Applying a public health approach to autism research: A framework for action / Diana SCHENDEL in Autism Research, 15-4 (April 2022)  

Public ISBD [article]  inAutism Research > 15-4 (April 2022) . - p.592-601 Titre : Applying a public health approach to autism research: A framework for action Type de document : texte imprimé Auteurs : Diana SCHENDEL, Auteur ; Anne M. ROUX, Auteur ; Elizabeth MCGHEE HASSRICK, Auteur ; Kristen LYALL, Auteur ; Lindsay L. SHEA, Auteur ; Giacomo VIVANTI, Auteur ; Andrea T. WIECKOWSKI, Auteur ; Craig J. NEWSCHAFFER, Auteur ; Diana L. ROBINS, Auteur Article en page(s) : p.592-601 Langues : Anglais (eng) Mots-clés : Autism Spectrum Disorder/diagnosis  Autistic Disorder/diagnosis  Humans  Public Health  Quality of Life  autism spectrum disorder  communication  knowledge Index. décimale : PER Périodiques Résumé : Most published autism research, and the funding that supports it, remains focused on basic and clinical science. However, the public health impact of autism drives a compelling argument for utilizing a public health approach to autism research. Fundamental to the public health perspective is a focus on health determinants to improve quality of life and to reduce the potential for adverse outcomes across the general population, including in vulnerable subgroups. While the public health research process can be conceptualized as a linear, 3-stage path consisting of discovery - testing - translation/dissemination/implementation, in this paper we propose an integrated, cyclical research framework to advance autism public health objectives in a more comprehensive manner. This involves discovery of primary, secondary and tertiary determinants of health in autism; and use of this evidence base to develop and test detection, intervention, and dissemination strategies and the means to implement them in 'real world' settings. The proposed framework serves to facilitate identification of knowledge gaps, translational barriers, and shortfalls in implementation; guides an iterative research cycle; facilitates purposeful integration of stakeholders and interdisciplinary researchers; and may yield more efficient achievement of improved health and well-being among persons on the autism spectrum at the population-level. LAY SUMMARY: Scientists need better ways to identify and address gaps in autism research, conduct research with stakeholders, and use findings to improve the lives of autistic people. We recommend an approach, based in public health science, to guide research in ways that might impact lives more quickly. En ligne : https://dx.doi.org/10.1002/aur.2689 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=473 [article] Applying a public health approach to autism research: A framework for action [texte imprimé] / Diana SCHENDEL, Auteur ; Anne M. ROUX, Auteur ; Elizabeth MCGHEE HASSRICK, Auteur ; Kristen LYALL, Auteur ; Lindsay L. SHEA, Auteur ; Giacomo VIVANTI, Auteur ; Andrea T. WIECKOWSKI, Auteur ; Craig J. NEWSCHAFFER, Auteur ; Diana L. ROBINS, Auteur . - p.592-601. Langues : Anglais (eng) in Autism Research > 15-4 (April 2022) . - p.592-601 Mots-clés : Autism Spectrum Disorder/diagnosis  Autistic Disorder/diagnosis  Humans  Public Health  Quality of Life  autism spectrum disorder  communication  knowledge Index. décimale : PER Périodiques Résumé : Most published autism research, and the funding that supports it, remains focused on basic and clinical science. However, the public health impact of autism drives a compelling argument for utilizing a public health approach to autism research. Fundamental to the public health perspective is a focus on health determinants to improve quality of life and to reduce the potential for adverse outcomes across the general population, including in vulnerable subgroups. While the public health research process can be conceptualized as a linear, 3-stage path consisting of discovery - testing - translation/dissemination/implementation, in this paper we propose an integrated, cyclical research framework to advance autism public health objectives in a more comprehensive manner. This involves discovery of primary, secondary and tertiary determinants of health in autism; and use of this evidence base to develop and test detection, intervention, and dissemination strategies and the means to implement them in 'real world' settings. The proposed framework serves to facilitate identification of knowledge gaps, translational barriers, and shortfalls in implementation; guides an iterative research cycle; facilitates purposeful integration of stakeholders and interdisciplinary researchers; and may yield more efficient achievement of improved health and well-being among persons on the autism spectrum at the population-level. LAY SUMMARY: Scientists need better ways to identify and address gaps in autism research, conduct research with stakeholders, and use findings to improve the lives of autistic people. We recommend an approach, based in public health science, to guide research in ways that might impact lives more quickly. En ligne : https://dx.doi.org/10.1002/aur.2689 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=473

