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Prenatal exposure to paternal smoking and likelihood for autism spectrum disorder in Autism, 25-7 (October 2021)
[article]
Titre : Prenatal exposure to paternal smoking and likelihood for autism spectrum disorder Type de document : Texte imprimé et/ou numérique Article en page(s) : p.1946-1959 Langues : Anglais (eng) Mots-clés : Autism Spectrum Disorder/epidemiology/etiology Causality Family Female Humans Pregnancy Prenatal Exposure Delayed Effects/epidemiology/etiology Risk Factors Smoking/adverse effects autism spectrum disorders environmental factors risk factor epidemiology to the research presented in this manuscript. Index. décimale : PER Périodiques Résumé : What is Already Known about This Subject: Genetics, (including de novo mutations), environmental factors (including toxic exposures), and their interactions impact autism spectrum disorder etiology. Paternal smoking is a candidate risk for autism spectrum disorder due to biological plausibility, high prevalence, and potential intervention.What This Study Adds: This original study and its replication confirms that paternal factors can substantially contribute to autism spectrum disorder risk for their offspring. It specifically indicates that paternal smoking both before and during pregnancy contributes significantly to autism spectrum disorder risk.Implications for practice, research, or policy: Smoking prevention, especially in pregnancy planning, may decrease autism spectrum disorder risk in offspring. En ligne : http://dx.doi.org/10.1177/13623613211007319 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=451
in Autism > 25-7 (October 2021) . - p.1946-1959[article] Prenatal exposure to paternal smoking and likelihood for autism spectrum disorder [Texte imprimé et/ou numérique] . - p.1946-1959.
Langues : Anglais (eng)
in Autism > 25-7 (October 2021) . - p.1946-1959
Mots-clés : Autism Spectrum Disorder/epidemiology/etiology Causality Family Female Humans Pregnancy Prenatal Exposure Delayed Effects/epidemiology/etiology Risk Factors Smoking/adverse effects autism spectrum disorders environmental factors risk factor epidemiology to the research presented in this manuscript. Index. décimale : PER Périodiques Résumé : What is Already Known about This Subject: Genetics, (including de novo mutations), environmental factors (including toxic exposures), and their interactions impact autism spectrum disorder etiology. Paternal smoking is a candidate risk for autism spectrum disorder due to biological plausibility, high prevalence, and potential intervention.What This Study Adds: This original study and its replication confirms that paternal factors can substantially contribute to autism spectrum disorder risk for their offspring. It specifically indicates that paternal smoking both before and during pregnancy contributes significantly to autism spectrum disorder risk.Implications for practice, research, or policy: Smoking prevention, especially in pregnancy planning, may decrease autism spectrum disorder risk in offspring. En ligne : http://dx.doi.org/10.1177/13623613211007319 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=451 Prenatal exposure to paternal smoking and likelihood for autism spectrum disorder / Bora KIM in Autism, 26-7 (October 2022)
[article]
Titre : Prenatal exposure to paternal smoking and likelihood for autism spectrum disorder Type de document : Texte imprimé et/ou numérique Auteurs : Bora KIM, Auteur ; Mina HA, Auteur ; Young Shin KIM, Auteur ; Yun-Joo KOH, Auteur ; Shan DONG, Auteur ; Ho-Jang KWON, Auteur ; Young-Suk KIM, Auteur ; Myung-Ho LIM, Auteur ; Ki-Chung PAIK, Auteur ; Seung-Jin YOO, Auteur ; Hosanna KIM, Auteur ; Patricia S. HONG, Auteur ; Stephan J. SANDERS, Auteur ; Bennett L. LEVENTHAL, Auteur Article en page(s) : p.1946-1959 Langues : Anglais (eng) Mots-clés : Autism Spectrum Disorder/epidemiology/etiology Causality Family Female Humans Pregnancy Prenatal Exposure Delayed Effects/epidemiology/etiology Risk Factors Smoking/adverse effects autism spectrum disorders environmental factors risk factor epidemiology relation to the research presented in this manuscript. Index. décimale : PER Périodiques Résumé : What is Already Known about This Subject: Genetics, (including de novo mutations), environmental factors (including toxic exposures), and their interactions impact autism spectrum disorder etiology. Paternal smoking is a candidate risk for autism spectrum disorder due to biological plausibility, high prevalence, and potential intervention.What This Study Adds: This original study and its replication confirms that paternal factors can substantially contribute to autism spectrum disorder risk for their offspring. It specifically indicates that paternal smoking both before and during pregnancy contributes significantly to autism spectrum disorder risk.Implications for practice, research, or policy: Smoking prevention, especially in pregnancy planning, may decrease autism spectrum disorder risk in offspring. En ligne : http://dx.doi.org/10.1177/13623613211007319 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=484
