Childhood predictors of avoidant personality disorder traits in adolescence: a seven-wave birth cohort study / Theresa WILBERG ; Elfrida HARTVEIT KVARSTEIN ; Silje STEINSBEKK in Journal of Child Psychology and Psychiatry, 66-3 (March 2025)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 66-3 (March 2025) . - p.366-377 Titre : Childhood predictors of avoidant personality disorder traits in adolescence: a seven-wave birth cohort study Type de document : Texte imprimé et/ou numérique Auteurs : Theresa WILBERG, Auteur ; Elfrida HARTVEIT KVARSTEIN, Auteur ; Silje STEINSBEKK, Auteur Article en page(s) : p.366-377 Langues : Anglais (eng) Mots-clés : Adolescent  attachment  avoidant personality disorder  childhood  longitudinal Index. décimale : PER Périodiques Résumé : Background Although it is widely assumed that avoidant personality disorder (AvPD) originates in childhood, there is little prospective research to substantiate this claim. We therefore aimed to determine whether presumed childhood risk factors predict AvPD traits at 16?years. Methods A population-based sample (n?=?1,077; 50.9% female) from the 2003 and 2004 birth cohorts in Trondheim, Norway was examined biennially from 4 to 16?years. The number of AvPD traits at the age of 16 was assessed with the structured clinical interview for DSM-5 personality disorders and regressed on the intercept and growth in child risk and protective factors until the age of 14. Results The prevalence of AvPD at the age of 16 was 3.2% (95% CI: 2.2?4.1). Higher levels and an increased number of social anxiety symptoms over time, as well as increased negative affectivity/neuroticism, predicted a higher number of AvPD traits. When the levels and changes in these factors were adjusted for, less and decreasing extraversion forecasted more AvPD traits, as did declining self-worth, higher levels of parental AvPD traits, and increased onlooking behavior. Conclusions Neuroticism, low extraversion, social anxiety symptoms, passive onlooking behavior, and low self-worth predicted a higher number of AvPD traits in adolescence, as did more AvPD traits in parents. Efforts to enhance self-worth, reduce social anxiety, and promote peer interaction among onlooking children may reduce the development of AvPD traits in adolescence. En ligne : https://doi.org/10.1111/jcpp.14064 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=548 [article] Childhood predictors of avoidant personality disorder traits in adolescence: a seven-wave birth cohort study [Texte imprimé et/ou numérique] / Theresa WILBERG, Auteur ; Elfrida HARTVEIT KVARSTEIN, Auteur ; Silje STEINSBEKK, Auteur . - p.366-377. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 66-3 (March 2025) . - p.366-377 Mots-clés : Adolescent  attachment  avoidant personality disorder  childhood  longitudinal Index. décimale : PER Périodiques Résumé : Background Although it is widely assumed that avoidant personality disorder (AvPD) originates in childhood, there is little prospective research to substantiate this claim. We therefore aimed to determine whether presumed childhood risk factors predict AvPD traits at 16?years. Methods A population-based sample (n?=?1,077; 50.9% female) from the 2003 and 2004 birth cohorts in Trondheim, Norway was examined biennially from 4 to 16?years. The number of AvPD traits at the age of 16 was assessed with the structured clinical interview for DSM-5 personality disorders and regressed on the intercept and growth in child risk and protective factors until the age of 14. Results The prevalence of AvPD at the age of 16 was 3.2% (95% CI: 2.2?4.1). Higher levels and an increased number of social anxiety symptoms over time, as well as increased negative affectivity/neuroticism, predicted a higher number of AvPD traits. When the levels and changes in these factors were adjusted for, less and decreasing extraversion forecasted more AvPD traits, as did declining self-worth, higher levels of parental AvPD traits, and increased onlooking behavior. Conclusions Neuroticism, low extraversion, social anxiety symptoms, passive onlooking behavior, and low self-worth predicted a higher number of AvPD traits in adolescence, as did more AvPD traits in parents. Efforts to enhance self-worth, reduce social anxiety, and promote peer interaction among onlooking children may reduce the development of AvPD traits in adolescence. En ligne : https://doi.org/10.1111/jcpp.14064 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=548