Associations between accelerated parental biologic age, autism spectrum disorder, social traits, and developmental and cognitive outcomes in their children / Ashley Y. SONG in Autism Research, 15-12 (December 2022)  

Public ISBD [article]  inAutism Research > 15-12 (December 2022) . - p.2359-2370 Titre : Associations between accelerated parental biologic age, autism spectrum disorder, social traits, and developmental and cognitive outcomes in their children Type de document : texte imprimé Auteurs : Ashley Y. SONG, Auteur ; Kelly M. BAKULSKI, Auteur ; Jason I. FEINBERG, Auteur ; Craig J. NEWSCHAFFER, Auteur ; Lisa A. CROEN, Auteur ; Irva HERTZ-PICCIOTTO, Auteur ; Rebecca J. SCHMIDT, Auteur ; Homayoon FARZADEGAN, Auteur ; Kristen LYALL, Auteur ; M. Daniele FALLIN, Auteur ; Heather E. VOLK, Auteur ; Christine LADD-ACOSTA, Auteur Article en page(s) : p.2359-2370 Langues : Anglais (eng) Mots-clés : Child  Male  Pregnancy  Female  Humans  Autism Spectrum Disorder/epidemiology/genetics  Prospective Studies  Parents  Cognition  Biological Products  Epigenesis, Genetic  DNA methylation  age acceleration  autism spectrum disorder  autism-related traits  biologic age  epigenetic age  parental age Index. décimale : PER Périodiques Résumé : Parental age is a known risk factor for autism spectrum disorder (ASD), however, studies to identify the biologic changes underpinning this association are limited. In recent years, "epigenetic clock" algorithms have been developed to estimate biologic age and to evaluate how the epigenetic aging impacts health and disease. In this study, we examined the relationship between parental epigenetic aging and their child's prospective risk of ASD and autism related quantitative traits in the Early Autism Risk Longitudinal Investigation study. Estimates of epigenetic age were computed using three robust clock algorithms and DNA methylation measures from the Infinium HumanMethylation450k platform for maternal blood and paternal blood specimens collected during pregnancy. Epigenetic age acceleration was defined as the residual of regressing chronological age on epigenetic age while accounting for cell type proportions. Multinomial logistic regression and linear regression models were completed adjusting for potential confounders for both maternal epigenetic age acceleration (n = 163) and paternal epigenetic age acceleration (n = 80). We found accelerated epigenetic aging in mothers estimated by Hannum's clock was significantly associated with lower cognitive ability and function in offspring at 12 months, as measured by Mullen Scales of Early Learning scores (ÃŽ2 = -1.66, 95% CI: -3.28, -0.04 for a one-unit increase). We also observed a marginal association between accelerated maternal epigenetic aging by Horvath's clock and increased odds of ASD in offspring at 36 months of age (aOR = 1.12, 95% CI: 0.99, 1.26). By contrast, fathers accelerated aging was marginally associated with decreased ASD risk in their offspring (aOR = 0.83, 95% CI: 0.68, 1.01). Our findings suggest epigenetic aging could play a role in parental age