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Infant temperament prospectively predicts general psychopathology in childhood / Santiago MORALES in Development and Psychopathology, 34-3 (August 2022)
[article]
Titre : Infant temperament prospectively predicts general psychopathology in childhood Type de document : Texte imprimé et/ou numérique Auteurs : Santiago MORALES, Auteur ; Alva TANG, Auteur ; Maureen E. BOWERS, Auteur ; Natalie V. MILLER, Auteur ; George A. BUZZELL, Auteur ; Elizabeth SMITH, Auteur ; Kaylee SEDDIO, Auteur ; Heather A. HENDERSON, Auteur ; Nathan A. FOX, Auteur Article en page(s) : p.774-783 Langues : Anglais (eng) Mots-clés : general factor motor activity p factor psychopathology temperament Index. décimale : PER Périodiques Résumé : Recent models of psychopathology suggest the presence of a general factor capturing the shared variance among all symptoms along with specific psychopathology factors (e.g., internalizing and externalizing). However, few studies have examined predictors that may serve as transdiagnostic risk factors for general psychopathology from early development. In the current study we examine, for the first time, whether observed and parent-reported infant temperament dimensions prospectively predict general psychopathology as well as specific psychopathology dimensions (e.g., internalizing and externalizing) across childhood. In a longitudinal cohort (N = 291), temperament dimensions were assessed at 4 months of age. Psychopathology symptoms were assessed at 7, 9, and 12 years of age. A bifactor model was used to estimate general, internalizing, and externalizing psychopathology factors. Across behavioral observations and parent-reports, higher motor activity in infancy significantly predicted greater general psychopathology in mid to late childhood. Moreover, low positive affect was predictive of the internalizing-specific factor. Other temperament dimensions were not related with any of the psychopathology factors after accounting for the general psychopathology factor. The results of this study suggest that infant motor activity may act as an early indicator of transdiagnostic risk. Our findings inform the etiology of general psychopathology and have implications for the early identification for children at risk for psychopathology. En ligne : http://dx.doi.org/10.1017/S0954579420001996 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=484
in Development and Psychopathology > 34-3 (August 2022) . - p.774-783[article] Infant temperament prospectively predicts general psychopathology in childhood [Texte imprimé et/ou numérique] / Santiago MORALES, Auteur ; Alva TANG, Auteur ; Maureen E. BOWERS, Auteur ; Natalie V. MILLER, Auteur ; George A. BUZZELL, Auteur ; Elizabeth SMITH, Auteur ; Kaylee SEDDIO, Auteur ; Heather A. HENDERSON, Auteur ; Nathan A. FOX, Auteur . - p.774-783.
Langues : Anglais (eng)
in Development and Psychopathology > 34-3 (August 2022) . - p.774-783
Mots-clés : general factor motor activity p factor psychopathology temperament Index. décimale : PER Périodiques Résumé : Recent models of psychopathology suggest the presence of a general factor capturing the shared variance among all symptoms along with specific psychopathology factors (e.g., internalizing and externalizing). However, few studies have examined predictors that may serve as transdiagnostic risk factors for general psychopathology from early development. In the current study we examine, for the first time, whether observed and parent-reported infant temperament dimensions prospectively predict general psychopathology as well as specific psychopathology dimensions (e.g., internalizing and externalizing) across childhood. In a longitudinal cohort (N = 291), temperament dimensions were assessed at 4 months of age. Psychopathology symptoms were assessed at 7, 9, and 12 years of age. A bifactor model was used to estimate general, internalizing, and externalizing psychopathology factors. Across behavioral observations and parent-reports, higher motor activity in infancy significantly predicted greater general psychopathology in mid to late childhood. Moreover, low