Development of symptoms of oppositional defiant disorder from preschool to adolescence: the role of bullying victimization and emotion regulation / Silje STEINSBEKK ; Lars WICHSTRØM in Journal of Child Psychology and Psychiatry, 65-3 (March 2023)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 65-3 (March 2023) . - p.343-353 Titre : Development of symptoms of oppositional defiant disorder from preschool to adolescence: the role of bullying victimization and emotion regulation Type de document : Texte imprimé et/ou numérique Auteurs : Silje STEINSBEKK, Auteur ; Lars WICHSTRØM, Auteur Article en page(s) : p.343-353 Index. décimale : PER Périodiques Résumé : Background Childhood oppositional defiant disorder (ODD) is associated with adverse outcomes which can continue to impair life well into adulthood. Identifying modifiable etiological factors of ODD is therefore essential. Although bullying victimization and poor emotion regulation are assumed to be risk factors for the development of ODD symptoms, little research has been conducted to test this possibility. Methods A sample (n?=?1,042) from two birth cohorts of children in the city of Trondheim, Norway, was assessed biennially from age 4 to 14?years. Parents and children (from age 8) were assessed with clinical interviews to determine symptoms of ODD, children reported on their victimization from bullying, and teachers reported on children's emotion regulation. Results Oppositional defiant disorder symptoms increased from age 4 to 6, from age 8 to 10, and then started to wane as children entered adolescence. A Random Intercept Cross-Lagged Panel Model revealed that increased emotion regulation predicted a reduced number of ODD symptoms across development (??=??.15 to ?.13, p? En ligne : https://doi.org/10.1111/jcpp.13845 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=520 [article] Development of symptoms of oppositional defiant disorder from preschool to adolescence: the role of bullying victimization and emotion regulation [Texte imprimé et/ou numérique] / Silje STEINSBEKK, Auteur ; Lars WICHSTRØM, Auteur . - p.343-353. in Journal of Child Psychology and Psychiatry > 65-3 (March 2023) . - p.343-353 Index. décimale : PER Périodiques Résumé : Background Childhood oppositional defiant disorder (ODD) is associated with adverse outcomes which can continue to impair life well into adulthood. Identifying modifiable etiological factors of ODD is therefore essential. Although bullying victimization and poor emotion regulation are assumed to be risk factors for the development of ODD symptoms, little research has been conducted to test this possibility. Methods A sample (n?=?1,042) from two birth cohorts of children in the city of Trondheim, Norway, was assessed biennially from age 4 to 14?years. Parents and children (from age 8) were assessed with clinical interviews to determine symptoms of ODD, children reported on their victimization from bullying, and teachers reported on children's emotion regulation. Results Oppositional defiant disorder symptoms increased from age 4 to 6, from age 8 to 10, and then started to wane as children entered adolescence. A Random Intercept Cross-Lagged Panel Model revealed that increased emotion regulation predicted a reduced number of ODD symptoms across development (??=??.15 to ?.13, p? En ligne : https://doi.org/10.1111/jcpp.13845 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=520

Food fussiness and food neophobia share a common etiology in early childhood / Andrea D. SMITH in Journal of Child Psychology and Psychiatry, 58-2 (February 2017)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 58-2 (February 2017) . - p.189-196 Titre : Food fussiness and food neophobia share a common etiology in early childhood Type de document : Texte imprimé et/ou numérique Auteurs : Andrea D. SMITH, Auteur ; Moritz HERLE, Auteur ; Alison FILDES, Auteur ; Lucy COOKE, Auteur ; Silje STEINSBEKK, Auteur ; Clare H. LLEWELLYN, Auteur Année de publication : 2017 Article en page(s) : p.189-196 Langues : Anglais (eng) Mots-clés : Eating behavior  twin design  behavioral genetics  food fussiness  food neophobia