in Autism > 26-7 (October 2022) . - p.1946-1959[article] Prenatal exposure to paternal smoking and likelihood for autism spectrum disorder [Texte imprimé et/ou numérique] / Bora KIM, Auteur ; Mina HA, Auteur ; Young Shin KIM, Auteur ; Yun-Joo KOH, Auteur ; Shan DONG, Auteur ; Ho-Jang KWON, Auteur ; Young-Suk KIM, Auteur ; Myung-Ho LIM, Auteur ; Ki-Chung PAIK, Auteur ; Seung-Jin YOO, Auteur ; Hosanna KIM, Auteur ; Patricia S. HONG, Auteur ; Stephan J. SANDERS, Auteur ; Bennett L. LEVENTHAL, Auteur . - p.1946-1959.
Langues : Anglais (eng)
in Autism > 26-7 (October 2022) . - p.1946-1959
Mots-clés : Autism Spectrum Disorder/epidemiology/etiology Causality Family Female Humans Pregnancy Prenatal Exposure Delayed Effects/epidemiology/etiology Risk Factors Smoking/adverse effects autism spectrum disorders environmental factors risk factor epidemiology relation to the research presented in this manuscript. Index. décimale : PER Périodiques Résumé : What is Already Known about This Subject: Genetics, (including de novo mutations), environmental factors (including toxic exposures), and their interactions impact autism spectrum disorder etiology. Paternal smoking is a candidate risk for autism spectrum disorder due to biological plausibility, high prevalence, and potential intervention.What This Study Adds: This original study and its replication confirms that paternal factors can substantially contribute to autism spectrum disorder risk for their offspring. It specifically indicates that paternal smoking both before and during pregnancy contributes significantly to autism spectrum disorder risk.Implications for practice, research, or policy: Smoking prevention, especially in pregnancy planning, may decrease autism spectrum disorder risk in offspring. En ligne : http://dx.doi.org/10.1177/13623613211007319 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=484 Alcohol and Tobacco use While Breastfeeding and Risk of Autism Spectrum Disorder or Attention Deficit/Hyperactivity Disorder / L. GIBSON in Journal of Autism and Developmental Disorders, 52-3 (March 2022)
[article]
Titre : Alcohol and Tobacco use While Breastfeeding and Risk of Autism Spectrum Disorder or Attention Deficit/Hyperactivity Disorder Type de document : Texte imprimé et/ou numérique Auteurs : L. GIBSON, Auteur ; M. PORTER, Auteur Article en page(s) : p.1223-1234 Langues : Anglais (eng) Mots-clés : Attention Deficit Disorder with Hyperactivity/epidemiology/etiology Autism Spectrum Disorder/epidemiology/etiology Breast Feeding Child Female Humans Infant Pregnancy Retrospective Studies Tobacco Use Alcohol Attention deficit/hyperactivity disorder Autism spectrum disorder Breastfeeding Tobacco Index. décimale : PER Périodiques Résumé : Research has linked prenatal alcohol and tobacco use with Attention Deficit Hyperactivity Disorder (ADHD), and variably with Autism Spectrum Disorder (ASD). Lactational use has been scantly considered. This study examined whether it may alter ADHD or ASD risk. Participants were 5107 infants recruited in 2004 and assessed longitudinally for the Growing Up in Australia Study. Logistic regression did not find any associations between maternal alcohol and tobacco use while breastfeeding and ADHD or ASD diagnosis at ages 6-7 or 10-11 years. Alcohol and tobacco use during lactation may not increase ADHD or ASD risk. Abstaining from alcohol and tobacco, however, may still be the safest option. Analyses were limited by lack of alcohol timing and retrospective variables that future research should address. En ligne : http://dx.doi.org/10.1007/s10803-021-05027-3 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=455
in Journal of Autism and Developmental Disorders > 52-3 (March 2022) . - p.1223-1234[article] Alcohol and Tobacco use While Breastfeeding and Risk of Autism Spectrum Disorder or Attention Deficit/Hyperactivity Disorder [Texte imprimé et/ou numérique] / L. GIBSON, Auteur ; M. PORTER, Auteur . - p.1223-1234.
