Dépouillements

Editorial: For crying out loud: Infant signaling and parental responsiveness / Charles H. ZEANAH in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1171-1172 Titre : Editorial: For crying out loud: Infant signaling and parental responsiveness Type de document : Texte imprimé et/ou numérique Auteurs : Charles H. ZEANAH, Auteur Article en page(s) : p.1171-1172 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Crying represents a powerful signal for human newborns. How parents should respond has been a matter of debate, with some urging prompt responding and others expressing concern about spoiling. Despite the fervor of the debate, few longitudinal observational studies have addressed the question. A new study in this issue by Bilgin and Wolke provides important data about ignoring crying in early infancy and its predictive correlates. The results of their investigation are discussed in the context of attachment and behavioral theories. En ligne : http://dx.doi.org/10.1111/jcpp.13338 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Editorial: For crying out loud: Infant signaling and parental responsiveness [Texte imprimé et/ou numérique] / Charles H. ZEANAH, Auteur . - p.1171-1172. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1171-1172 Index. décimale : PER Périodiques Résumé : Crying represents a powerful signal for human newborns. How parents should respond has been a matter of debate, with some urging prompt responding and others expressing concern about spoiling. Despite the fervor of the debate, few longitudinal observational studies have addressed the question. A new study in this issue by Bilgin and Wolke provides important data about ignoring crying in early infancy and its predictive correlates. The results of their investigation are discussed in the context of attachment and behavioral theories. En ligne : http://dx.doi.org/10.1111/jcpp.13338 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Research Review: The strength of the genetic overlap between ADHD and other psychiatric symptoms - a systematic review and meta-analysis / Anneli ANDERSSON in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1173-1183 Titre : Research Review: The strength of the genetic overlap between ADHD and other psychiatric symptoms - a systematic review and meta-analysis Type de document : Texte imprimé et/ou numérique Auteurs : Anneli ANDERSSON, Auteur ; Catherine TUVBLAD, Auteur ; Qi CHEN, Auteur ; Ebba DU RIETZ, Auteur ; Samuele CORTESE, Auteur ; Ralf KUJA-HALKOLA, Auteur ; Henrik LARSSON, Auteur Article en page(s) : p.1173-1183 Langues : Anglais (eng) Mots-clés : Adhd  externalizing  genetic  internalizing  neurodevelopmental  overlap  twins Index. décimale : PER Périodiques Résumé : BACKGROUND: Attention-deficit/hyperactivity disorder (ADHD) frequently co-occurs with other psychiatric disorders. Twin studies have established that these co-occurrences are in part due to shared genetic risks. However, the strength of these genetic overlaps and the potential heterogeneity accounted for by type of psychiatric symptoms, age, and methods of assessment remain unclear. We conducted a systematic review to fill this gap. METHODS: We searched PubMed, PsycINFO, Embase, and Web of Science until March 07, 2019. Genetic correlations (r(g) ) were used as effect size measures. RESULTS: A total of 31 independent studies fulfilled the inclusion criteria. The pooled estimates showed that the associations between ADHD and other psychiatric symptoms were partly explained by shared genetic factors, with a pooled genetic correlation of 0.50, 95% confidence interval: 0.46-0.60. The genetic correlations (r(g) ) between ADHD and externalizing (r(g)  = .49 [0.37-0.61]), internalizing (r(g)  = .50 [0.39-0.69]), and neurodevelopmental (r(g)  = .56 [0.47-0.66]) symptoms were similar in magnitude. The genetic correlations in childhood and adulthood were r(g)  = .53 (0.43-0.63) and r(g)  = .51 (0.44-0.56), respectively. For methods of assessment, the genetic correlations were also similar in strength, self-reports r(g)  = .52 (0.47-0.58), other informants r(g)  = .55 (0.41-0.69), and combined raters r(g)  = .50 (0.33-0.65). CONCLUSIONS: These findings indicate that the co-occurrence of externalizing, internalizing, and neurodevelopmental disorder symptoms in individuals with ADHD symptoms in part is due to a shared genetic risk. En ligne : http://dx.doi.org/10.1111/jcpp.13233 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Research Review: The strength of the genetic overlap between ADHD and other psychiatric symptoms - a systematic review and meta-analysis [Texte imprimé et/ou numérique] / Anneli ANDERSSON, Auteur ; Catherine TUVBLAD, Auteur ; Qi CHEN, Auteur ; Ebba DU RIETZ, Auteur ; Samuele CORTESE, Auteur ; Ralf KUJA-HALKOLA, Auteur ; Henrik LARSSON, Auteur . - p.1173-1183. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1173-1183 Mots-clés : Adhd  externalizing  genetic  internalizing  neurodevelopmental  overlap  twins Index. décimale : PER Périodiques Résumé : BACKGROUND: Attention-deficit/hyperactivity disorder (ADHD) frequently co-occurs with other psychiatric disorders. Twin studies have established that these co-occurrences are in part due to shared genetic risks. However, the strength of these genetic overlaps and the potential heterogeneity accounted for by type of psychiatric symptoms, age, and methods of assessment remain unclear. We conducted a systematic review to fill this gap. METHODS: We searched PubMed, PsycINFO, Embase, and Web of Science until March 07, 2019. Genetic correlations (r(g) ) were used as effect size measures. RESULTS: A total of 31 independent studies fulfilled the inclusion criteria. The pooled estimates showed that the associations between ADHD and other psychiatric symptoms were partly explained by shared genetic factors, with a pooled genetic correlation of 0.50, 95% confidence interval: 0.46-0.60. The genetic correlations (r(g) ) between ADHD and externalizing (r(g)  = .49 [0.37-0.61]), internalizing (r(g)  = .50 [0.39-0.69]), and neurodevelopmental (r(g)  = .56 [0.47-0.66]) symptoms were similar in magnitude. The genetic correlations in childhood and adulthood were r(g)  = .53 (0.43-0.63) and r(g)  = .51 (0.44-0.56), respectively. For methods of assessment, the genetic correlations were also similar in strength, self-reports r(g)  = .52 (0.47-0.58), other informants r(g)  = .55 (0.41-0.69), and combined raters r(g)  = .50 (0.33-0.65). CONCLUSIONS: These findings indicate that the co-occurrence of externalizing, internalizing, and neurodevelopmental disorder symptoms in individuals with ADHD symptoms in part is due to a shared genetic risk. En ligne : http://dx.doi.org/10.1111/jcpp.13233 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Parental use of 'cry it out' in infants: no adverse effects on attachment and behavioural development at 18 months / Ayten BILGIN