Index. décimale : PER Périodiques Résumé : Background ‘Food fussiness’ (FF) is the tendency to be highly selective about which foods one is willing to eat, and emerges in early childhood; ‘food neophobia’ (FN) is a closely related characteristic but specifically refers to rejection of unfamiliar food. These behaviors are associated, but the extent to which their etiological architecture overlaps is unknown. The objective of this study was to quantify the relative contribution of genetic and environmental influences to variation in FF and FN in early childhood; and to establish the extent to which they share common genetic and environmental influences. Method Participants were 1,921 families with 16-month-old twins from the Gemini birth cohort. Parents completed the Child Eating Behaviour Questionnaire which included three FF items and four FN items. Bivariate quantitative genetic modeling was used to quantify: (a) genetic and environmental contributions to variation in FF and FN; and (b) the extent to which genetic or environmental influences on FF and FN are shared across the traits. Results Food fussiness and FN were strongly correlated (r = .72, p < .001). Proportions of variation in FF were equally explained by genetic (.46; 95% CI: 0.41–0.52) and shared environmental influences (.46; 95% CI: 0.41–0.51). Shared environmental effects accounted for a significantly lower proportion of variation in FN (.22; 95% CI: 0.14–0.30), but genetic influences were not significantly different from those on FF (.58, 95% CI: 0.50–0.67). FF and FN largely shared a common etiology, indicated by high genetic (.73; 95% CI: 0.67–0.78) and shared environmental correlations (.78; 95% CI: 0.69–0.86) across the two traits. Conclusions Food fussiness and FN both show considerable heritability at 16 months but shared environmental factors, for example the home environment, influenced more interindividual differences in the expression of FF than in FN. FF and FN largely share a common etiology. En ligne : http://dx.doi.org/10.1111/jcpp.12647 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=299 [article] Food fussiness and food neophobia share a common etiology in early childhood [Texte imprimé et/ou numérique] / Andrea D. SMITH, Auteur ; Moritz HERLE, Auteur ; Alison FILDES, Auteur ; Lucy COOKE, Auteur ; Silje STEINSBEKK, Auteur ; Clare H. LLEWELLYN, Auteur . - 2017 . - p.189-196. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 58-2 (February 2017) . - p.189-196 Mots-clés : Eating behavior  twin design  behavioral genetics  food fussiness  food neophobia Index. décimale : PER Périodiques Résumé : Background ‘Food fussiness’ (FF) is the tendency to be highly selective about which foods one is willing to eat, and emerges in early childhood; ‘food neophobia’ (FN) is a closely related characteristic but specifically refers to rejection of unfamiliar food. These behaviors are associated, but the extent to which their etiological architecture overlaps is unknown. The objective of this study was to quantify the relative contribution of genetic and environmental influences to variation in FF and FN in early childhood; and to establish the extent to which they share common genetic and environmental influences. Method Participants were 1,921 families with 16-month-old twins from the Gemini birth cohort. Parents completed the Child Eating Behaviour Questionnaire which included three FF items and four FN items. Bivariate quantitative genetic modeling was used to quantify: (a) genetic and environmental contributions to variation in FF and FN; and (b) the extent to which genetic or environmental influences on FF and FN are shared across the traits. Results Food fussiness and FN were strongly correlated (r = .72, p < .001). Proportions of variation in FF were equally explained by genetic (.46; 95% CI: 0.41–0.52) and shared environmental influences (.46; 95% CI: 0.41–0.51). Shared environmental effects accounted for a significantly lower proportion of variation in FN (.22; 95% CI: 0.14–0.30), but genetic influences were not significantly different from those on FF (.58, 95% CI: 0.50–0.67). FF and FN largely shared a common etiology, indicated by high genetic (.73; 95% CI: 0.67–0.78) and shared environmental correlations (.78; 95% CI: 0.69–0.86) across the two traits. Conclusions Food fussiness and FN both show considerable heritability at 16 months but shared environmental factors, for example the home environment, influenced more interindividual differences in the expression of FF than in FN. FF and FN largely share a common etiology. En ligne : http://dx.doi.org/10.1111/jcpp.12647 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=299