risks on child brain development. En ligne : http://dx.doi.org/10.1002/aur.2822 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=488 [article] Associations between accelerated parental biologic age, autism spectrum disorder, social traits, and developmental and cognitive outcomes in their children [texte imprimé] / Ashley Y. SONG, Auteur ; Kelly M. BAKULSKI, Auteur ; Jason I. FEINBERG, Auteur ; Craig J. NEWSCHAFFER, Auteur ; Lisa A. CROEN, Auteur ; Irva HERTZ-PICCIOTTO, Auteur ; Rebecca J. SCHMIDT, Auteur ; Homayoon FARZADEGAN, Auteur ; Kristen LYALL, Auteur ; M. Daniele FALLIN, Auteur ; Heather E. VOLK, Auteur ; Christine LADD-ACOSTA, Auteur . - p.2359-2370. Langues : Anglais (eng) in Autism Research > 15-12 (December 2022) . - p.2359-2370 Mots-clés : Child  Male  Pregnancy  Female  Humans  Autism Spectrum Disorder/epidemiology/genetics  Prospective Studies  Parents  Cognition  Biological Products  Epigenesis, Genetic  DNA methylation  age acceleration  autism spectrum disorder  autism-related traits  biologic age  epigenetic age  parental age Index. décimale : PER Périodiques Résumé : Parental age is a known risk factor for autism spectrum disorder (ASD), however, studies to identify the biologic changes underpinning this association are limited. In recent years, "epigenetic clock" algorithms have been developed to estimate biologic age and to evaluate how the epigenetic aging impacts health and disease. In this study, we examined the relationship between parental epigenetic aging and their child's prospective risk of ASD and autism related quantitative traits in the Early Autism Risk Longitudinal Investigation study. Estimates of epigenetic age were computed using three robust clock algorithms and DNA methylation measures from the Infinium HumanMethylation450k platform for maternal blood and paternal blood specimens collected during pregnancy. Epigenetic age acceleration was defined as the residual of regressing chronological age on epigenetic age while accounting for cell type proportions. Multinomial logistic regression and linear regression models were completed adjusting for potential confounders for both maternal epigenetic age acceleration (n = 163) and paternal epigenetic age acceleration (n = 80). We found accelerated epigenetic aging in mothers estimated by Hannum's clock was significantly associated with lower cognitive ability and function in offspring at 12 months, as measured by Mullen Scales of Early Learning scores (ÃŽ2 = -1.66, 95% CI: -3.28, -0.04 for a one-unit increase). We also observed a marginal association between accelerated maternal epigenetic aging by Horvath's clock and increased odds of ASD in offspring at 36 months of age (aOR = 1.12, 95% CI: 0.99, 1.26). By contrast, fathers accelerated aging was marginally associated with decreased ASD risk in their offspring (aOR = 0.83, 95% CI: 0.68, 1.01). Our findings suggest epigenetic aging could play a role in parental age risks on child brain development. En ligne : http://dx.doi.org/10.1002/aur.2822 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=488