in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1184-1193 Titre : Parental use of 'cry it out' in infants: no adverse effects on attachment and behavioural development at 18 months Type de document : Texte imprimé et/ou numérique Auteurs : Ayten BILGIN, Auteur ; Dieter WOLKE, Auteur Article en page(s) : p.1184-1193 Langues : Anglais (eng) Mots-clés : Crying  attachment  cry-it-out Index. décimale : PER Périodiques Résumé : BACKGROUND: Leaving infant to cry it out has been the subject of discussion among researchers and parents. Nevertheless, there is paucity of empirical research investigating the association between leaving infant to cry it out and consequent crying duration and frequency, mother-infant attachment and behavioural development. METHODS: The sample with complete longitudinal data comprised 178 infants and their caretakers. Parental use of 'leaving infant to cry out' and cry duration were assessed with maternal report at term, 3, 6 and 18 months, and frequency of crying was assessed at term, 3 and 18 months of age. Attachment was measured at 18 months using the strange situation procedure. Behavioural development of the infant was assessed with two observational measures and a parent-report questionnaire at 18 months. RESULTS: The use of 'leaving infant to crying' was rare at term and increased over the next 18 months. 'Leaving infants to cry it out' at term was associated with a decrease in crying frequency at 3 months. Furthermore, leaving infants to cry it out a few times at term and often at 3 months was associated with shorter fuss/cry duration at 18 months of age. No adverse impacts of leaving infants to cry it out in the first 6 months on infant-mother attachment and behavioural development at 18 months were found. CONCLUSIONS: Contemporary practice of some parents in the United Kingdom to occasionally or often 'leaving infant to cry it out' during the first 6 months was not associated with adverse behavioural development and attachment at 18 months. Increased use of 'leaving to cry it out' with age may indicate differential responding by parents related to infant self-regulation. En ligne : http://dx.doi.org/10.1111/jcpp.13223 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Parental use of 'cry it out' in infants: no adverse effects on attachment and behavioural development at 18 months [Texte imprimé et/ou numérique] / Ayten BILGIN, Auteur ; Dieter WOLKE, Auteur . - p.1184-1193. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1184-1193 Mots-clés : Crying  attachment  cry-it-out Index. décimale : PER Périodiques Résumé : BACKGROUND: Leaving infant to cry it out has been the subject of discussion among researchers and parents. Nevertheless, there is paucity of empirical research investigating the association between leaving infant to cry it out and consequent crying duration and frequency, mother-infant attachment and behavioural development. METHODS: The sample with complete longitudinal data comprised 178 infants and their caretakers. Parental use of 'leaving infant to cry out' and cry duration were assessed with maternal report at term, 3, 6 and 18 months, and frequency of crying was assessed at term, 3 and 18 months of age. Attachment was measured at 18 months using the strange situation procedure. Behavioural development of the infant was assessed with two observational measures and a parent-report questionnaire at 18 months. RESULTS: The use of 'leaving infant to crying' was rare at term and increased over the next 18 months. 'Leaving infants to cry it out' at term was associated with a decrease in crying frequency at 3 months. Furthermore, leaving infants to cry it out a few times at term and often at 3 months was associated with shorter fuss/cry duration at 18 months of age. No adverse impacts of leaving infants to cry it out in the first 6 months on infant-mother attachment and behavioural development at 18 months were found. CONCLUSIONS: Contemporary practice of some parents in the United Kingdom to occasionally or often 'leaving infant to cry it out' during the first 6 months was not associated with adverse behavioural development and attachment at 18 months. Increased use of 'leaving to cry it out' with age may indicate differential responding by parents related to infant self-regulation. En ligne : http://dx.doi.org/10.1111/jcpp.13223 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Sex differences in foetal origins of child emotional symptoms: a test of evolutionary hypotheses in a large, general population cohort / Elizabeth C. BRAITHWAITE in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1194-1202 Titre : Sex differences in foetal origins of child emotional symptoms: a test of evolutionary hypotheses in a large, general population cohort Type de document : Texte imprimé et/ou numérique Auteurs : Elizabeth C. BRAITHWAITE, Auteur ; Andrew PICKLES, Auteur ; Nicola WRIGHT, Auteur ; Helen SHARP, Auteur ; Jonathan HILL, Auteur Article en page(s) : p.1194-1202 Langues : Anglais (eng) Mots-clés : Sex differences  emotional disorder  maternal depression  postnatal  prenatal Index. décimale : PER Périodiques Résumé : BACKGROUND: Based on previous findings from the Wirral Child Health and Development Study (WCHADS), and on evolutionary hypotheses, we preregistered analyses of data from a large epidemiological sample (https://osf.io/fn5g9/register/564d31db8c5e4a7c9694b2be), to test for sex-dependent moderation by prenatal maternal depressive symptoms of the association between postnatal maternal depressive symptoms and child emotional problems. METHODS: A total of 8,354 mothers and children were followed from pregnancy to 3.5 years in the Avon Longitudinal Study of Parents and Children (ALSPAC). Self-report measures of prenatal and postnatal maternal depressive symptoms, and maternal report of child emotional symptoms were administered. RESULTS: There was a three-way interaction between maternal prenatal and postnatal depression, and child sex (Coeff .042 95% CI 0.015 to 0.068, p = .002). This arose from moderation by prenatal depression, in opposite directions in boys and in girls. In boys, the association between postnatal depression and child emotional symptoms was weaker following lower prenatal depressive symptoms (interaction term coeff = .030, p = .001), and in girls, to a lesser extent, the association was stronger following lower prenatal depressive symptoms (interaction term coeff = -.012, p = .221). CONCLUSIONS: We replicated the finding from the WCHADS that prenatal depression modifies the association between postnatal depression and children's emotional problems in a sex-dependent fashion. In ALSPAC, the sex difference was explained mainly by a protective effect of low prenatal depression in boys, while in WCHADS, it arose from greater vulnerability of girls to postnatal depression following low prenatal depression. In the light of these findings, in evaluating and implementing early interventions, there is need to consider that risks associated with postnatal depression may vary depending on maternal mood during pregnancy and may differ between boys and girls. En ligne : http://dx.doi.org/10.1111/jcpp.13229 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Sex differences in foetal origins of child emotional symptoms: a test of evolutionary hypotheses in a large, general population cohort [Texte imprimé et/ou numérique] / Elizabeth C. BRAITHWAITE, Auteur ; Andrew PICKLES, Auteur ; Nicola WRIGHT, Auteur ; Helen SHARP, Auteur ; Jonathan HILL, Auteur . - p.1194-1202. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1194-1202 Mots-clés : Sex differences  emotional disorder  maternal depression  postnatal  prenatal Index. décimale : PER Périodiques Résumé : BACKGROUND: Based on previous findings from the Wirral Child Health and Development Study (WCHADS), and on evolutionary hypotheses, we preregistered analyses of data from a large epidemiological sample (https://osf.io/fn5g9/register/564d31db8c5e4a7c9694b2be), to test for sex-dependent moderation by prenatal maternal depressive symptoms of the association between postnatal maternal depressive symptoms and child emotional problems. METHODS: A total of 8,354 mothers and children were followed from pregnancy to 3.5 years in the Avon Longitudinal Study of Parents and Children (ALSPAC). Self-report measures of prenatal and postnatal maternal depressive symptoms, and maternal report of child emotional symptoms were administered. RESULTS: There was a three-way interaction between maternal prenatal and postnatal depression, and child sex (Coeff .042 95% CI 0.015 to 0.068, p = .002). This arose from moderation by prenatal depression, in opposite directions in boys and in girls. In boys, the association between postnatal depression and child emotional symptoms was weaker following lower prenatal depressive symptoms (interaction term coeff = .030, p = .001), and in girls, to a lesser extent, the association was stronger following lower prenatal depressive symptoms (interaction term coeff = -.012, p = .221). CONCLUSIONS: We replicated the finding from the WCHADS that prenatal depression modifies the association between postnatal depression and children's emotional problems in a sex-dependent fashion. In ALSPAC, the sex difference was explained mainly by a protective effect of low prenatal depression in boys, while in WCHADS, it arose from greater vulnerability of girls to postnatal depression following low prenatal depression. In the light of these findings, in evaluating and implementing early interventions, there is need to consider that risks associated with postnatal depression may vary depending on maternal mood during pregnancy and may differ between boys and girls. En ligne : http://dx.doi.org/10.1111/jcpp.13229 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Intergenerational transmission of anxiety: linking parental anxiety to infant autonomic hyperarousal and fearful temperament / Wieke DE VENTE in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1203-1212 Titre : Intergenerational transmission of anxiety: linking parental anxiety to infant autonomic hyperarousal and fearful temperament Type de document : Texte imprimé et/ou numérique Auteurs : Wieke DE VENTE, Auteur ; Mirjana MAJDANDŽI?, Auteur ; Susan M. BOGELS, Auteur Article en page(s) : p.1203-1212 Langues : Anglais (eng) Mots-clés : Behavioural inhibition  fearful temperament  habituation  heart rate  heart rate variability Index. décimale : PER Périodiques Résumé : BACKGROUND: Autonomic hyperarousal has been proposed as a dispositional risk factor for anxiety disorders (ADs). Therefore, we studied physiological arousal in offspring of fathers and mothers with and without ADs and whether infant hyperarousal predicts subsequent fearful temperament. METHODS: Infants (N = 128; age = 4 months) did a novel stimuli task (exposure to visual, olfactory, and acoustic stimuli and an unfamiliar male) and a habituation task (exposure to a repeated acoustic stimulus). Heart rate (HR) and heart rate variability (HRV) were measured during baseline, stimuli and post-stimuli rest. Parents' AD status and severity were measured using a diagnostic interview and their fearful temperament using a questionnaire. Child fearful temperament was measured at 4 months, 1 year and 2.5 years with observations during structured tasks. RESULTS: Parents' fearful temperament (significant in the habituation task), AD status (significant in the novel stimuli task) and AD severity (significant in both tasks) predicted a higher HR in their infants. Infants' higher HR reactivity to novel stimuli and diminished HR recovery at 4 months predicted a more fearful temperament during infancy and toddlerhood. Infants' higher HR at 4 months predicted a more fearful temperament at 2.5 years. CONCLUSIONS: Parental prenatal anxiety (disorders) predicted infants' autonomic arousal, which in turn predicted later fearful temperament in children. Outcomes suggest that autonomic hyperarousal is a dispositional risk factor of ADs. En ligne : http://dx.doi.org/10.1111/jcpp.13208 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Intergenerational transmission of anxiety: linking parental anxiety to infant autonomic hyperarousal and fearful temperament [Texte imprimé et/ou numérique] / Wieke DE VENTE, Auteur ; Mirjana MAJDANDŽI?, Auteur ; Susan M. BOGELS, Auteur . - p.1203-1212. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1203-1212 Mots-clés : Behavioural inhibition  fearful temperament  habituation  heart rate  heart rate variability Index. décimale : PER Périodiques Résumé : BACKGROUND: Autonomic hyperarousal has been proposed as a dispositional risk factor for anxiety disorders (ADs). Therefore, we studied physiological arousal in offspring of fathers and mothers with and without ADs and whether infant hyperarousal predicts subsequent fearful temperament. METHODS: Infants (N = 128; age = 4 months) did a novel stimuli task (exposure to visual, olfactory, and acoustic stimuli and an unfamiliar male) and a habituation task (exposure to a repeated acoustic stimulus). Heart rate (HR) and heart rate variability (HRV) were measured during baseline, stimuli and post-stimuli rest. Parents' AD status and severity were measured using a diagnostic interview and their fearful temperament using a questionnaire. Child fearful temperament was measured at 4 months, 1 year and 2.5 years with observations during structured tasks. RESULTS: Parents' fearful temperament (significant in the habituation task), AD status (significant in the novel stimuli task) and AD severity (significant in both tasks) predicted a higher HR in their infants. Infants' higher HR reactivity to novel stimuli and diminished HR recovery at 4 months predicted a more fearful temperament during infancy and toddlerhood. Infants' higher HR