54 recherche sur le mot-clé 'Childhood'

Childhood high-frequency EEG activity during sleep is associated with incident insomnia symptoms in adolescence / Julio FERNANDEZ-MENDOZA in Journal of Child Psychology and Psychiatry, 60-7 (July 2019)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 60-7 (July 2019) . - p.742-751 Titre : Childhood high-frequency EEG activity during sleep is associated with incident insomnia symptoms in adolescence Type de document : texte imprimé Auteurs : Julio FERNANDEZ-MENDOZA, Auteur ; Yun LI, Auteur ; Jing FANG, Auteur ; Susan L. CALHOUN, Auteur ; Alexandros N. VGONTZAS, Auteur ; Duanping LIAO, Auteur ; Edward O BIXLER, Auteur Année de publication : 2019 Article en page(s) : p.742-751 Langues : Anglais (eng) Mots-clés : adolescence  beta  childhood  electroencephalogram  hyperarousal  incidence  insomnia symptoms Index. décimale : PER Périodiques Résumé : BACKGROUND: Insomnia has been associated in cross-sectional studies with increased beta (15-35 Hz) electroencephalogram (EEG) power during nonrapid eye movement (NREM) sleep, an index of cortical hyperarousal. However, it is unknown whether this cortical hyperarousal is present before individuals with insomnia develop the disorder. To fill this gap, we examined the association of childhood sleep high-frequency EEG activity with incident insomnia symptoms (i.e., absence of insomnia symptoms in childhood but presence in adolescence). METHODS: We studied a case-control subsample of 45 children (6-11 years) from the Penn State Child Cohort, a population-based random sample of 421 children, who were followed up after 8 years as adolescents (13-20 years). We examined low-beta (15-25 Hz) and high-beta (25-35 Hz) relative power at central EEG derivations during NREM sleep and, in secondary analyses, during sleep onset latency, sleep onset, and REM sleep. Incident insomnia symptoms were defined as the absence of parent-reported difficulty falling and/or staying asleep during childhood and a self-report of these insomnia symptoms during adolescence. RESULTS: Childhood high-beta power during NREM sleep was significantly increased in children who developed insomnia symptoms in adolescence (n = 25) as compared to normal sleeping controls (n = 20; p = .03). Multivariable-adjusted logistic regression models showed that increased childhood high-beta EEG power during NREM sleep was associated with a threefold increased odds (95% CI = 1.12-7.98) of incident insomnia symptoms in adolescence. No other significant relationships were observed for other sleep/wake states or EEG frequency bands. CONCLUSIONS: Increased childhood high-frequency EEG power during NREM sleep is associated with incident insomnia symptoms in adolescence. This study indicates that cortical hyperarousal during sleep may be a premorbid neurophysiological sign of insomnia, which may mediate the increased risk of psychiatric disorders associated with insomnia. En ligne : http://dx.doi.org/10.1111/jcpp.12945 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=401 [article] Childhood high-frequency EEG activity during sleep is associated with incident insomnia symptoms in adolescence [texte imprimé] / Julio FERNANDEZ-MENDOZA, Auteur ; Yun LI, Auteur ; Jing FANG, Auteur ; Susan L. CALHOUN, Auteur ; Alexandros N. VGONTZAS, Auteur ; Duanping LIAO, Auteur ; Edward O BIXLER, Auteur . - 2019 . - p.742-751. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 60-7 (July 2019) . - p.742-751 Mots-clés : adolescence  beta  childhood  electroencephalogram  hyperarousal  incidence  insomnia symptoms Index. décimale : PER Périodiques Résumé : BACKGROUND: Insomnia has been associated in cross-sectional studies with increased beta (15-35 Hz) electroencephalogram (EEG) power during nonrapid eye movement (NREM) sleep, an index of cortical hyperarousal. However, it is unknown whether this cortical hyperarousal is present before individuals with insomnia develop the disorder. To fill this gap, we examined the association of childhood sleep high-frequency EEG activity with incident insomnia symptoms (i.e., absence of insomnia symptoms in childhood but presence in adolescence). METHODS: We studied a case-control subsample of 45 children (6-11 years) from the Penn State Child Cohort, a population-based random sample of 421 children, who were followed up after 8 years as adolescents (13-20 years). We examined low-beta (15-25 Hz) and high-beta (25-35 Hz) relative power at central EEG derivations during NREM sleep and, in secondary analyses, during sleep onset latency, sleep onset, and REM sleep. Incident insomnia symptoms were defined as the absence of parent-reported difficulty falling and/or staying asleep during childhood and a self-report of these insomnia symptoms during adolescence. RESULTS: Childhood high-beta power during NREM sleep was significantly increased in children who developed insomnia symptoms in adolescence (n = 25) as compared to normal sleeping controls (n = 20; p = .03). Multivariable-adjusted logistic regression models showed that increased childhood high-beta EEG power during NREM sleep was associated with a threefold increased odds (95% CI = 1.12-7.98) of incident insomnia symptoms in adolescence. No other significant relationships were observed for other sleep/wake states or EEG frequency bands. CONCLUSIONS: Increased childhood high-frequency EEG power during NREM sleep is associated with incident insomnia symptoms in adolescence. This study indicates that cortical hyperarousal during sleep may be a premorbid neurophysiological sign of insomnia, which may mediate the increased risk of psychiatric disorders associated with insomnia. En ligne : http://dx.doi.org/10.1111/jcpp.12945 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=401

