé : Background Deficient empathic processing is thought to foster conduct disorder (CD). It is important to determine the extent to which neural response associated with perceiving harm to others predicts CD symptoms and callous disregard for others. Methods A total of 107 9- to 11-year-old children (52 female) were recruited from pediatric and mental health clinics, representing a wide range of CD symptoms. Children were scanned with functional magnetic resonance imaging while viewing brief video clips of persons being harmed intentionally or accidentally. Results Perceiving harm evoked increased hemodynamic response in the anterior insula (aINS), anterior cingulate cortex (ACC), amygdala, periaqueductal gray (PAG), caudate, and inferior parietal lobe (IPL) across all participants. Intentionally caused, relative to unintentional harm was associated with greater activity in the aINS, amygdala, and temporal pole. There was an inverse association of number of CD symptoms with right posterior insula in both the Harm > No Harm and the Intentional > Unintentional Harm contrasts. Furthermore, an inverse association between callousness and posterior insula activation was found in the Harm > No Harm contrast, with the opposite pattern for reactive aggression scores. An interaction revealed a stronger association in girls between CD symptoms and the right posterior superior temporal sulcus (pSTS) in the Intentional Harm versus Unintentional Harm contrast. Conclusions Children with greater CD and callousness exhibit dampened hemodynamic response to viewing others being harmed in the insula, a region which plays a key role in empathy and emotional awareness. Sex differences in the neural correlates of CD were observed. En ligne : http://dx.doi.org/10.1111/jcpp.12474 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=285 [article] Brain response to viewing others being harmed in children with conduct disorder symptoms [texte imprimé] / Kalina J. MICHALSKA, Auteur ; Thomas A. ZEFFIRO, Auteur ; Jean DECETY, Auteur . - p.510-519. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 57-4 (April 2016) . - p.510-519 Mots-clés : Conduct disorder  callousness  affective arousal  emotional empathy  insula  anterior cingulate cortex Index. décimale : PER Périodiques Résumé : Background Deficient empathic processing is thought to foster conduct disorder (CD). It is important to determine the extent to which neural response associated with perceiving harm to others predicts CD symptoms and callous disregard for others. Methods A total of 107 9- to 11-year-old children (52 female) were recruited from pediatric and mental health clinics, representing a wide range of CD symptoms. Children were scanned with functional magnetic resonance imaging while viewing brief video clips of persons being harmed intentionally or accidentally. Results Perceiving harm evoked increased hemodynamic response in the anterior insula (aINS), anterior cingulate cortex (ACC), amygdala, periaqueductal gray (PAG), caudate, and inferior parietal lobe (IPL) across all participants. Intentionally caused, relative to unintentional harm was associated with greater activity in the aINS, amygdala, and temporal pole. There was an inverse association of number of CD symptoms with right posterior insula in both the Harm > No Harm and the Intentional > Unintentional Harm contrasts. Furthermore, an inverse association between callousness and posterior insula activation was found in the Harm > No Harm contrast, with the opposite pattern for reactive aggression scores. An interaction revealed a stronger association in girls between CD symptoms and the right posterior superior temporal sulcus (pSTS) in the Intentional Harm versus Unintentional Harm contrast. Conclusions Children with greater CD and callousness exhibit dampened hemodynamic response to viewing others being harmed in the insula, a region which plays a key role in empathy and emotional awareness. Sex differences in the neural correlates of CD were observed. En ligne : http://dx.doi.org/10.1111/jcpp.12474 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=285

Brain structure abnormalities in adolescent girls with conduct disorder / Graeme FAIRCHILD in Journal of Child Psychology and Psychiatry, 54-1 (January 2013)  

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Cognitive and Familial Contributions to Conduct Disorder in Children / Jean TOUPIN in Journal of Child Psychology and Psychiatry, 41-3 (March 2000)

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Different brain responses during empathy in autism spectrum disorders versus conduct disorder and callous-unemotional traits / Eduard T. KLAPWIJK in Journal of Child Psychology and Psychiatry, 57-6 (June 2016)  

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Does comorbid anxiety counteract emotion recognition deficits in conduct disorder? / Roxanna M.L. SHORT in Journal of Child Psychology and Psychiatry, 57-8 (August 2016)  

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Empathy in children with autism and conduct disorder: group-specific profiles and developmental aspects / Christina SCHWENCK in Journal of Child Psychology and Psychiatry, 53-6 (June 2012)  

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