at 4 months predicted a more fearful temperament at 2.5 years. CONCLUSIONS: Parental prenatal anxiety (disorders) predicted infants' autonomic arousal, which in turn predicted later fearful temperament in children. Outcomes suggest that autonomic hyperarousal is a dispositional risk factor of ADs. En ligne : http://dx.doi.org/10.1111/jcpp.13208 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Parent-child neural synchrony: a novel approach to elucidating dyadic correlates of preschool irritability / Laura E. QUIÑONES-CAMACHO in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1213-1223 Titre : Parent-child neural synchrony: a novel approach to elucidating dyadic correlates of preschool irritability Type de document : Texte imprimé et/ou numérique Auteurs : Laura E. QUIÑONES-CAMACHO, Auteur ; Frank A. FISHBURN, Auteur ; M. Catalina CAMACHO, Auteur ; Christina O. HLUTKOWSKY, Auteur ; Theodore J. HUPPERT, Auteur ; Lauren S. WAKSCHLAG, Auteur ; Susan B. PERLMAN, Auteur Article en page(s) : p.1213-1223 Langues : Anglais (eng) Mots-clés : Neural synchrony  irritability  parent-child synchrony  prefrontal cortex  recovery Index. décimale : PER Périodiques Résumé : BACKGROUND: Research to date has largely conceptualized irritability in terms of intraindividual differences. However, the role of interpersonal dyadic processes has received little consideration. Nevertheless, difficulties in how parent-child dyads synchronize during interactions may be an important correlate of irritably in early childhood. Innovations in developmentally sensitive neuroimaging methods now enable the use of measures of neural synchrony to quantify synchronous responses in parent-child dyads and can help clarify the neural underpinnings of these difficulties. We introduce the Disruptive Behavior Diagnostic Observation Schedule: Biological Synchrony (DB-DOS:BioSync) as a paradigm for exploring parent-child neural synchrony as a potential biological mechanism for interpersonal difficulties in preschool psychopathology. METHODS: Using functional near-infrared spectroscopy (fNIRS) 4- to 5-year-olds (N = 116) and their mothers completed the DB-DOS:BioSync while assessing neural synchrony during mild frustration and recovery. Child irritability was measured using a latent irritability factor that was calculated from four developmentally sensitive indicators. RESULTS: Both the mild frustration and the recovery contexts resulted in neural synchrony. However, less neural synchrony during the recovery context only was associated with more child irritability. CONCLUSIONS: Our results suggest that recovering after a frustrating period might be particularly challenging for children high in irritability and offer support for the use of the DB-DOS:BioSync task to elucidate interpersonal neural mechanisms of developmental psychopathology. En ligne : http://dx.doi.org/10.1111/jcpp.13165 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Parent-child neural synchrony: a novel approach to elucidating dyadic correlates of preschool irritability [Texte imprimé et/ou numérique] / Laura E. QUIÑONES-CAMACHO, Auteur ; Frank A. FISHBURN, Auteur ; M. Catalina CAMACHO, Auteur ; Christina O. HLUTKOWSKY, Auteur ; Theodore J. HUPPERT, Auteur ; Lauren S. WAKSCHLAG, Auteur ; Susan B. PERLMAN, Auteur . - p.1213-1223. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1213-1223 Mots-clés : Neural synchrony  irritability  parent-child synchrony  prefrontal cortex  recovery Index. décimale : PER Périodiques Résumé : BACKGROUND: Research to date has largely conceptualized irritability in terms of intraindividual differences. However, the role of interpersonal dyadic processes has received little consideration. Nevertheless, difficulties in how parent-child dyads synchronize during interactions may be an important correlate of irritably in early childhood. Innovations in developmentally sensitive neuroimaging methods now enable the use of measures of neural synchrony to quantify synchronous responses in parent-child dyads and can help clarify the neural underpinnings of these difficulties. We introduce the Disruptive Behavior Diagnostic Observation Schedule: Biological Synchrony (DB-DOS:BioSync) as a paradigm for exploring parent-child neural synchrony as a potential biological mechanism for interpersonal difficulties in preschool psychopathology. METHODS: Using functional near-infrared spectroscopy (fNIRS) 4- to 5-year-olds (N = 116) and their mothers completed the DB-DOS:BioSync while assessing neural synchrony during mild frustration and recovery. Child irritability was measured using a latent irritability factor that was calculated from four developmentally sensitive indicators. RESULTS: Both the mild frustration and the recovery contexts resulted in neural synchrony. However, less neural synchrony during the recovery context only was associated with more child irritability. CONCLUSIONS: Our results suggest that recovering after a frustrating period might be particularly challenging for children high in irritability and offer support for the use of the DB-DOS:BioSync task to elucidate interpersonal neural mechanisms of developmental psychopathology. En ligne : http://dx.doi.org/10.1111/jcpp.13165 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Girls' brain structural connectivity in late adolescence relates to history of depression symptoms / Rajpreet CHAHAL in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1224-1233 Titre : Girls' brain structural connectivity in late adolescence relates to history of depression symptoms Type de document : Texte imprimé et/ou numérique Auteurs : Rajpreet CHAHAL, Auteur ; David G. WEISSMAN, Auteur ; Scott MAREK, Auteur ; Shawn A. RHOADS, Auteur ; Alison E. HIPWELL, Auteur ; Erika E. FORBES, Auteur ; Kate KEENAN, Auteur ; Amanda E. GUYER, Auteur Article en page(s) : p.1224-1233 Langues : Anglais (eng) Mots-clés : Adolescence  brain imaging  connectomics  depression  development Index. décimale : PER Périodiques Résumé : BACKGROUND: Girls' depressive symptoms typically increase in adolescence, with individual differences in course and severity being key risk factors for impaired emotional functioning in young adulthood. Given the continued brain white matter (WM) maturation that occurs in adolescence, the present study tested whether structural connectivity patterns in late adolescence are associated with variation in the course of depression symptom severity throughout adolescence. METHOD: Participants were girls (N = 115) enrolled in a multiyear prospective cohort study of risk for depression. Initial depression severity (intercept) at age 10 and change in severity (linear slope) across ages 10-19 were examined in relation to WM tractography collected at age 19. Network-based statistic analyses were used to identify clusters showing variation in structural connectivity in association with depressive symptom intercept, slope, and