Autism spectrum disorder and birth spacing: Findings from the study to explore early development (SEED) / Laura A. SCHIEVE in Autism Research, 11-1 (January 2018)  

Public ISBD [article]  inAutism Research > 11-1 (January 2018) . - p.81-94 Titre : Autism spectrum disorder and birth spacing: Findings from the study to explore early development (SEED) Type de document : texte imprimé Auteurs : Laura A. SCHIEVE, Auteur ; Lin H. TIAN, Auteur ; Carolyn DREWS-BOTSCH, Auteur ; Gayle C. WINDHAM, Auteur ; Craig J. NEWSCHAFFER, Auteur ; Julie L. DANIELS, Auteur ; Li-Ching LEE, Auteur ; Lisa A. CROEN, Auteur ; M. Daniele FALLIN, Auteur Article en page(s) : p.81-94 Langues : Anglais (eng) Mots-clés : autism spectrum disorder  developmental disabilities  birth spacing  epidemiology  risk factor Index. décimale : PER Périodiques Résumé : Previous studies of autism spectrum disorder (ASD) and birth spacing had limitations; few examined phenotypic case subtypes or explored underlying mechanisms for associations and none assessed whether other (non ASD) developmental disabilities (DDs) were associated with birth spacing. We assessed associations between inter?pregnancy interval (IPI) and both ASD and other DDs using data from the Study to Explore Early Development, a multi?site case?control study with rigorous case?finding and case?classification methods and detailed data collection on maternal reproductive history. Our sample included 356 ASD cases, 627 DD cases, and 524 population (POP) controls born in second or later births. ASD and DD cases were further sub?divided according to whether the child had intellectual disability (ID). ASD cases were also sub?divided by ASD symptom severity, and DD cases were subdivided by presence of some ASD symptoms (indicated on an autism screener). Odds ratios, adjusted for maternal?child sociodemographic factors, (aORs) and 95% confidence intervals were derived from logistic regression models. Among term births, ASD was associated with both IPI <18 months (aOR 1.5 [1.1 2.2]) and ≥60 months (1.5 [0.99 2.4]). Both short and long IPI associations were stronger among ASD cases with high severity scores (aORs 2.0 [1.3 3.3] and 1.8 [0.99 3.2], respectively). Associations were unchanged after adding several factors potentially related to the causal pathway to regression models. DD was not associated with either short or long IPI?overall, among term births, or in any subgroup examined. These findings extend those from previous studies and further inform recommendations on optimal pregnancy spacing. Autism Res 2018, 11: 81 94. ? 2017 International Society for Autism Research, Wiley Periodicals, Inc. Lay Summary We investigated whether the amount of time between pregnancies was associated autism spectrum disorder (ASD) or other developmental disabilities (DD) in children. ASD was increased in second and later?born children who were conceived less than 18 months or 60 or more months after the mother's previous birth. Other DDs were not associated with birth spacing. En ligne : https://doi.org/10.1002/aur.1887 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=333 [article] Autism spectrum disorder and birth spacing: Findings from the study to explore early development (SEED) [texte imprimé] / Laura A. SCHIEVE, Auteur ; Lin H. TIAN, Auteur ; Carolyn DREWS-BOTSCH, Auteur ; Gayle C. WINDHAM, Auteur ; Craig J. NEWSCHAFFER, Auteur ; Julie L. DANIELS, Auteur ; Li-Ching LEE, Auteur ; Lisa A. CROEN, Auteur ; M. Daniele FALLIN, Auteur . - p.81-94. Langues : Anglais (eng) in Autism Research > 11-1 (January 2018) . - p.81-94 Mots-clés : autism spectrum disorder  developmental disabilities  birth spacing  epidemiology  risk factor Index. décimale : PER Périodiques Résumé : Previous studies of autism spectrum disorder (ASD) and birth spacing had limitations; few examined phenotypic case subtypes or explored underlying mechanisms for associations and none assessed whether other (non ASD) developmental disabilities (DDs) were associated with birth spacing. We assessed associations between inter?pregnancy interval (IPI) and both ASD and other DDs using data from the Study to Explore Early Development, a multi?site case?control study with rigorous case?finding and case?classification methods and detailed data collection on maternal reproductive history. Our sample included 356 ASD cases, 627 DD cases, and 524 population (POP) controls born in second or later births. ASD and DD cases were further sub?divided according to whether the child had intellectual disability (ID). ASD cases were also sub?divided by ASD symptom severity, and DD cases were subdivided by presence of some ASD symptoms (indicated on an autism screener). Odds ratios, adjusted for maternal?child sociodemographic factors, (aORs) and 95% confidence intervals were derived from logistic regression models. Among term births, ASD was associated with both IPI <18 months (aOR 1.5 [1.1 2.2]) and ≥60 months (1.5 [0.99 2.4]). Both short and long IPI associations were stronger among ASD cases with high severity scores (aORs 2.0 [1.3 3.3] and 1.8 [0.99 3.2], respectively). Associations were unchanged after adding several factors potentially related to the causal pathway to regression models. DD was not associated with either short or long IPI?overall, among term births, or in any subgroup examined. These findings extend those from previous studies and further inform recommendations on optimal pregnancy spacing. Autism Res 2018, 11: 81 94. ? 2017 International Society for Autism Research, Wiley Periodicals, Inc. Lay Summary We investigated whether the amount of time between pregnancies was associated autism spectrum disorder (ASD) or other developmental disabilities (DD) in children. ASD was increased in second and later?born children who were conceived less than 18 months or 60 or more months after the mother's previous birth. Other DDs were not associated with birth spacing. En ligne : https://doi.org/10.1002/aur.1887 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=333

Brief Report: Examining the Association of Autism and Adverse Childhood Experiences in the National Survey of Children’s Health: The Important Role of Income and Co-occurring Mental Health Conditions / Connor M. KERNS in Journal of Autism and Developmental Disorders, 47-7 (July 2017)  