positive affect was predictive of the internalizing-specific factor. Other temperament dimensions were not related with any of the psychopathology factors after accounting for the general psychopathology factor. The results of this study suggest that infant motor activity may act as an early indicator of transdiagnostic risk. Our findings inform the etiology of general psychopathology and have implications for the early identification for children at risk for psychopathology. En ligne : http://dx.doi.org/10.1017/S0954579420001996 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=484 Brinp1(-/-) mice exhibit autism-like behaviour, altered memory, hyperactivity and increased parvalbumin-positive cortical interneuron density / S. R. BERKOWICZ in Molecular Autism, 7 (2016)
[article]
Titre : Brinp1(-/-) mice exhibit autism-like behaviour, altered memory, hyperactivity and increased parvalbumin-positive cortical interneuron density Type de document : Texte imprimé et/ou numérique Auteurs : S. R. BERKOWICZ, Auteur ; T. J. FEATHERBY, Auteur ; Z. QU, Auteur ; A. GIOUSOH, Auteur ; N. A. BORG, Auteur ; J. I. HENG, Auteur ; J. C. WHISSTOCK, Auteur ; P. I. BIRD, Auteur Article en page(s) : 22p. Langues : Anglais (eng) Mots-clés : Animals Attention Deficit Disorder with Hyperactivity/metabolism/pathology Autism Spectrum Disorder/metabolism/pathology Behavior, Animal Brain/metabolism/pathology Disease Models, Animal Female Genotype Glycoproteins/genetics/metabolism Interneurons/metabolism Male Memory, Short-Term Mice Mice, Inbred C57BL Mice, Knockout Motor Activity Nerve Tissue Proteins/deficiency/genetics/metabolism Parvalbumins/genetics/metabolism Phenotype Real-Time Polymerase Chain Reaction Vocalization, Animal Autism spectrum disorder Brinp1 Cortex Hyperactivity Interneuron Knock-out Neurodevelopment Parvalbumin Index. décimale : PER Périodiques Résumé : BACKGROUND: BMP/RA-inducible neural-specific protein 1 (Brinp1) is highly conserved in vertebrates, and continuously expressed in the neocortex, hippocampus, olfactory bulb and cerebellum from mid-embryonic development through to adulthood. METHODS: Brinp1 knock-out (Brinp1(-/-)) mice were generated by Cre-recombinase-mediated removal of the third exon of Brinp1. Knock-out mice were characterised by behavioural phenotyping, immunohistochemistry and expression analysis of the developing and adult brain. RESULTS: Absence of Brinp1 during development results in a behavioural phenotype resembling autism spectrum disorder (ASD), in which knock-out mice show reduced sociability and changes in vocalisation capacity. In addition, Brinp1(-/-) mice exhibit hyper-locomotor activity, have impaired short-term memory, and exhibit poor reproductive success. Brinp1(-/-) mice show increased density of parvalbumin-expressing interneurons in the adult mouse brain. Brinp1(-/-) mice do not show signs of altered neural precursor proliferation or increased apoptosis during late embryonic brain development. The expression of the related neuronal migration genes Astn1 and Astn2 is increased in the brains of Brinp1(-/-) mice, suggesting that they may ameliorate the effects of Brinp1 loss. CONCLUSIONS: Brinp1 plays an important role in normal brain development and function by influencing neuronal distribution within the cortex. The increased cortical PV-positive interneuron density and altered behaviour of Brinp1(-/-) mice resemble features of a subset of human neurological disorders; namely autism spectrum disorder (ASD) and the hyperactivity aspect of attention deficit hyperactivity disorder (ADHD). En ligne : http://dx.doi.org/10.1186/s13229-016-0079-7 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=328
in Molecular Autism > 7 (2016) . - 22p.[article] Brinp1(-/-) mice exhibit autism-like behaviour, altered memory, hyperactivity and increased parvalbumin-positive cortical interneuron density [Texte imprimé et/ou numérique] / S. R. BERKOWICZ, Auteur ; T. J. FEATHERBY, Auteur ; Z. QU, Auteur ; A. GIOUSOH, Auteur ; N. A. BORG, Auteur ; J. I. HENG, Auteur ; J. C. WHISSTOCK, Auteur ; P. I. BIRD, Auteur . - 22p.
Langues : Anglais (eng)
in Molecular Autism > 7 (2016) . - 22p.