Homotypic and heterotypic continuity of symptoms of psychiatric disorders from age 4 to 10 years: a dynamic panel model / Lars WICHSTRØM in Journal of Child Psychology and Psychiatry, 58-11 (November 2017)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 58-11 (November 2017) . - p.1239-1247 Titre : Homotypic and heterotypic continuity of symptoms of psychiatric disorders from age 4 to 10 years: a dynamic panel model Type de document : Texte imprimé et/ou numérique Auteurs : Lars WICHSTRØM, Auteur ; Jay BELSKY, Auteur ; Silje STEINSBEKK, Auteur Article en page(s) : p.1239-1247 Langues : Anglais (eng) Mots-clés : Attention-deficit/hyperactivity disorder  anxiety  conduct disorder  continuity  depression  fixed effects  heterotypic  homotypic  longitudinal  life-events  oppositional defiant disorder  prospective  psychiatric disorder  symptoms Index. décimale : PER Périodiques Résumé : Background Childhood psychiatric disorders and their symptoms evince both within-disorder (homotypic) and between-disorder (heterotypic) continuities. These continuities may be due to earlier symptoms causing later symptoms or, alternatively, that the same (unknown) causes (e.g., genetics) are operating across time. Applying a novel data analytic approach, we disentangle these two explanations. Methods Participants in a Norwegian community study were assessed biennially from 4 to 10 years of age with clinical interviews (n = 1,042). Prospective reciprocal relations between symptoms of disorders were analyzed with a dynamic panel model within a structural equation framework, adjusting for all unmeasured time-invariant confounders and time-varying negative life-events. Results Homotypic continuities in symptoms characterized all disorders; strongest for attention-deficit/hyperactivity disorder (ADHD) (r = .32–.62), moderate for behavioral disorders (r = .31–.48) and for anxiety and depression (r = .15–.40), and stronger between 8 and 10 than between 4 and 6 years. Heterotypic continuity also characterized all disorders. A dynamic panel model showed that most continuities were due to unmeasured time-invariant factors rather than effects of earlier symptoms on later symptoms, although symptoms of behavioral disorders, which evinced two-year homotypic continuity (B = .14, 95% CI: .04, .25), did influence later symptoms of ADHD (B = .13, CI: .03, .23), and earlier ADHD symptoms influenced later anxiety disorder symptoms (B = .07, CI: .01, .12). Conclusions Homotypic and heterotypic continuities of symptoms of childhood psychiatric disorders are mostly due to unobserved time-invariant factors. Nonetheless, symptoms of earlier behavioral disorders may affect later symptoms of such disorders and of ADHD, and ADHD may increase the risk of later anxiety. Thus, even if interventions do not alter basic etiological factors, symptom reduction may itself cause later symptom reduction. En ligne : http://dx.doi.org/10.1111/jcpp.12754 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=326 [article] Homotypic and heterotypic continuity of symptoms of psychiatric disorders from age 4 to 10 years: a dynamic panel model [Texte imprimé et/ou numérique] / Lars WICHSTRØM, Auteur ; Jay BELSKY, Auteur ; Silje STEINSBEKK, Auteur . - p.1239-1247. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 58-11 (November 2017) . - p.1239-1247 Mots-clés : Attention-deficit/hyperactivity disorder  anxiety  conduct disorder  continuity  depression  fixed effects  heterotypic  homotypic  longitudinal  life-events  oppositional defiant disorder  prospective  psychiatric disorder  symptoms Index. décimale : PER Périodiques Résumé : Background Childhood psychiatric disorders and their symptoms evince both within-disorder (homotypic) and between-disorder (heterotypic) continuities. These continuities may be due to earlier symptoms causing later symptoms or, alternatively, that the same (unknown) causes (e.g., genetics) are operating across time. Applying a