their interaction. RESULTS: Higher initial depressive severity and steeper positive slope (separately) were associated with greater structural connectivity between temporal, subcortical socioaffective, and occipital regions. Intercept showed more connectivity associations than slope. The interaction effect indicated that higher initial symptom severity and a steeper negative slope (i.e., alleviating symptoms) were related to greater connectivity between cognitive control regions. Moderately severe symptoms that worsened over time were followed by greater connectivity between self-referential and cognitive regions (e.g., posterior cingulate and frontal gyrus). CONCLUSIONS: Higher depressive symptom severity in early adolescence and increasing symptom severity over time may forecast structural connectivity differences in late adolescence, particularly in pathways involving cognitive and emotion-processing regions. Understanding how clinical course relates to neurobiological correlates may inform new treatment approaches to adolescent depression. En ligne : http://dx.doi.org/10.1111/jcpp.13184 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Girls' brain structural connectivity in late adolescence relates to history of depression symptoms [Texte imprimé et/ou numérique] / Rajpreet CHAHAL, Auteur ; David G. WEISSMAN, Auteur ; Scott MAREK, Auteur ; Shawn A. RHOADS, Auteur ; Alison E. HIPWELL, Auteur ; Erika E. FORBES, Auteur ; Kate KEENAN, Auteur ; Amanda E. GUYER, Auteur . - p.1224-1233. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1224-1233 Mots-clés : Adolescence  brain imaging  connectomics  depression  development Index. décimale : PER Périodiques Résumé : BACKGROUND: Girls' depressive symptoms typically increase in adolescence, with individual differences in course and severity being key risk factors for impaired emotional functioning in young adulthood. Given the continued brain white matter (WM) maturation that occurs in adolescence, the present study tested whether structural connectivity patterns in late adolescence are associated with variation in the course of depression symptom severity throughout adolescence. METHOD: Participants were girls (N = 115) enrolled in a multiyear prospective cohort study of risk for depression. Initial depression severity (intercept) at age 10 and change in severity (linear slope) across ages 10-19 were examined in relation to WM tractography collected at age 19. Network-based statistic analyses were used to identify clusters showing variation in structural connectivity in association with depressive symptom intercept, slope, and their interaction. RESULTS: Higher initial depressive severity and steeper positive slope (separately) were associated with greater structural connectivity between temporal, subcortical socioaffective, and occipital regions. Intercept showed more connectivity associations than slope. The interaction effect indicated that higher initial symptom severity and a steeper negative slope (i.e., alleviating symptoms) were related to greater connectivity between cognitive control regions. Moderately severe symptoms that worsened over time were followed by greater connectivity between self-referential and cognitive regions (e.g., posterior cingulate and frontal gyrus). CONCLUSIONS: Higher depressive symptom severity in early adolescence and increasing symptom severity over time may forecast structural connectivity differences in late adolescence, particularly in pathways involving cognitive and emotion-processing regions. Understanding how clinical course relates to neurobiological correlates may inform new treatment approaches to adolescent depression. En ligne : http://dx.doi.org/10.1111/jcpp.13184 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Associations between ADHD and emotional problems from childhood to young adulthood: a longitudinal genetically sensitive study / Adi STERN in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1234-1242 Titre : Associations between ADHD and emotional problems from childhood to young adulthood: a longitudinal genetically sensitive study Type de document : Texte imprimé et/ou numérique Auteurs : Adi STERN, Auteur ; Jessica AGNEW-BLAIS, Auteur ; Andrea DANESE, Auteur ; Helen L. FISHER, Auteur ; Timothy MATTHEWS, Auteur ; Guilherme V. POLANCZYK, Auteur ; Jasmin WERTZ, Auteur ; Louise ARSENEAULT, Auteur Article en page(s) : p.1234-1242 Langues : Anglais (eng) Mots-clés : Attention deficit/hyperactivity disorder  anxiety  depression  development  genetics Index. décimale : PER Périodiques Résumé : BACKGROUND: Attention deficit/hyperactivity disorder (ADHD) is associated with emotional problems, and their co-occurrence often leads to worse outcomes. We investigated the developmental associations between ADHD and emotional problems from childhood to early adolescence and examined the genetic and environmental contributions to their developmental link. We further tested whether this developmental association remained across the transition to young adulthood. METHODS: We used data from the Environmental Risk (E-Risk) Longitudinal Twin Study, a cohort of 2,232 British twins. In childhood, ADHD and emotional problems were assessed at ages 5, 7, 10 and 12 with mothers' and teachers' reports. At age 18, we used self-reported symptoms according to DSM-5 criteria for ADHD, and DSM-IV for anxiety and depression. RESULTS: Longitudinal analyses showed that earlier ADHD was associated with later emotional problems consistently across childhood. However, earlier emotional problems were not associated with later ADHD symptoms. The developmental association between ADHD and later emotional problems in childhood was entirely explained by common genetic factors. Consistent with results in childhood, earlier symptoms of ADHD were associated with later emotional problems during the transition to young adulthood. CONCLUSIONS: Our findings demonstrate that ADHD symptoms are predictors of the development of emotional problems, from childhood up to young adulthood, through shared genetic influences. Interventions targeting ADHD symptoms might prevent the development of emotional problems. Clinicians treating youth with ADHD must be aware of their risk for developing emotional problems and ought to assess, monitor and treat emotional problems alongside ADHD symptoms from childhood to adulthood. En ligne : http://dx.doi.org/10.1111/jcpp.13217 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Associations between ADHD and emotional problems from childhood to young adulthood: a longitudinal genetically sensitive study [Texte imprimé et/ou numérique] / Adi STERN, Auteur ; Jessica AGNEW-BLAIS, Auteur ; Andrea DANESE, Auteur ; Helen L. FISHER, Auteur ; Timothy MATTHEWS, Auteur ; Guilherme V. POLANCZYK, Auteur ; Jasmin WERTZ, Auteur ; Louise ARSENEAULT, Auteur . - p.1234-1242. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1234-1242 Mots-clés : Attention deficit/hyperactivity disorder  anxiety  depression  development  genetics Index. décimale : PER Périodiques Résumé : BACKGROUND: Attention deficit/hyperactivity disorder (ADHD) is associated with emotional problems, and their co-occurrence often leads to worse outcomes. We investigated the developmental associations between ADHD and emotional problems from childhood to early adolescence and examined the genetic and environmental contributions to their developmental link. We further tested whether this developmental association remained across the transition to young adulthood. METHODS: We used data from the Environmental Risk (E-Risk) Longitudinal Twin Study, a cohort of 2,232 British twins. In childhood, ADHD and emotional problems were assessed at ages 5, 7, 10 and 12 with mothers' and teachers' reports. At age 18, we used self-reported symptoms according to DSM-5 criteria for ADHD, and DSM-IV for anxiety and depression. RESULTS: Longitudinal analyses showed that earlier ADHD was associated with later emotional problems consistently across childhood. However, earlier emotional problems were not associated with later ADHD symptoms. The developmental association between ADHD and later emotional problems in childhood was entirely explained by common genetic factors. Consistent with results in childhood, earlier symptoms of ADHD were associated with later emotional problems during the transition to young adulthood. CONCLUSIONS: Our findings demonstrate that ADHD symptoms are predictors of the development of emotional problems, from childhood up to young adulthood, through shared genetic influences. Interventions targeting ADHD symptoms might prevent the development of emotional problems. Clinicians treating youth with ADHD must be aware of their risk for developing emotional problems and ought to assess, monitor and treat emotional problems alongside ADHD symptoms from childhood to adulthood. En ligne : http://dx.doi.org/10.1111/jcpp.13217 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Association between environmental tobacco smoke exposure across the first four years of life and manifestation of externalizing behavior problems in school-aged children / Lisa GATZKE-KOPP in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1243-1252 Titre : Association between environmental tobacco smoke exposure across the first four years of life and manifestation of externalizing behavior problems in school-aged children Type de document : Texte imprimé et/ou numérique Auteurs : Lisa GATZKE-KOPP, Auteur ; Michael T. WILLOUGHBY, Auteur ; Siri WARKENTIEN, Auteur ; Daniel PETRIE, Auteur ; Roger MILLS-KOONCE, Auteur ; Clancy BLAIR, Auteur Article en page(s) : p.1243-1252 Langues : Anglais (eng) Mots-clés : Disruptive behavior  dopamine  environmental exposures  externalizing disorder  tobacco exposure Index. décimale : PER Périodiques Résumé : BACKGROUND: Extensive literature in human and animal models has documented an association between maternal smoking during pregnancy and externalizing behavior in offspring. It remains unclear; however, the extent to which postnatal environmental smoke exposure is associated with behavioral development, particularly for children whose mothers did not smoke during pregnancy. The present study examined whether magnitude of exposure to environmental smoke across the first four years of life demonstrated a linear association with later externalizing symptoms. METHODS: Exposure was quantified through salivary cotinine measured when children were 6, 15, 24, and 48 months of age, providing a more accurate quantification of realized exposure than can be estimated from parental report of cigarettes smoked. Data were available for n = 1,096 (50% male; 44% African American) children recruited for the Family Life Project, a study of child development in areas of rural poverty. RESULTS: Analyses indicate a linear association between cotinine and children's symptoms of hyperactivity and conduct problems. This association remained significant after controlling for family poverty level, parental education, parental history of ADHD, hostility, depression, caregiver IQ, and obstetric complications. Furthermore, this association was unchanged when excluding mothers who smoked during pregnancy from the model. CONCLUSIONS: Findings are consistent with animal models demonstrating an effect of environmental exposure to nicotine on ongoing brain development in regions related to hyperactivity and impulsivity, and highlight the importance of mitigating children's exposure to environmental smoke, including sources that extend beyond the parents. En ligne : http://dx.doi.org/10.1111/jcpp.13157 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Association between environmental tobacco smoke exposure across the first four years of life and manifestation of externalizing behavior problems in school-aged children [Texte imprimé et/ou numérique] / Lisa GATZKE-KOPP, Auteur ; Michael T. WILLOUGHBY, Auteur ; Siri WARKENTIEN, Auteur ; Daniel PETRIE, Auteur ; Roger MILLS-KOONCE, Auteur ; Clancy BLAIR, Auteur . - p.1243-1252. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1243-1252 Mots-clés : Disruptive behavior  dopamine  environmental exposures  externalizing disorder  tobacco exposure Index. décimale : PER Périodiques Résumé : BACKGROUND: Extensive literature in human and animal models has documented an association between maternal smoking during pregnancy and externalizing behavior in offspring. It remains unclear; however, the extent to which postnatal environmental smoke exposure is associated with behavioral development, particularly for children whose mothers did not smoke during pregnancy. The present study examined whether magnitude of exposure to environmental smoke across the first four years of life demonstrated a linear association with later externalizing symptoms. METHODS: Exposure was quantified through salivary cotinine measured when children were 6, 15, 24, and 48 months of age, providing a more accurate quantification of realized exposure than can be estimated from parental report of cigarettes smoked. Data were available for n = 1,096 (50% male; 44% African American) children recruited for the Family Life Project, a study of child development in areas of rural poverty. RESULTS: Analyses indicate a linear association between cotinine and children's symptoms of hyperactivity and conduct problems. This association remained significant after controlling for family poverty level, parental education, parental history of ADHD, hostility, depression, caregiver IQ, and obstetric complications. Furthermore, this association was unchanged when excluding mothers who smoked during pregnancy from the model. CONCLUSIONS: Findings are consistent with animal models demonstrating an effect of environmental exposure to nicotine on ongoing brain development in regions related to