Childhood infections and autism spectrum disorders and/or intellectual disability: a register-based cohort study / HÃ¥kan KARLSSON in Journal of Neurodevelopmental Disorders, 14 (2022)  

Public ISBD [article]  inJournal of Neurodevelopmental Disorders > 14 (2022) Titre : Childhood infections and autism spectrum disorders and/or intellectual disability: a register-based cohort study Type de document : texte imprimé Auteurs : HÃ¥kan KARLSSON, Auteur ; Hugo SJÖQVIST, Auteur ; Martin BRYNGE, Auteur ; Renee GARDNER, Auteur ; Christina DALMAN, Auteur Langues : Anglais (eng) Mots-clés : Autism Spectrum Disorder/complications/diagnosis/epidemiology  Child  Cohort Studies  Female  Humans  Intellectual Disability/complications/diagnosis/epidemiology  Pregnancy  Siblings  Autism spectrum disorders  Childhood  Infection  Intellectual disability  Risk Index. décimale : PER Périodiques Résumé : OBJECTIVE: To explore the associations between childhood infections and subsequent diagnoses of autism spectrum disorder (ASD), intellectual disability (ID), and their co-occurrence. METHODS: The association between specialized care for any infection, defined by ICD-codes, and later ASD or ID was investigated in a register-based cohort of 556,732 individuals born 1987-2010, resident in Stockholm County, followed from birth to their 18th birthday or December 31, 2016. We considered as potential confounders children's characteristics, family socioeconomic factors, obstetric complications, and parental histories of treatment for infection and psychiatric disorders in survival analyses with extended Cox regression models. Residual confounding by shared familial factors was addressed in sibling analyses using within-strata estimation in Cox regression models. Sensitivity analyses with the exclusion of congenital causes of ASD/ID and documented risk for infections were also performed. RESULTS: Crude estimates indicated that infections during childhood were associated with later ASD and ID with the largest risks observed for diagnoses involving ID. Inclusion of covariates, exclusion of congenital causes of ASD/ID from the population, and sibling comparisons highlighted the potential for confounding by both heritable and non-heritable factors, though risks remained in all adjusted models. In adjusted sibling comparisons, excluding congenital causes, infections were associated with later "ASD without ID" (HR 1.24, 95%CI 1.15-1.33), "ASD with ID" (1.57, 1.35-1.82), and "ID without ASD" (2.01, 1.76-2.28). Risks associated with infections varied by age at exposure and by age at diagnosis of ASD/ID. CONCLUSIONS: Infections during childhood may contribute to a later diagnosis of ID and ASD. En ligne : https://dx.doi.org/10.1186/s11689-022-09422-4 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=574 [article] Childhood infections and autism spectrum disorders and/or intellectual disability: a register-based cohort study [texte imprimé] / HÃ¥kan KARLSSON, Auteur ; Hugo SJÖQVIST, Auteur ; Martin BRYNGE, Auteur ; Renee GARDNER, Auteur ; Christina DALMAN, Auteur. Langues : Anglais (eng) in Journal of Neurodevelopmental Disorders > 14 (2022) Mots-clés : Autism Spectrum Disorder/complications/diagnosis/epidemiology  Child  Cohort Studies  Female  Humans  Intellectual Disability/complications/diagnosis/epidemiology  Pregnancy  Siblings  Autism spectrum disorders  Childhood  Infection  Intellectual disability  Risk Index. décimale : PER Périodiques Résumé : OBJECTIVE: To explore the associations between childhood infections and subsequent diagnoses of autism spectrum disorder (ASD), intellectual disability (ID), and their co-occurrence. METHODS: The association between specialized care for any infection, defined by ICD-codes, and later ASD or ID was investigated in a register-based cohort of 556,732 individuals born 1987-2010, resident in Stockholm County, followed from birth to their 18th birthday or December 31, 2016. We considered as potential confounders children's characteristics, family socioeconomic factors, obstetric complications, and parental histories of treatment for infection and psychiatric disorders in survival analyses with extended Cox regression models. Residual confounding by shared familial factors was addressed in sibling analyses using within-strata estimation in Cox regression models. Sensitivity analyses with the exclusion of congenital causes of ASD/ID and documented risk for infections were also performed. RESULTS: Crude estimates indicated that infections during childhood were associated with later ASD and ID with the largest risks observed for diagnoses involving ID. Inclusion of covariates, exclusion of congenital causes of ASD/ID from the population, and sibling comparisons highlighted the potential for confounding by both heritable and non-heritable factors, though risks remained in all adjusted models. In adjusted sibling comparisons, excluding congenital causes, infections were associated with later "ASD without ID" (HR 1.24, 95%CI 1.15-1.33), "ASD with ID" (1.57, 1.35-1.82), and "ID without ASD" (2.01, 1.76-2.28). Risks associated with infections varied by age at exposure and by age at diagnosis of ASD/ID. CONCLUSIONS: Infections during childhood may contribute to a later diagnosis of ID and ASD. En ligne : https://dx.doi.org/10.1186/s11689-022-09422-4 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=574