novel data analytic approach, we disentangle these two explanations. Methods Participants in a Norwegian community study were assessed biennially from 4 to 10 years of age with clinical interviews (n = 1,042). Prospective reciprocal relations between symptoms of disorders were analyzed with a dynamic panel model within a structural equation framework, adjusting for all unmeasured time-invariant confounders and time-varying negative life-events. Results Homotypic continuities in symptoms characterized all disorders; strongest for attention-deficit/hyperactivity disorder (ADHD) (r = .32–.62), moderate for behavioral disorders (r = .31–.48) and for anxiety and depression (r = .15–.40), and stronger between 8 and 10 than between 4 and 6 years. Heterotypic continuity also characterized all disorders. A dynamic panel model showed that most continuities were due to unmeasured time-invariant factors rather than effects of earlier symptoms on later symptoms, although symptoms of behavioral disorders, which evinced two-year homotypic continuity (B = .14, 95% CI: .04, .25), did influence later symptoms of ADHD (B = .13, CI: .03, .23), and earlier ADHD symptoms influenced later anxiety disorder symptoms (B = .07, CI: .01, .12). Conclusions Homotypic and heterotypic continuities of symptoms of childhood psychiatric disorders are mostly due to unobserved time-invariant factors. Nonetheless, symptoms of earlier behavioral disorders may affect later symptoms of such disorders and of ADHD, and ADHD may increase the risk of later anxiety. Thus, even if interventions do not alter basic etiological factors, symptom reduction may itself cause later symptom reduction. En ligne : http://dx.doi.org/10.1111/jcpp.12754 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=326

Longitudinal relations between impaired executive function and symptoms of psychiatric disorders in childhood / Marte HALSE in Journal of Child Psychology and Psychiatry, 63-12 (December 2022)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 63-12 (December 2022) . - p.1574-1582 Titre : Longitudinal relations between impaired executive function and symptoms of psychiatric disorders in childhood Type de document : Texte imprimé et/ou numérique Auteurs : Marte HALSE, Auteur ; Silje STEINSBEKK, Auteur ; ASA HAMMAR, Auteur ; Lars WICHSTRØM, Auteur Article en page(s) : p.1574-1582 Langues : Anglais (eng) Mots-clés : Child  Adolescent  Child, Preschool  Humans  Executive Function  Attention Deficit and Disruptive Behavior Disorders/epidemiology  Conduct Disorder  Attention Deficit Disorder with Hyperactivity  Anxiety Disorders  Adolescents  Brief  Capa  Papa  child development  developmental psychopathology  longitudinal  mental health  p factor  psychiatric disorder  self-regulation Index. décimale : PER Périodiques Résumé : BACKGROUND: Malfunctioning of executive functions correlates with psychopathology in children. However, the directionality, the extent to which the relation varies for various disorders, and whether prospective relations afford causal interpretations are not known. METHODS: A community sample of Norwegian children (n=874) was studied biennially from the age of 6 to 14 years. Executive functions were assessed using the Behavior Rating Inventory of Executive Function Teacher-report and symptoms of psychopathology were assessed using the Preschool Age Psychiatric Assessment (age 6; parents) and Child and Adolescent Psychiatric Assessment (ages 8-14; children and parents). Prospective reciprocal relations were examined using a random intercept cross-lagged panel model that adjusts for all unobserved time-invariant confounders. RESULTS: Even when time-invariant confounders were accounted for, reduced executive functions predicted increased symptoms of depressive disorders, anxiety disorders, attention-deficit hyperactivity disorder (ADHD), oppositional defiant disorder (ODD), and conduct disorder (CD) 2 years later, even when previous changes in these symptoms were adjusted for. The level of prediction (B=.83, 95% CI [.37, 1.3]) was not different for different disorders or ages. Conversely, reduced executive functions were predicted by increased symptoms of all disorders (B=.01, 95% CI [.01, .02]). CONCLUSIONS: Reduced executive functioning may be involved in the etiology of depression, anxiety, ADHD, and ODD/CD to an equal extent. Moreover, increased depression, anxiety, ADHD, and ODD/CD may negatively impact executive functioning. En ligne : http://dx.doi.org/10.1111/jcpp.13622 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=490 [article] Longitudinal relations between impaired executive function and symptoms of psychiatric disorders in childhood [Texte imprimé et/ou numérique] / Marte HALSE, Auteur ; Silje STEINSBEKK, Auteur ; ASA HAMMAR, Auteur ; Lars WICHSTRØM, Auteur . - p.1574-1582. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 63-12 (December 2022) . - p.1574-1582 Mots-clés : Child  Adolescent  Child, Preschool  Humans  Executive Function  Attention Deficit and Disruptive Behavior Disorders/epidemiology  Conduct Disorder  Attention Deficit Disorder with Hyperactivity  Anxiety Disorders  Adolescents  Brief  Capa  Papa  child development  developmental psychopathology  longitudinal  mental health  p factor  psychiatric disorder  self-regulation Index. décimale : PER Périodiques Résumé : BACKGROUND: Malfunctioning of executive functions correlates with psychopathology in children. However, the directionality, the extent to which the relation varies for various disorders, and whether prospective relations afford causal interpretations are not known. METHODS: A community sample of Norwegian children (n=874) was studied biennially from the age of 6 to 14 years. Executive functions were assessed using the Behavior Rating Inventory of Executive Function Teacher-report and symptoms of psychopathology were assessed using the Preschool Age Psychiatric Assessment (age 6; parents) and Child and Adolescent Psychiatric Assessment (ages 8-14; children and parents). Prospective reciprocal relations were examined using a random intercept cross-lagged panel model that adjusts for all unobserved time-invariant confounders. RESULTS: Even when time-invariant confounders were accounted for, reduced executive functions predicted increased symptoms of depressive disorders, anxiety disorders, attention-deficit hyperactivity disorder (ADHD), oppositional defiant disorder (ODD), and conduct disorder (CD) 2 years later, even when previous changes in these symptoms were adjusted for. The level of prediction (B=.83, 95% CI [.37, 1.3]) was not different for different disorders or ages. Conversely, reduced executive functions were predicted by increased symptoms of all disorders (B=.01, 95% CI [.01, .02]). CONCLUSIONS: Reduced executive functioning may be involved in the etiology of depression, anxiety, ADHD, and ODD/CD to an equal extent. Moreover, increased depression, anxiety, ADHD, and ODD/CD may negatively impact executive functioning. En ligne : http://dx.doi.org/10.1111/jcpp.13622 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=490

Prevalence and course of anxiety disorders and symptoms from preschool to adolescence: a 6-wave community study / Silje STEINSBEKK in Journal of Child Psychology and Psychiatry, 63-5 (May 2022)  

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Reciprocal relations between dimensions of attention-deficit/hyperactivity and anxiety disorders from preschool age to adolescence: sex differences in a birth cohort sample / Mojtaba HABIBI ASGARABAD ; Silje STEINSBEKK ; Cynthia M. HARTUNG ; Lars WICHSTRØM in Journal of Child Psychology and Psychiatry, 66-2 (February 2025)  

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Reciprocal relations between interparental aggression and symptoms of oppositional defiant and conduct disorders: a seven-wave cohort study of within-family effects from preschool to adolescence / Silje STEINSBEKK ; Lars WICHSTRØM in Journal of Child Psychology and Psychiatry, 65-6 (June 2024)  

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Social skills and symptoms of anxiety disorders from preschool to adolescence: a prospective cohort study / Mojtaba HABIBI ASGARABAD in Journal of Child Psychology and Psychiatry, 64-7 (July 2023)  

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The co-occurrence between symptoms of internet gaming disorder and psychiatric disorders in childhood and adolescence: prospective relations or common causes? / Beate WOLD HYGEN in Journal of Child Psychology and Psychiatry, 61-8 (August 2020)  

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