Langues : Anglais (eng)
in Journal of Autism and Developmental Disorders > 52-3 (March 2022) . - p.1223-1234
Mots-clés : Attention Deficit Disorder with Hyperactivity/epidemiology/etiology Autism Spectrum Disorder/epidemiology/etiology Breast Feeding Child Female Humans Infant Pregnancy Retrospective Studies Tobacco Use Alcohol Attention deficit/hyperactivity disorder Autism spectrum disorder Breastfeeding Tobacco Index. décimale : PER Périodiques Résumé : Research has linked prenatal alcohol and tobacco use with Attention Deficit Hyperactivity Disorder (ADHD), and variably with Autism Spectrum Disorder (ASD). Lactational use has been scantly considered. This study examined whether it may alter ADHD or ASD risk. Participants were 5107 infants recruited in 2004 and assessed longitudinally for the Growing Up in Australia Study. Logistic regression did not find any associations between maternal alcohol and tobacco use while breastfeeding and ADHD or ASD diagnosis at ages 6-7 or 10-11 years. Alcohol and tobacco use during lactation may not increase ADHD or ASD risk. Abstaining from alcohol and tobacco, however, may still be the safest option. Analyses were limited by lack of alcohol timing and retrospective variables that future research should address. En ligne : http://dx.doi.org/10.1007/s10803-021-05027-3 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=455 Association of maternal autoimmune disease and early childhood infections with offspring autism spectrum disorder: A population-based cohort study / Timothy C. NIELSEN in Autism Research, 15-12 (December 2022)
[article]
Titre : Association of maternal autoimmune disease and early childhood infections with offspring autism spectrum disorder: A population-based cohort study Type de document : Texte imprimé et/ou numérique Auteurs : Timothy C. NIELSEN, Auteur ; Natasha NASSAR, Auteur ; Antonia W. SHAND, Auteur ; Hannah F. JONES, Auteur ; Velda X. HAN, Auteur ; Shrujna PATEL, Auteur ; Adam J. GUASTELLA, Auteur ; Russell C DALE, Auteur ; Samantha J. LAIN, Auteur Article en page(s) : p.2371-2380 Langues : Anglais (eng) Mots-clés : Child Child, Preschool Humans Autism Spectrum Disorder/epidemiology/etiology Cohort Studies Odds Ratio Logistic Models Autoimmune Diseases/epidemiology/complications Australia autism Spectrum disorder autoimmune diseases infections pregnancy Index. décimale : PER Périodiques Résumé : The aim of this study was to examine potential synergistic effects between maternal autoimmune disease and early childhood infections and their association with autism spectrum disorder (ASD) in offspring. Both exposures have been associated with increased risk of ASD in previous studies, but potential synergistic effects remain underexplored. We conducted a population-based cohort study of singleton children born at term gestation (37-41 weeks) in New South Wales, Australia from January 2002 to December 2008. Maternal autoimmune diagnoses and childhood infections before age 2 years were identified from linked maternal and child hospital admissions, and ASD diagnoses by age 9 years were identified from linked disability services data. Multivariable logistic regression assessed the association between each exposure and ASD and additive interaction between exposures, controlling for potential confounders. A total of 18,451 children exposed to maternal autoimmune disease were propensity score matched (1:2) to 36,902 unexposed children. Any maternal autoimmune disease (adjusted odds ratio (aOR) 1.25, 95% confidence interval (CI) 1.07-1.47) and any childhood infection before age 2 years (aOR 1.38, 95% CI 1.15-1.67) were each associated with ASD. However, there was no evidence of additive interaction between the two exposures (relative excess risk due to interaction [RERI] 0.128, 95% CI -0.418-0.675) resulting in increased odds of ASD in offspring. Future studies could examine potential interactions between other sources of maternal immune activation and childhood infection and impact on ASD and other neurodevelopmental disorders. En ligne : http://dx.doi.org/10.1002/aur.2824 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=488
in Autism Research > 15-12 (December 2022) . - p.2371-2380[article] Association of maternal autoimmune disease and early childhood infections with offspring autism spectrum disorder: A population-based cohort study [Texte imprimé et/ou numérique] / Timothy C. NIELSEN, Auteur ; Natasha NASSAR, Auteur ; Antonia W. SHAND, Auteur ; Hannah F. JONES, Auteur ; Velda X. HAN, Auteur ; Shrujna PATEL, Auteur ; Adam J. GUASTELLA, Auteur ; Russell C DALE, Auteur ; Samantha J. LAIN, Auteur . - p.2371-2380.