Childhood predictors of avoidant personality disorder traits in adolescence: a seven-wave birth cohort study / Theresa WILBERG ; Elfrida HARTVEIT KVARSTEIN ; Silje STEINSBEKK in Journal of Child Psychology and Psychiatry, 66-3 (March 2025)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 66-3 (March 2025) . - p.366-377 Titre : Childhood predictors of avoidant personality disorder traits in adolescence: a seven-wave birth cohort study Type de document : texte imprimé Auteurs : Theresa WILBERG, Auteur ; Elfrida HARTVEIT KVARSTEIN, Auteur ; Silje STEINSBEKK, Auteur Article en page(s) : p.366-377 Langues : Anglais (eng) Mots-clés : Adolescent  attachment  avoidant personality disorder  childhood  longitudinal Index. décimale : PER Périodiques Résumé : Background Although it is widely assumed that avoidant personality disorder (AvPD) originates in childhood, there is little prospective research to substantiate this claim. We therefore aimed to determine whether presumed childhood risk factors predict AvPD traits at 16 years. Methods A population-based sample (n 1,077; 50.9% female) from the 2003 and 2004 birth cohorts in Trondheim, Norway was examined biennially from 4 to 16 years. The number of AvPD traits at the age of 16 was assessed with the structured clinical interview for DSM-5 personality disorders and regressed on the intercept and growth in child risk and protective factors until the age of 14. Results The prevalence of AvPD at the age of 16 was 3.2% (95% CI: 2.2 4.1). Higher levels and an increased number of social anxiety symptoms over time, as well as increased negative affectivity/neuroticism, predicted a higher number of AvPD traits. When the levels and changes in these factors were adjusted for, less and decreasing extraversion forecasted more AvPD traits, as did declining self-worth, higher levels of parental AvPD traits, and increased onlooking behavior. Conclusions Neuroticism, low extraversion, social anxiety symptoms, passive onlooking behavior, and low self-worth predicted a higher number of AvPD traits in adolescence, as did more AvPD traits in parents. Efforts to enhance self-worth, reduce social anxiety, and promote peer interaction among onlooking children may reduce the development of AvPD traits in adolescence. En ligne : https://doi.org/10.1111/jcpp.14064 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=548 [article] Childhood predictors of avoidant personality disorder traits in adolescence: a seven-wave birth cohort study [texte imprimé] / Theresa WILBERG, Auteur ; Elfrida HARTVEIT KVARSTEIN, Auteur ; Silje STEINSBEKK, Auteur . - p.366-377. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 66-3 (March 2025) . - p.366-377 Mots-clés : Adolescent  attachment  avoidant personality disorder  childhood  longitudinal Index. décimale : PER Périodiques Résumé : Background Although it is widely assumed that avoidant personality disorder (AvPD) originates in childhood, there is little prospective research to substantiate this claim. We therefore aimed to determine whether presumed childhood risk factors predict AvPD traits at 16 years. Methods A population-based sample (n 1,077; 50.9% female) from the 2003 and 2004 birth cohorts in Trondheim, Norway was examined biennially from 4 to 16 years. The number of AvPD traits at the age of 16 was assessed with the structured clinical interview for DSM-5 personality disorders and regressed on the intercept and growth in child risk and protective factors until the age of 14. Results The prevalence of AvPD at the age of 16 was 3.2% (95% CI: 2.2 4.1). Higher levels and an increased number of social anxiety symptoms over time, as well as increased negative affectivity/neuroticism, predicted a higher number of AvPD traits. When the levels and changes in these factors were adjusted for, less and decreasing extraversion forecasted more AvPD traits, as did declining self-worth, higher levels of parental AvPD traits, and increased onlooking behavior. Conclusions Neuroticism, low extraversion, social anxiety symptoms, passive onlooking behavior, and low self-worth predicted a higher number of AvPD traits in adolescence, as did more AvPD traits in parents. Efforts to enhance self-worth, reduce social anxiety, and promote peer interaction among onlooking children may reduce the development of AvPD traits in adolescence. En ligne : https://doi.org/10.1111/jcpp.14064 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=548