hyperactivity and impulsivity, and highlight the importance of mitigating children's exposure to environmental smoke, including sources that extend beyond the parents. En ligne : http://dx.doi.org/10.1111/jcpp.13157 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Prenatal exposure to polycyclic aromatic hydrocarbons modifies the effects of early life stress on attention and Thought Problems in late childhood / David PAGLIACCIO in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1253-1265 Titre : Prenatal exposure to polycyclic aromatic hydrocarbons modifies the effects of early life stress on attention and Thought Problems in late childhood Type de document : Texte imprimé et/ou numérique Auteurs : David PAGLIACCIO, Auteur ; Julie B. HERBSTMAN, Auteur ; Frederica PERERA, Auteur ; Deliang TANG, Auteur ; Jeff GOLDSMITH, Auteur ; Bradley S. PETERSON, Auteur ; Virginia RAUH, Auteur ; Amy E. MARGOLIS, Auteur Article en page(s) : p.1253-1265 Langues : Anglais (eng) Mots-clés : Attention deficit hyperactivity disorder  adversity  child behavior checklist  child development  toxicants Index. décimale : PER Périodiques Résumé : BACKGROUND: Risk for childhood psychopathology is complex and multifactorial, implicating direct and interacting effects of familial and environmental factors. The role of environmental neurotoxicants in psychiatric risk is of growing concern, including polycyclic aromatic hydrocarbons (PAH), common in air pollution. Prenatal PAH exposure is linked to adverse physical, behavioral, and cognitive outcomes as well as increasing psychiatric risk. It is unclear whether environmental exposures, like PAH, magnify the effects of exposure to early life stress (ELS), a critical risk factor for psychopathology. The current work aimed to test potential interactions between prenatal PAH exposure and psychosocial/socioeconomic stress on psychiatric symptoms in school-age children. METHODS: Data were from the Columbia Center for Children's Environmental Health Mothers and Newborns longitudinal birth cohort study. Prenatal PAH exposure was ascertained though air monitoring during pregnancy and maternal PAH-DNA adducts at delivery. Mothers reported on ELS (child age 5) and on child psychiatric symptoms across childhood (child age 5, 7, 9, and 11) using the Child Behavior Checklist (CBCL). RESULTS: Significant prenatal airborne PAH × ELS interactions (FDR-corrected) predicted CBCL Attention (? = 0.22, t(307) = 3.47, p < .001, p(fdr ) = .003) and Thought Problems T-scores (? = 0.21, t(307) = 3.29, p = .001, p(fdr ) = .004) at age 11 (n = 319). Relative to those with lower exposure, children with higher prenatal PAH exposure exhibited stronger positive associations between ELS and CBCL Attention and Thought Problem T-scores. This interaction was also significant examining convergent ADHD measures (Conners, DuPaul) and examining maternal PAH-DNA adducts (? = 0.29, t(261) = 2.48, p = .01; n = 273). A three-way interaction with assessment wave indicated that the PAH × ELS interaction on Attention Problems was stronger later in development (? = 0.03, t(1,601) = 2.19, p = .03; n = 477). CONCLUSIONS: Prenatal exposure to PAH, a common neurotoxicant in air pollution, may magnify or sustain the effects of early life psychosocial/socioeconomic stress on psychiatric outcomes later in child development. This work highlights the critical role of air pollution exposure on child mental health. En ligne : http://dx.doi.org/10.1111/jcpp.13189 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Prenatal exposure to polycyclic aromatic hydrocarbons modifies the effects of early life stress on attention and Thought Problems in late childhood [Texte imprimé et/ou numérique] / David PAGLIACCIO, Auteur ; Julie B. HERBSTMAN, Auteur ; Frederica PERERA, Auteur ; Deliang TANG, Auteur ; Jeff GOLDSMITH, Auteur ; Bradley S. PETERSON, Auteur ; Virginia RAUH, Auteur ; Amy E. MARGOLIS, Auteur . - p.1253-1265. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1253-1265 Mots-clés : Attention deficit hyperactivity disorder  adversity  child behavior checklist  child development  toxicants Index. décimale : PER Périodiques Résumé : BACKGROUND: Risk for childhood psychopathology is complex and multifactorial, implicating direct and interacting effects of familial and environmental factors. The role of environmental neurotoxicants in psychiatric risk is of growing concern, including polycyclic aromatic hydrocarbons (PAH), common in air pollution. Prenatal PAH exposure is linked to adverse physical, behavioral, and cognitive outcomes as well as increasing psychiatric risk. It is unclear whether environmental exposures, like PAH, magnify the effects of exposure to early life stress (ELS), a critical risk factor for psychopathology. The current work aimed to test potential interactions between prenatal PAH exposure and psychosocial/socioeconomic stress on psychiatric symptoms in school-age children. METHODS: Data were from the Columbia Center for Children's Environmental Health Mothers and Newborns longitudinal birth cohort study. Prenatal PAH exposure was ascertained though air monitoring during pregnancy and maternal PAH-DNA adducts at delivery. Mothers reported on ELS (child age 5) and on child psychiatric symptoms across childhood (child age 5, 7, 9, and 11) using the Child Behavior Checklist (CBCL). RESULTS: Significant prenatal airborne PAH × ELS interactions (FDR-corrected) predicted CBCL Attention (? = 0.22, t(307) = 3.47, p < .001, p(fdr ) = .003) and Thought Problems T-scores (? = 0.21, t(307) = 3.29, p = .001, p(fdr ) = .004) at age 11 (n = 319). Relative to those with lower exposure, children with higher prenatal PAH exposure exhibited stronger positive associations between ELS and CBCL Attention and Thought Problem T-scores. This interaction was also significant examining convergent ADHD measures (Conners, DuPaul) and examining maternal PAH-DNA adducts (? = 0.29, t(261) = 2.48, p = .01; n = 273). A three-way interaction with assessment wave indicated that the PAH × ELS interaction on Attention Problems was stronger later in development (? = 0.03, t(1,601) = 2.19, p = .03; n = 477). CONCLUSIONS: Prenatal exposure to PAH, a common neurotoxicant in air pollution, may magnify or sustain the effects of early life psychosocial/socioeconomic stress on psychiatric outcomes later in child development. This work highlights the critical role of air pollution exposure on child mental health. En ligne : http://dx.doi.org/10.1111/jcpp.13189 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Early pubertal timing predicts onset and recurrence of depressive episodes in boys and girls / Elissa J. HAMLAT in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1266-1274 Titre : Early pubertal timing predicts onset and recurrence of depressive episodes in boys and girls Type de document : Texte imprimé et/ou numérique Auteurs : Elissa J. HAMLAT, Auteur ; Kathleen