Langues : Anglais (eng)
in Autism Research > 15-12 (December 2022) . - p.2371-2380
Mots-clés : Child Child, Preschool Humans Autism Spectrum Disorder/epidemiology/etiology Cohort Studies Odds Ratio Logistic Models Autoimmune Diseases/epidemiology/complications Australia autism Spectrum disorder autoimmune diseases infections pregnancy Index. décimale : PER Périodiques Résumé : The aim of this study was to examine potential synergistic effects between maternal autoimmune disease and early childhood infections and their association with autism spectrum disorder (ASD) in offspring. Both exposures have been associated with increased risk of ASD in previous studies, but potential synergistic effects remain underexplored. We conducted a population-based cohort study of singleton children born at term gestation (37-41 weeks) in New South Wales, Australia from January 2002 to December 2008. Maternal autoimmune diagnoses and childhood infections before age 2 years were identified from linked maternal and child hospital admissions, and ASD diagnoses by age 9 years were identified from linked disability services data. Multivariable logistic regression assessed the association between each exposure and ASD and additive interaction between exposures, controlling for potential confounders. A total of 18,451 children exposed to maternal autoimmune disease were propensity score matched (1:2) to 36,902 unexposed children. Any maternal autoimmune disease (adjusted odds ratio (aOR) 1.25, 95% confidence interval (CI) 1.07-1.47) and any childhood infection before age 2 years (aOR 1.38, 95% CI 1.15-1.67) were each associated with ASD. However, there was no evidence of additive interaction between the two exposures (relative excess risk due to interaction [RERI] 0.128, 95% CI -0.418-0.675) resulting in increased odds of ASD in offspring. Future studies could examine potential interactions between other sources of maternal immune activation and childhood infection and impact on ASD and other neurodevelopmental disorders. En ligne : http://dx.doi.org/10.1002/aur.2824 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=488 The association of in utero tobacco smoke exposure, quantified by serum cotinine, and Autism Spectrum Disorder / K. BERGER in Autism Research, 14-9 (September 2021)
[article]
Titre : The association of in utero tobacco smoke exposure, quantified by serum cotinine, and Autism Spectrum Disorder Type de document : Texte imprimé et/ou numérique Auteurs : K. BERGER, Auteur ; M. PEARL, Auteur ; M. KHARRAZI, Auteur ; Y. LI, Auteur ; J. DEGUZMAN, Auteur ; J. SHE, Auteur ; P. BEHNIWAL, Auteur ; K. LYALL, Auteur ; G. WINDHAM, Auteur Article en page(s) : p.2017-2026 Langues : Anglais (eng) Mots-clés : Autism Spectrum Disorder/epidemiology/etiology Child Cotinine Female Humans Maternal Exposure/adverse effects Pregnancy Smoking Tobacco Smoke Pollution/adverse effects/analysis Autism Spectrum Disorder cotinine pregnancy smoking tobacco smoking Index. décimale : PER Périodiques Résumé : Previous studies on in utero exposure to maternal environmental tobacco smoke (ETS) or maternal active smoking and Autism Spectrum Disorder (ASD) have not been entirely consistent, and no studies have examined in utero cotinine concentrations as an exposure classification method. We measured cotinine in stored second trimester maternal serum for 498 ASD cases and 499 controls born in California in 2011-2012. We also obtained self-reported maternal cigarette smoking during and immediately prior to pregnancy, as well as covariate data, from birth records. Using unconditional logistic regression, we