Public ISBD [article]  inJournal of Autism and Developmental Disorders > 47-7 (July 2017) . - p.2275-2281 Titre : Brief Report: Examining the Association of Autism and Adverse Childhood Experiences in the National Survey of Children’s Health: The Important Role of Income and Co-occurring Mental Health Conditions Type de document : texte imprimé Auteurs : Connor M. KERNS, Auteur ; Craig J. NEWSCHAFFER, Auteur ; Steven J. BERKOWITZ, Auteur ; Brian K. LEE, Auteur Article en page(s) : p.2275-2281 Langues : Anglais (eng) Mots-clés : Autism spectrum disorder  Adverse childhood experiences  Comorbidity  Anxiety  Depression  Attention problems  Behavioral problems Index. décimale : PER Périodiques Résumé : Adverse childhood experiences (ACEs) are risk factors for mental and physical illness and more likely to occur for children with autism spectrum disorder (ASD). The present study aimed to clarify the contribution of poverty, intellectual disability and mental health conditions to this disparity. Data on child and family characteristics, mental health conditions and ACEs were analyzed in 67,067 youth from the 2011–2012 National Survey of Children’s Health. In an income-stratified sample, the association of ASD and ACEs was greater for lower income children and significantly diminished after controlling for child mental health conditions, but not intellectual disability. Findings suggest that the association of ACEs and ASD is moderated by family income and contingent on co-occurring mental health conditions. En ligne : https://doi.org/10.1007/s10803-017-3111-7 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=314 [article] Brief Report: Examining the Association of Autism and Adverse Childhood Experiences in the National Survey of Children’s Health: The Important Role of Income and Co-occurring Mental Health Conditions [texte imprimé] / Connor M. KERNS, Auteur ; Craig J. NEWSCHAFFER, Auteur ; Steven J. BERKOWITZ, Auteur ; Brian K. LEE, Auteur . - p.2275-2281. Langues : Anglais (eng) in Journal of Autism and Developmental Disorders > 47-7 (July 2017) . - p.2275-2281 Mots-clés : Autism spectrum disorder  Adverse childhood experiences  Comorbidity  Anxiety  Depression  Attention problems  Behavioral problems Index. décimale : PER Périodiques Résumé : Adverse childhood experiences (ACEs) are risk factors for mental and physical illness and more likely to occur for children with autism spectrum disorder (ASD). The present study aimed to clarify the contribution of poverty, intellectual disability and mental health conditions to this disparity. Data on child and family characteristics, mental health conditions and ACEs were analyzed in 67,067 youth from the 2011–2012 National Survey of Children’s Health. In an income-stratified sample, the association of ASD and ACEs was greater for lower income children and significantly diminished after controlling for child mental health conditions, but not intellectual disability. Findings suggest that the association of ACEs and ASD is moderated by family income and contingent on co-occurring mental health conditions. En ligne : https://doi.org/10.1007/s10803-017-3111-7 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=314

Brief Report: Service Use and Associated Expenditures Among Adolescents with Autism Spectrum Disorder Transitioning to Adulthood / Lindsay L. SHEA in Journal of Autism and Developmental Disorders, 48-9 (September 2018)  