C. MCCORMICK, Auteur ; Jami F. YOUNG, Auteur ; Benjamin L. HANKIN, Auteur Article en page(s) : p.1266-1274 Langues : Anglais (eng) Mots-clés : Depression  puberty  that they have no competing or potential conflicts of interest. Index. décimale : PER Périodiques Résumé : BACKGROUND: Recurrent depressive episodes during adolescence result in significant impairment and increased risk for subsequent adverse outcomes throughout the life span. Evidence suggests that early pubertal timing predicts the onset of depressive episodes (particularly for girls); however, it is not known if pubertal timing prospectively predicts recurrent depressive episodes in youth. METHODS: At baseline, 603 youth (56% female, at baseline: M(age)  = 12.09, SD = 2.35) reported on their pubertal development. Youth and their parents completed a semistructured diagnostic interview to assess depressive episodes at baseline and then evaluated for onset repeatedly every 6 months for a period of 36 months. RESULTS: Controlling for past history of depression, Cox proportional hazards models examined whether earlier pubertal timing predicted (a) days to first depressive episode from baseline and (b) days to a second (recurrent) depressive episode from the end of the first episode. Early pubertal timing predicted the onset of the first depressive episode after baseline (b = .19, Wald = 5.36, p = .02, HR = 1.21), as well as a recurrent episode during course of study follow-up episode (b = .32, Wald = 6.16, p = .01, HR = 1.38). CONCLUSIONS: Findings reinforce the importance of considering the impact of early pubertal timing on depression risk. Investigation on how pubertal timing interacts with other risk factors to predict depression recurrence is needed. En ligne : http://dx.doi.org/10.1111/jcpp.13198 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Early pubertal timing predicts onset and recurrence of depressive episodes in boys and girls [Texte imprimé et/ou numérique] / Elissa J. HAMLAT, Auteur ; Kathleen C. MCCORMICK, Auteur ; Jami F. YOUNG, Auteur ; Benjamin L. HANKIN, Auteur . - p.1266-1274. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1266-1274 Mots-clés : Depression  puberty  that they have no competing or potential conflicts of interest. Index. décimale : PER Périodiques Résumé : BACKGROUND: Recurrent depressive episodes during adolescence result in significant impairment and increased risk for subsequent adverse outcomes throughout the life span. Evidence suggests that early pubertal timing predicts the onset of depressive episodes (particularly for girls); however, it is not known if pubertal timing prospectively predicts recurrent depressive episodes in youth. METHODS: At baseline, 603 youth (56% female, at baseline: M(age)  = 12.09, SD = 2.35) reported on their pubertal development. Youth and their parents completed a semistructured diagnostic interview to assess depressive episodes at baseline and then evaluated for onset repeatedly every 6 months for a period of 36 months. RESULTS: Controlling for past history of depression, Cox proportional hazards models examined whether earlier pubertal timing predicted (a) days to first depressive episode from baseline and (b) days to a second (recurrent) depressive episode from the end of the first episode. Early pubertal timing predicted the onset of the first depressive episode after baseline (b = .19, Wald = 5.36, p = .02, HR = 1.21), as well as a recurrent episode during course of study follow-up episode (b = .32, Wald = 6.16, p = .01, HR = 1.38). CONCLUSIONS: Findings reinforce the importance of considering the impact of early pubertal timing on depression risk. Investigation on how pubertal timing interacts with other risk factors to predict depression recurrence is needed. En ligne : http://dx.doi.org/10.1111/jcpp.13198 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Editorial Perspective: Why I am now convinced that emotional disorders are increasingly common among young people in many countries / Tamsin FORD in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1275-1277 Titre : Editorial Perspective: Why I am now convinced that emotional disorders are increasingly common among young people in many countries Type de document : Texte imprimé et/ou numérique Auteurs : Tamsin FORD, Auteur Article en page(s) : p.1275-1277 Langues : Anglais (eng) Mots-clés : Epidemiologic studies  aetiology  implementation Index. décimale : PER Périodiques Résumé : This editorial perspective comments on the evidence that mental health is deteriorating among children and young people at the same time as the impact of poor mental health is more impairing. It discusses the methodological difficulties in detecting a 'true' change in prevalence. There are particular gaps in the evidence from low- and middle-income countries and also in relation to autism spectrum disorders. The evidence in high-income countries points to recent increases in anxiety, depression and self-harm in teenagers, particularly young women. We need more longitudinal data to disentangle the potential explanations for these changes. En ligne : http://dx.doi.org/10.1111/jcpp.13211 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Editorial Perspective: Why I am now convinced that emotional disorders are increasingly common among young people in many countries [Texte imprimé et/ou numérique] / Tamsin FORD, Auteur . - p.1275-1277. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1275-1277 Mots-clés : Epidemiologic studies  aetiology  implementation Index. décimale : PER Périodiques Résumé : This editorial perspective comments on the evidence that mental health is deteriorating among children and young people at the same time as the impact of poor mental health is more impairing. It discusses the methodological difficulties in detecting a 'true' change in prevalence. There are particular gaps in the evidence from low- and middle-income countries and also in relation to autism spectrum disorders. The evidence in high-income countries points to recent increases in anxiety, depression and self-harm in teenagers, particularly young women. We need more longitudinal data to disentangle the potential explanations for these changes. En ligne : http://dx.doi.org/10.1111/jcpp.13211 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434

Erratum in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1277 Titre : Erratum Type de document : Texte imprimé et/ou numérique Article en page(s) : p.1277 Langues : Anglais (eng) Index. décimale : PER Périodiques En ligne : http://dx.doi.org/10.1111/jcpp.13336 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 [article] Erratum [Texte imprimé et/ou numérique] . - p.1277. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 61-11 (November 2020) . - p.1277 Index. décimale : PER Périodiques En ligne : http://dx.doi.org/10.1111/jcpp.13336 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434