found no association between log10 cotinine concentrations and odds for developing ASD among children of non-smokers (aOR: 0.93 [95% CI: 0.69, 1.25] per ng/ml), which represents exposure to ETS, though there may be a possible interaction with race. We found no association between cotinine-defined smoking (?3.08?ng/ml vs. <3.08?ng/ml) (adjusted odds ratio [aOR]: 0.73 (95% confidence interval [95% CI]: 0.35, 1.54)) or self-reported smoking (aOR: 1.64 [95% CI: 0.65, 4.16]) and ASD. In one of the few studies of ETS and the first with measured cotinine, our results indicate no overall relationship between in utero exposure to tobacco smoke from maternal ETS exposure or active smoking, and development of ASD. LAY SUMMARY: This study found that women who smoke or are exposed to tobacco smoke during pregnancy are not more likely to have children with Autism Spectrum Disorder (ASD). This is the first ASD study to measure a chemical in the mother's blood during pregnancy to identify exposure to tobacco smoke. En ligne : http://dx.doi.org/10.1002/aur.2561 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=450
in Autism Research > 14-9 (September 2021) . - p.2017-2026[article] The association of in utero tobacco smoke exposure, quantified by serum cotinine, and Autism Spectrum Disorder [Texte imprimé et/ou numérique] / K. BERGER, Auteur ; M. PEARL, Auteur ; M. KHARRAZI, Auteur ; Y. LI, Auteur ; J. DEGUZMAN, Auteur ; J. SHE, Auteur ; P. BEHNIWAL, Auteur ; K. LYALL, Auteur ; G. WINDHAM, Auteur . - p.2017-2026.
Langues : Anglais (eng)
in Autism Research > 14-9 (September 2021) . - p.2017-2026
Mots-clés : Autism Spectrum Disorder/epidemiology/etiology Child Cotinine Female Humans Maternal Exposure/adverse effects Pregnancy Smoking Tobacco Smoke Pollution/adverse effects/analysis Autism Spectrum Disorder cotinine pregnancy smoking tobacco smoking Index. décimale : PER Périodiques Résumé : Previous studies on in utero exposure to maternal environmental tobacco smoke (ETS) or maternal active smoking and Autism Spectrum Disorder (ASD) have not been entirely consistent, and no studies have examined in utero cotinine concentrations as an exposure classification method. We measured cotinine in stored second trimester maternal serum for 498 ASD cases and 499 controls born in California in 2011-2012. We also obtained self-reported maternal cigarette smoking during and immediately prior to pregnancy, as well as covariate data, from birth records. Using unconditional logistic regression, we found no association between log10 cotinine concentrations and odds for developing ASD among children of non-smokers (aOR: 0.93 [95% CI: 0.69, 1.25] per ng/ml), which represents exposure to ETS, though there may be a possible interaction with race. We found no association between cotinine-defined smoking (?3.08?ng/ml vs. <3.08?ng/ml) (adjusted odds ratio [aOR]: 0.73 (95% confidence interval [95% CI]: 0.35, 1.54)) or self-reported smoking (aOR: 1.64 [95% CI: 0.65, 4.16]) and ASD. In one of the few studies of ETS and the first with measured cotinine, our results indicate no overall relationship between in utero exposure to tobacco smoke from maternal ETS exposure or active smoking, and development of ASD. LAY SUMMARY: This study found that women who smoke or are exposed to tobacco smoke during pregnancy are not more likely to have children with Autism Spectrum Disorder (ASD). This is the first ASD study to measure a chemical in the mother's blood during pregnancy to identify exposure to tobacco smoke. En ligne : http://dx.doi.org/10.1002/aur.2561 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=450