Childhood unpredictability research within the developmental psychopathology framework: Advances, implications, and future directions / Jenalee R. DOOM ; Deborah HAN ; Kenia M. RIVERA ; Tenzin TSETEN in Development and Psychopathology, 36-5 (December 2024)  

Public ISBD [article]  inDevelopment and Psychopathology > 36-5 (December 2024) . - p.2452-2463 Titre : Childhood unpredictability research within the developmental psychopathology framework: Advances, implications, and future directions : Development and Psychopathology Type de document : texte imprimé Auteurs : Jenalee R. DOOM, Auteur ; Deborah HAN, Auteur ; Kenia M. RIVERA, Auteur ; Tenzin TSETEN, Auteur Année de publication : 2024 Article en page(s) : p.2452-2463 Langues : Anglais (eng) Mots-clés : childhood  developmental psychopathology  interventions  unpredictability Index. décimale : PER Périodiques Résumé : Greater unpredictability in childhood from the level of the caregiver-child dyad to broader family, home, or environmental instability is consistently associated with disruptions in cognitive, socioemotional, behavioral, and biological development in humans. These findings are bolstered by experimental research in non-human animal models suggesting that early life unpredictability is an important environmental signal to the developing organism that shapes neurodevelopment and behavior. Research on childhood unpredictability has surged in the past several years, guided in part by theoretical grounding from the developmental psychopathology framework (shaped largely by Dr. Dante Cicchetti?s innovative work). The current review focuses on future directions for unpredictability research, including probing intergenerational effects, the role of predictability in resilience, cultural and contextual considerations, and novel developmental outcomes that should be tested in relation to childhood unpredictability. We urge the integration of multidisciplinary perspectives and collaborations into future research on unpredictability. We also provide ideas for translating this research to real-world practice and policy and encourage high-quality research testing whether incorporating predictability into interventions and policy improves developmental outcomes, which would support further dissemination of these findings. En ligne : https://dx.doi.org/10.1017/S0954579424000610 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=545 [article] Childhood unpredictability research within the developmental psychopathology framework: Advances, implications, and future directions : Development and Psychopathology [texte imprimé] / Jenalee R. DOOM, Auteur ; Deborah HAN, Auteur ; Kenia M. RIVERA, Auteur ; Tenzin TSETEN, Auteur . - 2024 . - p.2452-2463. Langues : Anglais (eng) in Development and Psychopathology > 36-5 (December 2024) . - p.2452-2463 Mots-clés : childhood  developmental psychopathology  interventions  unpredictability Index. décimale : PER Périodiques Résumé : Greater unpredictability in childhood from the level of the caregiver-child dyad to broader family, home, or environmental instability is consistently associated with disruptions in cognitive, socioemotional, behavioral, and biological development in humans. These findings are bolstered by experimental research in non-human animal models suggesting that early life unpredictability is an important environmental signal to the developing organism that shapes neurodevelopment and behavior. Research on childhood unpredictability has surged in the past several years, guided in part by theoretical grounding from the developmental psychopathology framework (shaped largely by Dr. Dante Cicchetti?s innovative work). The current review focuses on future directions for unpredictability research, including probing intergenerational effects, the role of predictability in resilience, cultural and contextual considerations, and novel developmental outcomes that should be tested in relation to childhood unpredictability. We urge the integration of multidisciplinary perspectives and collaborations into future research on unpredictability. We also provide ideas for translating this research to real-world practice and policy and encourage high-quality research testing whether incorporating predictability into interventions and policy improves developmental outcomes, which would support further dissemination of these findings. En ligne : https://dx.doi.org/10.1017/S0954579424000610 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=545