Public ISBD [article]  inJournal of Autism and Developmental Disorders > 48-9 (September 2018) . - p.3223-3227 Titre : Brief Report: Service Use and Associated Expenditures Among Adolescents with Autism Spectrum Disorder Transitioning to Adulthood Type de document : texte imprimé Auteurs : Lindsay L. SHEA, Auteur ; Ming XIE, Auteur ; Paul TURCOTTE, Auteur ; Steven C. MARCUS, Auteur ; Robert FIELD, Auteur ; Craig J. NEWSCHAFFER, Auteur ; David S. MANDELL, Auteur Article en page(s) : p.3223-3227 Langues : Anglais (eng) Mots-clés : Autism  Intellectual disability  Medicaid  Service use  Transition Index. décimale : PER Périodiques Résumé : This study compared Medicaid service utilization and expenditures among adolescents with autism spectrum disorder (ASD) to adolescents with intellectual disability (ID) as they aged into adulthood. Medicaid Analytic eXtract (MAX) data was used to identify a national cohort. Winsorization was utilized to control for expenditure outliers. A greater proportion of adolescents with ASD utilized most services. Decreases in the use of key services, including psychiatric outpatient services, were observed for both groups. Changes in medical services, such as increases in inpatient and long term care services, among the ASD cohort suggest medical needs of adolescents with ASD change as they age. Information remains lacking on changing ASD symptom presentation during the transition to adolescence. En ligne : http://dx.doi.org/10.1007/s10803-018-3563-4 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=368 [article] Brief Report: Service Use and Associated Expenditures Among Adolescents with Autism Spectrum Disorder Transitioning to Adulthood [texte imprimé] / Lindsay L. SHEA, Auteur ; Ming XIE, Auteur ; Paul TURCOTTE, Auteur ; Steven C. MARCUS, Auteur ; Robert FIELD, Auteur ; Craig J. NEWSCHAFFER, Auteur ; David S. MANDELL, Auteur . - p.3223-3227. Langues : Anglais (eng) in Journal of Autism and Developmental Disorders > 48-9 (September 2018) . - p.3223-3227 Mots-clés : Autism  Intellectual disability  Medicaid  Service use  Transition Index. décimale : PER Périodiques Résumé : This study compared Medicaid service utilization and expenditures among adolescents with autism spectrum disorder (ASD) to adolescents with intellectual disability (ID) as they aged into adulthood. Medicaid Analytic eXtract (MAX) data was used to identify a national cohort. Winsorization was utilized to control for expenditure outliers. A greater proportion of adolescents with ASD utilized most services. Decreases in the use of key services, including psychiatric outpatient services, were observed for both groups. Changes in medical services, such as increases in inpatient and long term care services, among the ASD cohort suggest medical needs of adolescents with ASD change as they age. Information remains lacking on changing ASD symptom presentation during the transition to adolescence. En ligne : http://dx.doi.org/10.1007/s10803-018-3563-4 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=368

Case-control meta-analysis of blood DNA methylation and autism spectrum disorder / Shan V. ANDREWS in Molecular Autism, 9 (2018)  

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Characterizing self-reported physical activity before and during a subsequent pregnancy among parents in a familial autism cohort / Megan G. BRAGG in Autism, 29-1 (January 2025)  

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Children with Autism: Quality of Life and Parental Concerns / Li-Ching LEE in Journal of Autism and Developmental Disorders, 38-6 (July 2008)  

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A Comparative Analysis of the Full and Short Versions of the Social Responsiveness Scale in Estimating an Established Autism Risk Factor Association in ECHO: Do we Get the Same Estimates? / Xuejuan NING ; Mina HOSSEINI ; Lisa A. CROEN ; Robert M. JOSEPH ; Margaret R. KARAGAS ; Christine LADD-ACOSTA ; Rebecca LANDA ; Daniel S. MESSINGER ; Craig J. NEWSCHAFFER ; Ruby NGUYEN ; Sally OZONOFF ; T. Michael O'SHEA ; Rebecca J. SCHMIDT ; Cindy O. TREVINO ; Kristen LYALL in Journal of Autism and Developmental Disorders, 55-6 (June 2025)  

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Development and validation of a streamlined autism case confirmation approach for use in epidemiologic risk factor research in prospective cohorts / Craig J. NEWSCHAFFER in Autism Research, 10-3 (March 2017)  

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Distributional Properties and Criterion Validity of a Shortened Version of the Social Responsiveness Scale: Results from the ECHO Program and Implications for Social Communication Research / Kristen LYALL in Journal of Autism and Developmental Disorders, 51-7 (July 2021)  

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Does a claims diagnosis of autism mean a true case? / James P. BURKE in Autism, 18-3 (April 2014)  

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Examining associations between prenatal biomarkers of oxidative stress and ASD-related outcomes using quantile regression / Meghan E. CAREY in Journal of Autism and Developmental Disorders, 53-8 (August 2023)  

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Expert consensus regarding indicators of a traumatic reaction in autistic youth: a Delphi survey / Connor M. KERNS in Journal of Child Psychology and Psychiatry, 64-1 (January 2023)  

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Exploring potential sources of childhood trauma: A qualitative study with autistic adults and caregivers / Connor M. KERNS in Autism, 26-8 (November 2022)  

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