Mots-clés : Animals Attention Deficit Disorder with Hyperactivity/metabolism/pathology Autism Spectrum Disorder/metabolism/pathology Behavior, Animal Brain/metabolism/pathology Disease Models, Animal Female Genotype Glycoproteins/genetics/metabolism Interneurons/metabolism Male Memory, Short-Term Mice Mice, Inbred C57BL Mice, Knockout Motor Activity Nerve Tissue Proteins/deficiency/genetics/metabolism Parvalbumins/genetics/metabolism Phenotype Real-Time Polymerase Chain Reaction Vocalization, Animal Autism spectrum disorder Brinp1 Cortex Hyperactivity Interneuron Knock-out Neurodevelopment Parvalbumin Index. décimale : PER Périodiques Résumé : BACKGROUND: BMP/RA-inducible neural-specific protein 1 (Brinp1) is highly conserved in vertebrates, and continuously expressed in the neocortex, hippocampus, olfactory bulb and cerebellum from mid-embryonic development through to adulthood. METHODS: Brinp1 knock-out (Brinp1(-/-)) mice were generated by Cre-recombinase-mediated removal of the third exon of Brinp1. Knock-out mice were characterised by behavioural phenotyping, immunohistochemistry and expression analysis of the developing and adult brain. RESULTS: Absence of Brinp1 during development results in a behavioural phenotype resembling autism spectrum disorder (ASD), in which knock-out mice show reduced sociability and changes in vocalisation capacity. In addition, Brinp1(-/-) mice exhibit hyper-locomotor activity, have impaired short-term memory, and exhibit poor reproductive success. Brinp1(-/-) mice show increased density of parvalbumin-expressing interneurons in the adult mouse brain. Brinp1(-/-) mice do not show signs of altered neural precursor proliferation or increased apoptosis during late embryonic brain development. The expression of the related neuronal migration genes Astn1 and Astn2 is increased in the brains of Brinp1(-/-) mice, suggesting that they may ameliorate the effects of Brinp1 loss. CONCLUSIONS: Brinp1 plays an important role in normal brain development and function by influencing neuronal distribution within the cortex. The increased cortical PV-positive interneuron density and altered behaviour of Brinp1(-/-) mice resemble features of a subset of human neurological disorders; namely autism spectrum disorder (ASD) and the hyperactivity aspect of attention deficit hyperactivity disorder (ADHD). En ligne : http://dx.doi.org/10.1186/s13229-016-0079-7 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=328 Priming Global Processing Strategy Improves the Perceptual Performance of Children with Autism Spectrum Disorders / G. SOROOR in Journal of Autism and Developmental Disorders, 52-3 (March 2022)
[article]
Titre : Priming Global Processing Strategy Improves the Perceptual Performance of Children with Autism Spectrum Disorders Type de document : Texte imprimé et/ou numérique Auteurs : G. SOROOR, Auteur ; S. MOKHTARI, Auteur ; H. POURETEMAD, Auteur Article en page(s) : p.1019-1029 Langues : Anglais (eng) Mots-clés : Autism Spectrum Disorder Child Humans Motor Activity Autism spectrum disorders (ASD) Global processing strategy Illusory shape Perceptual integration Perceptual priming Index. décimale : PER Périodiques Résumé : We investigated (1) if the perceptual integration performance is different in children with ASD in comparison with their typically developed (TD) counterparts; and (2) if activating- priming- the global processing strategy, could benefit the integration performance of children with ASD in the subsequent task. We observed that in comparison with the TD group, children with ASD had lower performance in an information integration task that required identification of illusory shapes. Additionally, we observed that priming the global processing strategy increased the correct identification of the illusory shapes in the subsequent task. We suggested that studies on priming effect shed light on the different aspects of perceptual properties of ASD, and could also be used in developing new rehabilitation plans. En ligne : http://dx.doi.org/10.1007/s10803-021-05007-7 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=455
in Journal of Autism and Developmental Disorders > 52-3 (March 2022) . - p.1019-1029[article] Priming Global Processing Strategy Improves the Perceptual Performance of Children with Autism Spectrum Disorders [Texte imprimé et/ou numérique] / G. SOROOR, Auteur ; S. MOKHTARI, Auteur ; H. POURETEMAD, Auteur . - p.1019-1029.
Langues : Anglais (eng)
in Journal of Autism and Developmental Disorders > 52-3 (March 2022) . - p.1019-1029
Mots-clés : Autism Spectrum Disorder Child Humans Motor Activity Autism spectrum disorders (ASD) Global processing strategy Illusory shape Perceptual integration Perceptual priming Index. décimale : PER Périodiques Résumé : We investigated (1) if the perceptual integration performance is different in children with ASD in comparison with their typically developed (TD) counterparts; and (2) if activating- priming- the global processing strategy, could benefit the integration performance of children with ASD in the subsequent task. We observed that in comparison with the TD group, children with ASD had lower performance in an information integration task that required identification of illusory shapes. Additionally, we observed that priming the global processing strategy increased the correct identification of the illusory shapes in the subsequent task. We suggested that studies on priming effect shed light on the different aspects of perceptual properties of ASD, and could also be used in developing new rehabilitation plans. En ligne : http://dx.doi.org/10.1007/s10803-021-05007-7 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=455