Allostasis and metastasis: The yin and yang of childhood self-regulation / Samuel V. WASS in Development and Psychopathology, 35-1 (February 2023)  

Public ISBD [article]  inDevelopment and Psychopathology > 35-1 (February 2023) . - p.179-190 Titre : Allostasis and metastasis: The yin and yang of childhood self-regulation Type de document : texte imprimé Auteurs : Samuel V. WASS, Auteur Article en page(s) : p.179-190 Langues : Anglais (eng) Mots-clés : attention control  childhood  emotion reactivity  emotion regulation  infancy  self-control  self-regulation Index. décimale : PER Périodiques Résumé : Most research has studied self-regulation by presenting experimenter-controlled test stimuli and measuring change between baseline and stimulus. In the real world, however, stressors do not flash on and off in a predetermined sequence, and there is no experimenter controlling things. Rather, the real world is continuous and stressful events can occur through self-sustaining interactive chain reactions. Self-regulation is an active process through which we adaptively select which aspects of the social environment we attend to from one moment to the next. Here, we describe this dynamic interactive process by contrasting two mechanisms that underpin it: the ''yin'' and ''yang'' of self-regulation. The first mechanism is allostasis, the dynamical principle underlying self-regulation, through which we compensate for change to maintain homeostasis. This involves upregulating in some situations and downregulating in others. The second mechanism is metastasis, the dynamical principle underling dysregulation. Through metastasis, small initial perturbations can become progressively amplified over time. We contrast these processes at the individual level (i.e., examining moment-to-moment change in one child, considered independently) and also at the inter-personal level (i.e., examining change across a dyad, such as a parent-child dyad). Finally, we discuss practical implications of this approach in improving the self-regulation of emotion and cognition, in typical development and psychopathology. En ligne : https://doi.org/10.1017/S0954579421000833 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=499 [article] Allostasis and metastasis: The yin and yang of childhood self-regulation [texte imprimé] / Samuel V. WASS, Auteur . - p.179-190. Langues : Anglais (eng) in Development and Psychopathology > 35-1 (February 2023) . - p.179-190 Mots-clés : attention control  childhood  emotion reactivity  emotion regulation  infancy  self-control  self-regulation Index. décimale : PER Périodiques Résumé : Most research has studied self-regulation by presenting experimenter-controlled test stimuli and measuring change between baseline and stimulus. In the real world, however, stressors do not flash on and off in a predetermined sequence, and there is no experimenter controlling things. Rather, the real world is continuous and stressful events can occur through self-sustaining interactive chain reactions. Self-regulation is an active process through which we adaptively select which aspects of the social environment we attend to from one moment to the next. Here, we describe this dynamic interactive process by contrasting two mechanisms that underpin it: the ''yin'' and ''yang'' of self-regulation. The first mechanism is allostasis, the dynamical principle underlying self-regulation, through which we compensate for change to maintain homeostasis. This involves upregulating in some situations and downregulating in others. The second mechanism is metastasis, the dynamical principle underling dysregulation. Through metastasis, small initial perturbations can become progressively amplified over time. We contrast these processes at the individual level (i.e., examining moment-to-moment change in one child, considered independently) and also at the inter-personal level (i.e., examining change across a dyad, such as a parent-child dyad). Finally, we discuss practical implications of this approach in improving the self-regulation of emotion and cognition, in typical development and psychopathology. En ligne : https://doi.org/10.1017/S0954579421000833 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=499

Commentary: A social environment approach to promotive and protective practice in childhood resilience – reflections on Ungar (2014) / Michael G. WESSELLS in Journal of Child Psychology and Psychiatry, 56-1 (January 2015)  

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Exploring potential sources of childhood trauma: A qualitative study with autistic adults and caregivers / Connor M. KERNS in Autism, 26-8 (November 2022)  

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Family income in early childhood and subsequent attention deficit/hyperactivity disorder: a quasi-experimental study / Henrik LARSSON in Journal of Child Psychology and Psychiatry, 55-5 (May 2014)  

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A genetically informed longitudinal study of early-life temperament and childhood aggression / Eric N. PENICHET in Development and Psychopathology, 37-2 (May 2025)  

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Impulsivity profiles across five harmonized longitudinal childhood preventive interventions and associations with adult outcomes / Natalie GOULTER in Development and Psychopathology, 37-2 (May 2025)  

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